ライフサイエンスコーパス: carious

1 ed as intact, moderately carious, or grossly carious.

2 were associated with the number of untreated carious and missing tooth surfaces in adulthood.

3 The number of untreated carious and missing tooth surfaces were associated with

4 32 y), oral health outcomes (e.g., number of carious and missing tooth surfaces), and oral health-rel

5 ess for components of hydrated dentin from 8 carious and non-carious human teeth.

6 Cervical composites treating root carious and noncarious cervical lesions usually extend s

7 Permanent carious and sound teeth indicated for extraction were ex

8 Carious and sound teeth indicated for extraction were ra

9 reparative mechanisms that operate following carious and traumatic dental injury are critical for pul

10 ansitions between healthy (sound), diseased (carious), and disease-controlled (arrested) states for 6

11 en comparing the amount of CFU isolated from carious biopsies from different colour and hardness cate

12 eth restored with crowns and presenting with carious cavitation were excluded.

13 o patients presenting 62 CDs (RT1 GR and non-carious cervical lesion (NCCLs) were randomly allocated

14 gingival recession (GR) associated with non-carious cervical lesion-combined defects (CDs).

15 gival recession (GR) RT1 associated with non-carious cervical lesions (NCCL) B+ partially restored us

16 ngival recession (GR) is associated with non-carious cervical lesions (NCCL), represent a clinical co

17 to assess the development/prevalence of non-carious cervical lesions (NCCLs) at sites that have and

18 ingival recessions (GRs) associated with non-carious cervical lesions (NCCLs) prior to surgical treat

19 gingival recession (GR) associated with non-carious cervical lesions (NCCLs) treated by connective t

20 t is, gingival recession associated with non-carious cervical lesions, were treated by partial resin

21 of disease states, ranging from sub-surface carious changes through to more advanced lesions.

22 , 16 slightly demineralized, and 29 distinct carious changes were mounted to take digital radiographs

23 xpression was also evident in dental pulp of carious compared with noncarious teeth.

24 on) annually averted 0.29 (95%CI: 0.16-0.42) carious coronal and 0.22 (95%CI: 0.08-0.37) carious root

25 sable mixture which could be used to restore carious defects in teeth followed a tortuous path of neg

26 ed this hypothesis by bonding to transparent carious dentin containing occluded dentinal tubules.

27 bide bur, and both were lower than flat, non-carious dentin controls.

28 Carious dentin from extracted human molars was removed w

29 e physical and microstructural properties of carious dentin in the 4 different zones to determine imp

30 veness were assessed by the working time for carious dentin removal and Knoop microhardness values, r

31 ents used for the chemomechanical removal of carious dentin.

32 gnize signals from TGF-beta1 and bacteria in carious dentin.

33 nated during bonding to occluded transparent carious dentin.

34 omposites have the potential to remineralize carious enamel lesions.

35 were placed in holders that exposed only the carious enamel surface.

36 lants were associated with decreased risk of carious first molars (odds ratio, 0.21 [95% CI, 0.16-0.2

37 Tooth slices were prepared from non-carious human molars and treated with 0-50 ng/mL rhVEGF(

38 Sixty extracted carious human teeth were treated with a conventional bur

39 th the presence of Gram-positive bacteria in carious human teeth.

40 ts of hydrated dentin from 8 carious and non-carious human teeth.

41 ls the direction of neurite outgrowth toward carious injuries by modulating the secretion of brain-de

42 ntal pulp fibroblasts, localized beneath the carious injury site, do express this receptor.

43 nerve fiber's terminal branches beneath the carious injury site.

44 l orchestrating pulp nerve sprouting beneath carious injury, a critical step in dentin-pulp regenerat

45 n of BDNF by pulp fibroblasts under sites of carious injury.

46 secretion of nerve growth factor (NGF) upon carious injury.

47 ucture, human dental pulp, following chronic carious insult.

48 h random permuted blocks): C+P, conventional carious lesion management (complete carious tooth tissue

49 sed based on the presence of a new cavitated carious lesion or a new pulpally involved lesion across

50 entine colour and hardness as indicators for carious lesion severity has never been assessed in a sys

51 c and reliable than colour to detect dentine carious lesion severity.

52 a severe recession defect and its associated carious lesion were managed using the combination of a l

53 with at least 1 primary molar with dentinal carious lesion were randomized across 3 arms (1:1:1 via

54 92.2% of the subjects presented at least one carious lesion, whereas 22.5% and 56.2% were diagnosed w

55 d to promote remineralization and harden the carious lesion.

56 Carious lesions (a proxy for dental health) were identif

57 lesions (CAE), and caries active with dentin carious lesions (CA).

58 ing or reversing noncavitated facial/lingual carious lesions (low certainty) and that 38% silver diam

59 practice, can arrest occlusal non-cavitated carious lesions (NCCLs); however, U.S. oral health provi

60 from enamel carious lesions (PE) and dentin carious lesions (PD) were collected.

61 ies-free tooth surfaces (PF) and from enamel carious lesions (PE) and dentin carious lesions (PD) wer

62 acted permanent molars with natural occlusal carious lesions (score > 4 following the International C

63 tal analysis also revealed zinc abundance in carious lesions and around the pulp chamber.

64 Candida albicans are often co-isolated from carious lesions and associated with increased disease se

65 all accuracy of visual methods for detecting carious lesions and to identify possible sources of hete

66 incidence of cavitated or pulpally involved carious lesions at follow-up and stunting (relative risk

67 n conclusion, treatment of symptomatic, deep carious lesions by ozone following partial removal of ca

68 For proximal carious lesions confined to enamel (not reaching the ena

69 The incidence of pulpally involved carious lesions had an effect on wasting prevalence (WHZ

70 ce that the development of pulpally involved carious lesions has an effect on WHZ scores.

71 ther Thio-GLU or GLU had significantly fewer carious lesions in the buccal enamel or dentinal surface

72 ronment caused by periodontitis and advanced carious lesions in women with unexplained infertility sh

73 In clinical practice, diagnosis of suspected carious lesions is verified by using conventional dental

74 hin the microbial communities in deep-dentin carious lesions may play a fundamental role in caries et

75 variety of effective interventions to treat carious lesions nonrestoratively.

76 t effective for arresting advanced cavitated carious lesions on any coronal surface (moderate to high

77 st or reversal of noncavitated and cavitated carious lesions on primary and permanent teeth and 2) ad

78 roximal, and noncavitated and cavitated root carious lesions on primary and/or permanent teeth, respe

79 Effects of carious lesions on stunting and wasting were assessed us

80 Here, for the first time, we report carious lesions preserved in specimens of A. simus, reco

81 aSNR and aHTMCNR was significantly higher in carious lesions than in healthy hard tissue (p < 0.001).

82 fabM strain exhibited fewer and less severe carious lesions than those observed in the wild-type str

83 ficantly lower number of enamel and dentinal carious lesions was observed for the mutant-infected rat

84 The depth of the artificial carious lesions was significantly affected by "adhesive

85 Sixteen deep-dentin carious lesions were obtained from the first permanent m

86 the in vivo caries model, enamel and dentin carious lesions were significantly reduced in rats that

87 llus casei, which often predominates in deep carious lesions where B cells and plasma cells predomina

88 ould intervene invasively (restoratively) on carious lesions where evidence and clinical recommendati

89 This study found that in the deep-dentin carious lesions, Actinobacteria (35.8%) and Firmicutes (

90 nuated surface and subsurface regions within carious lesions, and similar regions were not present in

91 Fourteen patients with clinically suspected carious lesions, verified by standardized dental examina

92 type c strain originally isolated from human carious lesions, which is extensively used as a laborato

93 f a microbial community of human deep-dentin carious lesions.

94 severity states by their number of untreated carious lesions.

95 at involves the same risk factors as primary carious lesions.

96 ologies or near-infrared imaging to identify carious lesions.

97 her known risk factors, including history of carious lesions.

98 sting or reversing noncavitated or cavitated carious lesions.

99 issue and reduced signal from de-mineralized carious lesions.

100 thogenic potential of S. mutans in advancing carious lesions.

101 aggressive onset of the disease with rampant carious lesions.

102 ence factor responsible for the formation of carious lesions.

103 invasive/nonrestorative treatment of "early" carious lesions: those confined to enamel or reaching th

104 Their source was: (1) carious material from advanced root lesions (ARL), (2) p

105 Exposed root surfaces frequently exhibit non-carious notches representing material loss by abrasion,

106 Teeth were categorized as intact, moderately carious, or grossly carious.

107 uited 534 children aged 6-10 yrs with >or= 2 carious posterior teeth.

108 Hall Technique (HT) is a method for managing carious primary molars.

109 Carious primary teeth fixed within 2 min of SDF applicat

110 Carious primary teeth without SDF application (no-SDF, n

111 urvival of vital pulp therapies (VPTs) after carious pulp exposure in adult teeth.

112 th with irreversible pulpitis as a result of carious pulp exposure.

113 long-term success/survival rates in treating carious pulp exposures.

114 nic whites (0.6) having the fewest untreated carious root surfaces.

115 carious coronal and 0.22 (95%CI: 0.08-0.37) carious root surfaces.

116 the genus level, only 25% of the deep-dentin carious samples showed Lactobacillus as the most abundan

117 obability of transitioning between sound and carious states in 6-year molars ranged from 0.0022 to 0.

118 The mean cost per carious surface avoided was estimated at pound251 (95% c

119 aces in MA users was quite distinctive, with carious surface involvement being highest for the maxill

120 es (95%CI = 1.0-1.9), or approximately 1 new carious surface per person per year.

121 The primary outcome was the number of carious surfaces that had a recurrence of caries.

122 The DE was 1.4 for the clustering of carious surfaces within teeth, 6.0 for carious teeth wit

123 ous teeth within an individual, and 38.0 for carious surfaces within the individual.

124 ure in the SDF group was 38.3% (2167 of 5651 carious surfaces) compared with 45.5% (2116 of 4647) in

125 outcomes were only detected with respect to carious surfaces.

126 n, is activated at the injured site of human carious teeth and plays an important role in dental-pulp

127 ting significantly lower aSNR and aHTMCNR in carious teeth compared to healthy teeth (p = 0.01).

128 molecules present in pulp cell lysates from carious teeth specifically activated PAR-2, but those fr

129 Twenty human extracted and non-carious teeth were divided into 4 groups: 5 primary and

130 ermine the clinical outcome of pulpotomy for carious teeth with irreversible pulpitis and it's predic

131 be isolated from inflamed pulp derived from carious teeth with symptomatic irreversible pulpitis (I-

132 ng of carious surfaces within teeth, 6.0 for carious teeth within an individual, and 38.0 for carious

133 l: < 1 and high-risk school: >/= 1 untreated carious teeth).

134 ne and treat patients with a matched pair of carious teeth, and each pair of teeth was treated in a r

135 The neural status of carious teeth, particularly those associated with a pain

136 of BDNF secretion by pulp fibroblasts under carious teeth.

137 nitial mechanisms regulating this process in carious teeth.

138 tensive caries, as measured by the number of carious teeth.

139 of caries progression in sealed vs. unsealed carious teeth.

140 g was 2.6% for sealed and 12.6% for unsealed carious teeth.

141 Cavities were prepared and carious tissue on pulpo-proximal walls selectively remov

142 e subjective (S) vs. objective (O) selective carious tissue removal using hand-excavation versus a se

143 The carious tissue was removed in a shorter time with CBG (m

144 sis was performed with the outcome variables carious tooth or surface.

145 new caries outcomes defined as the number of carious tooth surfaces within each cluster.

146 samples were collected from caries-free and carious tooth surfaces.

147 entional carious lesion management (complete carious tooth tissue removal and restoration placement)

148 tion; B+P, biological management (sealing in carious tooth tissue restoratively) with prevention; and

149 At the subject-level, the sound to carious transition probabilities were 0.07 and 0.12 afte