ライフサイエンスコーパス: caudate

1 efrontal cortex, inferior frontal gyrus, and caudate.

2 left HG, and left and right HG and the left caudate.

3 cortices and striata and in human postmortem caudate.

4 end level relative to baseline in the dorsal caudate.

5 e significantly increased fALFF in the right caudate.

6 ctivity between the dorsal ACC and bilateral caudate.

7 e type of learning processes mediated by the caudate.

8 -sensitive markers decreased within the left caudate.

9 elative to self were tracked in the thalamus/caudate.

10 thalamus, and putative target of DMFC in the caudate.

11 mbens as well as right frontal pole and left caudate.

12 prefrontal cortex (DLPFC), hippocampus, and caudate.

13 and low dopamine transporter binding in the caudate.

14 eraction in right anterior insula and dorsal caudate.

15 frontal white matter (-11.4%; P < .001), and caudate (-10.6%; P = .04), while the Cho:Cr ratio was hi

16 ith TBI and PD had reduced DaT levels in the caudate (12.2% and 18.7%, respectively) and putamen (9.0

17 ion of 20 ng/ml and D2/3 occupancies of 43% (caudate), 25% (putamen), 43% (thalamus).

18 unction was clearly lower in PSP than in PD (caudate: 34.1% difference, g = -1.08, 95% confidence int

19 on of 60 ng/ml, and D2/3 occupancies of 61% (caudate), 49% (putamen) and 69% (thalamus).

20 iated with high D2/3 occupancies (65 +/- 8%, caudate; 67 +/- 11%, thalamus; 52 +/- 11%, putamen).

21 This was also true for the left caudate (95% CI = 0.002-0.099).

22 tribution volume ratio (DVR) relative to the caudate (a pseudoreference region).

23 We previously reported elevated caudate activation under haloperidol in this sample of p

24 These results suggest that, as indexed by caudate activity, successful service dogs generalize ass

25 utamen, p<0.0001) and (18)F-FDOPA uptake (in caudate: age </=50 years, p=0.0002; all other age ranges

26 Using anatomically defined ROIs in the caudate, amygdala, and visual cortex, we developed a cla

27 These viruses affect caudates, an order in which information regarding Ranavi

28 rior lateral occipital cortex, and the right caudate and anterior supramarginal gyrus were correlated

29 shape variation of the left amygdala, right caudate and bilateral putamen (corrected P = 0.05).

30 functional connectivity between the ventral caudate and clusters within the mid-cingulate cortex and

31 ght hippocampus, on average, but lower right caudate and corpus callosum volume, relative to 22q-del

32 t, functional connectivity between the right caudate and cuneus was correlated with the Tinnitus Func

33 Both caudate and dorsolateral prefrontal cortex demonstrated

34 Recording from downstream neurons in the caudate and from thalamic neurons projecting to the medi

35 motor area fibers (M2/M3/M4) arched over the caudate and lateral motor area fibers (M1/LPMCv) curved

36 n levels in increasing activity in the right caudate and left anterior insula during anticipation of

37 sed functional connectivity between the left caudate and left HG, and left and right HG and the left

38 activated bilateral insula, bilateral dorsal caudate and left precentral gyrus.

39 Greater caudate and medial prefrontal cortex reactivity to gain

40 functional connectivity between the ventral caudate and medial temporal cortex increased as a functi

41 on errors showed a positive correlation with caudate and nucleus accumbens activity during placebo, w

42 In contrast, lithium administration reduced caudate and nucleus accumbens activity during reward out

43 erent phases of reward processing within the caudate and nucleus accumbens.

44 r within the left uncinate fasciculus, right caudate and occipital regions (p < 0.05).

45 (anterior insula), and learning and memory (caudate and parahippocampal gyrus).

46 wnregulated and 822 upregulated genes in the caudate and putamen (striatum) of TS individuals.

47 Caudate and putamen DaT, putamen to caudate ratios and l

48 (SMD) of PET uptakes in the whole striatum, caudate and putamen in manifest and premanifest HDGECs c

49 ls from the anterior cingulate cortex (ACC), caudate and putamen of 16 RC BD-I, 34 non-RC BD-I and 44

50 y to the DA presynaptic terminals postmortem caudate and putamen of 3 healthy individuals, 4 PD cases

51 nglia transcriptome by RNA sequencing in the caudate and putamen of nine TS and nine matched normal c

52 , and ventral striatum) and with ex-smokers (caudate and putamen).

53 n adult macaques following delivery into the caudate and putamen, brain regions which comprise the st

54 For the caudate and putamen, LL showed higher DRD2 availability

55 amine transporter binding (all age ranges in caudate and putamen, p<0.0001) and (18)F-FDOPA uptake (i

56 2 groups, with good discrimination for both caudate and putamen.

57 e) of the subcortex, namely of the amygdala, caudate and putamen; a functional brain network related

58 d, functional connectivity between the right caudate and superior lateral occipital cortex, and the r

59 r neural activation in the bilateral ventral caudate and the nucleus accumbens during reward receipt

60 Reduced functional connectivity between the caudate and the ventrolateral prefrontal cortex was sele

61 and later in dorsolateral prefrontal cortex, caudate and ventral striatum, and c) contribute to featu

62 gdala volumes and greater lateral ventricle, caudate, and accumbens volumes (Cohen's d values, -0.90

63 areas, including the frontoparietal cortex, caudate, and cerebellum.

64 ne showed increased GBCr in the lateral PFC, caudate, and insula.

65 dial OFC (BA11), lateral OFC (BA47), head of caudate, and nucleus accumbens (NAc).

66 inferior frontal and superior temporal gyri, caudate, and other structures is affirmed.

67 e frontal gyri, precuneus, cingulate cortex, caudate, and postcentral gyrus (all regions: p < .001, e

68 Local atrophy of the left hippocampus, right caudate, and right pallidum had a positive correlation w

69 atal D2/3R availability, particularly in the caudate, and this relationship mediated the relationship

70 In fMRI analyses, during inhibition, right caudate anomalies reflected a childhood ADHD history and

71 dities, particularly in the anterior insula, caudate, anterior cingulate, medial frontal gyrus, and d

72 This was not caused by altered size of the caudate, as its cross-sectional surface areas were simil

73 or cingulate cortices and bilateral thalamus/caudate, as well as the right anterior insula/frontal op

74 trongly expressed in the corpus callosum and caudate axon fibers.

75 s, amygdala, insular, cingulate, cerebellum, caudate, basal-forebrain, and thalamus areas (p < 0.01).

76 Dopamine synthesis was 16% higher in the caudate body, 17% higher in the dorsal putamen, and 17%

77 right frontal subcallosal gyrus (BA 34) and caudate body, and in the cerebellar tonsils (P < 0.001).

78 right frontal subcallosal gyrus (BA 34) and caudate body, and in the cerebellar tonsils (p<0.001).

79 and 3) to unexpected reward omission in the caudate body.

80 .02 ppm +/- 0.02 and 19.65 sec(-1) +/- 3.6), caudate (case participants: -0.1 ppm +/- 0.04 and 18.21

81 activation in multiple regions (e.g., in the caudate, cingulate, and precentral gyrus) and decreased

82 levels in the putamen (12.8%) but not in the caudate compared with controls.

83 involvement, 22.4% had bilaterally impaired caudate.Compared with those with a baseline normal cauda

84 riatal connectivity, which included abnormal caudate connections with a distributed set of associativ

85 n the GD group, activity in the thalamus and caudate correlated negatively with gambling severity.

86 mbens (Cohen's d=-0.15), amygdala (d=-0.19), caudate (d=-0.11), hippocampus (d=-0.11), putamen (d=-0.

87 0.19 vs -0.10), amygdala (d=-0.18 vs -0.14), caudate (d=-0.13 vs -0.07), hippocampus (d=-0.12 vs -0.0

88 To directly examine the specific role of caudate D2-type receptors in reversal performance, the D

89 Hippocampal and caudate D2DR availability were interrelated, and functio

90 l temporal cortex increased as a function of caudate D2DR availability.

91 ing and was linked to OT, whereas a stronger caudate-dACC connectivity was associated with increase i

92 rate/severe TBI showed similar reductions in caudate DaT binding, but patients with PD showed a great

93 Patients with low caudate dopamine transporter binding showed improvement

94 t baseline, 51.6% of 397 patients had normal caudate dopamine transporter binding, 26.0% had unilater

95 n injury were only seen in patients with low caudate dopamine transporter levels.

96 Early significant caudate dopaminergic denervation was found in half of th

97 ugh not typical of Parkinson's disease (PD), caudate dopaminergic dysfunction can occur in early stag

98 We investigated the occurrence of caudate dopaminergic dysfunction in the very early phase

99 hium administration enhanced activity in the caudate during reward anticipation compared to placebo.

100 We defined a clinically significant caudate dysfunction as (123)I-FP-CIT binding <-2 SDs com

101 e striatal value representations by applying caudate electrical stimulation in macaque monkeys (n = 3

102 er, choice-selective neurons in FEF, but not caudate, encoded behaviorally derived biases in the accu

103 on of thalamus (F(1,59) = 34.85; p < 0.001), caudate (F(1,59) = 59.35; p < 0.001), putamen (F(1,59) =

104 ype on DRD2 availability at baseline for the caudate (F(2,90) = 8.2, p = 0.001) and putamen (F(2,90)

105 03 ppm +/- 0.02 and 19.73 sec(-1) +/- 3.01), caudate (first MRI: -0.09 ppm +/- 0.05 and 21.38 sec(-1)

106 igue and brain activation was evident in the caudate for this task as well.

107 e.Compared with those with a baseline normal caudate function, at the4-year follow-up patients with a

108 Furthermore, caudate functional connectivity patterns differentiated

109 bcortical structures examined (the amygdala, caudate, globus pallidus, putamen, and thalamus).

110 ial (BP(ND)) and MPH dose in the head of the caudate (hCd), demonstrating increased extracellular DA

111 lutamine (Gln) metabolites (Glx) in the left caudate head (P = 0.027).

112 lidal volume (PV) in males, and (2) relative caudate head expansion and ventral striatum contraction

113 synthesis capacity in the dorsal putamen and caudate head was positively correlated with gambling dis

114 d the same Task X Fatigue interaction in the caudate head.

115 ratios compared with healthy controls in the caudate (healthy controls 2.98 [SD 0.63] vs GBA 3.26 [0.

116 ngulate cortex, thalamus, putamen, pallidum, caudate, hippocampus, and brain stem.

117 cal structures (nucleus accumbens, amygdala, caudate, hippocampus, pallidum, putamen, thalamus, and l

118 onnectivity between the dorsolateral PFC and caudate in pre-HD participants.

119 follow-up patients with a baseline bilateral caudate involvement showed a higher frequency of cogniti

120 Baseline bilateral caudate involvement was associated with increased risk o

121 Significant caudate involvement was observed in 83.9% of the populat

122 ne transporter binding, 26.0% had unilateral caudate involvement, 22.4% had bilaterally impaired caud

123 nt induction in the number of GAD67-cells in caudate-kindled rats in the dentate gyrus and CA3 hippoc

124 upt the excitation/inhibition balance in the caudate leading to dysfunctional corticostriatal circuit

125 neural responses to smoking cues in the left caudate, left inferior frontal gyrus, and left frontal p

126 n in the striatum, especially in the rostral caudate, manifesting as excess synthesis and release.

127 cess (Fan et al., 2018) that was affected by caudate microstimulation (Doi et al., 2020).

128 We argue that caudate microstimulation can differentially increase sti

129 wn to induce neural plasticity [10, 11], and caudate microstimulation in primates has been shown to a

130 t effect, power of >80% was achieved for the caudate (n = 661), pallidum (n = 687), and nonventricula

131 Caudate neural recordings (n = 1) show that changes in v

132 symmetric reward-choice associations, single caudate neurons encoded both sources of information.

133 ults imply distinct contributions of FEF and caudate neurons to reward-biased decision-making and put

134 vity network, with decreased connectivity in caudate nuclei and thalami and increased connectivity in

135 of subcortical heterotopia posterior to the caudate nuclei, "ribbon-like" heterotopia in the posteri

136 utamina and globi pallidi, dysgenesis of the caudate nuclei, olfactory bulbs hypoplasia, and anomaly

137 Dense bilateral connections were to caudate nuclei.

138 The basal ganglia, especially the caudate nucleus 'head' (CDh) of the striatum, receive in

139 5 vs. 1.285 x 10(-3) mm(2)/s, p = 0.031) and caudate nucleus (CN) (1.319 vs. 1.394 x 10(-3) mm(2)/s,

140 of 3 separate striatal areas (putamen (PT), caudate nucleus (CN) and accumbens nucleus (NAC)) from p

141 = 10), superior cerebellar peduncle (n = 7), caudate nucleus (n = 4), whole thalamus (n = 3), and put

142 performance correlated with lower CBF in the caudate nucleus (P = .01), thalamus (P = .04), frontal c

143 The medial caudate nucleus also shows hyperactivity in humans lacki

144 1) the lateral prefrontal cortex and medial caudate nucleus and 2) the supplementary motor area, sup

145 hese unassigned voxels were localised to the caudate nucleus and anterior putamen, overlapping with e

146 tivity between the right DLPFC and the right caudate nucleus and bilateral (para)cingulate gyrus incr

147 ectivity differences by placing seeds in the caudate nucleus and Heschl's Gyrus (HG) of both hemisphe

148 or, middle and posterior cingulate cortices, caudate nucleus and nucleus accumbens (corrected P = 0.0

149 ate cortices (anterior, mid- and posterior), caudate nucleus and nucleus accumbens.

150 opamine D2-type receptor (D2R) levels in the caudate nucleus and performance in a discrimination reve

151 n methamphetamine users from controls in the caudate nucleus and putamen and higher D1-receptor densi

152 Caudate nucleus and putamen DAT function was clearly low

153 The caudate nucleus and putamen underwent severe neuronal lo

154 The neuropil of both the caudate nucleus and putamen was invaded with reactive as

155 based methods in both the DAT-rich striatum (caudate nucleus and putamen) and the SERT-rich extrastri

156 ce of multipolar ChAT-ir interneurons in the caudate nucleus and putamen, whereas monkeys have a more

157 ported a cognition-action separation between caudate nucleus and putamen-a striatal sub-division uniq

158 nt differences were seen in the hippocampus, caudate nucleus and thalamus of the preterm rabbits.

159 rmed) centrality values in OCD for volume of caudate nucleus and thalamus, and surface area of parace

160 Higher grey-matter volumes in the caudate nucleus and the left cerebellum at age 14 years

161 ergic input from the substantia nigra to the caudate nucleus and the putamen.

162 l connectivity of orbito-frontal cortex with caudate nucleus and to structural changes within limbic

163 The results revealed the caudate nucleus as the key brain structure involved in s

164 er, model-based fMRI analyses identified the caudate nucleus as the key structure involved in selecti

165 y stimuli led to increased activation of the caudate nucleus associated with reward processing, angry

166 between the right orbito-frontal cortex with caudate nucleus bilaterally.

167 sed connectivity between the DN and the left caudate nucleus could play a role in balance impairment

168 otonergic degeneration in human ventromedial caudate nucleus from individuals with an APOE epsilon4 a

169 teromers was next demonstrated in postmortem caudate nucleus from schizophrenic subjects, even though

170 Similar results were seen in mouse caudate nucleus homozygous for APOE epsilon4 via targete

171 ese results underscore the importance of the caudate nucleus in relation to cognitive fatigue.

172 nd was associated with a reduced dentate and caudate nucleus iron content compared to placebo.

173 has demonstrated that neural activity in the caudate nucleus is modulated by task-relevant action val

174 The caudate nucleus may be involved in the repetitive moveme

175 striatal (123)I-FP-CIT binding ratios in the caudate nucleus of PSP patients than in that of both PD

176 specific bottom-up cues, and they place the caudate nucleus of the dorsal striatum at the center of

177 re was no evidence of altered variability of caudate nucleus or frontal lobe volumes.

178 re was no evidence of altered mean volume of caudate nucleus or putamen.

179 These results imply that the caudate nucleus plays causal roles in coordinating decis

180 te cortex and between the right amygdala and caudate nucleus predicted the magnitude of reduction in

181 Caudate nucleus recordings (n = 4) revealed multi-scale

182 ofrontal cortex, anterior temporal lobe, and caudate nucleus than PCA, and PCA showed more asymmetric

183 1907 ranged from 3.7 +/- 1.5 mL/cm(3) in the caudate nucleus to 14.5 +/- 5.3 mL/cm(3) in the occipita

184 nal capsule adjacent to the head of the left caudate nucleus was found in PD-ICB, but not surviving c

185 ents, the decreased connectivity in the left caudate nucleus was related with worse balance performan

186 peduncle and pontine tegmentum alongside the caudate nucleus were implicated as critical structures.

187 subset of these regions (PCC, thalamus, and caudate nucleus) covaried with the level of arousal.

188 gered endogenous opioid release in thalamus, caudate nucleus, and anterior insula.

189 ght lateral geniculate nucleus [LGN]), right caudate nucleus, and bilateral prefrontal regions.

190 refrontal cortex (vmPFC), nucleus accumbens, caudate nucleus, and putamen.

191 tified for ADHD and volumes of the amygdala, caudate nucleus, and putamen.

192 ates include the frontal eye field (FEF) and caudate nucleus, but their distinct roles are not unders

193 timulations of the hippocampus, amygdala, or caudate nucleus, followed by sacrifice and immunohistoch

194 the nucleus accumbens, amygdala, brainstem, caudate nucleus, globus pallidus, putamen and thalamus,

195 ned brain areas with low FA values including caudate nucleus, globus pallidus, putamen, superior temp

196 volumes of the nucleus accumbens, amygdala, caudate nucleus, hippocampus, and putamen are smaller in

197 ties with a predilection for the head of the caudate nucleus, insula, and cortical spread to the limb

198 ing in functional connectivity involving the caudate nucleus, insula, medial prefrontal cortex and ot

199 ajor input station of the basal ganglia, the caudate nucleus, plays a causal role in integrating unce

200 Differences were detected in the dorsal caudate nucleus, putamen, and globus pallidus but the ob

201 Binding potential for caudate nucleus, putamen, and substantia nigra was evalu

202 the following areas: level of basal ganglia (caudate nucleus, putamen, corpus callosum, posterior lim

203 l subcortical gray matter volumes (thalamus, caudate nucleus, putamen, globus pallidus, hippocampus,

204 elated regions (nucleus accumbens, r = 0.29; caudate nucleus, r = 0.27) to unhealthy FF commercials p

205 d in the substantia nigra (SNc), dentate and caudate nucleus, red nucleus, putamen and globus pallidu

206 volumes of four deep grey matter structures (caudate nucleus, thalamus, subthalamic nucleus and lenti

207 best classification of ischemic core for the caudate nucleus, the lentiform nucleus, and the insula (

208 the striosomes than the matrix in the monkey caudate nucleus, the opposite was found in the mouse str

209 iosome and matrix compartments of the monkey caudate nucleus, with the exception of a small amount of

210 teral and dorsomedial prefrontal cortex, and caudate nucleus.

211 did not differ across species in the medial caudate nucleus.

212 rom -0.13 in the dorsal raphe to 0.88 in the caudate nucleus.

213 lt mode and multiple control networks in the caudate nucleus.

214 alterations in 3D chromatin structure in the caudate nucleus.

215 error regression within the caudate, ventral caudate/nucleus accumbens, and anterior and posterior in

216 mPFC and, for the first time, in the dorsal caudate of antipsychotic-naive patients with FEP, which

217 chronic indwelling cannulae into the medial caudate of male and female marmoset monkeys performing a

218 ere found both in the mPFC and in the dorsal caudate of patients with FEP compared with healthy contr

219 y elevated GPC+PC levels in ACC, putamen and caudate of RC BD-I patients compared to healthy controls

220 cipated in stimulation sessions during which caudate or putamen stimulation was delivered for some im

221 The TSPO VT was measured in the dorsal caudate, orbitofrontal cortex, thalamus, ventral striatu

222 ne whether TSPO VT is elevated in the dorsal caudate, orbitofrontal cortex, thalamus, ventral striatu

223 cortices; and in the subcortical regions of caudate (P < 0.001), putamen (P < 0.001) and thalamus (P

224 ip in left hemispheric amygdala (p = 0.010), caudate (p = 0.008), inferior frontal gyrus (p = 0.004),

225 , no dopamine increases were observed in the caudate (p = 0.1) or putamen (p = 0.8) following methylp

226 <0.0001) and dorsal (p=0.0002) raphe nuclei, caudate (p=0.00015), putamen (p=0.036), thalamus (p=0.00

227 umber on intracranial volume and on regional caudate, pallidum and putamen volumes (beta = -0.71 to -

228 nt negative dose-response for the accumbens, caudate, pallidum, putamen and ICV (P = 0.0032, 8.9 x 10

229 ced a significant reduction of volume in the caudate, pallidum, putamen and thalamus ipsilateral to t

230 off-target binding regions were considered: caudate, pallidum, putamen, thalamus, cerebellar white m

231 basal ganglia, in either their putaminal or caudate part; and (ii) at least two SEEG seizures were r

232 ing the mPFC and a second region, the dorsal caudate, patients with FEP were treated with oral risper

233 ndogenous dopamine levels in the head of the caudate predict changes in resting-state functional conn

234 a expansions within the striatum (i.e. motor caudate) predicted better lower extremity motor score at

235 Greater caudate prediction error response when underweight was a

236 nsitivity correlated positively with ventral caudate prediction error response.

237 distribution volume ratio with respect to a caudate pseudo-reference region.

238 , displayed down-regulated expression in the caudate putamen and NAc of morphine, but not cocaine, ab

239 activated regions include accumbens nucleus, caudate putamen, claustrum, bed nucleus of the stria ter

240 was also a prominent increase in GMD in the caudate putamen.

241 describe pharmacokinetic-occupancy curves in caudate, putamen and thalamus.

242 increases and decreases in subregions of the caudate, putamen, and hippocampus in 22q-dup relative to

243 Striatal volume (sum of caudate, putamen, and nucleus accumbens volumes) and ICV

244 actor analysis, and Pearson correlations for caudate, putamen, and pallidum (also correlated with age

245 patients in the amygdala, nucleus accumbens, caudate, putamen, and posterior ventral thalamus, while

246 The left and right SBRs for caudate, putamen, and striatum were evaluated with the m

247 he NHP experiment, binding potentials in the caudate, putamen, and substantia nigra (4.9, 4.9, and 1,

248 l D2R availability compared with nonsmokers (caudate, putamen, and ventral striatum) and with ex-smok

249 verage SBRs and z scores for whole striatum, caudate, putamen, anterior putamen, and posterior putame

250 est network-wide hyporeactivity of striatal (caudate, putamen, nucleus accumbens) and cortical (insul

251 in four basal ganglia regions, including the caudate, putamen, nucleus accumbens, and globus pallidus

252 olume and surface-based shape metrics of the caudate, putamen, pallidum, and nucleus accumbens in 53

253 ctive interfering particles (DIPs), into the caudate-putamen (CP) and scored for an innate immune res

254 choroid plexus, 0.29 ug . g(-1) +/- 0.05 in caudate-putamen, 0.26 ug . g(-1) +/- 0.05 in reticular n

255 ising the nucleus accumbens core, shell, and caudate-putamen, are instrumental for a wide-range of fu

256 cortical brain regions (cortex, hippocampus, caudate-putamen, nucleus accumbens, thalamus, and hypoth

257 onounced in the basal ganglia, including the caudate-putamen, striatum and substantia nigra.

258 nked as follows: cingulate cortex > insula > caudate/putamen > frontal cortex > temporal cortex > tha

259 (TRAP) to conduct transcriptomic analyses of caudate/putamen (striatal) cell type-specific gene expre

260 he amygdala, and decreased metabolism in the caudate/putamen and medial geniculate nucleus.

261 s treated with methadone exhibited increased caudate/putamen metabolism, whereas buprenorphine produc

262 The caudate/putamen was rich in alpha1, alpha2, alpha5, all

263 However, intermediate doses of intra-caudate quinpirole produced significant improvement in r

264 ioral sensitivity to specific doses of intra-caudate quinpirole.

265 ity (r=-0.289, p=0.0264), and brain atrophy (caudate r=0.178, p=0.0087; whole-brain r=0.602, p<0.0001

266 rior cingulate cortex (r = -0.56, p = 0.02), caudate (r = -0.66, p < 0.01), frontal cortex (r = -0.52

267 uction in putamen DaT and a lower putamen to caudate ratio.

268 patients with PD showing a lower putamen to caudate ratio.

269 Caudate and putamen DaT, putamen to caudate ratios and left-right symmetry of DaT were compa

270 in posterior OFC extending to right insula, caudate region and frontal medial OFC respectively.

271 (occ;30-60) was significantly greater in the caudate region of subjects in the DD group (ES = 0.83, c

272 t amygdala/hippocampus and bilateral putamen/caudate relative to antipsychotic-treated patients and c

273 Exploratory analyses indicated that lower caudate response to tastants when hungry was associated

274 ed (behavioural variant, anterior insula and caudate; semantic variant, anterior temporal cortex; non

275 Electrical microstimulation at caudate sites during motion viewing affected the monkeys

276 Caudate stimulation enhanced learning.

277 se findings suggest that temporally specific caudate stimulation is a promising neuromodulation strat

278 Caudate stimulation was associated with increased dorsol

279 iatum, specifically the ventral striatum and caudate, striatal nodes implicated in motivational goal-

280 activity during memory encoding in the right caudate (t(977) = 2.992, p = 0.003) and five other corti

281 In the primate basal ganglia, the caudate tail (CDt) encodes the historical values (good o

282 ed two pathways originating from the primate caudate tail (CDt).

283 p showed a Task X Fatigue interaction in the caudate tail resulting from a positive correlation betwe

284 ty to limbic networks, associative networks, caudate, thalamus, and cerebellum was positively correla

285 ra hippocampus, amygdala, insula, cingulate, caudate, thalamus, and cerebellum) in T2DM patients.

286 ha3 and alpha5 were more concentrated in the caudate than in the putamen.

287 r density of calretinin+ interneurons in the caudate that was driven by loss of small calretinin+ neu

288 significantly dysregulated modules in the HD caudate, the most prominently affected brain region, and

289 e of cocaine dependence in human post-mortem caudate tissue.

290 the binding potential ranged from 22 in the caudate to 90 in the occipital cortex.

291 e compared single-neuron activity in FEF and caudate to each other and to accumulate-to-bound model p

292 analyses, the peak was located in the right caudate) to unexpected reward than unexpected punishment

293 airment (age, UPSIT, RBDSQ, CSF Abeta42, and caudate uptake on DAT imaging) allowed prediction of cog

294 in the nucleus accumbens, dorsal putamen and caudate using immunohistochemistry.

295 ) for prediction error regression within the caudate, ventral caudate/nucleus accumbens, and anterior

296 h-analysis model indicated that the parental caudate-vmPFC connectivity in infancy predicted lower ch

297 table coparental behavioral styles; stronger caudate-vmPFC connectivity was associated with more coll

298 The negative association between the caudate volume and ADHD symptoms was not moderated by a

299 The magnitude of response in the caudate was positively correlated with a successful outc

300 Because we had prior hypotheses about the caudate, we performed a confirmatory analysis of a separ