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1 ablish the basic mechanism of the eukaryotic cell division cycle.
2 olling periodic gene expression in the human cell division cycle.
3 unction in cytoskeletal organization and the cell division cycle.
4 ying microtubule cytoskeleton throughout the cell division cycle.
5 chromatin need to be duplicated during each cell division cycle.
6 other growth genes to promote entry into the cell division cycle.
7 y which cell growth is controlled during the cell division cycle.
8 htly coupled to a single complete eukaryotic cell division cycle.
9 both centrioles and centrosomes in a single cell division cycle.
10 ant volume fluctuations that occur over each cell division cycle.
11 maker is not influenced by the status of the cell division cycle.
12 odification, export, and assembly during the cell division cycle.
13 restricting DNA replication to once in every cell division cycle.
14 romosome replication and does not follow the cell division cycle.
15 tion of cell division starting at the second cell division cycle.
16 d interact with, but are independent of, the cell division cycle.
17 plays important roles in the control of the cell division cycle.
18 tion of the S phase program in the mammalian cell division cycle.
19 rescentus that controls an early step in the cell division cycle.
20 accurately copies billions of DNA bases each cell division cycle.
21 that is responsible for re-initiation of the cell division cycle.
22 complex on chromatin during G1 phase of the cell division cycle.
23 ribosome synthesis and specific steps in the cell division cycle.
24 at did not affect the G1/S transition of the cell division cycle.
25 ses (Cdks) are the central regulators of the cell division cycle.
26 genesis is likely to be under control of the cell division cycle.
27 at the core of the machinery that drives the cell division cycle.
28 try into, continuation of, and exit from the cell division cycle.
29 radation of p21, a negative regulator of the cell division cycle.
30 having progressed beyond mid-S phase in the cell division cycle.
31 origin to one round of DNA replication each cell division cycle.
32 c material is accurately passed through each cell division cycle.
33 to chromatin isolated from each phase of the cell division cycle.
34 that would have normally withdrawn from the cell division cycle.
35 novel mechanism that exerts control over the cell division cycle.
36 es Start, the irreversible commitment to the cell division cycle.
37 he effects of oncogenic Ras on arrest of the cell division cycle.
38 e critical for the proper progression of the cell division cycle.
39 ntry into the DNA synthetic phase (S) of the cell division cycle.
40 block pRb(+) cells in the G(1)-phase of the cell division cycle.
41 ial for cell viability and regulation of the cell division cycle.
42 nges characteristic of cells re-entering the cell division cycle.
43 lication machinery during the S phase of the cell division cycle.
44 ck entry into the DNA-synthetic phase of the cell division cycle.
45 incompletely understood events, arrests the cell division cycle.
46 he G1 to S phase transition of the mammalian cell division cycle.
47 not enter the DNA synthetic (S) phase of the cell division cycle.
48 EE788 prevents entry into the S-phase of the cell division cycle.
49 e p27 IDR controls entry into S phase of the cell division cycle.
50 tures of cells in the committed phase of the cell division cycle.
51 s either continue to proliferate or exit the cell division cycle.
52 metabolites change significantly through the cell division cycle.
53 ow metabolic fluxes are coordinated with the cell division cycle.
54 ptional activity per DNA copy throughout the cell division cycle.
55 tion of CDK1 expression had an impact on the cell division cycle.
56 sight into the regulation of CDK1 during the cell division cycle.
57 erwent reversible phosphorylation during the cell division cycle.
58 emble of interacting cells in a model of the cell division cycle.
59 th each other and with progression through a cell-division cycle.
60 iescent and, therefore, able to re-enter the cell-division cycle.
61 ade and controlled through the course of the cell-division cycle.
62 s have transient interactions throughout the cell-division cycle.
63 tosis is the last critical decision during a cell-division cycle.
64 veloped that assumes expanding volumes and a cell-division cycle.
65 tion of the centrosome is a key event in the cell-division cycle.
66 ecular link between the cilia life cycle and cell-division cycle.
67 state stably propagates through at least 14 cell division cycles.
68 in is stably transmitted through consecutive cell division cycles.
69 fore the morula stage after only two to four cell division cycles.
70 rochromatin is gradual and requires multiple cell division cycles.
71 sis of the aneuploid cells during subsequent cell division cycles.
72 ycle times that emerge as 12-hr synchronized cell division cycles.
73 rning circadian clock-dependent synchronized cell division cycles.
74 ith each other, resulting in circadian-gated cell division cycles.
75 protect chromosome ends from erosion during cell division cycles.
76 ion complex component, double-parked protein/cell division cycle 10-dependent transcript 1, is not on
77 tor 1 [RIF1], Replication Factor A 3 [RFA3], Cell Division Cycle 13 [CDC13], Pbp1p Binding Protein [P
78 conserved dual-specificity phosphatase human cell-division cycle 14A (hCDC14A) associates with the ac
80 predicted a possible phosphorylation site by cell division cycle 2 (Cdc2), which directly phosphoryla
82 g three genes, two nuclear (beta-tubulin and cell division cycle 2) and a gene from the plastid genom
88 s showed that induction of expression of the cell division cycle 20 gene (Cdc20), a key regulator of
91 e uncover the E3 ubiquitin ligase Cdc20-APC (cell division cycle 20-anaphase promoting complex) as a
94 G(2)/M phase, partially through induction of cell division cycle 25 (Cdc25) isoform C (Cdc25C) degrad
96 henotype that is linked to overexpression of cell division cycle 25 (Cdc25)A phosphatase and cell-cyc
98 nal REM (rat sarcoma exchange motif), CDC25 (cell division cycle 25), and PR (proline-rich) tail doma
99 phorylation of p27 at Thr187 was mediated by cell division cycle 25A (Cdc25A), confirmed using Cdc25A
103 ents also decreased the levels of Cdc25B and cell division cycle 25C (Cdc25C) phosphatases with an in
105 on of the let-7 target cell cycle regulators cell division cycle 34 (Cdc34) and E2F transcription fac
106 RNA synthetase domain containing 1 (Aarsd1), cell division cycle 37 (Cdc37), and stress induced phosp
110 at shock protein 90) and its cofactor Cdc37 (cell division cycle 37 protein) are crucial to prevent t
111 ly greater proportion of the two Rho GTPases cell division cycle 42 (CDC42) and Rac family small GTPa
113 s regulated by SRC-like adaptor 2 (Sla2) and cell division cycle 42 (Cdc42) independently of Sla2's r
119 GTPases have suggested that reduced Duo and cell division cycle 42 (Cdc42) transcript expression is
120 his idea, altered expression of genes in the cell division cycle 42 (CDC42)-CDC42 effector protein (C
122 inhibition of Rac family small GTPase 1 and cell division cycle 42 activation, as well as downstream
123 the nascent axon, and the Rho GTPase Cdc42 (cell division cycle 42) activates the mPar6alpha/Par3 (P
124 Here, we investigated the role of CDC42 (cell division cycle 42) during vascular morphogenesis an
129 s of the Rho family, including Rho, Rac, and cell division cycle 42, regulate the actin cytoskeleton.
131 1, resulting in the activation of downstream cell division cycle 42/Rac family small GTPase 1 signali
132 demonstrate that T cell-specific deletion of cell-division cycle 42 (Cdc42) GTPase causes a profound
133 ing replication, PP2A exists in complex with cell division cycle 45 (CDC45) and that increased PP2A a
134 ) during S phase in yeast, and Sld3 recruits cell division cycle 45 (Cdc45) to minichromosome mainten
135 and the replicative helicase containing the cell division cycle 45 (Cdc45)/minichromosome maintenanc
138 we explore the effect of phosphorylation of cell division cycle 6 (Cdc6), a DNA replication initiati
139 breast cancer cell cycle, is associated with Cell Division Cycle 6 (CDC6), Cyclin-dependent kinase 2
140 hat the origin-recognition complex (ORC) and cell-division cycle 6 (Cdc6) proteins recognize and enci
143 ion of CHK1i 18 h after gemcitabine elicited cell division cycle 7 (CDC7)- and cyclin-dependent kinas
147 of Ste7, thereby favoring the resumption of cell division cycling after pheromone-induced growth arr
148 gulate progression through each stage of the cell division cycle and as such are major targets for de
149 ciates with Cul4A/DDB1 during an unperturbed cell division cycle and both Chk1 phosphorylation and re
151 ells, it leads to a reversible exit from the cell division cycle and entry into G0, a cell cycle stat
152 abscission of the midbody at the end of the cell division cycle and for phosphorylation and activati
153 amined Scc2 chromatin association during the cell division cycle and found that the affinity of Scc2
154 on by RNase enzymes, different phases of the cell division cycle and growth rates, and the existence
155 ENP-A is deposited at centromeres during the cell division cycle and identify an evolutionally conser
156 hat the genome is completely duplicated each cell division cycle and in how the division of cells is
157 dilute aquatic environment, coordinates the cell division cycle and multiple cell differentiation ev
159 method to examine how cell size impacts the cell division cycle and reaffirm that there is a negativ
160 alysis of 53 conditional lethal mutations in cell division cycle and RNA synthesis related genes, rev
162 budding yeast Saccharomyces cerevisiae, the cell division cycle and sporulation are mutually exclusi
163 to specific subcellular addresses during the cell division cycle and sporulation, and proper localiza
164 ated destruction of Dup is necessary for the cell division cycle and suggest that Geminin inhibition
165 eting of Epac during different stages of the cell division cycle and the structural features that are
166 d us to explore the relationship between the cell division cycle and the yeast respiratory oscillatio
169 ate coordination with other processes during cell division cycles and response to environmental cues.
170 ates that metabolic cycling does not require cell division cycling and that metabolic synchrony does
171 ic clues about cooperative regulation of the cell-division cycle and apoptosis by these oncogenes.
172 e abnormal centriole numbers within a single cell-division cycle and provide insights into the regula
173 central role in coordinating the eukaryotic cell division cycle, and also serve to integrate diverse
174 s a key cellular system coordinated with the cell division cycle, and major efforts in systems biolog
175 gest subunit of ORC, is regulated during the cell division cycle, and thus ORC is a dynamic complex.
176 sing parasites have a life cycle with unique cell-division cycles, and a repertoire of divergent CDKs
178 xcept kinesin family member 18B (KIF18B) and cell division cycle associated 3 (CDCA3) were of confirm
180 olic cycling occurs during the phases of the cell division cycle associated with mass accumulation in
182 n a low-activity state during an unperturbed cell division cycle but at the same time keeps Chk1 prim
183 ordinates the serial events required for the cell division cycle, but no Cdk1 substrate has been iden
184 protein phosphatases positively regulate the cell division cycle by activating cyclin-dependent prote
185 s, p21 and p27, which regulate the mammalian cell division cycle by inhibiting cyclin-dependent kinas
187 mosome replication are triggered during each cell division cycle by the initiator protein, DnaA.
188 size primarily modulates the duration of the cell-division cycle by controlling the G1/S transition k
189 induce DNA damage, but also during aberrant cell-division cycles caused by activated oncogenes and i
190 ant decrease in protein levels of cyclin B1, cell division cycle (Cdc) 25B, and Cdc25C, leading to ac
192 died the interaction of two oscillators, the cell division cycle (CDC) and the yeast metabolic cycle
194 bution of immunohistochemical markers of the cell division cycle (CDC) in 5 of the 16 neurogenic nich
196 s 1970s landmark papers describing the first cell division cycle (CDC) mutants in budding yeast.
197 ng the actin cytoskeleton, and with selected cell division cycle (cdc) mutations affecting processes
198 ved in the cell cycle, notably cyclin, E2F1, cell division cycle (CDC), and minichromosome maintenanc
199 n the other hand, cyclins A1, A2, B1 and B2, cell division cycle (CDC)2 and its kinase, CDC25 A and B
200 in that it segregated at the very end of the cell division cycle: cells showed a single fluorescent f
201 black yeasts displayed highly unconventional cell division cycles compared to those of traditional mo
202 lthough Cdc7p levels are constant during the cell division cycle, Dbf4p and Cdc7p-Dbf4p kinase activi
205 hat cell growth during the G(1) phase of the cell division cycle dilutes the cell cycle inhibitor Ret
206 h types of oscillations are coupled with the cell division cycle, exhibit period determination by CK1
207 ylation by DDK to form an active CMG [Cdc45 (cell division cycle gene 45), Mcm2-7, GINS (Go, Ichi, Ni
208 s involved in cellular processes such as the cell division cycle, gene transcription, the DNA damage
209 e female germ-line clones of these potential cell division cycle genes and to identify those which ca
210 e-wide program of gene expression during the cell division cycle in a human cancer cell line (HeLa) w
213 of eukaryotic chromosomes occurs once every cell division cycle in normal cells and is a tightly con
214 evels remain relatively unchanged during the cell division cycle in primary human T lymphocytes and i
215 ly uncouples centrosome duplication from the cell division cycle in prostate cancer cells through CEP
216 ssion and failed to enter the S phase of the cell division cycle in response to CSF-1 stimulation.
217 ive agents of malaria, have evolved a unique cell division cycle in the clinically relevant asexual b
219 pond to mating pheromones by arresting their cell-division cycle in G1 and differentiating into a cel
220 /DDB1 ubiquitin ligase during an unperturbed cell division cycle, in response to replicative stress a
221 number of fundamental processes comprise the cell division cycle, including spindle formation, chromo
222 enome is transcribed periodically during the cell division cycle, indicating that properly timed gene
223 Gene transcription is a noisy process, and cell division cycle is an important source of gene trans
227 of apoptosis (programmed cell death) to the cell division cycle is essential for homeostasis and gen
230 ing organisms, divergence from the canonical cell division cycle is often necessary to ensure the pro
233 hat in such a state, progression through the cell division cycle is reversibly arrested in an orderly
237 ve cell fates; glucose, which stimulates the cell division cycle, is a potent inhibitor of sporulatio
238 hat drive photosynthetic cell growth and the cell division cycle; it also exhibits a highly choreogra
240 IIIalpha is phosphorylated on Ser123 by the cell division cycle kinase Cdk2 beginning early in S pha
241 ase 5 (Cdk5), a member of the Cdc2 family of cell division cycle kinases that has recently been copur
243 t regulatory nodes that govern both the host cell division cycle machinery and the initiation of cent
245 cdc123, homolog of mammalian D123, is a new cell division cycle mutant with a G2 delay at permissive
250 ng is associated with the G0/G1 phase of the cell division cycle of slowly growing budding yeast, tra
251 fection of cells by many viruses affects the cell division cycle of the host cell to favor viral repl
253 d, taking advantage of the rapid synchronous cell division cycles of Xenopus eggs and cell-free syste
257 h its Large T stabilization domain region to cell division cycle protein 20 (Cdc20) and, possibly, cd
258 Anaphase-promoting complex (APC/C) bound to Cell division cycle protein 20 (CDC20), and ends upon mi
266 ogen-activated protein kinase (MAPK) and the cell-division cycle protein kinase Cdc2, are known to be
268 rom Cdc42, a Rho family member important for cell-division cycle regulation, of lower eukaryotes, yet
269 22q11.2 was identical to the hCDCrel (human cell division cycle related) gene that maps to the regio
273 analysis, we found that MYC even within one cell division cycle resulted in a several-magnitude incr
275 ve BrdU labeling in vivo that the progenitor cell division cycle slows down markedly as their numbers
276 ypical periods of hours, are slower than the cell-division cycle, so the state of the oscillator has
277 transduction pathways that regulate an early cell division cycle step and the gain of motility later
278 can coincide exactly with the S phase of the cell division cycle, suggesting that oxidative metabolis
279 have found that, during the G2 phase of the cell division cycle, TFAP4 is targeted for proteasome-de
280 ar organisms and in meiosis, the specialized cell division cycle that gives rise to haploid gametes.
282 To understand the genetic control of algal cell division cycle that pertains to phytoplankton bloom
283 these cleavage cycles are replaced by longer cell division cycles that include gap phases and checkpo
284 ved yeasts reveal alternative mechanisms for cell division cycles that seem likely to expand the repe
285 rapid, early cleavage cycles are replaced by cell-division cycles that lengthen and acquire G (gap) p
286 rawal of nutrients triggers an exit from the cell division cycle, the induction of autophagy, and eve
287 n of one and only one daughter centriole per cell division cycle, the prevailing view is that centrio
288 ed and stimulated to synchronously enter the cell division cycle, they entered S phase 2-3 h earlier
290 ing a DNA checkpoint kinase that couples the cell division cycle to the circadian cycle abolishes syn
291 oach by coupling transcription kinetics with cell division cycles to delineate how they are combined
292 subfamily A) and the AAA-type ATPase Cdc48 (cell division cycle), Ubr1 directs the substrate to prot
293 Gap 1/DNA synthesis)-phase transition of the cell division cycle, ultimately resulting in decreased c
294 ined impacts of the cht7 mutation during the cell division cycle under nutrient deficiency in light-d
295 ribution of membranous structures during the cell division cycle underlie the cell division defects i
297 onse that uncouples DNA replication from the cell division cycle, which is reinforced in metazoans by
299 e poles during prophase and metaphase of the cell division cycle, with diminished staining of anaphas
300 ulation of cells in the mitosis phase of the cell division cycle, with some cells in the G(0)/G(1) ph