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1 tonin, and dopamine or by stimulation of the cauda equina.
2 e before giving rise to a long and extensive cauda equina.
3  stimulated through their motor roots in the cauda equina.
4 dehyde and removal of the sciatic nerves and cauda equina.
5 ficits at admission and 17.3% presented with cauda equina.
6 c spinal cord and nodular enhancement of the cauda equina.
7 vidence of leptomeningeal enhancement in the cauda equina although CSF cytology was negative, and one
8 ging studies demonstrated enhancement of the cauda equina and leptomeninges of the lower spinal cord.
9 6) had metastatic cancer with spinal cord or cauda equina compression, life expectancy greater than 8
10 5% CI 0.85-2.75; p=0.15), and spinal cord or cauda equina compressions were reported in seven (2%) of
11 anaesthetized rats spinalized at T10-T11 and cauda equina cut).
12                      Conus medullaris and/or cauda equina forms of spinal cord injury commonly result
13 on that future repair of brachial plexus and cauda equina injuries will include spinal cord surgery t
14 n of avulsed roots after brachial plexus and cauda equina injuries.
15 unctional outcomes after brachial plexus and cauda equina injuries.
16 clinical interest after conus medullaris and cauda equina injuries.
17 eration in future attempts of treating acute cauda equina injuries.
18                         Here, we developed a cauda equina injury and repair rodent model to investiga
19 sacral ventral root avulsion injury model of cauda equina injury to investigate the lesion effect in
20 peated, they demonstrated enhancement of the cauda equina nerve roots, trigeminal nerve, and pachymen
21                              Injuries to the cauda equina of the spinal cord result in autonomic and
22  patients with bowel dysfunction in complete cauda equina syndrome (CES).
23 rotoxicity, including seizures, papilledema, cauda equina syndrome (n = 2), and encephalitis after a
24                  Compression associated with cauda equina syndrome must be reversed within 48 hours t
25 s presented with meningitis alone (73%), the cauda equina syndrome or focal infection (15%), or poste
26  osteomyelitis, cholecystitis, osteoporosis, cauda equina syndrome, and osseous defect), resulting in
27 anglia and brainstem, spinal cord, conus and cauda equina to disorders of peripheral innervation.
28 e ventral tail artery in rats that had their cauda equina transected to eliminate the somatic innerva
29  (17%) of the 18 lesions, enhancement in the cauda equina was similar to that seen in Guillain-Barre