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1 nications between the carotid system and the cavernous sinus.
2 scular lesions mainly found in the spine and cavernous sinus.
3 uperior ophthalmic vein thrombosis and bulky cavernous sinus.
4 icular and spiny endings at the level of the cavernous sinus.
6 cluding tumour activity, relationship to the cavernous sinus and patient predisposition to headache.
7 ulas are abnormal communications between the cavernous sinus and the external or internal carotid art
8 t structures, including the optic nerves and cavernous sinuses, and may result in more profound visua
9 bers that terminate in the trabeculae of the cavernous sinus as an ending that resembles a Golgi tend
10 in postmortem human brain samples, meninges, cavernous sinus (cavum trigeminale), and cranial nerves
11 tion of OCTA in a patient with dural carotid-cavernous sinus fistula (CCF), which was complicated by
12 t or in certain cases as primary treatment), cavernous sinus fistulae, parasellar syndromes, and pitu
13 superior orbital fissure, orbital apex, and cavernous sinus have been used to define the anatomic lo
19 hat tumor invasion of the medial wall of the cavernous sinus may explain the relatively low biochemic
21 a longitudinal study of patients with benign cavernous sinus meningiomas localized immediately adjace
23 ns a compelling treatment for lesions of the cavernous sinus, pineal, and sellar regions and offers i
24 vibrissae capsule, adjacent to the ring and cavernous sinuses (the areas adjacent to blood and lymph
25 In cases of severe dural AV fistula in the cavernous sinus, the pituitary gland is enlarged, which
28 erative inspection of the medial wall of the cavernous sinus, which was surgically removed when invas
29 an extended anterior MLV network around the cavernous sinus, with exit routes through the foramina o