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1  cell-envelope proteins without experimental cell disruption.
2 epresentative strains that respond poorly to cell disruption.
3 ive rise to the UPB precursor membranes upon cell disruption.
4 to virtual homogeneity within a few hours of cell disruption.
5 ility disorder associated with enteric glial-cell disruption.
6 s in combination with high pressure nitrogen cell disruption.
7 h bacterial invasion, colonization, and host cell disruption.
8  ciliary beat frequency and caused ependymal cell disruption.
9 5AC mucin transcription only after bacterial cell disruption.
10  a carrier protein and is released only upon cell disruption.
11 sion conformation-specific antibody prior to cell disruption.
12 tructural modifications, indicating enhanced cell disruption.
13 antibody prevented virus-induced perineurial cell disruption.
14  electron microscope confirmed the effective cell disruption.
15 iour differently, with striatal and Purkinje cell disruptions affecting the variability and the speed
16 exes were sensitive to acid washing prior to cell disruption and caveolae enrichment.
17 onist BK or the antagonist NPC17731 prior to cell disruption and caveolae enrichment.
18 n contrast, the Zaire GP induced endothelial cell disruption and cytotoxicity in both nonhuman primat
19 tionally, the new extraction method combines cell disruption and extraction in one step, and the appr
20  processes and rather attributable to Muller cell disruption and iatrogenic damage.
21         PKD activation was maintained during cell disruption and immunopurification and was associate
22 h hydrostatic pressure (HHP), known to cause cell disruption and modify enzymatic activity and food t
23  and proline showed increased, likely due to cell disruption and release.
24 mproved, and in particular the procedures of cell disruption and storage were optimized.
25 sed bioavailability generated by the vegetal cell disruption and the release of the bioactive compoun
26 lassical autolysis, ultrasound led to a high cell disruption, and after 20h of ultrasonic treatment,
27 en by thorough optimization of cell sorting, cell disruption, and PCR conditions.
28 e state after stroke and identify systemic B-cell disruption as a target for therapeutic manipulation
29  in the hippocampus, possibly linked to grid cell disruption, as circuit mechanisms underlying spatia
30  was determined to be a lytic virus, causing cell disruption beginning at 30 hpi.
31                       After fermentation and cell disruption, BoNT/A(Hc) was purified by using a thre
32  The initial step in this technique involves cell disruption by homogenization, followed by clearance
33 pressure homogenization (HPH) for mechanical cell disruption, by enzymatic treatment for cell wall po
34 ic disruption reagents and note that genetic cell disruption can be used for functional analyses of s
35 cterized by lipid accumulation that leads to cell disruption, can be found in hair follicle-associate
36                  Recent studies suggest that cell disruption caused by cryoablation may increase the
37  activity of a second gene, mec-6, rendering cell disruption conditionally dependent on genetic backg
38 ons including extraction, brewing, microbial cell disruption, dairy processing, emulsification, ferme
39 s shown that cold plasma treatment can cause cell disruption due to reactive species generation and e
40 o tissue type due to varying efficiencies of cell disruption during organelle isolation procedures or
41       Here we report on genetically mediated cell disruption effected by the toxic Caenorhabditis ele
42 ting with small CM indentations, which after cell disruption give rise to a membrane fraction that ca
43  rapid DNA isolation method using high-speed cell disruption (HSCD) incorporating chaotropic reagents
44 sion conformation-specific antibody prior to cell disruption, indicating that the introduced disulfid
45 ure change just below that which would cause cell disruption leading to death.
46                                        After cell disruption, M-phase Golgi was recovered in two size
47 st that the changes in selectivity seen with cell disruption may be due to a loss of cellular factors
48                    Ultra Turrax was the best cell disruption method, and refrigeration was the best p
49 en cavitation) with those obtained using the cell-disruption method employed by others (homogenizatio
50 ction buffers were compared along with three cell disruption methods including bead beating, probe so
51 Yeast biomass was subjected to six different cell disruption methods prior to SFE-CO(2).
52 hat seem to leak from acidic organelles upon cell disruption, microspheres are retained within these
53  Furthermore, acid washing of cells prior to cell disruption minimally affected the CLR-associated B2
54 treated human lung microvascular endothelial cells, disruption of B1R-CPM heterodimers inhibited B1R-
55                          Compared to control cells, disruption of PRC2 in HCMV-infected THP1 or NT2D1
56                            In normal NIH 3T3 cells, disruption of Stat3 signaling with dominant-negat
57 d organization of actin filaments in Sertoli cells, disruption of the blood-testis barrier and sperma
58                                    In native cells, disruption of the C terminus-syntrophin gamma 2 P
59                              In intact yeast cells, disruption of the TRM9 gene results in the comple
60                                In human stem cells, disruption of these condensates leads to genome-w
61  replication in tumor-associated endothelial cells, disruption of tumor blood flow, and hypoxia withi
62 ed and membrane-bound proteins in eukaryotic cells, disruption of which is the basis of the congenita
63 h a novel mechanism mitigated in transformed cells: disruption of proteasome-targeted degradation of
64 as hyperphosphorylated in PTP-PEST-deficient cells; disruption of the c-Abl-PSTPIP1-PEST-type PTP ter
65 cell press or Glucanex digestion followed by cell disruption removed the capsule and produced cell wa
66 with fine-resolution microscopy and gene and cell disruptions, reveal a coherent picture of an organi
67 han other stimuli that decrypt TF, including cell disruption, TF pathway inhibitor inhibition, and ca
68 tection of microorganisms requires microbial cell disruption to release nucleic acids.
69 analysis of the biomass before and after the cell disruption treatments revealed a more efficient cel
70 iscs for cancer treatment through mechanical cell disruption under an applied rotating magnetic field
71                                              Cell disruptions using ultrasonic energy transmitted thr
72                                         Host cell disruption was measured quantitatively by a trypan
73 s noted around the iStent inject head and SC cell disruption was observed at the iStent inject insert
74 displacement close to but below the point of cell disruption yielded calibrated signal amplitudes as