ライフサイエンスコーパス: carotid occlusion

1 of brain ischemia and 4 were excluded due to carotid occlusion.

2 peripheral artery disease, and contralateral carotid occlusion.

3 ge test results will do well after permanent carotid occlusion.

4 ing a cohort of 45 symptomatic patients with carotid occlusion.

5 redicted stroke in patients with symptomatic carotid occlusion.

6 accurately predicts stroke in patients with carotid occlusion.

7 l disease, and 28 (12%) having contralateral carotid occlusion.

8 may be an unusual presentation of impending carotid occlusion.

9 ct injury, and to 12.3 +/- 5.6 mmol/L before carotid occlusion.

10 t (velocity 4 m/sec) injury followed by sham carotid occlusion.

11 ham operated or subjected to 5 min bilateral carotid occlusion.

12 ombogenicity as reflected by delayed time to carotid occlusion.

13 fered carotid stenting for the prevention of carotid occlusion.

14 eptides in response to muscle contraction or carotid occlusions.

15 mice were subjected to two vessel (bilateral carotid) occlusion (2VO) or 2VO plus systemic hypotensio

16 teral hemispheric stroke following permanent carotid occlusion after the superficial temporal artery

17 The impact injury alone and bilateral carotid occlusion alone caused minimal neuronal loss in

18 n gerbils were submitted to 30 min bilateral carotid occlusion and 2 h of reperfusion at 37 degreesC

19 One of them underwent permanent carotid occlusion and did not develop any delayed ischem

20 In patients with chronic carotid occlusion and increased OEF, increased CBV may i

21 f normothermic forebrain ischemia (bilateral carotid occlusion and MABP=30 mmHg) and allowed to recov

22 end of the cut tibial nerve, brief repeated carotid occlusions and carotid sinus nerve stimulations

23 prespecified subgroups of age, contralateral carotid occlusion, and baseline surgical risk.

24 inical or animal studies have evaluated mild carotid occlusion, and few examined unilateral occlusion

25 ina, previous ipsilateral CEA, contralateral carotid occlusion, and other severe comorbid illnesses.

26 ipsilateral stroke and TIA in patients with carotid occlusion, and to a lesser extent in asymptomati

27 (108.2 +/- 1.4 mm Hg) was unaffected by the carotid occlusions, and was similar among animals and co

28 pact injury followed by 40 mins of bilateral carotid occlusion; and c) 2.5-mm deformation impact (vel

29 al arterial thrombosis, as tested in vivo by carotid occlusion assays.

30 sec) injury followed by 40 mins of bilateral carotid occlusion; b) sham impact injury followed by 40

31 Brief carotid occlusions caused a release of irNPYs from the l

32 Only the brief carotid occlusions caused a similar increase in MABP and

33 The presence of a contralateral carotid occlusion (CCO) is an established high-risk feat

34 > or =60% ACS in patients with contralateral carotid occlusion (CCO).

35 d CT imaging revealed thoracic aortitis with carotid occlusion, coronary artery stenosis, ischemic st

36 A new carotid occlusion during annual monitoring with carotid

37 iewed data from 81 patients with symptomatic carotid occlusion enrolled in a prospective study of hae

38 During global cerebral ischemia induced by carotid occlusion, flow to all regions was reduced by ne

39 Using rats with unilateral carotid occlusion followed by hypoxia at postnatal day 7

40 duces delayed post-ischemic (5 min bilateral carotid occlusion) hippocampal CA1 neuronal degeneration

41 After a single 6-min bilateral carotid occlusion, histological damage was evident in th

42 n mice with a dose-dependent protection from carotid occlusion in a ferric chloride-induced thrombosi

43 of developing an infarct following permanent carotid occlusion in the course of brain surgery.

44 Carotid occlusions induced c-Fos-ir expression in the ar

45 We have found that transient carotid occlusion induces a wide variation in histologic

46 The risk of progression to carotid occlusion is well below the risk of carotid sten

47 Preventing carotid occlusion may not be a valid indication for sten

48 n dioxide in air in 107 patients with either carotid occlusion (n = 48) or asymptomatic carotid steno

49 estar rats underwent bilateral or unilateral carotid occlusion of 28-45%.

50 sk patients such as those with contralateral carotid occlusion or bilateral severe stenosis.

51 N attenuated the cardiovascular responses to carotid occlusions, or altered the pattern of release of

52 in the whole group (P: < 0.00001) and in the carotid occlusion (P: = 0.019) and carotid stenosis (P:

53 cle 1 h before (but not 6 h after) bilateral carotid occlusion prevented the ischemia-induced decreas

54 sed in cases with demonstrated contralateral carotid occlusion, prior cerebrovascular accident (CVA),

55 surgical or endovascular treatment such as a carotid occlusion procedure or stent-graft placement.

56 Bilateral carotid occlusion produces extensive neuronal damage in

57 occlusion, 5 minutes of transient bilateral carotid occlusion (purported to cause negligible adenosi

58 These patients with symptomatic carotid occlusion should not be discouraged from air tra

59 The St. Louis Carotid Occlusion Study (STLCOS) demonstrated that incre

60 Records from the Carotid Occlusion Surgery Study (COSS), a randomised tri

61 ese patients underwent permanent therapeutic carotid occlusion; three patients had subsequent infarct

62 nt for both MRP-14 and CD36 failed to reduce carotid occlusion times, indicating that CD36 is require

63 Seventy-seven patients with symptomatic carotid occlusion travelled by aeroplane to a single PET

64 les River Laboratories to 5 min of bilateral carotid occlusion under continuous striatal temperature

65 of cerebral hemodynamics, 110 patients with carotid occlusion underwent (a) positron emission tomogr

66 ly enhanced with antecedent coronary-but not carotid-occlusion versus controls.

67 ereas DNase or FVII knockdown had no effect, carotid occlusion was abrogated with RNase or FXII knock

68 Transient carotid occlusion was elicited with a hydraulic occluder

69 tolerance group, in which a 2-min bilateral carotid occlusion was followed 3 days later by a 6-min i

70 of stroke in patients with symptoms and with carotid occlusion was repeated by substituting a count-b

71 sustained attention that persisted until the carotid occlusion was reversed.

72 Both muscle contractions and carotid occlusions were performed 3, 6 and 12 h after th

73 radical output, and thrombus formation, and carotid occlusion, while tail hemostasis was unaffected.

74 ines a subgroup of patients with symptomatic carotid occlusion who are at high risk for subsequent st

75 cost-effective in patients with symptomatic carotid occlusion who have increased OEF.

76 In the past two decades unilateral carotid occlusion with 8% hypoxia has been used to study

77 ult rats were exposed to 10 min of bilateral carotid occlusion with simultaneous hypotension.