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1 tion may provide valuable insights into IUGR cardiovascular physiology.
2 INTS: Wave travel plays an important role in cardiovascular physiology.
3 s (betaARs) are critical regulators of acute cardiovascular physiology.
4 gh altitude and developed the field of fetal cardiovascular physiology.
5 nsive role of these small regulatory RNAs in cardiovascular physiology.
6 ant regulators of renal sodium transport and cardiovascular physiology.
7 r (GPCR) Agtrl1b, a known regulator of adult cardiovascular physiology.
8 he wider context of vascular development and cardiovascular physiology.
9 re consequences of fundamental principles of cardiovascular physiology.
10 multimodal assessment of cerebrovascular and cardiovascular physiology.
11  effects of mouse ADCY9 gene inactivation on cardiovascular physiology.
12 tic machinery that is normally implicated in cardiovascular physiology.
13 talk with a specific emphasis on cardiac and cardiovascular physiology.
14 od pressure, and diagnostic codes related to cardiovascular physiology.
15 fer considerable advantages for the field of cardiovascular physiology.
16 , an important regulator of reproductive and cardiovascular physiology.
17  aortic valve is an important determinant of cardiovascular physiology and anatomic location of commo
18 ous-flow LVADs alter blood flow dynamics and cardiovascular physiology and can induce progression of
19 gical molecules that play important roles in cardiovascular physiology and contribute to disease init
20 fine the key roles that mitochondria play in cardiovascular physiology and disease and provide insigh
21 iotensin II (Ang II) plays a central role in cardiovascular physiology and disease.
22 ectronics have transformed investigations of cardiovascular physiology and disease.
23 nt statins were shown to improve biomarkers, cardiovascular physiology and lung function in animal mo
24 ing the effects exerted by mTOR signaling in cardiovascular physiology and pathological states.
25 R pathway plays a key regulatory function in cardiovascular physiology and pathology.
26 icate that sex has an important influence on cardiovascular physiology and pathology.
27 , content, and transmission, with a focus on cardiovascular physiology and pathophysiology.
28 dase (Nox) enzymes are critical mediators of cardiovascular physiology and pathophysiology.
29 ones have emerged as important modulators of cardiovascular physiology and pathophysiology.
30 be biologically inactive - have key roles in cardiovascular physiology and pathophysiology.
31 indered studies on the role of endothelin in cardiovascular physiology and pathophysiology.
32 use technology provide novel models to study cardiovascular physiology and pathophysiology.
33 model provide a foundation for understanding cardiovascular physiology and pathophysiology.
34 urnals that publish peer-reviewed studies in cardiovascular physiology and pharmacology in the year 2
35 ese pathways, RGS proteins are regulators of cardiovascular physiology and potentially novel drug tar
36 nderstood how altered ACTC structure affects cardiovascular physiology and results in the development
37 tability, hypothalamic-pituitary signalling, cardiovascular physiology and sympathetic function.
38 r actions of PARs that play a role in normal cardiovascular physiology and that are likely to contrib
39 Changes in other domains, such as cognition, cardiovascular physiology, and metabolic indicators, wer
40 he short-term effects of estrogen central to cardiovascular physiology are mediated by ERalpha functi
41 findings suggest that normal early-pregnancy cardiovascular physiology, as assessed by NT-proBNP conc
42 role of exosomes and microvesicles in normal cardiovascular physiology, as well as during cardiovascu
43                                              Cardiovascular physiology at rest and during acute stres
44 ions or polymorphisms in genes that regulate cardiovascular physiology, blood coagulation, lipid meta
45  Starling are not only famous as pioneers in cardiovascular physiology, but also responsible for the
46 ssociated with acute, unfavorable effects on cardiovascular physiology, but few studies have examined
47                                              Cardiovascular physiology changes during embryonic devel
48 of these events and their role in regulating cardiovascular physiology during health and disease.
49   It is, therefore, not surprising that both cardiovascular physiology (eg, heart rate and blood pres
50 ct evidence for a role of Cyp 4a isoforms in cardiovascular physiology, establish Cyp 4a14 (-/-) mice
51 x differences are apparent at every level of cardiovascular physiology from action potential duration
52 nsation), sound (hearing), and shear stress (cardiovascular physiology); however, the identity of the
53  hours every day) on neural, peripheral, and cardiovascular physiology in Drosophila melanogaster.
54  inflammation and thrombosis and measures of cardiovascular physiology in healthy young persons.
55 pothyroidism often cause opposing effects on cardiovascular physiology in the short term.
56 to enable fully conscious in vivo studies of cardiovascular physiology in transgenic rodent disease m
57 ing PKA phosphorylation at this site impairs cardiovascular physiology in vivo, leading to reduced ex
58  cellular iron homeostasis in the context of cardiovascular physiology, iron deficiency, and iron ove
59 xpress estrogen receptors, but their role in cardiovascular physiology is not well understood.
60  clear that, in addition to effects on adult cardiovascular physiology, lipids also play key roles in
61                          Changes in renal or cardiovascular physiology or metabolism cannot explain t
62 kinases (PKGs) play important roles in human cardiovascular physiology, regulating vascular tone and
63      The role played by nitric oxide (NO) in cardiovascular physiology remains highly controversial.
64 y have high aerobic capacity and distinctive cardiovascular physiology resembling that of endothermic
65 (miR30a-5p, miR-145-5p) which are related to cardiovascular physiology, showed a significant systemic
66 de (NO) signaling regulates key processes in cardiovascular physiology, specifically vasodilation, pl
67 y of methods have been used to measure basic cardiovascular physiology, spinal oxygenation, mitochond
68 hibits pharmacological properties related to cardiovascular physiology that are distinct from nitric
69                   NO is a major regulator of cardiovascular physiology that reduces vascular and card
70  To understand their impact on metabolic and cardiovascular physiology, we tested the effects of thes