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1 ntaining the cDNA for LL-37/hCAP-18, a human cathelicidin antimicrobial peptide.
2 ability of PilB to mediate GBS resistance to cathelicidin antimicrobial peptides.
3 ate immune responses, such as the release of cathelicidin antimicrobial peptides.
4 deI and ompD are important for resistance to cathelicidin antimicrobial peptide, a mouse AMP produced
5                                              Cathelicidin antimicrobial peptides are effectors of inn
6                                              Cathelicidin antimicrobial peptides are essential for th
7                                              Cathelicidin antimicrobial peptides are present at sites
8 L-10R activation repressed the expression of cathelicidin antimicrobial peptide as well as antigen-pr
9                                          The cathelicidin antimicrobial peptide (Camp) gene was upreg
10  its analogs induced expression of the human cathelicidin antimicrobial peptide (CAMP) gene.
11                                              Cathelicidin antimicrobial peptide (CAMP) is a naturally
12 r Typhi and S. Typhimurium will induce human cathelicidin antimicrobial peptide (CAMP) production, an
13           The circulating senescence markers cathelicidin antimicrobial peptide (CAMP), B-galactosida
14  (S1P), regulates production of a major AMP, cathelicidin antimicrobial peptide (CAMP), in response t
15                   The innate immune element, cathelicidin antimicrobial peptide (CAMP), is vital in t
16 uction of a cutaneous innate immune element, cathelicidin antimicrobial peptide (CAMP), through endop
17 of the multifunctional antimicrobial peptide cathelicidin antimicrobial peptide (CAMP).
18 lls to produce an innate immune element, the cathelicidin antimicrobial peptide (CAMP).
19 or (VDR)-dependent mechanisms regulate human cathelicidin antimicrobial peptide (CAMP)/LL-37 in vario
20 motaxis, including elevated myeloperoxidase, cathelicidin antimicrobial peptide, complement C3, and c
21  CD14 and TLR2, complementing an increase in cathelicidin antimicrobial peptide expression.
22 ction was mediated through the production of cathelicidin antimicrobial peptide from adipocytes becau
23                                          The Cathelicidin Antimicrobial Peptide gene (CAMP), which pr
24                            These include the cathelicidin antimicrobial peptide gene and the TLR core
25 )D) enhances innate immunity by inducing the cathelicidin antimicrobial peptide (hCAP).
26    The genes encoding lysozyme, lactoferrin, cathelicidin antimicrobial peptide (hCAP18/LL-37), cathe
27 ifferentiating into adipocytes and producing cathelicidin antimicrobial peptide in response to Staphy
28 triking bactericidal interactions with human cathelicidin antimicrobial peptide LL-37, a frontline co
29 ly localizes with elevated expression of the cathelicidin antimicrobial peptide LL-37.
30 let-derived antimicrobials in serum, and the cathelicidin antimicrobial peptide LL-37.
31 h AD, but not psoriasis, is deficient in the cathelicidin antimicrobial peptide (LL-37) and human bet
32 esponding to residues 7-27 of the only human cathelicidin antimicrobial peptide, LL37, is shown to ex
33     These observations provide evidence that cathelicidin antimicrobial peptides mediate an anti-infl
34 mycin, the susceptibility of the bacteria to cathelicidin antimicrobial peptides or serum complement
35                              The presence of cathelicidin antimicrobial peptides provides an importan
36                      Mast cells (MC) express cathelicidin antimicrobial peptides that act as broad-sp
37 , organelles that also contain precursors of cathelicidins, antimicrobial peptides that undergo prote
38                            Administration of cathelicidin antimicrobial peptides to mice with late-st
39     Using MCs derived from mice deficient in cathelicidin antimicrobial peptide, we showed that antim
40 tibility to reactive oxygen species and host cathelicidin antimicrobial peptides, which correlated wi