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1 provider-induced demand in the management of carotid artery stenosis.
2 mp index and SP are related to contralateral carotid artery stenosis.
3 Included patients underwent TCAR or CEA for carotid artery stenosis.
4 Sjogren's syndrome may be a risk factor for carotid artery stenosis.
5 ery blood flow, in persons with asymptomatic carotid artery stenosis.
6 atment for patients with asymptomatic severe carotid artery stenosis.
7 darterectomy (CEA) for certain patients with carotid artery stenosis.
8 ical guideline on screening for asymptomatic carotid artery stenosis.
9 most meaningful risk factor was ipsilateral carotid artery stenosis.
10 arotid endarterectomy for severe symptomatic carotid artery stenosis.
11 agnostic criteria used to classify degree of carotid artery stenosis.
12 gher pulse pressure and higher prevalence of carotid artery stenosis.
13 reatment for patients presenting symptomatic carotid artery stenosis.
14 scularization for patients with asymptomatic carotid artery stenosis.
15 ts, and stroke alone, in symptomatic >/= 50% carotid artery stenosis.
16 ons about optimal management of asymptomatic carotid artery stenosis.
17 atients with symptomatic versus asymptomatic carotid artery stenosis.
18 /low (0-15 years) SHS exposure and < or =25% carotid artery stenosis.
19 method of revascularization for extracranial carotid artery stenosis.
20 oppler US for assistance in the diagnosis of carotid artery stenosis.
21 my (CEA) for the prevention of stroke due to carotid artery stenosis.
22 ffective as CEA for treatment of symptomatic carotid artery stenosis.
23 ents that are impacting on the management of carotid artery stenosis.
24 symptomatic or asymptomatic atherosclerotic carotid-artery stenosis.
26 Of 10579 individuals with a diagnosis of carotid artery stenosis (4615 women and 5964 men; mean [
27 patients who underwent revascularization for carotid artery stenosis, 7664 patients (8.9%) underwent
28 endarterectomy are both options for treating carotid-artery stenosis, an important cause of stroke.
29 e if they had severe unilateral or bilateral carotid artery stenosis and both doctor and patient agre
30 ic velocity ratio for assessment of internal carotid artery stenosis and decrease some of the reporte
33 nt and decline associated with left internal carotid artery stenosis and intima-media thickness, afte
35 teries of 120 patients with suspected severe carotid artery stenosis and previous acute cerebral isch
36 peripheral artery disease (PAD) and incident carotid artery stenosis and progression to the first maj
37 recently symptomatic patients with suspected carotid artery stenosis and to compare this with their c
38 utcome in a clinical subset of patients with carotid artery stenosis and transient systemic hypotensi
39 ars, range, 59.4-69.7) with ipsilateral >70% carotid artery stenosis and who underwent carotid endart
42 iabetes mellitus, blood lipids, asymptomatic carotid artery stenosis), and 4 lifestyle factors (cigar
43 omy for symptomatic patients with high-grade carotid artery stenosis, and a marginal benefit for asym
45 attack, diabetes mellitus, or hypertension; carotid artery stenosis; ankle-arm blood pressure index;
46 and in those with a history of hypertension, carotid artery stenosis, aortic valve disease, smoking,
52 7BL/6J stroke mouse models (bilateral common carotid artery stenosis [BCCAS] and middle cerebral arte
53 y was queried for individuals diagnosed with carotid artery stenosis between October 1, 2006, and Sep
55 an alternative to carotid endarterectomy for carotid artery stenosis but there are scarce long-term e
56 ternative to endarterectomy for treatment of carotid artery stenosis, but long-term efficacy is uncer
57 orders including coronary artery disease and carotid artery stenosis, but their association with Alzh
58 F1a deficiency restrains the pathogenesis of carotid artery stenosis by rewiring inflammatory and met
59 uld be offered CAS as a treatment option for carotid artery stenosis by vascular surgeons or interven
60 ry disease (PAD), and ultrasound imaging for carotid artery stenosis (CAS) >50% and abdominal aortic
64 ific hazards models, hypertension, diabetes, carotid artery stenosis, coronary artery disease, and pr
65 tions: atrial fibrillation, Crohn's disease, carotid artery stenosis, coronary artery disease, multip
67 d need to improve selection of patients with carotid artery stenosis for carotid endarterectomy (CEA)
68 and transfemoral carotid artery stenting for carotid artery stenosis, from September 2016 to April 20
69 usion, and presence of extracranial internal carotid artery stenosis (>50%) demonstrated on pre-MT co
71 ects with >25 years of SHS exposure and >25% carotid artery stenosis had a 3-fold increase (hazard ra
72 to reduce the stroke impact of asymptomatic carotid artery stenosis has proved difficult over the la
73 rgence of percutaneous revascularization for carotid artery stenosis has raised further questions abo
74 arterectomy, medical therapy of asymptomatic carotid artery stenosis has reduced rates of stroke to a
75 ndomized trial for treatment of asymptomatic carotid artery stenosis have helped to establish the ind
77 t hazard ratios for 1-year mortality whereas carotid artery stenosis, hyperlipidemia, and hypertensio
78 efficacious alternative for the treatment of carotid artery stenosis in a veteran population and outc
79 and cost-effective screening examination for carotid artery stenosis in asymptomatic populations.
81 ecommends against screening for asymptomatic carotid artery stenosis in the general adult population.
84 itant diseases such as diabetes mellitus and carotid artery stenosis influences the risk of severe in
85 raft, lower extremity revascularization, and carotid artery stenosis interventions and higher rates o
88 edical therapy in patients with asymptomatic carotid artery stenosis is predicated upon a life expect
89 monocular blindness associated with internal-carotid-artery stenosis is a risk factor for stroke.
90 stroke ipsilateral to severe atherosclerotic carotid-artery stenosis is offset by complications durin
91 ugh many factors influence the management of carotid artery stenosis, it is not well understood wheth
92 n significant variation in classification of carotid artery stenosis, likely leading to differences i
94 o findings were further confirmed in a mouse carotid artery stenosis model, where the administration
95 tool to identify patients with asymptomatic carotid artery stenosis most likely to benefit from caro
96 rent cerebral ischemic events in symptomatic carotid artery stenosis (odds ratio = 12.2, 95% CI = 5.5
97 with established coronary artery disease or carotid artery stenosis of 15% or greater determined by
98 endarterectomy who had either a symptomatic carotid artery stenosis of at least 50% of the luminal d
99 tid artery revascularisation or asymptomatic carotid artery stenosis of at least 50%), or coronary ar
102 arterectomy and who had either a symptomatic carotid-artery stenosis of at least 50 percent of the lu
103 tigated the impact of contralateral internal carotid artery stenosis on carotid artery stump pressure
106 l/6J mice were subjected to bilateral common carotid artery stenosis or a sham operation and fed norm
107 hemia (OR, 7.67; CI, 5.31-11.07; P < 0.001), carotid artery stenosis (OR, 7.52; CI, 6.22-9.09; P < 0.
109 ndard surgical risk with severe asymptomatic carotid artery stenosis randomly assigned to carotid art
113 1.09, 1.29), stroke (RR = 1.35, 1.20, 1.53), carotid artery stenosis (RR = 1.32, 1.06, 1.64), and isc
115 on Recommendation Statement on screening for carotid artery stenosis summarizes published evidence on
116 tion of stroke for patients with symptomatic carotid artery stenosis, surgical intervention as a part
117 itant diseases such as diabetes mellitus and carotid artery stenosis, the presence of diabetic retino
118 agreement between the tests for symptomatic carotid artery stenosis; to compare ipsilateral with con
119 pective analysis of patients with high-grade carotid artery stenosis treated with CEA or CAS by a vas
120 outcomes in high-surgical-risk patients with carotid artery stenosis treated with the Carotid WALLSTE
121 comparable hypoperfusion in adult mice using carotid artery stenosis triggered a similar tissue patho
122 Thirty-one patients with 50%-99% internal carotid artery stenosis underwent dynamic contrast-enhan
124 stenting for symptomatic > or =70% internal carotid artery stenosis were randomized in a double-blin
126 ith cerebral embolic protection for internal carotid artery stenosis were randomly assigned to proxim
130 were modeled for patients with asymptomatic carotid artery stenosis with 70%-89% and 50%-69% carotid
131 s, the benefits of treatment of asymptomatic carotid artery stenosis with carotid endarterectomy (CEA
132 ich the subset of patients with asymptomatic carotid artery stenosis with IPH on MR images would unde