ライフサイエンスコーパス: cell elongation

1 th through effects on cell proliferation and cell elongation.

2 ment, oriented cell division, and apicobasal cell elongation.

3 ockade all reduced MT coherence and/or tumor cell elongation.

4 ell wall softening at normal stages of rapid cell elongation.

5 ing dependent on both cell proliferation and cell elongation.

6 arily known for its role in controlling root cell elongation.

7 be predominantly attributed to variation in cell elongation.

8 rotein transduces GA signal to promote fibre cell elongation.

9 the regulation of root growth by controlling cell elongation.

10 BL1), that also antagonized BR responses and cell elongation.

11 gen peroxide and a corresponding decrease in cell elongation.

12 ittle is known about the regulation of fibre cell elongation.

13 cell shape-determining actin homolog MreB in cell elongation.

14 lacking both enzymes have a lethal block in cell elongation.

15 ns-Golgi network (TGN) is required for plant cell elongation.

16 owth inhibition, and this was due to reduced cell elongation.

17 eduction in spur length due to a decrease in cell elongation.

18 is to repress JA signaling and allow normal cell elongation.

19 d allow the expression of genes required for cell elongation.

20 of periaxin, a protein required for Schwann cell elongation.

21 type III carboxypeptidase in the control of cell elongation.

22 Binding of BR to BRI1 primarily promotes cell elongation.

23 icating that HBI1 is a positive regulator of cell elongation.

24 cal wall of the cell, supporting its role in cell elongation.

25 hange of SLR1 to promote its degradation for cell elongation.

26 further demonstrate roles in BR promotion of cell elongation.

27 ith actin cytoskeletal assembly during fiber cell elongation.

28 pporting a role of HERK1/THE1 in BR-mediated cell elongation.

29 ly independent of the BR pathway to regulate cell elongation.

30 f the mechanism that brings about controlled cell elongation.

31 , extensin arabinosylation--is important for cell elongation.

32 nam) and compound A22, both of which inhibit cell elongation.

33 otype with defects in both cell division and cell elongation.

34 n of both interdivision time and the rate of cell elongation.

35 xpansin (EXP) that are involved in promoting cell elongation.

36 arrays of microtubules that drive apicobasal cell elongation.

37 plants were dwarf, due primarily to reduced cell elongation.

38 that are likely to regulate the response of cell elongation.

39 nd might be a general mechanism in polarized cell elongation.

40 l before cell constriction and contribute to cell elongation.

41 ucleus, which mediate chilling tolerance and cell elongation.

42 that a distinct regulatory pathway promotes cell elongation.

43 und that these molecules are dispensable for cell elongation.

44 e Rho-kinase (Rok) and myosin II in anaphase cell elongation.

45 Depletion of CgtAV did not result in cell elongation.

46 agA to gastric epithelial cells and initiate cell elongation.

47 stalk synthesis may be a specialized form of cell elongation.

48 ortant roles in various processes, including cell elongation.

49 some of which have roles in cell division or cell elongation.

50 , plants rely on oriented cell divisions and cell elongation.

51 drive lateral PGN incorporation, leading to cell elongation.

52 in coordinating chromosome segregation with cell elongation.

53 litates chromosome segregation and modulates cell elongation.

54 LN229 cells was also correlated to increased cell elongation.

55 ganized cortical microtubules and suppressed cell elongation.

56 dependent (non-genomic) activity of auxin in cell elongation.

57 he cleavage site by undergoing two phases of cell elongation.

58 of MapZ and FtsZ positioning and subsequent cell elongation.

59 f secondary cell wall synthesis during plant cell elongation.

60 a scaffolding protein thought to orchestrate cell elongation.

61 a common transcriptional network to promote cell elongation.

62 ificial proton pump activation inhibits root cell elongation.

63 ificial proton pump activation inhibits root cell elongation.

64 re dependent on actomyosin contractility and cell elongation.

65 eral root cap associated with the control of cell elongation.

66 These included lack of fiber cell elongation, abnormal proliferation in prospective l

67 ittle consideration has been given as to how cell elongation affects the distribution of the key regu

68 ing both a transient decrease in the rate of cell elongation after 1.5 h but also a longer term gradu

69 ral root initiation in roots showing reduced cell elongation after auxin treatment.

70 utation that results in constitutive swarmer cell elongation also increased wosA transcription.

71 Overexpression of srk1 induces cell elongation, an indication of cell cycle G(2) delay,

72 appear to correlate with differences between cell elongation (anaerobic conditions) versus cell divis

73 e set of genes including those implicated in cell elongation and 16% of the genes affected in herk1 t

74 abidopsis thaliana, resulting in anisotropic cell elongation and a weak cell wall.

75 remodeling the basement membrane, promoting cell elongation and actin cytoskeletal reorganization, a

76 ibutes more strongly than the PKA pathway to cell elongation and adhesion, whereas nutrient limitatio

77 7, a novel cell polarity protein, the normal cell elongation and alignment upon leaving the primitive

78 d shade by the cotyledons triggers hypocotyl cell elongation and auxin target gene expression.

79 ex triggers extensive and unexpected Schwann cell elongation and branching to form long, parallel pro

80 Intrinsically disordered proteins control cell elongation and carbon reserves via an order-by-diso

81 This accumulation promotes asymmetrical cell elongation and causes differential growth between t

82 ls show aberrant morphology characterized as cell elongation and cell body rounding, loss of lamellip

83 well established roles of BRs in regulating cell elongation and cell division events, BRs also gover

84 ct modes of cell wall synthesis, involved in cell elongation and cell division, which are believed to

85 g involves coordinated adjustment of spatial cell elongation and cell flux.

86 Depletion of oMAP4 impairs cell elongation and cell-cell fusion.

87 processing in MarP-deficient cells leads to cell elongation and chain formation, a hallmark of proge

88 stomatal responses require reversible guard cell elongation and contraction.

89 , whereas ROCK1 depletion selectively led to cell elongation and defective tail retraction.

90 omatids from the cleavage site in a two-step cell elongation and demonstrate the role of myosin efflu

91 n is not as severe but also results in polar cell elongation and differentiation defects.

92 hape of the ocular lens depend on epithelial cell elongation and differentiation into fiber cells, fo

93 proteins are core cell wall synthases of the cell elongation and division machinery, and represent at

94 ansglycosylases RodA and FtsW, which support cell elongation and division respectively.

95 lts are consistent with a model in which the cell elongation and division systems are in competition

96 Auxin is a phytohormone involved in cell elongation and division.

97 ivities of cytoskeleton complexes that drive cell elongation and division.

98 ly understood morphogenetic functions during cell elongation and division.

99 s from the regulation of enzymes involved in cell elongation and division.

100 morphogenic processes that are distinct from cell elongation and division?

101 al due to the confounding interdependence of cell elongation and establishment of apico-basal polarit

102 stress for 24 h resulted in flow adaptation: cell elongation and formation of actin stress fibers ali

103 on to discover kinesins that are involved in cell elongation and found Gh KINESIN-4A expressed abunda

104 lant growth regulators, yet both can promote cell elongation and growth.

105 both the transcription of genes involved in cell elongation and hypocotyl growth.

106 oteolytic activity correlated with increased cell elongation and increased single-cell migration.

107 e retraction, but can support some germ band cell elongation and is thus not a full phenocopy of ush

108 r cell polarity (PCP) proteins that regulate cell elongation and mediolateral alignment.

109 During development, epidermal cell elongation and microtubule alignment occur simultan

110 genes appear to be specifically required for cell elongation and nectary maturation in the Aquilegia

111 to the midcell plane where it promotes zonal cell elongation and normal morphology.

112 n due to GA3-stimulation or TE-inhibition of cell elongation and production rate in leaves for both c

113 r endogenous GA4 content in leaf and greater cell elongation and production rates under the untreated

114 the individual contributions of differential cell elongation and proliferation to defining the apical

115 endent actin remodeling promotes endothelial cell elongation and proper organization of VE-cadherin i

116 to BR-related phenotypes, including impaired cell elongation and reduced expression of BR target gene

117 gulates shootward auxin transport to control cell elongation and root growth.

118 in and ethylene in control of root epidermal cell elongation and root hair elongation.

119 cking ampD, to analyze PG degradation during cell elongation and septation.

120 ed microtubules during polarized endothelial cell elongation and that depletion of FMNL3 retards elon

121 ive model for the coupling between bacterial cell elongation and the accumulation of an essential div

122 The model mimics the cell elongation and the Poisson effect (necking) that oc

123 Here, we examine the role of PME in cell elongation and the regulation of its secretion and

124 urther emphasizes the importance of RG-II in cell elongation and the utility of glycosyltransferase i

125 n of cell wall pectin is a key to regulating cell elongation and ultimately the shape of the plant bo

126 an important role in balancing cotton fiber cell elongation and wall synthesis.

127 icity and porosity, important parameters for cell elongation and water uptake.

128 s, rearrange the actin cytoskeleton to cause cell elongation, and alter junctional zona occludens 1 a

129 equatorial myosin II recruitment, prevented cell elongation, and caused a remarkable spindle defect

130 ctivity is required for cell adhesion, polar cell elongation, and cell differentiation.

131 pathways were involved in lignan synthesis, cell elongation, and fatty acid biosynthesis, all of whi

132 s, controlling the extent and orientation of cell elongation, and hence organ growth.

133 quired for normal cell production and normal cell elongation, and its natural genetic variation is in

134 as necessary for persistent cell protrusion, cell elongation, and stable cell orientation in 3D colla

135 for the maintenance of junctional integrity, cell elongation, and suppression of proliferation, pheno

136 play redundant roles in cell wall mechanics, cell elongation, and the axial growth of various vegetat

137 ing increasing stress-fiber polarization and cell elongation; and 2) entropic guidance on larger stri

138 diameter) nanotubes elicited a dramatic stem cell elongation (approximately 10-fold increased), which

139 We show that cortical changes underlying cell elongation are more sensitive to depletion of Rok a

140 ating growth to predawn, when conditions for cell elongation are optimal.

141 re short and thick, have reduced anisotropic cell elongation, are suppressed in a root-waving phenoty

142 domain oriented to the cortical cells during cell elongation as well as subsequent polar nuclear move

143 egulators of apical constriction-independent cell elongation, as alpha-Spectrin and integrin mutant c

144 ults from differential growth (i.e. enhanced cell elongation at the proximal abaxial side of the peti

145 3D) array that is oriented transverse to the cell elongation axis in wild-type plants and is oblique

146 lied either parallel or perpendicular to the cell elongation axis.

147 here bacterial cell division is arrested and cell elongation begins.

148 ysis showed that decreased cell division and cell elongation both contributed to the shortened leaves

149 ve regulator in light-mediated inhibition of cell elongation but a positive regulator in light-regula

150 affects the ability of cytokinin to inhibit cell elongation but not cell proliferation.

151 iginally characterized for their function in cell elongation, but it is becoming clear that they play

152 lates Arabidopsis root growth by controlling cell elongation, but it is currently unknown whether GA

153 ponsive MAP kinases, Fus3 and Kss1, promotes cell elongation, but only Fus3 promotes chemotropic grow

154 's abilities to regulate gene expression and cell elongation, but these defects are rescued by TPL fu

155 Consistent with a model in which CagA causes cell elongation by inhibiting the disassembly of adhesiv

156 elix-loop-helix (HLH) factors, which promote cell elongation by interacting antagonistically with ano

157 crescentus, FtsZ also plays a major role in cell elongation by spatially regulating the location of

158 t Cell Elongation posits that auxin promotes cell elongation by stimulating cell wall acidification a

159 t cell elongation posits that auxin promotes cell elongation by stimulating cell wall acidification a

160 esulting in differential gene expression and cell elongation causing the organ to bend.

161 gical changes in susceptible GNBs, including cell elongation, cell swelling, or lysis, at 90 min.

162 The degree of cell elongation correlated with the length of the traili

163 ally expressed, which is consistent with the cell elongation defect phenotype and the changes in the

164 y, and the yip4a yip4b double mutants have a cell elongation defect.

165 II activation, only partially suppressed the cell-elongation defect and the furrowing delay, but prev

166 ygalacturonase gene NIMNA (NMA) that lead to cell elongation defects in the early embryo and markedly

167 S-A causes apical surface irregularities and cell elongation defects.

168 emergence of lateral root primordia and root cell elongation depended on the rootward auxin stream an

169 Hertwig's rule, which holds that cell elongation determines division orientation, is ther

170 a possible association of this process with cell elongation/division interference.

171 in diverse processes, including PG turnover, cell elongation/division, and antibiotic resistance.

172 hain of antagonistic switches that regulates cell elongation downstream of multiple external and endo

173 m distachyon with dramatically enhanced root cell elongation due to increased cellular auxin levels h

174 We show that differential cell elongation during apical hook development is defect

175 CEK3, a Cho-specific kinase, is involved in cell elongation during root development.

176 This is caused by reduced cell elongation during the cold photoperiod.

177 int to a major role for HPATs in influencing cell elongation during tip growth in plants.

178 dies demonstrated that they are required for cell elongation during vegetative growth as herk1 the1 d

179 lization of apical junctions, which promotes cell elongation, during epithelial morphogenesis.

180 SMCs, suggesting that NOR-1 is a mediator of cell elongation effects.

181 Phototropism, or the differential cell elongation exhibited by a plant organ in response t

182 s from the surface ectoderm, as evidenced by cell elongation, exit from the cell cycle, and expressio

183 xin and brassinosteroid signaling as well as cell elongation/expansion, and increased expression of A

184 One of the most abundant proteins in the cell, elongation factor Tu, was found to be more oxidati

185 root elongation pom2 mutants are impaired in cell elongation, fertility, and microtubule-related func

186 t responds to exogenous strain by undergoing cell elongation, forming polarized apical microtubules,

187 AP cell elongation forms a gradient culminating at the post

188 The AP cell elongation gradient remains when mesoderm invaginat

189 of cell cycle, cell wall, organ initiation, cell elongation, hormone homeostasis, and meristem activ

190 n DivIVA (Wag31), responsible for asymmetric cell elongation, however the biological role of this int

191 cytoskeleton is understood to participate in cell elongation; however, a detailed description and mol

192 EPIYA-D motif impacted early changes in host cell elongation; however, the degree of elongation was c

193 to be non-responsive to gibberellin (GA) for cell elongation, hypersensitive to the GA synthesis inhi

194 al abiotic stress conditions could stimulate cell elongation in an ethylene-dependent manner.

195 arriers mediates the control of differential cell elongation in apical hook development.

196 environmental, and developmental controls of cell elongation in Arabidopsis hypocotyl.

197 id-dependent molecular circuit that promotes cell elongation in Arabidopsis hypocotyls.

198 ng lipid changes alter cellulose content and cell elongation in Arabidopsis.

199 ory network by which cytokinin inhibits root cell elongation in concert with the hormones auxin and e

200 putative transcription factor that regulates cell elongation in developing styles.

201 K6 suppresses ectopic outgrowth and promotes cell elongation in different regions of the hypocotyl.

202 cal microtubules and cell wall mechanics and cell elongation in flowering plants.

203 Mutations in CSI1 cause defective cell elongation in hypocotyls and roots and reduce cellu

204 The induction of cell elongation in hypocotyls requires temperature sensi

205 capable of intrinsically promoting hypocotyl cell elongation in light-grown plants, independently of

206 in biosynthesis pathway and thereby enhances cell elongation in low R:FR.

207 uxin, which regulates both cell division and cell elongation in plant development, are controlled by

208 Cell elongation in plants is controlled by environmental

209 se, the trehalose-degrading enzyme, inhibits cell elongation in response to heat and geldanamycin.

210 ctively, to integrate chilling tolerance and cell elongation in rice (Oryza sativa) (FSD2, Fe-superox

211 nsporter is required for ROS homeostasis and cell elongation in roots and that this balance is pertur

212 re it triggers local brassinosteroid-induced cell elongation in seedling stems, which depends upon a

213 Cell elongation in the basal part of the hypocotyl under

214 growth and developmental processes including cell elongation in the hypocotyl, whether or not it modu

215 a1, but not bif2, also function in promoting cell elongation in the inflorescence.

216 E, encoding a DL-endopeptidase important for cell elongation, in the ugtP mutant background produced

217 Effectors of cell elongation include microtubules and microtubule-ass

218 xin, showing that periaxin regulates Schwann cell elongation independent of its role in the dystrogly

219 n we found that DdcA prevents YneA-dependent cell elongation independent of YneA localization.

220 yB-interacting capacity to control hypocotyl cell elongation, independently of its ability to bind DN

221 tip involve cellular mechanisms that include cell elongation, intercalation, convergent extension, pr

222 Growth by cell elongation is a morphological process that transcen

223 Bacterial cell elongation is controlled by actin-MreB while cell d

224 mechanism of plant growth and morphogenesis, cell elongation is controlled by many hormonal and envir

225 ) primary roots to water deficit showed that cell elongation is maintained preferentially toward the

226 Cell elongation is promoted by different environmental a

227 o a highly mobile ring-like structure during cell elongation is quickly followed by the recruitment o

228 he epigenomic state and cell division versus cell elongation is suggested, as no differences in DNA m

229 Polarized cell elongation is triggered by small molecule cues duri

230 er, similar to Brachypodium distachyon, root cell elongation is, in general, robustly buffered agains

231 Moreover, similar to Brachypodium, root cell elongation is, in general, robustly buffered agains

232 steroid (BR), and gibberellin (GA), regulate cell elongation largely by influencing the expression of

233 1 cKO lenses displayed delayed primary fiber cell elongation, lenses from both Rac1 cKO strains were

234 onal laccase enzymes play important roles in cell elongation, lignification and pigmentation in plant

235 t necessary for glycan polymerization by the cell elongation machinery, as is commonly believed.

236 ant suggests that cell wall insertion during cell elongation normally occurs along two helices of opp

237 t tip where transition from cell division to cell elongation occurs.

238 n African rice, which regulates longitudinal cell elongation of the outer and inner glumes.

239 LF (rapid alkalinization factor), suppresses cell elongation of the primary root by activating the ce

240 gs is required for the specific processes of cell elongation or division, while the cell wall synthes

241 th, possibly repressing cell division and/or cell elongation or the length of time that cells elongat

242 Here we further characterized the AP cell elongation patterns during GBE, by tracking cells a

243 ways were important in the H. pylori-induced cell elongation phenotype.

244 proposed to cause cell crowding, leading to cell elongation (placode formation).

245 he long-standing Acid Growth Theory of Plant Cell Elongation posits that auxin promotes cell elongati

246 he long-standing Acid Growth Theory of plant cell elongation posits that auxin promotes cell elongati

247 Protrusion occurs through differential cell elongation, probably mediated by Islet, as we find

248 Z, MurG fails to accumulate near midcell and cell elongation proceeds unperturbed in appearance by in

249 In this study we have used the differential cell elongation process during apical hook development t

250 It was hypothesized that AQPs contribute to cell elongation processes by allowing water influx acros

251 aterial precedes and predicts the process of cell elongation provide support for the idea that the in

252 show that the GA-induced boost of hypocotyl cell elongation rate is not dependent upon the maintenan

253 the doubling time, rather than in the single-cell elongation rate, (iii) the division rate increases

254 During compression, the cell elongation rate, proliferation rate, DNA replicatio

255 ient segrosomes separation and increased the cell elongation rate.

256 ggested that the observed changes in abaxial cell elongation rates during ethylene treatment should r

257 nversion to a physiological phenotype namely cell elongation, reduced proliferation, lowered angiogen

258 pose that activation of UNC-82 kinase during cell elongation regulates thick filament attachment or g

259 3 genes preferentially expressed in fiber, a cell elongation regulator, PRE1, is strikingly At biased

260 regulating expression of photosynthetic and cell elongation-related genes in etiolated seedlings.

261 In Escherichia coli, cell elongation requires crosslink cleavage by PG endope

262 The coordination of cell elongation requires E-cadherin-mediated cell-cell a

263 systematic, parallel analysis of endothelial cell elongation response to different fluidic shearing p

264 Spirogyra produces ethylene and exhibits a cell elongation response to ethylene.

265 PG synthesis complexes that function during cell elongation (RodA-PBP2) and cell division (FtsW-FtsI

266 he roles of two other proteins important for cell elongation, RodA and MreB.

267 l processes, including cell differentiation, cell elongation, seed germination, and response to abiot

268 idoglycan hydrolysis, a process required for cell elongation, separation of progeny cells, and cell w

269 n pollen tubes, both the Ca(2+) gradient and cell elongation show oscillatory behavior, reinforcing t

270 rase-transpeptidase PBP1A interacts with the cell elongation-specific transpeptidase PBP2 in vitro an

271 2, which is commonly branded as an essential cell elongation-specific transpeptidase, switches its lo

272 tage, a stomata differentiation stage, and a cell elongation stage.

273 BH1 causes a severe dwarf phenotype, but the cell elongation suppression mechanism is still not well

274 lobacter crescentus is a specialized form of cell elongation that confers to the cell substantial adv

275 rated apical constriction and Rac1-dependent cell elongation that controls cell shape and thus curvat

276 ck revealed that although plasmolysis slowed cell elongation, the cells nevertheless "stored" growth

277 trient limitation the MAPK pathway regulates cell elongation, the PKA pathway regulates unipolar budd

278 GhHOX3 function antagonistically to regulate cell elongation, thereby establishing temporal control o

279 The cell wall modification suppresses root cell elongation, thereby generating short roots, whereas

280 Cytokinin regulates root cell elongation through ethylene-dependent and -independ

281 these conditions, by concomitantly promoting cell elongation through intrinsic transcriptional-regula

282 teract to consolidate the phase of hypocotyl cell elongation to peak at dawn under diurnal cycles in

283 The marked sensitivity of cell elongation to Rok depletion was highlighted by RNAi

284 ting the developmental transition from rapid cell elongation to secondary cell wall synthesis.

285 n antibiotic which specifically inhibits the cell elongation transpeptidase penicillin binding protei

286 Cell elongation triggered by human body temperature invo

287 verexpression of PUN1 results in exaggerated cell elongation under conditions of nitrogen stress.

288 scales with cell apical area to limit larger cell elongation under mechanical stress.

289 ng-term, phy-imposed inhibition of hypocotyl cell elongation under prolonged, continuous irradiation.

290 PCP) signaling is essential for mediolateral cell elongation underlying these movements, but how this

291 BC transporter, FtsEX, which is required for cell elongation, unlike cell division as in Escherichia

292 ds (BRs), play essential roles in modulating cell elongation, vascular differentiation, senescence an

293 or instance, brassinosteroids (BRs) regulate cell elongation, vascular differentiation, senescence an

294 w that bacterial cell division is coupled to cell elongation via a direct and essential interaction b

295 shion to Vpr; however, no additive effect of cell elongation was observed when srk1 and vpr were coex

296 y prior to the colonization of land and that cell elongation was possibly an ancestral ethylene respo

297 ontrast to its family members, which promote cell elongation when overexpressed in cells, PEAK3 overe

298 zones of cell proliferation and the zone of cell elongation where differentiation begins.

299 e focused on the earliest cell shape change, cell elongation, which occurs during anaphase B and prio

300 Parallel flow caused cell elongation with enhanced stress fibers and p-FAK, a