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1 fatty acid oxidation (acetylCoA carboxylase; carnitine-palmitoyltransferase).
2 ng enzyme activities for arylsulfatase A and carnitine palmitoyltransferase.
3     Both residues are replaced by glycine in carnitine palmitoyltransferases.
4  complex I was associated with a decrease in carnitine palmitoyltransferase 1 (cPT1) and cPT2 levels.
5 c remodeling, with an early isoform shift of carnitine palmitoyltransferase 1 (CPT1) toward increased
6  the content of malonyl-CoA, an inhibitor of carnitine palmitoyltransferase 1 (CPT1), and, thus, mito
7                                   The enzyme carnitine palmitoyltransferase 1 (CPT1), which is anchor
8 yl-CoA concentrations are known to derepress carnitine palmitoyltransferase 1 (CPT1).
9 ated with changes in ACSL1 (R(2) = 0.39) and carnitine palmitoyltransferase 1 (R(2) = 0.30) expressio
10 nthase, acetyl coenzyme A carboxylase 2, and carnitine palmitoyltransferase 1 alpha) in both WT and A
11 tty acid oxidation through the inhibition of carnitine palmitoyltransferase 1 by its product malonyl-
12 phorylated acetyl-coenzyme A carboxylase and carnitine palmitoyltransferase 1 in the liver.
13  of the lipogenic pool but diminution of the carnitine palmitoyltransferase 1 inhibitory pool under c
14 R) delta to the PPAR response element on the carnitine palmitoyltransferase 1 promoter.
15  acids (LCFAs) at the level of reduced CPT1 (carnitine palmitoyltransferase 1) activity at the outer
16                                        CPT1 (carnitine palmitoyltransferase 1) is a rate-limiting enz
17 pyruvate carboxykinase, fatty acid synthase, carnitine palmitoyltransferase 1, and glucokinase among
18  short-chain fatty acids (SCFAs) that bypass carnitine palmitoyltransferase 1, could similarly suppor
19 ated receptor alpha and induction of hepatic carnitine palmitoyltransferase 1, suggesting increased e
20 unction through its allosteric inhibition of carnitine palmitoyltransferase 1, the enzyme that normal
21  human mitochondria-specific protein and the carnitine palmitoyltransferase 1.
22 cid oxidation by stimulating the activity of carnitine palmitoyltransferase-1 (CPT-1) and inhibiting
23                                 In addition, carnitine palmitoyltransferase-1 (CPT-1) inhibitor up-re
24 atty acids (LCFAs) into the mitochondria via carnitine palmitoyltransferase-1 (CPT-1) is inhibited by
25             Remarkably, 30 does not activate carnitine palmitoyltransferase-1 (CPT-1) nor induces in
26 l downstream effects including inhibition of carnitine palmitoyltransferase-1 (CPT-1) with resultant
27 ns, C75 inhibits FAS activity and stimulates carnitine palmitoyltransferase-1 (CPT-1), consistent wit
28                                              Carnitine palmitoyltransferase-1 (CPT1) is a rate-limiti
29                                   The enzyme carnitine palmitoyltransferase-1 (CPT1) regulates long-c
30   Malonyl-CoA is an established inhibitor of carnitine palmitoyltransferase-1 (CPT1), an outer mitoch
31 o this end, we targeted the liver isoform of carnitine palmitoyltransferase-1 (encoded by the CPT1A g
32 r-activated receptor alpha protein and liver-carnitine palmitoyltransferase-1 (l-CPT-1) mRNA increase
33  lipogenesis and decrease in the activity of carnitine palmitoyltransferase-1 and total energy expend
34 quent treatment of mice for 4 weeks with the carnitine palmitoyltransferase-1 inhibitor, oxfenicine (
35 omir) consumed diets containing 0.01% of the carnitine palmitoyltransferase-1 inhibitor, R-etomoxir,
36             Using a skeletal muscle-specific carnitine palmitoyltransferase-1 KO model, we show that
37                                   Similarly, carnitine palmitoyltransferase-1 was inhibited after rep
38 genes of fatty acid oxidation such as Cpt-1 (carnitine palmitoyltransferase-1) as well as Pgc-1alpha
39                                  Activity of carnitine palmitoyltransferase-1, a key enzyme controlli
40 l-CoA is a potent inhibitor of mitochondrial carnitine palmitoyltransferase-1, a key enzyme involved
41 reflect a metabolic bottleneck downstream of carnitine palmitoyltransferase-1, a mitochondrial enzyme
42 lcohol-induced liver injury due to increased carnitine palmitoyltransferase-1, phosphorylated 5'AMP-a
43 chain fatty acid oxidation that depends upon carnitine palmitoyltransferase 1a (CPT1a) and hydroxyacy
44 the levels of the IMP2 client mRNAs encoding carnitine palmitoyltransferase 1A (CPT1A) and peroxisome
45           In addition, a missense SNP in the carnitine palmitoyltransferase 1A (CPT1A) gene was assoc
46 , cg01082498, and cg09737197) in intron 1 of carnitine palmitoyltransferase 1A (CPT1A) were strongly
47              Here we show that expression of carnitine palmitoyltransferase 1a (CPT1a), a mtLCFAO rat
48 hat FGFR blockade promotes the expression of carnitine palmitoyltransferase 1A (CPT1A), a rate-limiti
49 ess the Cpt1a gene, which encodes the enzyme carnitine palmitoyltransferase 1A (CPT1A), an enzyme tha
50  metabolism-acyl-CoA thioesterase 1 (Acot1), carnitine palmitoyltransferase 1a (Cpt1a), and perilipin
51 onent of mitochondrial fatty acid transport, carnitine palmitoyltransferase 1A (CPT1A), as a direct H
52                                    Increased carnitine palmitoyltransferase 1A (CPT1A), identified as
53 nduces activation of FAO and upregulation of carnitine palmitoyltransferase 1A (CPT1A), the rate-limi
54  SCs and myogenesis, we examined the role of carnitine palmitoyltransferase 1A (CPT1A), the rate-limi
55                                We found that carnitine palmitoyltransferase 1A (CPT1A), the rate-limi
56 e in malonyl-CoA levels and desinhibition of carnitine palmitoyltransferase 1A (CPT1A), which increas
57 cy of NOX4 resulted in reduced expression of carnitine palmitoyltransferase 1A (CPT1A), which is a ke
58              Surprisingly, the inhibition of carnitine palmitoyltransferase 1a (CPT1a), which is elev
59 sing expression of the beta-oxidation enzyme carnitine palmitoyltransferase 1A (CPT1A).
60 rile (PCN) down-regulated the mRNA levels of carnitine palmitoyltransferase 1A (in beta-oxidation) an
61 ation of transmembrane domain 2 (TM2) of rat carnitine palmitoyltransferase 1A (rCPT1A), to elucidate
62 ration of metabolic inputs is underpinned by carnitine palmitoyltransferase 1A and adenosine tri-phos
63 desaturase 1, cluster of differentiation 36, carnitine palmitoyltransferase 1A and the perilipin fami
64 metabolism genes acyl coenzyme A oxidase and carnitine palmitoyltransferase 1A in livers of alcohol-f
65 hosphoenolpyruvate carboxykinase and CPT-1a (carnitine palmitoyltransferase 1a) genes.
66                                       CPT1a (carnitine palmitoyltransferase 1a) in the liver mitochon
67 n, including carnitine O-octaniltransferase, carnitine palmitoyltransferase 1A, hydroxyacyl-CoA-dehyd
68 es and cholesterol and altered expression of carnitine palmitoyltransferase 1a, sterol regulatory ele
69  and gain-of-function experiments identified carnitine palmitoyltransferase -1a (CPT1a), a key regula
70 d the cg00574958 DNA methylation site at the carnitine palmitoyltransferase-1A (CPT1A) gene to be ass
71 tor-activated receptor-gamma (PPARgamma) and carnitine palmitoyltransferase 1alpha (CPT1alpha).
72 oactivator 1alpha, uncoupling protein 1, and carnitine palmitoyltransferase 1alpha, were increased by
73 ce with skeletal muscle-specific deletion of carnitine palmitoyltransferase 1b (Cpt1b(M-/-)), which l
74 ondrial matrix, which requires the action of carnitine palmitoyltransferase 1B (CPT1B) in striated mu
75  malonyl-CoA with simultaneous inhibition of carnitine palmitoyltransferase 1b and 2) catalyze the pa
76 in amino acids (BCAA) and fatty acids (e.g., carnitine palmitoyltransferase 1B).
77     These effects, coupled with an increased carnitine palmitoyltransferase 1b, led to increased fatt
78 imiting for glucose oxidation and suppresses carnitine palmitoyltransferase-1B (CPT-1B), a key enzyme
79                Transcriptional regulation of carnitine palmitoyltransferase-1beta (CPT-1beta) is coor
80                   The brain-specific isoform carnitine palmitoyltransferase 1C (CPT1C) has been impli
81            We demonstrate in HeLa cells that carnitine palmitoyltransferase 1C (CPT1C) senses malonyl
82  of the constituents of the AMPAR complex is carnitine palmitoyltransferase 1C (CPT1C), a brain-speci
83 ndrial long-chain fatty acid oxidation using carnitine palmitoyltransferase 2 (Cpt2(L-/-)) mice.
84 AC18:1)/AC2:0, an index for the diagnosis of carnitine palmitoyltransferase 2 (CPT2) deficiency, was
85 drial stress, using a cardiomyocyte-specific carnitine palmitoyltransferase 2 (CPT2) knockout model.
86 36 in the pyruvate dehydrogenase complex and carnitine palmitoyltransferase 2 (CPT2) lysine 457/8, in
87 e, CB-839-resistant TNBC cells had increased carnitine palmitoyltransferase 2 (CPT2) protein and CPT1
88 cluding acetyl-CoA carboxylase A (ACACA) and carnitine palmitoyltransferase 2 (CPT2), leading to the
89 tochondrial long-chain fatty acid oxidation, carnitine palmitoyltransferase 2 (CPT2), on muscle and h
90                                  Deletion of Carnitine palmitoyltransferase 2 (Cpt2), the rate-limiti
91 rotein upregulated in liver-specific KOs for carnitine palmitoyltransferase 2 and pyruvate carboxylas
92 asting, which is amplified in liver-specific carnitine palmitoyltransferase 2 knockout mice (Cpt2(L-/
93 O) mouse incapable of FAO due to the loss of carnitine palmitoyltransferase 2, the product of an obli
94 52 to alanine resulted in 50 and 93% loss in carnitine palmitoyltransferase activity, respectively.
95                  Functional assays show that carnitine palmitoyltransferase acylates homocarnitine to
96  whereas, in obese Zucker rat hearts, muscle carnitine palmitoyltransferase and medium-chain acyl-CoA
97 al membranes express two active but distinct carnitine palmitoyltransferases: carnitine palmitoyltran
98 oefficients for mitochondrial outer-membrane carnitine palmitoyltransferase (CPT I) over hepatic keto
99                                              Carnitine palmitoyltransferase (CPT) 1A adopts a polytop
100                                              Carnitine palmitoyltransferase (CPT) I catalyzes the con
101 ng-chain fatty acids in the early 1960s, the carnitine palmitoyltransferase (CPT) system has since co
102  with some component(s) of the mitochondrial carnitine palmitoyltransferase (CPT) system.
103  set out to determine if the cDNA encoding a carnitine palmitoyltransferase (CPT)-like protein recent
104                                 Depletion of carnitine palmitoyltransferase (CPT)2 activity through p
105 idea that malonyl-coenzyme A (CoA)-sensitive carnitine palmitoyltransferase (CPT-I) is localized on t
106 ated with increased expression of the muscle carnitine palmitoyltransferase (CPT-I) isoform as measur
107 nction and altered lipid metabolism and that carnitine palmitoyltransferases (CPT) have a major role
108 the metabolic channeling of acyl-CoA through carnitine palmitoyltransferases (CPT-1/2) and attenuated
109 tradiol inhibited hypothalamic expression of carnitine palmitoyltransferase (CPT1a and CPT1c) and pyr
110 iminished the T(3) induction of the Pdk4 and carnitine palmitoyltransferase (Cpt1a) genes.
111      We reported that T(3) induces genes for carnitine palmitoyltransferase (cpt1a), pyruvate dehydro
112 s were transduced with adenoviruses encoding carnitine palmitoyltransferase I (CPT I) isoforms or bet
113 nd an inhibitor of the two known isoforms of carnitine palmitoyltransferase I (CPT I), which control
114                                              Carnitine palmitoyltransferase I (CPT-I) catalyzes the r
115                                              Carnitine palmitoyltransferase I (CPT-I) catalyzes the r
116                                              Carnitine palmitoyltransferase I (CPT-I) catalyzes the t
117                                              Carnitine palmitoyltransferase I (CPT-I) is a key enzyme
118 oxidase], and mitochondrial differentiation [carnitine palmitoyltransferase I (CPT-I) isoforms] were
119 ptake of fatty acids and their expression of carnitine palmitoyltransferase I (CPT1A), a critical enz
120 d by the outer mitochondrial membrane enzyme carnitine palmitoyltransferase I (CPTI) and inhibited by
121                                              Carnitine palmitoyltransferase I (CPTI) catalyzes the co
122 ut distinct carnitine palmitoyltransferases: carnitine palmitoyltransferase I (CPTI), which is malony
123     Using deletion mutants of rat liver-type carnitine palmitoyltransferase I (L-CPT I) expressed in
124  N-terminal amino acid residues of rat liver carnitine palmitoyltransferase I (L-CPTI) are essential
125  N-terminal amino acid residues of rat liver carnitine palmitoyltransferase I (L-CPTI) on malonyl-CoA
126 n catalytic activity in the liver isoform of carnitine palmitoyltransferase I (L-CPTI), we separately
127 xidative flux, the expression of muscle-type carnitine palmitoyltransferase I (M-CPT I) was character
128 n the expression of the gene encoding muscle carnitine palmitoyltransferase I (M-CPT I), an enzyme in
129 induced accumulation of mRNA encoding muscle carnitine palmitoyltransferase I (M-CPT I), an enzyme th
130                        Heart/skeletal muscle carnitine palmitoyltransferase I (M-CPTI) is 30-100-fold
131 t in the heart, but the liver isoform (liver carnitine palmitoyltransferase I [L-CPT1]) is elevated i
132 lated to the role of ACC-beta in controlling carnitine palmitoyltransferase I activity and fatty acid
133 myotubes without affecting the activities of carnitine palmitoyltransferase I and II.
134 uscle suppress the activity of mitochondrial carnitine palmitoyltransferase I and thus fatty acid oxi
135 etabolic enzymes, pyruvate dehydrogenase and carnitine palmitoyltransferase I by modulating the level
136                                              Carnitine palmitoyltransferase I catalyzes the conversio
137                                       Muscle carnitine palmitoyltransferase I is predominant in the h
138 a shift toward increased expression of the L-carnitine palmitoyltransferase I isoform.
139 chain free fatty acids into mitochondria via carnitine palmitoyltransferase I relative to overall oxi
140 tricarboxylic acid cycle rates, flux through carnitine palmitoyltransferase I was 23% lower in hypert
141 Adv.cmv.L-CPT1 infusion (P<0.05), but muscle carnitine palmitoyltransferase I was unaffected.
142 ndrial (medium-chain acyl-CoA dehydrogenase, carnitine palmitoyltransferase I) and extramitochondrial
143 (+) or without (-) etomoxir (an inhibitor of carnitine palmitoyltransferase I).
144 tty acid oxidation through the inhibition of carnitine palmitoyltransferase I, a mitochondrial compon
145 oncentration of malonyl-CoA, an inhibitor of carnitine palmitoyltransferase I, have been linked to th
146            CPT-IA and CPT-IB are isoforms of carnitine palmitoyltransferase I, of which CPT-IA is exp
147 activated receptor alpha target, muscle-type carnitine palmitoyltransferase I, providing a second mec
148 me for fatty acid oxidation in mitochondria, carnitine palmitoyltransferase I; and by reduction of su
149                                              Carnitine palmitoyltransferase-I (CPT-I) catalyzes the r
150 FAO by pretreatment of fasting rats with the carnitine palmitoyltransferase-I (CPT-I) inhibitor reduc
151 ent, endogenous, and allosteric inhibitor of carnitine palmitoyltransferase-I (CPT-I), a key enzyme f
152  sequence coverage for the membrane proteins carnitine palmitoyltransferase-I (CPT-I), long-chain acy
153 ons in intramuscular pH (acidosis) attenuate carnitine palmitoyltransferase-I (CPT-I)-supported bioen
154                                      Hepatic carnitine palmitoyltransferase-I (CPT-IL) isolated from
155 idative responses to fasting are maintained; carnitine palmitoyltransferase-I induction and glucose l
156 tic expression of enzymes of fat catabolism (carnitine palmitoyltransferase-I, acyl-CoA oxidase, and
157                        Finally, knockdown of carnitine palmitoyltransferase IA in an AML patient-deri
158 rowth and differentiation factor 15), CPT1B (carnitine palmitoyltransferase IB)-protein and oral anti
159  analysis of the 5'-flanking sequence of the carnitine palmitoyltransferase Ibeta (CPT-Ibeta) gene de
160 onyl coA-sensitive and detergent-labile; and carnitine palmitoyltransferase II (CPTII), which is malo
161 is presentation closely resembles adult-type carnitine palmitoyltransferase II deficiency except that
162            Biallelic mutations in CPT2 cause carnitine palmitoyltransferase II deficiency, sometimes

 
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