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1 ll and cell membrane components required for cell enlargement.
2 nce, describing water uptake and vacuole and cell enlargement.
3 vacuole and limits the rate and direction of cell enlargement.
4 he apical surface may contribute to umbrella cell enlargement.
5 ne of roots supported a role for V-ATPase in cell enlargement.
6 followed by wall surface expansion and plant cell enlargement.
7 ary fresh weight appears to be the result of cell enlargement.
8 ll polysaccharides, permitting turgor-driven cell enlargement.
9 wth, a developmental process involving rapid cell enlargement.
10 plants is characterized by highly regulated cell enlargement.
11 determined by the orientation and extent of cell enlargement.
12 ead in eukaryotes and required for regulated cell enlargement.
13 ivates the cAMP/PKA signal pathway to induce cell enlargement.
14 ighly developed bundle sheath and subsequent cell enlargement.
16 h induced by SAG deletion was accompanied by cell enlargement and abnormal cell cycle profiling with
19 have observed that inactivating EXT1 induces cell enlargement and enhances metabolic switches such as
20 .3 blocker, inhibited DTH and suppressed Tem cell enlargement and motility in inflamed tissue but had
21 e intra-ER crystal growth was accompanied by cell enlargement and multinucleation and continued until
25 ssary for establishing senescence-associated cell enlargement and pro-inflammatory senescence-associa
26 the one hand, we found that the kinetics of cell enlargement and the final size of the tracheids wer
27 ound 10 d after pollination, coincident with cell enlargement and the onset of starch and storage pro
28 s induces hallmarks of senescence--including cell enlargement, and greater glucose uptake and mitocho
29 algal cultures, RMI caused loss of motility, cell enlargement, and vacuolization in the algal cells.
30 pal primordia had accelerated cell division, cell enlargement, anisotropic growth, and decoupling of
32 or pharmacological interventions that reduce cell enlargement attenuate SASP with minimal effect on s
33 rofibrils in the primary cell wall, limiting cell enlargement by restricting the ability of microfibr
34 cell function in vivo and propose that stem cell enlargement contributes to their functional decline
35 IS subjects demonstrated significant adipose cell enlargement; decrease in the percentage of small ad
36 ulates cell growth by specifically affecting cell enlargement, endoreduplication and/or cell division
39 By 12 wk, cell cycling subsided; instead, cell enlargement (hypertrophy) was seen, without fibrosi
40 nduce cardiomyocyte growth, characterized by cell enlargement (hypertrophy), increased protein synthe
43 highlights an unexpected instructive role of cell enlargement in modulating the SASP and reveals a me
44 yo development is accompanied by significant cell enlargement in some mutants (ttn1 and ttn5) but not
48 de range of effects provoked by NCRs such as cell enlargement, membrane alterations and permeabilizat
49 romosome II-deficient cells undergo dramatic cell enlargement, nucleoid condensation and degradation,
50 non-cell-autonomous manner to induce apical cell enlargement on both sides of their expression borde
51 ted to the function of the auxin hormones in cell enlargement such as protein disulfide-thiol interch
52 nt primary cell wall acts as a constraint to cell enlargement, this process may be integral to plant
53 ZmTIP1 expression in meristems and zones of cell enlargement: tips of primary and lateral roots, lea
54 cally, actin cytoskeleton remodeling couples cell enlargement to the nuclear localization of GATA4 an
56 ne the molecular mechanisms regulating plant cell enlargement, we have conducted a molecular genetic