コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 segregation, mitotic spindle formation, and cell cycle progression.
2 s the regulatory T-loop of CDKs that control cell cycle progression.
3 on and apoptosis inhibition for promotion of cell cycle progression.
4 mal ciliogenesis, and changes in mitosis and cell cycle progression.
5 virtue of its dual role in transcription and cell cycle progression.
6 aled a SMILR-dependent network essential for cell cycle progression.
7 l migration and invasion, without changes in cell cycle progression.
8 ical for controlled MPS1 activity and timely cell cycle progression.
9 ezrin suggest a potential role for Merlin in cell cycle progression.
10 D-4 appear to coordinate metabolic state and cell cycle progression.
11 M that coordinates nuclear morphogenesis and cell cycle progression.
12 e developed a novel approach to characterize cell cycle progression.
13 romosome replication, enabling robustness of cell cycle progression.
14 pool expansion by repressing an inhibitor of cell cycle progression.
15 while treatment with siSMPD4 caused delayed cell cycle progression.
16 esses histone gene expression independent of cell cycle progression.
17 l interconnectivity between ciliogenesis and cell cycle progression.
18 pregulation of IFN targets and inhibitors of cell cycle progression.
19 combination with cytoskeletal tension during cell cycle progression.
20 ucleotide, tRNA-derived fragments during the cell cycle progression.
21 k2 activity, p27 stability, and, ultimately, cell cycle progression.
22 perturbation of the endocrine system and of cell cycle progression.
23 l adaptation to the metabolic demands during cell cycle progression.
24 in kinase that is essential to viability and cell cycle progression.
25 ving E2F1 and Cyclin D1 in the regulation of cell cycle progression.
26 he MDM2 locus that suppresses p53 levels and cell cycle progression.
27 ific functions by differentially controlling cell cycle progression.
28 n-dependent kinase (CDK) complexes and halts cell cycle progression.
29 dies concluded that CCNB2 is dispensable for cell cycle progression.
30 to increased levels of p21 and inhibition of cell cycle progression.
31 (scRNA-seq) combined with gene signatures of cell cycle progression.
32 bition of Ca(2+) influx that is required for cell cycle progression.
33 criptional blockade of genes associated with cell cycle progression.
34 , suggesting intrinsic circadian patterns of cell cycle progression.
35 s known about how NAP activity is coupled to cell cycle progression.
36 ated by Ipl1/Aurora B phosphorylation during cell cycle progression.
37 s of cancer, and its down-regulation impairs cell cycle progression.
38 ays critical roles in signaling pathways and cell cycle progression.
39 es to transiently suppress DNA synthesis and cell cycle progression.
40 rease in the doubling time and impairment of cell cycle progression.
41 s and resulted in enhanced DNA synthesis and cell cycle progression.
42 utside of their canonical role in regulating cell cycle progression.
43 ellular processes, including cell growth and cell cycle progression.
44 en-induced DNA damage and maintaining proper cell cycle progression.
45 ical for extracellular matrix remodeling and cell cycle progression.
46 and associated effects on DNA synthesis and cell cycle progression.
47 ences in how the DNA damage response affects cell cycle progression.
48 to cell division or birth yet independent of cell cycle progression.
49 3 ubiquitin ligase and critical regulator of cell cycle progression.
50 ogene, promoting tumor growth by influencing cell cycle progression.
51 vating AKT signaling cascades and augmenting cell cycle progression.
52 embly and disassembly are tightly coupled to cell cycle progression.
53 erely inhibited CD4 T cell proliferation and cell cycle progression.
54 as cytoskeletal dynamics, transcription, and cell cycle progression.
55 plays a critical role in regulating meiotic cell cycle progression.
56 levels/activity of USP13 affects unperturbed cell-cycle progression.
57 hy centrioles can duplicate independently of cell-cycle progression.
58 mes very dense, almost quiescent, and ceases cell-cycle progression.
59 ulated efficient DNA replication and S-phase cell-cycle progression.
60 integrity by regulating DNA replication and cell-cycle progression.
61 inst aberrant hypoxic signaling and abnormal cell-cycle progression.
62 he cell-cycle protein cyclin A for efficient cell-cycle progression.
63 thful chromosome segregation and unperturbed cell-cycle progression.
64 nscription of hundreds of genes critical for cell-cycle progression.
65 s are necessary for kinetochore assembly and cell-cycle progression.
66 nal regulation, cellular growth control, and cell-cycle progression.
67 king nitrate assimilation and signaling with cell-cycle progression.
68 d reveals insights into the requirements for cell-cycle progression.
69 ion and tumor growth, highlighting a role in cell-cycle progression.
70 osphorylates the T-loop of CDKs that control cell-cycle progression.
71 y inhibiting RB1 function, thereby promoting cell-cycle progression.
72 lar role of lncRNAs (long noncoding RNAs) in cell-cycle progression.
73 h regulates gene expression, metabolism, and cell-cycle progression.
74 se of the cell cycle, where it is needed for cell-cycle progression.
75 this, our model captures the role of Plk1 in cell cycle progression and accurately reproduces multipl
77 lso show that the tetraspanin CD82 regulates cell cycle progression and can be used as a cell surface
78 leads to decreased forebrain size, abnormal cell cycle progression and cell death during brain devel
80 myogenesis through its important effects on cell cycle progression and cell proliferative signaling.
81 own of LXRbeta, but not ABCA1, had decreased cell cycle progression and cell survival, and decreased
82 isingly, IRX3/5 are required for appropriate cell cycle progression and chromatid segregation during
83 es (PPPs) by protein kinases is essential to cell cycle progression and control, particularly during
86 ween chromatids has the potential to disrupt cell cycle progression and genome integrity, so it is hi
87 se observations suggest a connection between cell cycle progression and Hippo pathway target expressi
88 t inhibitors cooperate to prevent viability, cell cycle progression and in vivo growth of melanomas h
89 ical defects, (iii) pUL97 expression impairs cell cycle progression and induces cell death, (iv) acti
90 hat loss of MCM10 function leads to impaired cell cycle progression and induction of DNA damage-respo
91 ls, miR-1205 promoted cell proliferation and cell cycle progression and inhibited hydrogen peroxide-i
92 Complex (APC) coactivator Cdh1 drives proper cell cycle progression and is implicated in the suppress
94 f KRT19 knockout cells identified defects in cell cycle progression and levels of target genes of E2F
96 lly, we found that DBC1 KO mice have altered cell cycle progression and liver regeneration after part
98 miR-10a-5p inhibited many genes that affect cell cycle progression and only a few inflammation-relat
99 adation at mitotic exit accelerates G1-phase cell cycle progression and perturbs global DNA methylati
100 gh matriglycan up-regulation does not affect cell cycle progression and proliferation of the cancer c
102 These data demonstrate the importance of cell cycle progression and pseudo-G(2) arrest in effecti
104 is an E2 that plays a key role in regulating cell cycle progression and requires unique structural el
105 d) and plays a key role in the regulation of cell cycle progression and root elongation, but its mech
106 signaling pathway plays an important role in cell cycle progression and stress-induced apoptosis.
107 l outcomes have been attributed to increased cell cycle progression and the development of genomic in
108 cell cycle can provide further insights into cell cycle progression and the uncontrolled proliferatio
109 control the expression of key regulators of cell cycle progression and therefore can synchronize the
110 Cyclin-dependent kinases (CDKs) regulate cell cycle progression and transcriptional processes, an
113 ng extracellular pilus filaments accelerates cell-cycle progression and cellular differentiation.
114 ch as E. coli and B. subtilis, regulation of cell-cycle progression and cellular organization achieve
115 inated role for RB1 and CDKN2a in regulating cell-cycle progression and differentiation during human
116 ion during pollen development and for normal cell-cycle progression and endoreplication during the di
117 phocytes that are associated with control of cell-cycle progression and genomic stability as well as
118 that CDK7 inhibition predominately disrupts cell-cycle progression and induces DNA replication stres
119 hosphorylates pRB and NRF1 thereby governing cell-cycle progression and mitochondrial metabolism.
120 essing either miRNA showed downregulation of cell-cycle progression and mitosis-associated proteins.
121 istic studies revealed that AS4583 inhibited cell-cycle progression and reduced DNA replication by di
123 chemically synthesized CIP peptide initiates cell-cycle progression and simultaneously inhibits surfa
124 unctions non-cell-autonomously to facilitate cell-cycle progression and stem cell proliferation.
125 tein 3 (IGFBP3), and that this regulates SCC cell-cycle progression and tumor growth in vivo Furtherm
128 vation of CDK2 mediates helicase activation, cell cycle progression, and both replication and mitotic
131 that mechanisms regulating mRNA translation, cell cycle progression, and gene expression differ from
132 ruption of genome fragile sites, inefficient cell cycle progression, and increased DNA damage if apop
133 ny formation, elicited S phase arrest during cell cycle progression, and induced both intrinsic and e
134 inds to Rb, releasing it from E2F to promote cell cycle progression, and inducing ubiquitination of R
136 additively to promote degradation of Cdkn1b, cell cycle progression, and proliferation of beta-select
137 egulated kinase (ERK) signaling, delayed ESC cell cycle progression, and repressed ESC differentiatio
138 iR-34 families have been reported to inhibit cell cycle progression, and their loss has been linked t
140 ay crucial roles in cell differentiation and cell-cycle progression, and kinase dysregulation is asso
142 these, the molecular pathways that restrict cell cycle progression are critical to the maintenance o
144 Although the major events in prokaryotic cell cycle progression are likely to be coordinated with
145 cyclin-dependent kinase 2 (CDK2), regulates cell cycle progression as cells exit quiescence and ente
146 O-Vanillin also affected networks related to cell cycle progression as well as connective tissue deve
147 of cellular processes, including apoptosis, cell cycle progression, as well as stem cell renewal.
148 st that DHX9-CIZ1 are required for efficient cell cycle progression at the G1/S transition and that n
149 cell lines by inducing DNA damage, arresting cell cycle progression at the G2/M phase, triggering apo
153 nt that modulates inflammatory signaling and cell cycle progression, but its molecular regulation is
154 the CaMKK-like protein kinase Ssp1 promotes cell cycle progression by activating the ARK Cdr2 accord
155 t kinase (CDK) inhibitor p27, which inhibits cell cycle progression by binding cyclin A- and cyclin E
156 oting complex/cyclosome (APC/C) orchestrates cell cycle progression by controlling the temporal degra
157 lization of Mad1 is tightly coordinated with cell cycle progression by kinetochore-extrinsic activity
158 Furthermore, PLCdelta4 and InsP(3) modulated cell cycle progression by regulating the expression of c
159 s a lower mass density than cytoplasm during cell cycle progression by scaling its volume to match th
160 ion, we find that surface contact stimulates cell-cycle progression by demonstrating that surface-sti
161 phase-upregulated lncRNA, SUNO1, facilitates cell-cycle progression by promoting YAP1-mediated gene e
163 cal tumor cell-intrinsic functions including cell-cycle progression, chromosomal stability, and cellu
164 s, FRMD6 inhibited viability, proliferation, cell cycle progression, colony formation, 3D spheroid gr
165 n the liver induces cyclin D1 and hepatocyte cell cycle progression; concurrent cyclin D1 ablation bl
166 Among the different cyclins controlling cell cycle progression, cyclin F does not partner with a
167 activity, checkpoint-regulated inhibition of cell cycle progression, defects in growth, and nuclear g
168 lso leads to chromosome congression defects, cell cycle progression delay, and defective chromosome s
170 s the core transcriptional machinery driving cell cycle progression, dictating the timing and fidelit
171 ic domain, and hence interfering with normal cell-cycle progression, did not affect virus replication
172 , and identify its role in the regulation of cell cycle progression, DNA repair and signal transducti
174 sists of downregulation of genes involved in cell cycle progression, DNA replication and repair and i
175 wo lesion types to the signals that regulate cell cycle progression, DNA replication, and cell surviv
176 or of the DNA damage response that regulates cell-cycle progression, DNA damage repair, and DNA repli
177 chanisms of PI3Kalpha and CDK4/6 blockade on cell-cycle progression, DNA damage response, and immune-
179 of FOXM1, a key transcriptional regulator of cell cycle progression during TCPOBOP-mediated hepatocyt
180 F interaction in regulating p53 activity and cell-cycle progression during conditions of glucose depr
181 ll, Siah2 regulation of Treg recruitment and cell cycle progression effectively controls melanoma dev
183 haracteristic HMGB4-dependent differences in cell cycle progression following cisplatin treatment.
184 g a cell cycle checkpoint inhibitor to drive cell cycle progression following ionizing radiation.
188 hat PHF2 controls the expression of critical cell cycle progression genes, particularly those related
189 A), leading to inhibition of cell viability, cell cycle progression, glucose uptake, and metabolism.
191 tigation demonstrates that Fbxl8 antagonizes cell cycle progression, hematopoietic cell proliferation
192 ar localization play key roles in regulating cell cycle progression; however, a deep mechanistic unde
193 gical contexts including cancer progression, cell cycle progression, human T cell activation, and hum
194 ions among downregulated DEG were related to cell cycle progression, immune response activation and b
196 effector T cells: mTORC1 inhibition impaired cell cycle progression in activated naive cells, but not
197 and the relationship between cell growth and cell cycle progression in bacterial systems have been pr
199 olytic virotherapy, suggesting that frequent cell cycle progression in cancer cells could make them m
200 ular phenotype was observed while monitoring cell cycle progression in cells lacking MCPH1 function.
206 the dynamics of chromosome condensation and cell cycle progression in MCPH1 deficient cells under un
209 ed the role of miR-181c in the regulation of cell cycle progression in relation to HCV infection.
210 g metabolic and biosynthetic activity during cell cycle progression in single cells, we found that ce
211 within minutes) activates Erk1/2 to modulate cell cycle progression in stem cells, which is mediated
212 component of this response, which regulates cell cycle progression in the face of replication stress
213 hepatectomy, confirming its pivotal role in cell cycle progression in this in vivo model, and enhanc
215 (Cdk)1, one of the master regulators of G2/M cell cycle progression in U. maydis, interacts and contr
216 back loop between appressorium formation and cell cycle progression in U. maydis, which serves as a "
218 C1/DN-DBC1 transitions play a role in normal cell cycle progression in vivo after cells leave quiesce
221 prolongs mitotic length and interferes with cell-cycle progression in human cells, and it causes cel
222 how bacteria colonize surfaces and regulate cell-cycle progression in response to cellular adhesion
225 cal role that this pathway has in regulating cell cycle progression, inhibiting CDK4/6 is an attracti
226 ing pathways pertinent to DNA damage repair, cell cycle progression, insulin-like growth factor signa
228 deregulated in most cancers and uncontrolled cell cycle progression is a hallmark of cancer cell.
229 s DNA damage, which induces filamentation as cell cycle progression is blocked to allow for repair.
231 rowth, and the extent of growth required for cell cycle progression is proportional to growth rate.
234 e regarding the role of FBXO31 during normal cell-cycle progression is restricted to its functions du
235 of Rb phosphorylation by cyclin D-Cdk4,6 in cell-cycle progression is unclear because Rb can be phos
236 erine/threonine kinase that is essential for cell cycle progression, is a novel and major regulator o
237 scription factor FoxM1 plays a vital role in cell cycle progression, is activated in numerous human m
238 sion plane orientation, together with proper cell-cycle progression, is critical for plant growth.
239 to DZ proliferation rather than for allowing cell-cycle progression itself and must be regulated dyna
240 cilium disassembly, long cilium, and delayed cell cycle progression leading to decreased proliferatio
242 ion of G2/M cell-cycle checkpoint allows the cell-cycle progression manifested as a phenotype resista
243 oss of pigmentation, and melanoblasts showed cell-cycle progression, migration, and cytokinesis defec
244 sion of LIN9, a transcriptional regulator of cell-cycle progression, occurs in 65% of patients with t
246 restores B-cell follicle formation, impedes cell cycle progression of pretumor B cells, and eventual
247 een interkinetic nuclear migration (INM) and cell-cycle progression of apical progenitors in the vent
252 reatment does not affect cellular viability, cell cycle progression or global cellular transcription.
254 contrast to the role of ASF1 in sporophytic cell cycle progression, our data indicate that during re
255 cation, far from being only a consequence of cell cycle progression, plays a key role in coordinating
257 eton and membrane organization to coordinate cell-cycle progression, polarized cell growth and multic
258 g the N-myristoyltransferase NMT1 suppresses cell-cycle progression, proliferation, and malignant gro
259 at impaired growth is due to perturbation of cell cycle progression rather than induction of apoptosi
260 SKP2 expression and impaired retinoblastoma cell cycle progression, re-expression of TRbeta2-46 but
262 letal metabolism, intracellular trafficking, cell cycle progression, repair/maintenance, bioenergetic
263 generation of the injured tissues: disrupted cell cycle progression, repressed renewal of Mist1-posit
264 cation, telomere maintenance and elongation, cell cycle progression, signal transduction and cell pro
265 processes, including cytoskeletal dynamics, cell-cycle progression, signal transduction, gene expres
267 ent-naive mice, MYCN overexpression promoted cell cycle progression, suppressed infiltration of cytot
268 by synchronizing PAR network activation with cell cycle progression, thereby ensuring that PAR protei
271 ily conserved ubiquitin ligase that controls cell cycle progression through spatiotemporally regulate
272 s an indirect regulator of transcription and cell cycle progression through the regulation of specifi
274 dence that CK2alpha depletion causes delayed cell cycle progression through the S-phase and defective
275 progression ensures that smaller cells delay cell cycle progression to accumulate more biomass than l
276 We used incorporation of EdU as a marker of cell cycle progression to analyze the response of severa
277 , our understanding of the molecules linking cell cycle progression to developmental time is incomple
278 thylation was functionally required to adapt cell cycle progression to the early stress response.
280 he retinoblastoma protein Rb, which inhibits cell-cycle progression until its inactivation by phospho
282 GH3 cell proliferation through induction of cell cycle progression via activation of the Akt-GSK-3be
283 the role of a S-phase up-regulated lncRNA in cell-cycle progression via modulating the expression of
284 nance of haESCs, and highlighted that proper cell cycle progression was critical for the maintenance
287 at chromosome condensation is uncoupled from cell cycle progression when MCPH1 function is lacking, r
288 gressively accumulates in the nucleus during cell cycle progression, where it interacts with class I
290 2 (CDK2) and cyclin-A expression, arresting cell cycle progression, whereas overexpression of miR-18
291 gene sets associated with cell division and cell-cycle progression, whereas TAZ preferentially regul
292 n 3 and IL13, and downregulated the genes in cell-cycle progression, which may explain the inferior p
293 n the control of stem cell proliferation and cell-cycle progression, which may have implications for
294 roliferation by suppressing CDKN1A, enabling cell cycle progression with a concomitant phosphorylatio
295 gly, expression of matriglycan is related to cell cycle progression with highest levels in S and G2 p
296 ndogenous or exogenous factors and regulates cell-cycle progression with DNA repair to minimize DNA d
298 t hepatocytes show a severe dysregulation of cell cycle progression, with incomplete mitoses, and a p