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1  influx via the mGluR1-mediated nonselective cation conductance.
2 electivity and gain of a depolarizing inward cation conductance.
3 longer than the glutamate-evoked increase in cation conductance.
4 dies and investigations of single-channel or cation conductance.
5 hat some mutant band 3 protein can mediate a cation conductance.
6 independent, Ca(2+)-regulated, non-selective cation conductance.
7 dney contain an ATP-activated, non-selective cation conductance.
8 epolarization arise from the activation of a cation conductance.
9 o NK3-mediated activation of a non-selective cation conductance.
10 as associated with a Ca2+- and Na+-permeable cation conductance.
11  that results in the observed alterations in cation conductance.
12  activation of a hyperpolarization-activated cation conductance.
13 duces an L-canavanine-activated nonselective cation conductance.
14 egmental area primarily by increasing such a cation conductance.
15  mediated by either chloride or nonselective cation conductances.
16 ocker of the calcium-activated non-selective cation conductance, abolished the effect on eupnoeic act
17  outward currents mediated by a nonselective cation conductance and a calcium-activated potassium con
18 nductance, which may include a non-selective cation conductance and a K+ conductance, appears to be i
19  rest of a hyperpolarization-activated mixed-cation conductance and a low-threshold, depolarization-a
20 n of the OoC activated the nonselective ChR2 cation conductance and depolarized DCs when measured in
21 vide a molecular explanation for p7-mediated cation conductance and its inhibition by adamantane deri
22 obilization and activation of a nonselective cation conductance; and cAMP increases mediated by G(s).
23 ocytes, vasopressin activated a nonselective cation conductance at concentrations that enhanced slow
24 er and by the calcium-activated nonselective cation conductance blocker flufenamic acid.
25 tion of a voltage-independent, non-selective cation conductance, but the timing mechanism responsible
26  (10 microM) caused inhibition of whole-cell cation conductance by approximately 55 %.
27 zation of the neurons initiated by increased cation conductance by cAMP-gated cation channels.
28 l 2 (TPC2) as the first reported melanosomal cation conductance by directly patch-clamping skin and e
29  key role of a Ca(2+)-activated nonselective cation conductance ((CaN)) in generating the plateau pot
30  Cl(-) solutions, and an inwardly rectifying cation conductance (cationic I(ATP)).
31             Our data suggest that a specific cation conductance composed of acid-sensing ion channels
32              In smooth muscle, non-selective cation conductances contribute to agonist-evoked depolar
33 tassium channels and that the cAMP-dependent cation conductance does not contribute significantly to
34                                 The block of cation conductances during the internal dialysis of Cs(+
35  for cation binding, cation selectivity, and cation conductance efficiency.
36 ts, the free-energy barriers and the maximum cation conductance for the permeation of various cations
37 (GIRK/K(G)) conductance and a cAMP-dependent cation conductance have both been implicated in this eff
38 e K+, and hyperpolarization-activated, mixed cation conductances have varying distributions in hippoc
39 th potassium conductance (I (Kir)) and mixed cation conductance (I (h)), engaging opposing influences
40 mediated by a calcium-activated nonselective cation conductance (I(CAN)) that is activated by calcium
41 tively active Ca(2+)-permeable non-selective cation conductance (I(cat)) in rabbit ear artery smooth
42 the hyperpolarization-activated nonselective cation conductance (I(h)) attenuates EPSPs mainly by red
43 t, I(T), and the hyperpolarization-activated cation conductance, I(sag).
44  presence of the hyperpolarization-activated cation conductance, I(sag).
45 nist for the calcium-activated non-selective cation conductance (ICAN ) abolished the effect of PGE2
46 n glucocorticoid-free medium by activating a cation conductance identical to the glucocorticoid-induc
47 yl borate (2-APB) to activate a nonselective cation conductance in its STIM1-independent mode.
48 fied that is essential for the light-induced cation conductance in photoreceptor cells.
49  production of a PLC-dependent, nonselective cation conductance in pontine neurons.
50 imulus for catfish, activated a nonselective cation conductance in some bilayers, which was antagoniz
51 bitors transiently increase the paracellular cation conductance in the thick ascending limb.
52 muscarinic receptor responsive non-selective cation conductance in Trp6 cells that was absent in cont
53 dy is to investigate the effect of Ang II on cation conductances in freshly dispersed rabbit mesenter
54  indicate that Ang II activates two distinct cation conductances in mesenteric artery myocytes by sti
55 trated that 5-HT and SP modulated background cation conductances in pre-BotC and motor neurons, inclu
56 ecrease and a Cs+-insensitive, non-selective cation conductance increase may account for the two type
57  activation of a hyperpolarization-activated cation conductance is a key mechanism through which sync
58 tion is Ca2+, indicating that influx via the cation conductance is another source of the increases in
59 polarization and so an amiloride-insensitive cation conductance is present.
60  suggesting they were due to a non-selective cation conductance (NSCC) carried by TRPM7.
61 group of ICC expressed a basal non-selective cation conductance (NSCC) that was inhibited by an incre
62 nderlie the diversity of amiloride-sensitive cation conductances observed in a wide variety of tissue
63 his probably explains the reduced monovalent cation conductance of the mutant channel.
64                           Yet any changes in cation conductance or pump activity are associated with
65                            Slow nonselective cation conductances play a central role in determining t
66 alysis of the novel P2Y1 receptor-associated cation conductance revealed that the open channel curren
67 -100 microM) stimulates a novel nonselective cation conductance seen only in oocytes expressing human
68 ent entering the cell through a non-specific cation conductance that continuously varied in amplitude
69 4 channel agonist, activated a non-selective cation conductance that coupled to activation of SK chan
70 nhibited, voltage-independent, non-selective cation conductance that has similar properties to the co
71 ill via a novel non-selective store-operated cation conductance that is blocked by nifedipine.
72   In voltage clamp, high [Ca2+]0 activates a cation conductance that underlies the depolarization.
73 gramicidins form well-defined channels, with cation conductances that are approximately 60% of those
74 MP, evoked a robust activation of whole-cell cation conductance to 220 % of control.
75                               A non-specific cation conductance was reversibly induced upon deoxygena
76 ompanied by up-regulation of a non-selective cation conductance with TRPM4-like properties and appare