ライフサイエンスコーパス: carotid artery

1 ima, media, or atherosclerotic plaque in the carotid artery.

2 were implanted unilaterally in the internal carotid artery.

3 s and maturation of a plexus surrounding the carotid artery.

4 tion followed by ligation of the left common carotid artery.

5 hest prevalence in the intracranial internal carotid artery.

6 or pediatric cardiac catheterization via the carotid artery.

7 creased 90% 14 d after balloon injury in rat carotid artery.

8 differential gene expression profiles in the carotid artery.

9 M1-/proximal M2-segment or terminal internal carotid artery.

10 mm) wide neck aneurysm of the right internal carotid artery.

11 d by temporarily occluding the contralateral carotid artery.

12 ft middle cerebral artery and right internal carotid artery.

13 minal imaging was performed in healthy swine carotid arteries.

14 ical properties in phantom and human cadaver carotid arteries.

15 l carotid artery) in both the left and right carotid arteries.

16 ed macrophage content and lesion size in the carotid arteries.

17 lar cell adhesion molecule 1 in wire-injured carotid arteries.

18 order of 100 mm Hg at the cranial end of the carotid arteries.

19 therosclerosis has been mainly researched in carotid arteries.

20 out mice showed enhanced constriction in the carotid arteries.

21 ma compared with normal tissue from the same carotid arteries.

22 ery conditions, were observed in stented rat carotid arteries.

23 ing, coronary CT angiography, and MRI of the carotid arteries.

24 an important role in the remodelling of the carotid arteries.

25 th young mice in both ligated and un-ligated carotid arteries.

26 ion and induction of disturbed flow in mouse carotid arteries.

27 velocities and low blood volume flow in the carotid arteries after ligation in FVB mice.

28 TSPAN2 expression is attenuated in mouse carotid arteries after ligation injury and in failed hum

29 scently-labeled solutes injected through the carotid artery after tDCS.

30 underwent magnetic resonance imaging of the carotid arteries and aorta.

31 light-sheet microscopy were applied to image carotid arteries and brachiocephalic arteries, allowing

32 Moreover, studies of experimental murine carotid arteries and cultured EC revealed that TWIST1 wa

33 liac arteries but not the brachial or common carotid arteries and not correlated significantly with b

34 expression of 5-HTT was elevated in injured carotid arteries and over-expression of 5-HTT induced pr

35 alizes with SMCs in the neointima of injured carotid arteries and promotes neointima formation in the

36 ine solution was used to inject the external carotid artery and a collagen sponge was positioned over

37 induced by endothelial abrasion of the right carotid artery and abdominal aorta of 7 rabbits fed an a

38 abetic rhesus monkeys with CGM probes in the carotid artery and collected glucose values at a frequen

39 for GPIbalpha have delayed thrombosis after carotid artery and cremaster microvascular injury withou

40 Carotid artery and echocardiographic abnormalities, and

41 cine model of synthetic graft placed between carotid artery and ipsilateral jugular vein was used to

42 with stenotic, aneurysmal, dissection of the carotid artery and its branches?

43 ifferent geometries were observed within the carotid artery and scored by volumetric analysis.

44 Brain uptake of (89)Zr-BVDFO on carotid artery and tail vein infusion with an intact BBB

45 e of the atherosclerotic plaques in both the carotid artery and the aortic root.

46 nching of the internal intracranial internal carotid artery and the basilar artery.

47 he proximal and distal portion of the common carotid artery and the mean value was taken.

48 cclusion after stroke (intracranial internal carotid artery and/or middle cerebral artery M1 and/or M

49 ng of the large elastic arteries (aortic and carotid arteries) and endothelial dysfunction.

50 hoGEF tyrosine phosphorylation in rat common carotid arteries, and siRNA-mediated down-regulation of

51 catheter sheath was inserted into the common carotid artery, and a 0.035-cm (0.014-in) guidewire was

52 large or giant wide-necked proximal internal carotid artery aneurysms.

53 ents (nine men, two women) undergoing common carotid artery angiography.

54 ing those the endothelium experiences in the carotid artery are responsible for determining the fate

55 tic plaques of the abdominal aorta and right carotid artery as compared with normal control arteries

56 tic plaques of the abdominal aorta and right carotid artery as compared with normal control arteries

57 nonclassical monocytes patrol inside healthy carotid arteries at a velocity of 36 mum/min, 3x faster

58 Extracranial internal carotid artery atherosclerotic occlusive disease is a co

59 th burst incidence was less sensitive as the carotid artery became stiffer in older men and women, wh

60 ed with burst area was more sensitive as the carotid artery became stiffer in older women but not in

61 ar carotid interventions on the extracranial carotid artery between 2009 and 2014.

62 anatomic and functional status of the human carotid artery bifurcation noninvasively.

63 of the intima-media thickness of both common carotid arteries blinded from the randomization arm.

64 Chronic reductions in carotid artery blood flow are associated with increased

65 n end-tidal PCO2 and blood pressure External carotid artery blood flow increased by ~43% during both

66 sive heat stress, with no change in internal carotid artery blood flow Neurovascular coupling (i.e. t

67 hibited endothelial recovery in wire-injured carotid arteries, but this effect was also abrogated by

68 he carotid plaques and contralateral plaques/carotid arteries by an experienced radionuclide radiolog

69 Intracranial carotid artery calcifications (ICACs) are one type of ca

70 delivery to the cortex from the ipsilateral carotid artery can be improved by temporarily occluding

71 ow increased mean blood pressure measured by carotid artery cannulation and increased microvascular r

72 on of endovascular therapy as a component of carotid artery care.

73 otid IMT was measured at 3 locations (common carotid artery, carotid bulb, internal carotid artery) i

74 of anti-ICAM-1 and anti-ICAM-1/liposomes via carotid artery catheter provided an additional respectiv

75 Common carotid artery (CCA) balloon angioplasty injury was perf

76 Bilateral common carotid artery (CCA) ligation (n = 24) or sham-operation

77 coil filter directly placed into both common carotid arteries (CCAs) was designed to capture emboli >

78 eudo-occlusion (PO) of the cervical internal carotid artery (cICA) can be caused by distal ICA occlus

79 er RNA and protein levels in plaque-invested carotid arteries compared with control arteries.

80 neointima and adventitia of the ligated left carotid arteries compared with the right.

81 in expression were observed in ligated mouse carotid arteries, correlating closely with expression of

82 es (CSE activity high) and plaque-containing carotid arteries (CSE activity low); (2) cultured human

83 ciency lowered the time to thrombosis in the carotid artery despite platelet function not being affec

84 AR, which involves ultrasound measurement of carotid artery diameter during sympathetic stimulation p

85 ndothelial dysfunction [area-under-the-curve carotid artery dilatation to acetylcholine in young: 345

86 Moyamoya disease, characterised by internal carotid artery dilatation, terminal segment stenosis and

87 bal cerebral tissue injury, and cognition in carotid artery disease (CAD).

88 To compare prevalence of carotid artery disease and its various types of lesions

89 ress the risks and benefits of screening for carotid artery disease as well as how to apply the guide

90 ntly, advances in percutaneous therapies for carotid artery disease have been reported and provide a

91 olism and hemodynamic disturbances caused by carotid artery disease in these disorders is discussed.

92 Ten patients with carotid artery disease underwent high-resolution MR imag

93 tal (10 symptomatic and 10 with asymptomatic carotid artery disease) had ferumoxytol-enhanced MR imag

94 ers for noninvasive label-free assessment of carotid artery disease.

95 in the care of the patient with asymptomatic carotid artery disease.

96 gnificantly more likely to have coronary and carotid artery diseases.

97 the detection of structural deterioration in carotid artery diseases.

98 ss, telomere elongation, genome instability, carotid artery distension and increased intima-media thi

99 iPhone camera was held in a cradle over the carotid artery during iPhone measurements.

100 quires a revision procedure such as external carotid artery (ECA) ligation or embolisation.

101 ter Embozene(R) embolization of the external carotid artery (ECA).

102 improved per-patient rate of weight gain or carotid artery echodensity; 71.0% of participants succee

103 We assessed carotid artery EDD and aPWV across the lifespan in mice

104 iveness of carotid artery stenting (CAS) and carotid artery endarterectomy (CEA) for the prevention o

105 ry, comprehensive ophthalmic evaluation, and carotid artery evaluation (by Doppler/angiography) on th

106 1 was enriched in calcified regions of human carotid arteries, examined by immunohistochemistry.

107 erformed the tail-vein bleeding test and the carotid artery ferric chloride-induced thrombosis model.

108 in adults: intima-media thickness of common carotid artery ([Formula: see text]), carotid plaque (CP

109 Microarrays of carotid arteries from Pcsk6(-/-) versus control mice rev

110 Three weeks post-PCL, ligated carotid arteries had greater intima media thickness, neo

111 BackgroundStenosis of the internal carotid artery has a higher risk for stroke.

112 umber and size of atherosclerotic plaques in carotid artery, heart, aortic arch and aorta in acute an

113 Blood flow (Q) in the internal and external carotid arteries (ICA and ECA, respectively) and vertebr

114 lood flow (CBF) was measured at the internal carotid artery (ICA) and vertebral artery (VA) and CBF v

115 neurysm in the cavernous segment of internal carotid artery (ICA) is a relatively rare entity.

116 graphy to distinguish true cervical internal carotid artery (ICA) occlusion from pseudo-occlusion (de

117 eginning at the cervical segment of internal carotid artery (ICA) together with a dissecting aneurysm

118 >18 years with vasospasm>50% of the internal carotid artery (ICA), anterior cerebral artery (ACA), an

119 randomized, first-in-human clinical CAPTURE (Carotid Artery Implant for Trapping Upstream Emboli for

120 (Carotid Artery Implant for Trapping Upstream Emboli for

121 sion of Abeta-activated platelets to injured carotid arteries in mice.

122 y partial carotid ligation (PCL) of the left carotid artery in young and old, normal chow (NC) or WD

123 ommon carotid artery, carotid bulb, internal carotid artery) in both the left and right carotid arter

124 Here we show that vascular injury in rodent carotid arteries induces YY1 expression along with reduc

125 d with higher risk of stroke due to internal carotid artery injuries, but monitoring was not useful f

126 Among those with isolated internal carotid artery injuries, five of nine with delayed strok

127 g for ipsilateral and contralateral internal carotid artery injury grade (adjusted risk ratio, 2.91;

128 ient mice have reduced thrombus growth after carotid artery injury relative to conventionally raised

129 Among patients who sustained an internal carotid artery injury with or without additional vessel

130 hrombosis was measured in vivo after FeCl(3) carotid artery injury, endothelial COX1 deletion acceler

131 Using 3 complementary mouse models of carotid artery injury, we demonstrated that both tamoxif

132 ponse is necessary for EC regeneration after carotid artery injury.

133 ition and between-group comparison of common carotid arteries intima-media thickness change.

134 The change in common carotid arteries intima-media thickness was -2.69 um (95

135 Data on common carotid artery intima-media thickness (CCA-IMT) were ava

136 We investigated the association of common carotid artery intima-media thickness (CCA-IMT) with sno

137 D) and evaluated the association with common carotid artery intima-media thickness (cCIMT) using mult

138 erosis, as measured by progression of common carotid artery intima-media thickness (cIMT), in adults

139 Mean common carotid artery intima-media thickness (IMT) and pulse wa

140 Mean common carotid artery intima-media thickness (IMT) and pulse-wa

141 gression of coronary artery calcium and mean carotid artery intima-media thickness and long-term expo

142 ected adults and evaluated associations with carotid artery intima-media thickness and plaque.

143 variables were significantly associated with carotid artery intima-media thickness estimates except f

144 Subclinical atherosclerosis was defined as carotid artery intima-media thickness of 780 mum or high

145 er high-density lipoprotein cholesterol with Carotid artery intima-media thickness strengthened with

146 METHODS AND Carotid artery intima-media thickness was measured at ba

147 h subclinical atherosclerosis as assessed by carotid artery intima-media thickness, while controlling

148 brachial artery flow-mediated dilatation and carotid artery intima-medial hyperplasia.

149 he primary outcome was the rate of change in carotid-artery intima-media thickness (CIMT), which was

150 rial was to determine the safety of internal carotid artery, intra-arterially delivered autologous bo

151 (control) starting 7 days before end-to-side carotid artery-jugular vein fistula creation and for up

152 l (3D) profiling of the vascular response to carotid artery ligation and induction of atherosclerosis

153 formation, we induced neointima formation by carotid artery ligation in Nf1(+/-) and WT mice with gen

154 ), reduced neointimal formation in the mouse carotid artery ligation model.

155 Furthermore, in Tcad/ApoE-DKO mice, carotid artery ligation resulted in a significant increa

156 ted in an atherosclerosis model generated by carotid artery ligation.

157 compared with the contralateral asymptomatic carotid arteries (median: 1.4; Q1-Q3, 1.3-1.6).

158 Five ex vivo porcine carotid artery models (n = 6 each) were compared-native,

159 En face staining of the murine aorta or carotid arteries modified with flow-altering cuffs demon

160 sion and VaD was induced by bilateral common carotid artery occlusion (BCCAO) in adult male Sprague D

161 Permanent bilateral common carotid artery occlusion (BCCAO) was used as a model of

162 term experimental permanent bilateral common carotid artery occlusion (BCCAO).

163 ation and brain infarct injury in the middle carotid artery occlusion ischemia/reperfusion model.

164 hemic injury was induced by bilateral common carotid artery occlusion, whereas severe focal stroke in

165 method to monitor retinopathy in a bilateral carotid artery occlusion-induced ocular ischemia, we obs

166 diet and modeled VCID via unilateral common carotid artery occlusion.

167 before and after ischemia induced by middle carotid artery occlusion.

168 monocrotaline, whereas it was upregulated in carotid arteries of Macaca fascicularis subjected to ath

169 Significant differences were detected in the carotid arteries of normal patients and those with aneur

170 sly with a fast intra-vascular sensor in the carotid artery of anaesthetized, mechanically ventilated

171 e left external and internal branches of the carotid artery of male FVB mice and performed sham opera

172 A catheter was inserted into the right carotid artery of mice, which acted as a vascular graft.

173 nderwent high-resolution MR imaging of their carotid arteries on a 1.5 T MR system.

174 was restricted to the territory of a single carotid artery on brain magnetic resonance imaging (MRI)

175 vein opacification in patients with internal carotid artery or middle cerebral artery (MCA) stroke an

176 All consecutive patients with internal carotid artery or middle cerebral artery occlusions tran

177 with occlusion of the intracranial internal carotid artery or proximal middle cerebral artery who ha

178 In 24 patients with terminal internal carotid artery or the proximal middle cerebral artery oc

179 s and the extracircular part of the internal carotid artery (p < 0.05).

180 Internal carotid artery patency was 100%, while the overall final

181 Internal carotid artery peak systolic velocity was used by all ce

182 d coronary artery calcification (CARDIA) and carotid artery plaque burden (BioImage).

183 ient was greater than 0.8 in all measures of carotid artery/plaque uptake (SUV) and greater than 0.6

184 art Association-lesion type VI) nonstenosing carotid artery plaques (CAPs) in cryptogenic stroke (CS)

185 carriers showed reduced CXCR4 expression in carotid artery plaques (n=188), which was furthermore as

186 features of both T cells and macrophages in carotid artery plaques of patients with clinically sympt

187 Presence of carotid artery plaques on the involved side was signific

188 c plaques and the asymptomatic contralateral carotid arteries/plaques showed no significant differenc

189 i-viral delivery of miR-30c into injured rat carotid arteries prevented the injury-induced increase i

190 idence of peripheral artery disease), of the carotid arteries (previous carotid artery revascularisat

191 pulse pressure, systolic blood pressure, and carotid artery procedures, implicating modulation of the

192 Our aim was to examine whether carotid artery reactivity (CAR; a novel, simple procedur

193 te stiffening of the aorta compared with the carotid arteries, reducing protective impedance mismatch

194 disease), of the carotid arteries (previous carotid artery revascularisation or asymptomatic carotid

195 e short-term, outcomes of patients following carotid artery revascularization.

196 hyroid due to its proximity to the pulsating carotid artery significantly impacts the visualization o

197 P values were adjusted for age, sex, carotid artery site, and family relations.

198 ular tissues (aortic valves, atherosclerotic carotid artery specimens, abdominal aortic aneurysms) we

199 tid disease was defined as cervical internal carotid artery stenosis (>50%) or occlusion.

200 es and Canada who underwent TCAR and CEA for carotid artery stenosis (2016- 2019) were included.

201 Of 10579 individuals with a diagnosis of carotid artery stenosis (4615 women and 5964 men; mean [

202 Here we used a bilateral common carotid artery stenosis (BCAS) mouse model of VaD to inv

203 The incidence of carotid artery stenosis and plaques, cardiac embolic sou

204 ars, range, 59.4-69.7) with ipsilateral >70% carotid artery stenosis and who underwent carotid endart

205 y was queried for individuals diagnosed with carotid artery stenosis between October 1, 2006, and Sep

206 to reduce the stroke impact of asymptomatic carotid artery stenosis has proved difficult over the la

207 tid artery revascularisation or asymptomatic carotid artery stenosis of at least 50%), or coronary ar

208 The degree of carotid artery stenosis on histology correlated well wit

209 Carotid artery stenosis on the involved side was worse i

210 l/6J mice were subjected to bilateral common carotid artery stenosis or a sham operation and fed norm

211 ndard surgical risk with severe asymptomatic carotid artery stenosis randomly assigned to carotid art

212 ompared CAS against CEA for the treatment of carotid artery stenosis were selected.

213 and in those with a history of hypertension, carotid artery stenosis, aortic valve disease, smoking,

214 Female sex, hypertension, carotid artery stenosis, aortic valve disease, smoking,

215 and transfemoral carotid artery stenting for carotid artery stenosis, from September 2016 to April 20

216 ugh many factors influence the management of carotid artery stenosis, it is not well understood wheth

217 gher pulse pressure and higher prevalence of carotid artery stenosis.

218 my (CEA) for the prevention of stroke due to carotid artery stenosis.

219 provider-induced demand in the management of carotid artery stenosis.

220 ical guideline on screening for asymptomatic carotid artery stenosis.

221 arotid endarterectomy for severe symptomatic carotid artery stenosis.

222 The impact of carotid artery stent fractures on the incidence of adver

223 lict regarding the relative effectiveness of carotid artery stenting (CAS) and carotid artery endarte

224 four randomised controlled trials comparing carotid artery stenting (CAS) and carotid endarterectomy

225 epresentative 30-day readmissions data after carotid artery stenting (CAS) and carotid endarterectomy

226 h following carotid endarterectomy (CEA) and carotid artery stenting (CAS) on a national level in Ger

227 ports periprocedural outcomes in a cohort of carotid artery stenting (CAS) performed for asymptomatic

228 riprocedural stroke or death is higher after carotid artery stenting (CAS) than carotid endarterectom

229 utcome after carotid endarterectomy (CEA) or carotid artery stenting (CAS).

230 75.8 years; 43% women) and 231077 underwent carotid artery stenting (mean age, 75.4 years; 49% women

231 th symptomatic carotid stenosis treated with carotid artery stenting (n=2326) or carotid endarterecto

232 reas no significant decrease was found after carotid artery stenting (unadjusted odds ratio, 0.96; 95

233 Carotid endarterectomy and carotid artery stenting are the leading approaches to re

234 perioperative stroke following transfemoral carotid artery stenting compared with carotid endarterec

235 Of 1021 patients treated with carotid artery stenting during a mean follow-up of 3.1+/

236 id artery revascularization and transfemoral carotid artery stenting for carotid artery stenosis, fro

237 g-term effectiveness in stroke prevention by carotid artery stenting in a large number of patients in

238 ed from controlled clinical trials undergoes carotid artery stenting in daily clinical practice.

239 ggest that independent modular filter use in carotid artery stenting in high surgical risk patients i

240 nrandomized, open-label, single-arm study of carotid artery stenting in high surgical risk patients w

241 rom 1999 to 2014, whereas the performance of carotid artery stenting increased until 2006 and then de

242 carotid artery stenosis randomly assigned to carotid artery stenting or carotid endarterectomy (Abbot

243 rocedural stroke or death risk compared with carotid artery stenting patients, and the difference sig

244 Embolic protection during carotid artery stenting reduces the rate of thromboembol

245 gnificantly lower stroke rates compared with carotid artery stenting via the transfemoral approach.

246 .02 to 2.61]; P = .04), whereas transfemoral carotid artery stenting was associated with more radiati

247 tid artery revascularization or transfemoral carotid artery stenting were identified (transcarotid ap

248 ural intervention (carotid endarterectomy or carotid artery stenting) compared with medical managemen

249 er, its outcomes, compared with transfemoral carotid artery stenting, are not well characterized.

250 evascularization, compared with transfemoral carotid artery stenting, was significantly associated wi

251 ion and 6640 patients underwent transfemoral carotid artery stenting.

252 tid artery revascularization vs transfemoral carotid artery stenting.

253 platelet therapy, carotid endarterectomy and carotid artery stenting.

254 lic Protection System in patients undergoing carotid artery stenting.

255 Carotid endarterectomy and carotid artery stenting.

256 ata outside of controlled clinical trials in carotid artery stenting.

257 Carotid artery stiffness (high-resolution echo tracking)

258 ore, we investigated the association between carotid artery stiffness and incident depressive symptom

259 Older women had greater carotid artery stiffness compared with older men, while

260 burst occurrence in older women with greater carotid artery stiffness to regulate BP similar to that

261 stensibility coefficient (indicating greater carotid artery stiffness) compared with those in the hig

262 The effects of HIV on carotid artery structure may differ across the lifespan,

263 rcle of Willis upon stenosis of the internal carotid arteries, supply blood to the anterior cerebral

264 ersus control, 2.12+/-0.27; P=0.001) and the carotid arteries (TBRmax: CKD, 2.45+/-0.65 versus contro

265 ive adventitial plexus after ligation of the carotid artery that evolved and matured over time.

266 Intravital microscopy of the carotid artery, the jugular vein, and cremasteric arteri

267 in is more potent than hirulog-1 in a murine carotid artery thrombosis model.

268 s, we also performed ferric chloride-induced carotid artery thrombosis.

269 del of ferric chloride induced non-occlusive carotid artery thrombosis.

270 In vivo, reperfusion of carotid artery thrombotic occlusion was also enhanced.

271 nd neutrophilia that precedes development of carotid artery thrombus formation.

272 injected with microembolic materials in the carotid artery to serve as positive controls.

273 In mice, the carotid artery to the ipsilateral jugular vein was conne

274 were quantified in 3,392 participants using carotid artery ultrasound imaging acquired over a mean o

275 S Cohort Study who underwent repeated B-mode carotid artery ultrasound imaging in 2004-2013.

276 ligible if they included bilaterally scanned carotid arteries using ultrasonography and defined incre

277 ic stroke propensity through the left common carotid artery using an idealized aortic arch model.

278 ic plaque on carotid bifurcation or internal carotid artery using the Mannheim consensus definition a

279 nt-Protected Percutaneous Angioplasty of the Carotid Artery versus Endarterectomy trial, Internationa

280 els, vessel grade, and vessel type (internal carotid artery, vertebral artery) with BCVI-associated s

281 ke Scale score), site of occlusion (internal carotid artery vs M1 segment of middle cerebral artery v

282 ; P < 0.05), catheterization route (internal carotid artery vs. external carotid or posterior communi

283 Median carotid artery wall echodensity and carotid-femoral puls

284 and intima-media-adventitia thickness], and carotid artery wall stress [CAWS]).

285 s was measured based on displacements of the carotid artery wall, and Young's modulus was 2-fold grea

286 The endothelial damage in atherosclerotic carotid arteries was assessed by electron microscopy and

287 s grafting of the jugular vein to the common carotid artery was performed 2 weeks after starting diet

288 otion associated with the thyroid due to the carotid artery was primarily in the lateral direction, w

289 e anesthetized, and the right or left common carotid artery was surgically exposed.

290 Conclusion The human carotid artery was visualized by using handheld volumetr

291 the rigid trachea, thyroid and the pulsating carotid artery, we hypothesize that imaging of thyroid m

292 Carotid arteries were isolated from newborn (postnatal d

293 Extradural aneurysms in the internal carotid artery were included; fusiform aneurysms, infund

294 and a high number of microembolic signals in carotid artery were observed during sheath/catheter mani

295 rotid plaques and contralateral asymptomatic carotid arteries, which was corrected for background act

296 rein, we show that neointima formation after carotid artery wire injury reduces markedly in CD40(-/-)

297 METHODS AND In mouse carotid arteries with established intimal lesions tailor

298 decellularized vessels obtained from porcine carotid arteries with poly (ethylmethacrylate-co-diethyl

299 IUGR group was seen in the iliac but not the carotid arteries without between-sex differences.

300 the lumen of the proximal cervical internal carotid artery without evidence of calcification.