コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 milar levels of insulin sensitivity and beta-cell function.
2 ental stage and that is adjusted for optimal cell function.
3 analysis and providing valuable insight into cell function.
4 immunosuppressive capabilities and promote T cell function.
5 l endothelial, macrophage, and smooth muscle cell function.
6 potential role for this factor in progenitor cell function.
7 meristem is essential for understanding stem cell function.
8 type-specific cell density is important for cell function.
9 sulinemia, and deteriorating pancreatic beta-cell function.
10 ates inflammation and endothelial progenitor cell function.
11 ing that directly contributes to enhanced NK cell function.
12 ia associated with continued decline in beta-cell function.
13 bolic model of the microvascular endothelial cell function.
14 milar levels of insulin sensitivity and beta-cell function.
15 liferation but does not appear to alter beta-cell function.
16 cantly reduced, resulting in decreased CD8 T cell function.
17 s, rather than by supplying GALC for Schwann cell function.
18 provide new strategies for reinvigorating T cell function.
19 hat impair, but do not completely abolish, T-cell function.
20 n or defective development) or impaired beta-cell function.
21 or maintenance without affecting normal stem cell function.
22 ging showed the greatest improvement in beta cell function.
23 maintaining Wnt signaling and canonical stem cell function.
24 or growth without negatively affecting CD8 T cell function.
25 , T regulatory cell depletion, or effector T cell function.
26 ial role of a 3-D environment for testing NK cell function.
27 oliferation without negative effects on beta-cell function.
28 recurrence in the genome or essentiality for cell function.
29 by reduced beta-cell number or impaired beta-cell function.
30 etion, as a manifestation of pancreatic beta-cell function.
31 Galpha(z)) is an important modulator of beta-cell function.
32 women was driven by improved pancreatic beta-cell function.
33 phosphorylation of Tyr-1510 of IQGAP1 alters cell function.
34 vents positive effects of bile acids on beta cell function.
35 powerfully modify multiple aspects of immune cell function.
36 's role in the regulation of pancreatic beta-cell function.
37 target for therapeutic manipulation of Treg cell function.
38 +)) signals play fundamental roles in immune cell function.
39 remodelling, cellular senescence and immune cell function.
40 splicing, antigen presentation, and CD8(+) T cell function.
41 -1 signaling in the regulation of human beta-cell function.
42 f epigenetic modulation on antitumour immune cell function.
43 MS-like disease and how these agents alter T cell function.
44 Fgfr2 expression both of which control stem cell function.
45 apies for the long-term preservation of beta cell function.
46 n opportunity to selectively regulate immune cell function.
47 ) is an essential signal for pancreatic beta-cell function.
48 he consequences of this with respect to beta-cell function.
49 processes, is essential for ensuring proper cell function.
50 this dynamism can be exploited to regulate B cell function.
51 type 1 and type 2 diabetes with reduced beta-cell function.
52 artmentalisation is necessary for eukaryotic cell function.
53 environment, the fibrotic response, and stem cell function.
54 D enrichment could be impactful to T2D islet cell function.
55 odulation of the TME to enhance antitumor NK cell function.
56 on of key transcription factors ensures beta-cell function.
57 mation, T(H)17 signaling, and natural killer cell function.
58 nd LN compared to NCs, but had similar CD8 T cell function.
59 s and the role of the molecular landscape in cell function.
60 actor eIF4G1 on glucose homeostasis and beta-cell function.
61 ta is a key regulator of regulatory T (Treg) cell function.
62 f osteoblasts and alterations in endothelial cell function.
63 ancreatic beta cell number or impairing beta cell function.
64 can be used to non-destructively determine T-cell function.
65 ffecting not only adipocytes but also immune cell function.
66 ymopoiesis generating long-term peripheral T-cell function.
67 ontinued essential roles in mature endocrine cell function.
68 e, which retained normal CD4(+) and CD8(+) T cell function.
69 se duration, in parallel with declining beta-cell function.
70 ling targets involved in the control of beta cell function.
71 proteins and lipids to regulate a variety of cell functions.
72 xpressed in a gene network related to immune cell functions.
73 the biochemical processes underlying various cell functions.
74 T cell and increasing monocyte and cytotoxic cell functions.
75 RNA sensing, cytokine responses, and immune cell functions.
76 ganismal development, homeostasis and single-cell functions.
77 tant regulator of a wide range of biological cell functions.
78 can be expected to have direct effects on NK cell functions.
79 to reveal mechanisms regulating fundamental cell functions.
80 ertoli cell epithelium and pertinent Sertoli cell functions.
81 ights into the control of stromal and immune cell functions.
82 re key for regulation of numerous eukaryotic cell functions.
83 specialized endosomes that are crucial for T cell functions.
84 controls intestinal inflammation through NK cell functions.
85 becomes particularly valuable for sustaining cell functions.
86 e of ATXN1 native interactions for correct B cell functioning.
87 absolute insulin secretion but impairs beta-cell function, 2) causes insulin resistance, and 3) redu
88 her age- and sex-dependent changes in immune cell function account for this effect remains unknown.
90 senting genetic determinants of reduced beta-cell function and abnormal hepatic lipid metabolism were
91 pex yield insight into the processes of stem-cell function and cell-fate acquisition in the maize see
93 P3(+) Tregs in vivo and suppressing effector cell function and could be the basis of effective tolera
95 ts role in the regulation of intestinal stem cell function and differentiation, however, has not been
98 re critical to the acquisition of effector T-cell function and ensuing secretion of pathogenic cytoki
99 gnificant association between T(CM) and beta-cell function and extended this to other T cell subsets.
100 ensitive ion channels are crucial for normal cell function and facilitate physiological function, suc
101 of excitotoxicity and overnutrition on beta-cell function and gene expression, we analyzed the impac
102 hypothesis that eIF4G1 is critical for beta-cell function and glucose homeostasis by genetically abl
103 hypothesis that eIF4G1 is critical for beta-cell function and glucose homeostasis by genetically abl
105 ic acid (ATRA) on wild-type NK and CD38KO NK cell function and highlighted potential benefits and dra
106 ndividually and together, impaired both beta-cell function and identity by reducing expression of gen
108 attributed mainly to increased regulatory T-cell function and increased allergen-specific IgG(4) , y
110 therefore, has an important contribution to cell function and integrity, which extends to the whole
111 (miR-199) negatively impacts pancreatic beta-cell function and its expression is highly increased in
112 tional approaches to define expression-based cell function and maturity profiles, herein called trans
114 irtuin1/EZH2 axis reduces cytolytic CD8(+) T cell function and might be targeted to overcome incidenc
115 underlying the toxic effects of As on neural cell function and neurodevelopment and identifies miR-12
117 letal muscle MHC expression on maintaining T cell function and pathogen control and argue that the no
125 icine and metformin) that alter inflammatory cell function and signalling pathways characteristic of
126 s have recently been shown to control T(reg) cell function and stability in different disease setting
127 nhibitory molecules in the control of T(reg) cell function and stability, with a focus on their roles
128 evated glucose and fatty acid levels on beta-cell function and survival, contributes to T2D-associate
129 -7, a cytokine that is critical for normal T cell function and that plays a well-established role in
130 genetic models for the study of regulatory B cell function and their potential for therapeutic interv
131 numerous organs now known to modulate immune cell function and therefore dictate immunological outcom
134 on is a novel strategy to improve multiple T cell functions and enhance ACT against solid tumors and
136 al genetic variation is associated with beta-cell functions and incident DM in non-Hispanic, Black wo
138 regions resulted in enhanced region-specific cell functions and maintained region-specific cell heter
139 ovel PD-1 substrates that modulate diverse T cell functions and may serve as future therapeutic targe
140 one marrow-derived dendritic cell-mediated T cell functions and reduce serum anti-dsDNA autoantibody
142 he biological underpinnings of leukemia stem cell function, and highlight the Sdc1-Itgbeta7 signaling
144 mmation, recovery of pathogen-specific CD4 T-cell function, and lung injury prior to and after ART in
145 elationships between chemical heterogeneity, cell function, and phenotype are not always understood.
146 et the degree of peptide specificity of Treg-cell function, and whether Treg ligands can be used to m
147 of geometric effects for a range of crucial cell functions, and are of relevance for efforts to deve
149 ockade of inhibitory receptors that limit NK cell functions, and therapeutic modulation of the TME to
151 networks, label-free tools that can modulate cell function are needed to evaluate the role of astrocy
153 domain of BTNL2, which was able to inhibit T cell function as well, exhibits distinctive structural f
154 e signaling, skin barrier function, and mast cell function, as well as pathways that have not yet bee
155 lize mTORC1 to prepare for subsequent plasma cell function, before the onset of antibody synthesis.
156 ess, additional investigations into effector cell function between KS and asymptomatic individuals ar
157 t fundamental differences in gingival immune cell function between PD and T2D-potentiated PD may acco
158 argeting ligand still permitted redirected T-cell function but significantly compromised antitumor fu
159 1 is an inhibitory receptor that regulates T cell function, but a role for PD-1 in regulating NK cell
160 tors and nucleosomes, that are necessary for cell function, but that also have the potential to imped
161 out life is critical for proper somatic stem cell function, but the complexities of the stem cell res
162 ization of RNAs and proteins is critical for cell function, but we still lack global maps and concept
163 utarate) relates mitochondrial metabolism to cell function by modulating the activity of 2-OG depende
164 xistence of a novel pathway in which myeloid cell function can be enhanced, with a key feature being
166 Mutations disrupting regulatory T (Treg) cell function can cause IPEX and IPEX-related disorders,
167 s normal testicular architecture and somatic cell function capable of supporting allogeneic donor ste
168 n and if not maintained results in defective cell function caused by the abnormal distribution of gen
169 s important in epigenetic modification, stem cell function, cell reprogramming and other processes.
170 allo-HSCT, ATG5-dependent dissociation of T-cell functions contributed to significant reduction in g
171 nowledge not previously implicated in T(reg) cell function, coregulates another gene network with SAT
172 he factors that adversely affect endothelial cell function could contribute to the development of new
174 ed bacteria as a biological probe of E. coli cell function during nitrogen starvation, we demonstrate
178 asure BCM, routine clinical measures of beta-cell function (e.g., C-peptide release) may not reflect
179 he regulation of metabolism, pancreatic beta-cell function, energy homeostasis, mood and behaviour in
182 questions otherwise intractable and engineer cell functions for future synthetic biology applications
184 ycans on cell surfaces can critically affect cell function, for example, by preventing close contact
185 interrelationship between ER stress and beta-cell function, here we treated insulin-secreting INS-1(8
186 nd the homeostatic model assessments of beta-cell function (HOMA-B) and insulin resistance (HOMA-IR),
188 e know little about how cell density affects cell function, how it is controlled, and how it sometime
189 e been previously shown to suppress effector cell function; however, their ability to treat pre-exist
190 nophenotyping, T-cell receptor clonotypes, T-cell function, immune responses to transgenes and autoan
199 irals (DAAs) and addition of RBV improves NK cell function in liver transplant (LTx) recipients.
201 roves metabolic parameters and promotes beta-cell function in mouse models of beta-cell failure actin
205 s expression changes of genes involved in NK-cell function in skin lesions of patients with atopic de
206 eptor's effect on intercalated and principal cell function in the cortical collecting duct (CCD).
207 diet (HFD)-induced obesity impairs CD8(+) T cell function in the murine TME, accelerating tumor grow
209 We show how apparent viscosity relates to cell function in the root, how the growth of cellular pr
212 ogical markers for predicting change in beta-cell function in type 1 diabetes, the finding that abata
221 a transcription activator that promotes stem cell functions in post-development mammalian cells; supp
222 f H5N1 (2:6) was due to the suppression of T cell functions in vivo Importantly, H5N1 (2:6)-infected
226 ac has emerged as a key regulator of various cell functions, including cell division, and has a patho
227 evels correlate with insulin levels and beta-cell function, indices of healthy glucose metabolism, in
229 biomarkers, anthropometry, measures of beta-cell function, insulin sensitivity, and lifestyle) data
230 Virus (HCV)-specific T cells and abnormal B cell function is a hallmark of chronic HCV infection.
233 articles show, the emerging picture of alpha-cell function is one of increased complexity that needs
238 genetic variation shapes haematopoietic stem cell function, leading to CHIP through mechanisms that a
239 nd mechanistic understanding of NONO in stem cell functions, lineage commitment and specification.
240 w that exogenous nutrients regulate the stem cell function, little is known about the effects of L-ar
246 idually had any effect on hematopoietic stem cell function or the development of mature lymphoid or m
249 en associated with innate immune and myeloid cell function pathways, whereas AA-dominant pathways mir
250 wnregulation of critical genes involved in B cell function (PAX5, CD79A, CD22, and FCRL1) and upregul
252 eatic progenitors compromised postnatal beta cell function, proliferation, and survival, preceded by
253 lar apparatus, regulating fundamental tendon cell functions relevant to exercise-induced adaptation a
260 ane fluidity plays an important role in many cell functions such as cell adhesion, and migration.
261 ular microenvironment continuously modulates cell functions such as growth, survival, apoptosis, diff
262 n factor, which is involved in critical mast cell functions such as synthesis of mediators and granul
263 volved a multitude of mechanisms to evade NK cell function, such as the targeting of pathways for NK
264 int association of 25(OH)D and PTH with beta-cell function, systemic inflammation, and kidney functio
266 p between changes in T cell subsets and beta-cell function that is a component of the natural history
267 d cell clones and induces changes in myeloid cell functions that promote atherosclerosis via inflamma
268 n with lymphopenia and alterations of CD4+ T-cell functions that renders the host prone to secondary
269 l membranes play a vital role in controlling cell functions that require local reorganization of the
271 ntracellular pH influence cancer and stromal cell function, their mutual interplay, and their interac
274 ings indicate that B cells modulate CD4(+) T cell function through an MHCII-dependent mechanism which
275 a novel role of C4S and C6S in human glioma cell functions, thus possibly representing targets in gl
276 ith aging such as reduced hematopoietic stem cell function, thymic involution and decreased lymphoid
279 ese diseases is focused on modulating T(reg) cell function to increase or decrease suppressive activi
280 vel evidence that cytokine signaling in beta-cells functions to limit viral replication and subsequen
281 MYC up-regulation in luminal prostate cancer cells functions to oppose the terminally differentiating
282 upregulate genes involved in mature pericyte cell function, together with a remarkable decrease in PI
283 muscle oxidative capacity and improves beta cell function.TRIAL REGISTRATIONClinicaltrials.gov NCT02
284 e molecular mechanism of HuR regulating Th17 cell functions, underscoring the therapeutic value of Hu
285 g future studies probing how proteins impact cell function, using transgenic variant libraries tested
287 at seek to subvert T cell and natural killer cell function via a remarkable array of mechanisms.
288 the role of NK cells in PD pathogenesis, NK cell function was investigated in a preformed fibril alp
291 To understand how these compounds alter T cell function, we assessed their therapeutic activity in
292 rm application of mechanical signals on stem cell function, we investigated the effect of LIV during
293 een speed, sensitivity, and specificity of T cell functioning, which are the main characteristics of
296 heterogeneity in NK cell age diversifies NK cell function, with "older" NK cells exhibiting more pot
297 inhibit cAMP production and downstream beta-cell function, with both activities being dependent on t
298 also produced in the pancreas, affects beta-cell function, with particular attention to the role of
299 Learning rules by which cell shape impacts cell function would enable control of cell physiology an
300 important modulators of oxidative stress and cell function, yet how mitochondrial dysfunction affects