ライフサイエンスコーパス: catalase

1 position of the hydrogen peroxide residue by catalase).

2 ioxidant enzymes (glutathione peroxidase and catalase).

3 larly in the presence of a competing enzyme (catalase).

4 tantial increase in production of KatB, a Mn-catalase.

5 ugs in 7H9 broth-oleic acid albumin dextrose catalase.

6 s enzymes, superoxide dismutase 2 (SOD2) and catalase.

7 arrier, compared to the "dry" active site in catalase.

8 ss-induced DNA damage, which was reversed by catalase.

9 he decomposition of H(2)O(2) by bovine liver catalase.

10 al antioxidant enzyme (AOE) genes, including catalase.

11 .e., glycolate oxidase, oxalate oxidase, and catalase.

12 roxide (H2O2)-scavenging pathways, including catalases.

13 a Compound II-like intermediate, similar to catalases.

14 Catalase 2 (CAT2) plays an important role in the detoxif

15 BIBR-1532 69.9+/-4.0, L-NAME 84.7+/-2.2, PEG-catalase 36.5+/-6.9*).

16 AGS 499 78.5+/-3.9; L-NAME 10.9+/-17.5*; PEG-catalase 79.2+/-4.9).

17 vehicle 74.6+/-4.1, L-NAME 37.0+/-2.0*, PEG-catalase 82.1+/-2.8; BIBR-1532 69.9+/-4.0, L-NAME 84.7+/

18 Catalase, a conserved and abundant enzyme found in all d

19 In catalase, a diminished potential would increase the sele

20 Our results reveal epigenetic silencing of Catalase, a key regulator of oxidative stress and DNA da

21 al progression because ectopic expression of catalase, a ROS scavenger, halted the in vitro-induced d

22 Pretreatment of cells with catalase, a scavenger of H2O2, or DUOX1 down-regulation

23 Pretreatment with catalase abrogated these effects, indicating a key role

24 sent in the superoxidized manganese (III/IV) catalase active site is determined by comparing experime

25 Decreased glycolate oxidase and catalase activities together with accumulation of glycol

26 tudy combines the use of glucose oxidase and catalase activities with the aim of rapid conversion of

27 a number of nanozymes showing peroxidase or catalase activities.

28 tosan enhanced total phenols, ascorbic acid, catalase activity and free radical scavenging activity d

29 Insulin-resistant iPSC also showed reduced catalase activity and increased susceptibility to oxidat

30 on-oxidizable phenylalanine exhibited higher catalase activity and less accumulation of off-pathway h

31 Mutant nrx1 plants displayed reduced catalase activity and were hypersensitive to oxidative s

32 alis with heme increased growth and restored catalase activity but resulted in decreased survival in

33 imarily of Trp-321, and PxEDs stimulate KatG catalase activity by preventing the accumulation of inac

34 The examination of catalase activity in response to oxidative stress reveal

35 Further, we demonstrate that the catalase activity is reduced at acidic pH, as compared w

36 gentamicin-resistant SCVs displayed greater catalase activity than wild-type bacteria, which contrib

37 f the electron transport chain); 3) increase catalase activity that is involved in H2O2 breakdown; an

38 er in endothelium of Old mice while vascular catalase activity was reduced by nearly half.

39 raction with O2 However, Rv2633c did exhibit catalase activity with a kcat of 1475 s(-1) and Km of 10

40 1 +/- 1.7 mm Cyanide and azide inhibited the catalase activity with Ki values of 3.8 mum and 37.7 mum

41 eased antioxidant capacity (MnSOD, CuSOD and Catalase activity).

42 acity for maintaining potassium homeostasis, CATALASE activity, and reduced ASCORBIC ACID OXIDASE (AA

43 , inadequate activation of heme enzymes, low catalase activity, defective clearance of H(2)O(2) and a

44 n weight loss, total soluble sugar, protein, catalase activity, peroxidase activity and increased in

45 ciated with reduction of both PPAR-gamma and catalase activity, which are reversed by both ACEA and t

46 xidatic electron donors (PxEDs) enhance KatG catalase activity.

47 ition by leptin, with subsequent increase of catalase activity.

48 agents, and extend the operable pH range for catalase activity.

49 ls more resilient toward ROS by coexpressing catalase along with a tumor specific chimeric Ag recepto

50 cistronic vector that concurrently expresses catalase, along with the CAR coexpressing catalase (CAR-

51 ion because pretreatment of eosinophils with catalase (an extracellular superoxide scavenger) or NSC

52 Herein it is a therapeutic based on catalase, an antioxidant enzyme that can effectively bre

53 Catalase, an enzyme decomposing peroxide, was found to s

54 ified by mitochondria-targeted expression of catalase and a mitochondria-targeted peptide antioxidant

55 l indices more effectively than non-targeted catalase and anti-ICAM-1 antibody alone.

56 reatment showed higher superoxide dismutase, catalase and ascorbate peroxidase activities as compared

57 The susceptibility of catalase and general stress response sigma factor mutant

58 A stimulatory effect on catalase and glutathione oxidase activities induced by M

59 significantly improved superoxide dismutase, catalase and glutathione peroxidase activities in H2O2 t

60 ting defense mechanisms (increased levels of catalase and glutathione peroxidase expression), observe

61 low levels of antioxidant enzymes, including catalase and GSH peroxidases, have supported a model in

62 ivator, Sulforaphane (SFN), augmented Prdx6, catalase and GSTpi expression in dose-dependent fashion,

63 hey showed increased levels of intracellular catalase and had a reduced oxidative state with less ROS

64 iopsy specimens with 8Br-cGMP also activated catalase and manganese superoxide dismutase expression,

65 utant strain (DeltaMakatG1) showed decreased catalase and peroxidase activities and significantly inc

66 Contrary to the expectation that catalase and peroxidase activities should be mutually an

67 able acidity, protein content, activities of catalase and peroxidase, chlorophyll and capsaicin conte

68 e, which may have been due to the absence of catalase and peroxiredoxin in the purified Hb.

69 Based on these data, we conclude that catalase and peroxiredoxin-glutaredoxin are determinants

70 se an increase in the enzymatic (peroxidase, catalase and phenylalanine ammonium lyase (PAL)) and non

71 xidase and approximately 3-fold increases in catalase and phenylalanine ammonium lyase activity.

72 cted cells, with a decrease in the levels of catalase and PRDX6 in exosomes derived from HIV-1-infect

73 ibited significantly increased activities of catalase and superoxide dismutase (SOD), compared to the

74 oncurrently GABA caused further increases in catalase and superoxide dismutase activities, which led

75 cells up-regulates the antioxidant proteins catalase and superoxide dismutase as well as the antiapo

76 Activities of catalase and superoxide dismutase enzymes, levels of tot

77 Islets size, number, and mRNA levels of catalase and superoxide dismutase were increased, wherea

78 otal glutathione contents, and activities of catalase and superoxide dismutase were significantly det

79 pidaecin and defensin-1, stress-related gene catalase and two genes linked to memory formation, pka a

80 Initially, homocysteine reacts with native catalase and/or redox-active transition metal ions to ge

81 y to target tissues to repress expression of catalases and other hydrogen peroxide defenses.

82 tioxidant enzymes superoxide dismutase 1 and catalase, and activation of the pro-oxidant protein kina

83 c is the first example of a non-heme di-iron catalase, and conclude that it is a member of a subset o

84 higher activities of glutathione reductase, catalase, and lipid peroxidation, indicating increased a

85 oduction, increased superoxide dismutase and catalase, and suppressed NADPH oxidase and reactive oxyg

86 ced activities of superoxide dismutase 2 and catalase, and were hypersensitive to hydrogen peroxide.

87 eme-containing enzymes (P-450s, peroxidases, catalases, and cytochrome c oxidase) involved in biologi

88 of extracellular vesicles containing active catalase; and 3) selective secretion of interleukin-6, i

89 A concomitant increase in catalase antioxidant activity and decreased DNA oxidativ

90 As in other eukaryotes, the Neurospora catalases are the main enzymes responsible for ROS clear

91 Some enzymes (dehydrogenases, oxidase, and catalase) are valuable products with high conversion eff

92 cosidase, invertase, beta-galactosidase, and catalase, are encapsulated in ZIF-8, UiO-66-NH(2), or Zn

93 R) gamma-coactivator 1alpha], PPARalpha, and catalase as key factors in antioxidant response, as well

94 drogenase 1, aldehyde dehydrogenase 1A1, and catalase, as well as the microsomal triglyceride transfe

95 acid and the enzymes glutathione reductase, catalase, ascorbate peroxidase and superoxide dismutase

96 Catalase assists to regulate production of cytokines, pr

97 ition metal homeostasis and an inhibition of catalase bioactivity have been reported.

98 verexpression of antioxidant enzymes such as catalase, by 73.4 fold was also observed.

99 e efficiency of heme peroxidases compared to catalases can be directly attributed to the different di

100 es catalase, along with the CAR coexpressing catalase (CAR-CAT), performed superior over CAR T cells

101 nt response to different sizes of CeO2 while catalase (CAT) activity was not affected by either size

102 mutase (SOD) activity, associated with lower catalase (CAT) and ascorbate peroxidase (APX) activities

103 lent antioxidant capacity (TEAC) levels, and catalase (CAT) and glutathione peroxidase (GPx) activiti

104 Activities of superoxide dismutase (SOD), catalase (CAT) and peroxidase (POD) decreased at a later

105 , we show that GLO physically interacts with catalase (CAT) in rice leaves, and that the interaction

106 ant enzymes - superoxide dismutase (SOD) and catalase (CAT), encapsulated in biodegradable nanopartic

107 utathione (GSH), superoxide dismutase (SOD), catalase (CAT), matrix metalloproteinase-9 (MMP-9), and

108 glutathione (GT), reduced glutathione (GSH), catalase (CAT), peroxidase (POD), superoxide dismutase (

109 ng levels of endogenous antioxidants such as catalase (CAT), superoxide dismutase (SOD) and glutathio

110 oxidants such as ascorbate peroxidase (APX), catalase (CAT), superoxide dismutase (SOD) and peroxidas

111 usion of bacterial lactate oxidase (LOX) and catalase (CAT), which irreversibly converts lactate and

112 hione (GSH), superoxide dismutase (SOD), and catalase (CAT).

113 At the site of inactivated catalase, cell-generated H(2)O(2) enters the cell via aq

114 e ternary polyplexes core for SR-A siRNA and catalase complexation, and a phosphatidylserine-modified

115 the protonation state of Helicobacter pylori catalase compound II.

116 st-mediated oxidant stress exposure, and its catalase contains oxidizable methionine (Met) residues.

117 dry weight, chlorophyll content, lipids, and catalase content when cells were exposed to P25 and 15.6

118 not only do endogenous antioxidants such as catalase contribute to ROS-induced cell death, but also

119 Calibrations using a catalase control to scavenge residual hydrogen peroxide

120 Wild type (live vaccine strain) or catalase-deficient F. tularensis (DeltakatG) show distin

121 were much more resistant to oxidants than a catalase-deletion mutant strain.

122 wth through AppBCX-mediated respiration in a catalase-dependent manner.

123 or kidneys and uniquely attenuated HPV via a catalase-dependent mechanism.

124 ay the major role in inactivating protective catalase, depleting glutathione and establishing apoptos

125 ER catalase did not effect the slow residual disulfide bond

126 oal of this study was to define the roles of catalase (encoded by hktE) and a bifunctional peroxiredo

127 roxide can be decomposed to oxygen by 0.44 U catalase enzyme and semiquantified in the range up to ap

128 Unlike the closely related catalase enzymes, it exhibits a low activity to dispropo

129 duced activities of superoxide dismutase and catalase enzymes.

130 Catalases, enzymes that detoxify H2O2, are widely distri

131 H2O2 treatments induced the activity of catalase - especially for 2-day-old sprouts treated with

132 observe a significant increase in endogenous catalase expression in SMA iPSCs.

133 eactive oxygen species through activation of catalase expression via SAPK/JNK phosphorylation and Nrf

134 Sirtuin-1, Foxo3alpha, and catalase expression were significantly decreased in RPTC

135 nuclear localization of FOXO3A and decreases catalase expression.

136 of HRP compensates less efficiently than in catalases for the energetic cost required to reorient th

137 two-electron chemistry that is critical for catalase function.

138 We find that the levels of catalase fused to GFP, both before and after a threat of

139 ess response elements were identified in the catalase gene and were shown to bind ATF6 in cardiac myo

140 first assembled MGII metagenome containing a catalase gene, which might be involved in scavenging rea

141 All investigated E. coli strains had catalase genes (katG, katE), genes coding for receptors

142 idant enzymes superoxide dismutase 2 (SOD2), catalase, glutathione peroxidase 1 (GPX1), and heme oxyg

143 was demonstrated by decreased expression of catalase, glutathione peroxidase 4, and glutathione synt

144 +WP diet increased the activities of hepatic catalase, glutathione peroxidase and glutathione S trans

145 The activities of catalase, glutathione peroxidase and superoxide dismutas

146 and oxidative stress (superoxide dismutase, catalase, glutathione peroxidase, lipidic and protein pe

147 pecies (ROS) scavenging machinery, including catalase, glutathione synthetase, glutathione reductase,

148 sociated with the physiological responses in catalase, guaiacol peroxidase, superoxide dismutase, sol

149 D, whereas polyethylene glycol-catalase (PEG-catalase; hydrogen peroxide scavenger) had no effect.

150 n (superoxide dismutase), hydrogen peroxide (catalase), hydroxyl radicals (mannitol) and singlet oxyg

151 n that the katA gene, encoding a cytoplasmic catalase in A. actinomycetemcomitans, was responsible fo

152 Our findings unravel the role of Mn-catalase in acclimatization to salt/oxidative stress and

153 hox) or STAT3 and overexpression of PDCD4 or catalase in BEAS-2B cells markedly inhibited the arsenic

154 as dependent on PCa epithelial inhibition of catalase in CAF.

155 more, we showed CSC-mediated upregulation of catalase in exosomes from uninfected cells, with a decre

156 2 cannot be ruled out due to the presence of catalase in flour and the fast reaction of H2O2 with glu

157 rum of issues involving the function of skin catalase in particular, and the native biological antiox

158 ellular addition of the inactive versions of catalase, in contrast to cells receiving HOCl alone.

159 Overexpression of catalase, in vivo, restored ATF6 knockout mouse heart fu

160 With some molecules of their protective catalase inactivated, these tumor cells allow locally su

161 g of secondary singlet oxygen generation and catalase inactivation within the cell population is thus

162 reaction with H2O2 Our results suggest that catalase-inactive intermediates accumulate because of of

163 ic) enzyme, indicating that they represented catalase-inactive intermediates.

164 human prostate carcinoma DU-145 cells, since catalase increased the survival of the cells subjected t

165 subunit gp91 was significantly increased in catalase-induced MDSC as determined by quantitative PCR

166 lomerase activator AGS 499 converted the PEG-catalase-inhibitable dilation to one mediated by nitric

167 eered protein form of the antioxidant enzyme catalase-inhibited cancer stem-like cells (CSCs), and tr

168 l cell line, and addition of the competitive catalase inhibitor hydroxylamine resulted in a dose-depe

169 cells treated with 3-amino-1,2,4-triazole (a catalase inhibitor) were the most sensitive.

170 We then encapsulated catalase inside the mesopores, observing that they are h

171 Since catalase is a key enzyme for the dismutation of virus-me

172 Because catalase is immobilized and sheltered by the ZIF-90 crys

173 position-dependent oxidant-quenching role of catalase is more important than the well described H2O2-

174 The H2O2 hydrolysing activity of purified catalase is necessary in order to stabilize glucose oxid

175 We show that mitochondrial targeting of catalase is sufficient to rescue redox transients, SOCE,

176 genes encoding heme biosynthesis enzymes and catalase (kat) were required for L. monocytogenes aerobi

177 aconitase (AcnB) and the detoxifying enzyme catalase (KatA).

178 changes, including the downregulation of the catalase KatG, an activating enzyme required for isoniaz

179 esults in dramatic loss of survival, whereas catalase (katG) overexpression suppresses the hypersensi

180 , we examined the presence of genes encoding catalases (katG and katE) and proteins participating in

181 While catalase knockdown in SMA iPSCs increased ROS production

182 lation experiments of Ras overexpression and catalase knockout in conjunction with the literature.

183 ATHase, the GDH, a monoamine oxidase, and a catalase leads to the production of enantiopure amines.

184 Incubation of CD14(+) monocytes with catalase led to a significant induction of functional MD

185 ) reversed astrocyte activation and restored catalase levels to normal, without changing mitochondria

186 pseudohalogenation (MPO/SCN(-)/H(2)O(2)) and catalase-like (MPO/H(2)O(2)) cycles.

187 shell nanostructures are expected to exhibit catalase-like activity to efficiently catalyze oxygen ge

188 produce synzyme protocells (Ru(4)PCVs) with catalase-like activity.

189 affect the affinity of MPO for H(2)O(2), (b) catalase-like cycle was slower, and (c) MPO retained enz

190 Therefore, the lower catalase-like efficiency of heme peroxidases compared to

191 In vitro and in vivo studies verify that the catalase-like nanohybrid significantly enhances the radi

192 to the lack of atomistic information on the catalase-like reaction in HRP.

193 Specifically, catalase, lipase, or alkaline phosphatase-filled colloid

194 Recently we cloned two catalase-lipoxygenase fusion protein genes (a and b) fro

195 The size of the obtained catalase-loaded exosomes (exoCAT) was in the range of 10

196 The catalase may be a potential drug target to aid in the pr

197 We hypothesized catalase may play a large stress-combating role independ

198 e viral expression of mitochondrial-targeted catalase (mCAT) produced similar functional benefits in

199 hat overexpress mitochondrial-targeted human catalase (mCAT).

200 LADR(low/-) suppressive cells are induced by catalase-mediated depletion of hydrogen peroxide (H2O2).

201 he antioxidants superoxide dismutase (SOD2), catalase, methionine sulfoxide reductase A, and the 20S

202 more, the synthetic superoxide dismutase and catalase mimetic EUK-134 also ablated the effects of hyp

203 ed in the presence of a superoxide dismutase/catalase mimetic.

204 we demonstrated that the H(2)O(2) scavenger catalase mimics the effect of Duox1 deficiency by enhanc

205 nfocal microscopy to confirm that the ~10 nm catalase molecules are embedded in 2 mum single-crystall

206 -concept design is demonstrated by embedding catalase molecules into uniformly sized ZIF-90 crystals

207 2) then inactivates some membrane-associated catalase molecules on at least a few tumor cells.

208 percentage of protective membrane associated catalase molecules on tumor cells.

209 al inactivation of a few membrane-associated catalase molecules.

210 The mechanism by which this catalase-negative bacterium can withstand endogenous hyd

211 xpression of antioxidant proteins, including catalase, NF-E2-related factor 2 (Nrf2), and heme oxygen

212 In contrast to a catalase-null strain, both site-change mutants proficien

213 These species continue to inactivate catalase on the originally triggered cells and on adjace

214 in, adsorption of proteins, transferrin, and catalase on the polystyrene (PS) or iron oxide (IO) NPs

215 n peroxide was injected and catalyzed by the catalases on the MNP-bacteria-PS complexes.

216 pathways for Compound II reduction found in catalases, only one is operative in HRP.

217 calisation towards the cell body in cheAY2-, catalase- or aconitase-deficient bacteria or in bacteria

218 OX2 knockout mice) and from NOX4-silenced or catalase-overexpressing INS-1E cells.

219 y the antioxidant vitamin E or mitochondrial catalase overexpression.

220 cts without CAD, whereas polyethylene glycol-catalase (PEG-catalase; hydrogen peroxide scavenger) had

221 me and that do not require activation by the catalase peroxidase KatG, thus circumventing the majorit

222 The activity of the antioxidant enzymes catalase, peroxidase and ascorbate peroxidase was also i

223 bit InhA, isoniazid must be activated by the catalase-peroxidase KatG.

224 TAML activators of peroxides are functional catalase-peroxidase mimics.

225 Here, we demonstrate that a bifunctional catalase-peroxidase, MakatG1, in the locust-specific fun

226 ostly based on two kinds of redox chemistry, catalases/peroxidases that depend on the heme prosthetic

227 ed by aquaporins and H(2) O(2) is removed by catalase, peroxiredoxin, glutathione peroxidase-like enz

228 Expressions of those genes encoding catalase, peroxiredoxin, thioredoxin and glutathione wer

229 ementation of polyethylene glycol-conjugated catalase (PG-CAT) for RSV-infected mice.

230 The majority of these isolates are catalase-positive Gram-positive rods from multiple gener

231 and subsequent catalytic generation of O2 by catalase present in the underlying viable epidermis and

232 h loading efficiency, sustained release, and catalase preservation against proteases degradation.

233 n mCAT mice expressing mitochondria-targeted catalase prevented Sirt3 and SOD2 impairment and attenua

234 ly, mitochondrial-targeted overexpression of catalase prevented the HFD-induced ischemic limb necrosi

235 -photon microscopy revealed that anti-ICAM-1/catalase prevents the transition of microglia to an acti

236 We found that catalases produced by Escherichia coli, the nematode's f

237 The existence of catalase producers may support the colonization of patho

238 ulation of H2O2-resistant SCVs with enhanced catalase production.

239 nd ATF6 in cardiac myocytes, which increased catalase promoter activity.

240 The binding of Nrf2 at the catalase promoter was analyzed by ChIP.

241 nd that Trp-321 is the preferred site of off-catalase protein oxidation in the native enzyme.

242 s, but was clearly overcome at a higher NQO1/catalase ratio consistent with cancer cells.

243 , was found to suppress DNA damage at a NQO1/catalase ratio found in healthy cells, but was clearly o

244 shows that this compound interferes with the catalase reaction.

245 Treatment with catalase reduced the ascorbate radical contents by as mu

246 lene oxide synthase and the newly identified catalase-related hydroperoxide lyase and given the role

247 ing catalases, the physiological roles of Mn-catalases remain inadequately characterized.

248 ovide compelling evidence that F. tularensis catalase restricts reactive oxygen species to temper mac

249 The Nrf2 inducer resveratrol, as opposed to catalase, reversed oxidative stress in lung epithelial c

250 OS) serine 1179 phosphorylation, whereas PEG-catalase scavenging of intracellular hydrogen peroxide o

251 iciently large to adsorb and host the enzyme catalase (size: 6-10 nm).

252 , or engineered to express the ROS scavenger catalase specifically within the mitochondria.

253 ant enzymes (i.e., superoxide dismutase, and catalase), strong DPPH-scavenging activity, and increasi

254 orial treatment with N-acetyl-l-cysteine and catalase substantially inhibited the ROS upsurge and PIN

255 findings were reversed with the addition of catalase suggesting that the effect of P-AscH(-) on meta

256 revented by the antioxidants glutathione and catalase, suggesting that HDM-induced reactive oxygen an

257 Furthermore, Sch A lowered DON-induced catalase, superoxide dismutase and glutathione peroxidas

258 tamine synthetase, alanine aminotransferase, catalase, superoxide dismutase, ornithine decarboxylase,

259 rf2, and Phase II enzymes (heme oxygenase-1, catalase, superoxide dismutase-1) in a time-dependent ma

260 Cu(5.4)O USNPs simultaneously possessing catalase-, superoxide dismutase-, and glutathione peroxi

261 In conclusion, catalase supplementation may represent a novel pharmacol

262 An inducible catalase system, to test causality for the role of hydro

263 tion of the CTT1 gene that encodes cytosolic catalase T.

264 Results indicate that catalase targeted to ICAM-1 reduces markers of oxidative

265 man chondrocytes by adenoviral expression of catalase targeted to the mitochondria (MCAT) and in cart

266 Biochemically, KatB was an efficient, robust catalase that remained active in the presence of high co

267 Unlike the heme-containing catalases, the physiological roles of Mn-catalases remai

268 Optimal inactivation of catalase then allows efficient apoptosis induction throu

269 Over 100 years after the discovery of catalase, these findings reveal a new non-enzymatic prot

270 We conjugated catalase to anti-ICAM-1 antibodies and administered the

271 Furthermore, expression of an ER-adapted catalase to degrade lumenal H2O2 attenuated PRDX4-mediat

272 neous expression of mitochondrially targeted catalase to reduce cellular ROS levels significantly sup

273 ed delivery system for a potent antioxidant, catalase, to treat Parkinson's disease (PD).

274 causative agent for these responses, as both catalase-treated and pyruvate oxidase-deficient bacteria

275 Similarly, combined N-acetyl-l-cysteine and catalase treatment also suppressed VDAC1-induced redistr

276 g the activity of superoxide dismutase-2 and catalase, two antioxidant enzymes that protect the cells

277 This dicobalt system also functions as a catalase upon treatment with H2O2.

278 genes, MnSOD, CuZnSOD, Nrf2, Keap1, GPx4 and Catalase was also examined.

279 Catalase was detected in primary HSC and a stromal cell

280 Catalase was induced by the canonical ER stressor, tunic

281 Catalase was loaded into exosomes ex vivo using differen

282 Remarkably, catalase was maintained in a reduced state by substrate-

283 transcription factor Nrf2 was activated, and catalase was upregulated via Nrf2.

284 2, levels of oPMN superoxide dismutase 1 and catalase were decreased in severe CP, despite increased

285 mponents related to either beta-oxidation or catalase were present.

286 translocation of Nrf2 and the expression of catalase were shown via Western blot.

287 Expression levels of genes encoding catalases were examined by real-time PCR.

288 dified with the detection antibodies and the catalases were simultaneously used to react with the tar

289 ), TH2 immunity (IL4), and oxidative stress (catalase) were validated in an independent asthmatic coh

290 thione peroxidase, superoxide dismutase, and catalase, were evaluated in each of the infected, treate

291 es such as superoxide dismutase 2 (SOD2) and catalase, which directly detoxify ROS.

292 hat mitochondrial targeted overexpression of catalase, which is established to mitigate oxidative str

293 tified here reside outside the ER, including catalase, which is known to decrease damaging reactive o

294 constructed with the aim to study the enzyme catalase, which is part of the biological antioxidative

295 e that skin contains a substantial amount of catalase, which is sufficient to detoxify H2O2 that reac

296 Furthermore, addition of catalase, which neutralizes H2O2, greatly suppresses S.

297 collected Postflight, with the exception of Catalase, which show no change.

298 ery of NRF2 was more effective than SOD2 and catalase, while expression of PGC1a accelerated photorec

299 ains (katA(H56A) and katA(Y339A)) containing catalase without enzyme activity but that retain all Met

300 iomarkers (body weight, protein, chitobiase, catalase, xenobiotic metabolism, and acetylcholinesteras