ライフサイエンスコーパス: carotid artery disease

1 in the care of the patient with asymptomatic carotid artery disease.

2 older and more complex patients present with carotid artery disease.

3 senescence (CD28(-)CD57(+)) with subclinical carotid artery disease.

4 Ultrasound microimaging detected carotid artery disease.

5 and stroke risk in the setting of occlusive carotid artery disease.

6 range, 26-56 years) with no known history of carotid artery disease.

7 otid artery disease than in those with right carotid artery disease.

8 (FS069) was evaluated in human subjects with carotid artery disease.

9 ood flow reserve in six patients with severe carotid artery disease.

10 oor vascular reserve in patients with severe carotid artery disease.

11 logies: abdominal aortic aneurysms (AAA) and carotid artery disease.

12 ted transcript (MIAT) to atherosclerosis and carotid artery disease.

13 oing carotid endarterectomy for asymptomatic carotid artery disease.

14 ers for noninvasive label-free assessment of carotid artery disease.

15 cularization strategy than endarterectomy in carotid-artery disease.

16 the detection of structural deterioration in carotid artery diseases.

17 gnificantly more likely to have coronary and carotid artery diseases.

18 We prospectively enrolled 61 patients with carotid artery disease (22 with occlusion, 39 with sever

19 Extracranial carotid artery disease accounts for approximately 25% of

20 three murine and one porcine animal model of carotid artery disease and AAA limited disease progressi

21 To compare prevalence of carotid artery disease and its various types of lesions

22 sment of clinically significant coronary and carotid artery disease and the guidance of treatment.

23 , history of myocardial infarction, previous carotid artery disease, and hypertension.

24 y in women with asymptomatic and symptomatic carotid artery disease, and physicians should expect com

25 t of abdominal aortic aneurysmal disease and carotid artery disease, and relevant issues for the anes

26 major treatment options for his symptomatic carotid artery disease are optimizing medical treatment,

27 ress the risks and benefits of screening for carotid artery disease as well as how to apply the guide

28 carotid endarterectomy for the treatment of carotid artery disease at 30 days and at 1 year.

29 an hospital and having giant cell arteritis, carotid artery disease, atrial fibrillation, cardiac val

30 bal cerebral tissue injury, and cognition in carotid artery disease (CAD).

31 In patients with carotid artery disease, carotid endarterectomy (CEA) and

32 ucible modeling in stroke-prone Tg25 rats of carotid artery disease, cerebral hemorrhagic infarctions

33 tal (10 symptomatic and 10 with asymptomatic carotid artery disease) had ferumoxytol-enhanced MR imag

34 ntly, advances in percutaneous therapies for carotid artery disease have been reported and provide a

35 brain ischemia caused by severe preocclusive carotid artery disease in the neck.

36 olism and hemodynamic disturbances caused by carotid artery disease in these disorders is discussed.

37 Whether carotid artery disease is a cause of cognitive impairmen

38 Potential mechanisms may include carotid artery disease or cardiac valvular disease.

39 ary revascularization, cardiovascular death, carotid artery disease, peripheral artery surgery, and u

40 n should be more common in persons with left carotid artery disease than in those with right carotid

41 After a TIA or stroke caused by carotid-artery disease, there is an increase in cerebral

42 aterial and blood samples from patients with carotid artery disease undergoing endarterectomy.

43 Ten patients with carotid artery disease underwent high-resolution MR imag

44 Carotid artery disease was evaluated (carotid ultrasound

45 n PMS studies had lower rates of symptomatic carotid artery disease within the preceding 6 months, pr