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1 tervention group (46 patients experienced 65 cardiopulmonary arrests).
2 o restore oxygenation and reduce the risk of cardiopulmonary arrest.
3 mary outcome was survival to discharge after cardiopulmonary arrest.
4 or with mild deficits following in-hospital cardiopulmonary arrest.
5 limitation of resuscitative measures only on cardiopulmonary arrest.
6 unique global ischemia/reperfusion insult of cardiopulmonary arrest.
7 tance of a respiratory etiology of pediatric cardiopulmonary arrest.
8 arge in hospitalized patients suffering from cardiopulmonary arrest.
9 in improving survival of patients suffering cardiopulmonary arrest.
10 to be declared dead before the occurrence of cardiopulmonary arrest.
11 ning for parents of infants at high risk for cardiopulmonary arrest.
12 to 2 groups based on the occurrence of donor cardiopulmonary arrest.
13 f the liver damage is induced by brief donor cardiopulmonary arrest.
14 se 32 patients had a total of 38 episodes of cardiopulmonary arrest.
15 roup A consisted of 37 donors who suffered a cardiopulmonary arrest.
16 l to patients with DNR orders who experience cardiopulmonary arrest.
17 5 (1.8%) pediatric admissions, experienced a cardiopulmonary arrest.
18 oral performance during subsequent simulated cardiopulmonary arrests.
19 lonic activity (22.7% vs 4.2%; P < .001) and cardiopulmonary arrest (9.7% vs 0.5%, P < .001) vs patie
20 ic therapy during pulmonary embolism-induced cardiopulmonary arrest and discuss the role of thromboly
21 ere is no reported correlation between donor cardiopulmonary arrest and hepatic allograft dysfunction
22 g intestinal grafts from donors who suffered cardiopulmonary arrest and resuscitation and to evaluate
23 n the venglustat group owing to an unrelated cardiopulmonary arrest and there were no deaths in the p
25 inpatients experiencing an index episode of cardiopulmonary arrest and undergoing cardiopulmonary re
26 atients were all children who presented with cardiopulmonary arrest and who were administered CPR in
27 d intubation increases the risk of hypoxemic cardiopulmonary arrest and/or pulmonary aspiration, resu
31 apid response team was effective in reducing cardiopulmonary arrests and total hospital mortality for
32 tween April and July 2009, all patients with cardiopulmonary arrests and unplanned intensive care uni
33 control group (123 patients experienced 183 cardiopulmonary arrests) and between July 1, 2010, and J
35 ge resulting from circulatory failure during cardiopulmonary arrest before organ procurement in group
37 ealth care teams participated in 2 simulated cardiopulmonary arrests, before and after debriefing.
39 s a guide for therapeutic decisions during a cardiopulmonary arrest but also as a surrogate for broad
40 erapies designed to enhance perfusion during cardiopulmonary arrest by emphasizing chest compressions
41 cted hyponatremic encephalopathy and died of cardiopulmonary arrest caused by brainstem herniation.
43 e been shown in adult inpatients to decrease cardiopulmonary arrest (code) rates outside of the inten
44 ther survival to discharge after in-hospital cardiopulmonary arrest could be improved by a program en
46 were observed in rapid response activation, cardiopulmonary arrest, death in hospital, ICU readmissi
47 rapid response team implementation, non-ICU cardiopulmonary arrests decreased from 1.4 to 0.9 per 1,
49 lticenter study of children with in-hospital cardiopulmonary arrest, ETCO(2) >=20 mm Hg was associate
50 er, n = 11), including advance warning of 27 cardiopulmonary arrest events (26%) for which return of
52 tients were followed for the occurrence of a cardiopulmonary arrest (external cardiac massage for at
53 ement in outcomes from in-hospital pediatric cardiopulmonary arrest following the introduction of a p
54 ing lifesaving resuscitation in a patient in cardiopulmonary arrest greatly outweighs the risk for se
55 eived chest compressions of any duration for cardiopulmonary arrest, had an endotracheal or tracheost
56 ining, survival to discharge after pediatric cardiopulmonary arrest improved, as did code team perfor
57 and treatment of cardiovascular collapse or cardiopulmonary arrest in an adult or pediatric patient
59 r-success" situations, including a witnessed cardiopulmonary arrest in which the initial cardiac rhyt
60 cation outcomes, total telemetry census, and cardiopulmonary arrests in comparison with the previous
62 grafts from donors who have suffered a brief cardiopulmonary arrest may sustain ischemic damage befor
64 er, patients successfully resuscitated after cardiopulmonary arrest often have a favorable prognosis.
65 Patients who were in a state of continuous cardiopulmonary arrest on admission, or who never achiev
66 ve was to study the influence of brief donor cardiopulmonary arrest on hepatic allograft outcome in h
67 citation for people who suffer an unexpected cardiopulmonary arrest or for whom resuscitation may hav
68 RT) has been shown to decrease mortality and cardiopulmonary arrests outside of the intensive care un
69 withdrawal of life-sustaining therapies and cardiopulmonary arrest, patients are cannulated onto ext
71 de mortality rates and code (respiratory and cardiopulmonary arrests) rates outside of the ICU settin
72 ith witnessed, nontraumatic, out-of-hospital cardiopulmonary arrests regardless of initial electrocar
73 oratory and clinical investigations of adult cardiopulmonary arrest, resuscitation guidelines recomme
76 he notion that a majority of out-of-hospital cardiopulmonary arrest survivors die after a decision to
78 with a decrease in the composite endpoint of cardiopulmonary arrests, unplanned ICU admissions, and m
79 ith those of patients who did not experience cardiopulmonary arrest using propensity score matching w
80 believe that care changes beyond response to cardiopulmonary arrest, varying from increased attention
82 ent in clinical performance during simulated cardiopulmonary arrests was not significantly different
84 atose survivors of witnessed out-of-hospital cardiopulmonary arrests who were admitted to an ICU duri