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1 ly control different aspects of the in vitro cell microenvironment.
2 ibits tumor invasion by inducing suppressive cell microenvironment.
3 tion of both the cytokine and cellular tumor cell microenvironment.
4  reprogrammed by inductive cues in the tumor cell microenvironment.
5 y can be suppressed by factors in the tumour-cell microenvironment.
6 chain and, thus, the remodeling of the tumor cell microenvironment.
7 cosphingolipids that are shed into the tumor cell microenvironment.
8 ates a favorable reprogramming of the immune cell microenvironment.
9  regulation sensitive to the mechanochemical cell microenvironment.
10  dysfunctions of MSCs in an in vitro 3D-stem cell microenvironment.
11 ited culture conditions that mimic the tumor cell microenvironment.
12 upon continuous guidance from thymus stromal cell microenvironments.
13 erentiation within complex and heterogeneous cell microenvironments.
14 ess damaged than 2D culture due to different cell microenvironments.
15  biophysical and biochemical cues within the cells' microenvironment.
16 e ratio of protease and antiproteases in the cells' microenvironment.
17 erited and mutation-driven genotypes and the cells' microenvironment.
18 uantitative and spatial aberrations of the T cell microenvironment across and within B-NHL entities.
19                            However, how host cell microenvironments affect antibiotic accumulation an
20 on of PTN through stimulation of the stromal cell microenvironment alone may be sufficient to account
21 ist of cell models that accurately mimic the cells' microenvironment, along with flexibly prototyped
22 of ExoS ADPRT activity within the eukaryotic cell microenvironment and into possible modulatory roles
23 ormation about protein expression within the cell microenvironment and is one of the most common tech
24 rtant for a better understanding of lymphoid cell microenvironment and migrating capacities and their
25 With precise control over small volumes, the cell microenvironment and other biological agents can be
26 y linked to its ability to modulate the host cell microenvironment and to facilitate efficient produc
27 ular matrix mimetics that imitate niche stem cell microenvironments and support cell growth for techn
28 ell adhesion cues that distinguish each stem cell microenvironment, and that are critically important
29 M molecules expressed in many different stem cell microenvironments, and their corresponding receptor
30                           Bioengineered stem cell microenvironments are used to specify the initial g
31                             Heterogeneous 3D cell microenvironment arrays are rapidly assembled by co
32 sh triggers the formation of an inflammatory cell microenvironment at the implant site through comple
33 egulator of EMT and preserves thymic stromal cell microenvironment by controlling age-related adipocy
34 utations that extrinsically disrupt the stem cell microenvironment can spread in adult intestine with
35 els to emulate the complexity of the natural cell microenvironment during embryogenesis, particularly
36 modulation of JAK/STAT signaling in the stem cell microenvironment (EBs and VMs).
37 concentrations of asparagine in the leukemic cell microenvironment - for the protective effects we ob
38 ility to re-programme the local inflammatory cell microenvironment from a 'hostile' to an 'instructiv
39 e use of core-shell microgel ink to decouple cell microenvironments from the structural shell for fur
40 iated with various factors, including immune cells, microenvironment, gut microbiome, and interaction
41                      Targeting of the tumour-cell microenvironment has not been investigated as a tre
42 ches for controlling the embryonic stem (ES) cell microenvironment have been developed for regulating
43 and promote protease activation in the tumor cell microenvironment; however, uPAR also regulates cell
44 thelial cell interactions in the parathyroid cell microenvironment in both human and NHP parathyroid
45  tissue chips in space to monitor and assess cell microenvironment in real-time.
46 contribute to wound healing, the role of the cell microenvironment in tissue repair remains elusive.
47 ssociated with increased CD3(+) and CD8(+) T cell microenvironments in precancer (mostly CD3(+), link
48 l receptor (BCR) signaling and on a specific cell microenvironment, including T cells, macrophages, a
49 ological processes, such as 1) cell-cell and cell-microenvironment interaction; 2) transdifferentiati
50 targets, stem cells and disruption of plasma cell microenvironment interactions.
51 n ex vivo culture that conserves key cell-to-cell microenvironment interactions.
52 n NTNBC; (2) non-neural functions related to cell-microenvironment interactions and intracellular dam
53 ng developed that target these complex tumor cell-microenvironment interactions and target the signal
54 , the data highlight the importance of tumor cell-microenvironment interactions and the necessity to
55 multi-cellular in nature where cell-cell and cell-microenvironment interactions determine the emergen
56  deposition and explain how Bves facilitates cell-microenvironment interactions in the regulation of
57  can control precisely individual aspects of cell-microenvironment interactions is presented and they
58 itical role in prosurvival CLL cell-cell and cell-microenvironment interactions with this agent.
59 cy as readouts, which may be useful to probe cell-microenvironment interactions.
60 ther tensile or compressive depending on the cell-microenvironment interactions.
61                      Acidification of cancer cell microenvironments is a hallmark of malignant solid
62 ymph nodes (LNs) and formation of LN stromal cell microenvironments is dependent on lymphotoxin-beta
63 xtensive remodeling of the MCF-7-Ptn/NIH 3T3 cell microenvironment; it up-regulated expression of mar
64 ation of the negative charges present in the cell microenvironment led to faster peak kinetics.
65 croscale principal strains for a majority of cell microenvironments located across diverse microstruc
66  This work provides new insight into how the cell microenvironment may influence Raf-1 expression to
67 ese attributes include colocalization with B cell microenvironments, MHC class II expression, dendrit
68 t further investigation of the tumour-immune cell microenvironment might yield insights into further
69 regulates adult GSC behavior within the stem cell microenvironment (niche).
70 gain insight into the histopathologic immune cell microenvironment of arthrofibrosis, we assessed the
71 wever, the precise composition of the immune cell microenvironment of MCL, its possible relationship
72 trients) and inner (immune cells and stromal cells) microenvironment of the gut.
73 y are revealing the importance of the cancer cell microenvironment on tumorigenesis and cancer progre
74                  Certain aspects of the stem cell microenvironment, or niche, are conserved between t
75 pproach to therapy is to manipulate the stem cell microenvironment, or niche, to facilitate repair by
76               The follicular lymphoma (FL) T-cell microenvironment plays a critical role in the biolo
77 tional phenotypes are defined by the current cell microenvironment, rather than an amalgamation of pa
78 athology of the disease in response to the T-cell microenvironment remains unclear.
79 gens; however, physical constrainment by the cell microenvironment represents an additional mechanism
80  novel components of the haematopoietic stem cell microenvironment, revealing that haematopoietic ste
81                        To test the non-tumor cell microenvironment role of RAGE, we performed syngene
82 cations in basic research such as studies of cell microenvironments, stem cell niches, metaplasia, or
83 rmation and organ regeneration to engineered cell microenvironments, synthetic biomaterials and artif
84 the extracellular biochemical milieu of stem cell microenvironments that regulate pluripotent cell fa
85  activities are controlled by the local stem cell microenvironment, the "niche." Wnt signals emanatin
86 lture conditions do not simulate the natural cell microenvironment, thus hampering in vivo translatio
87                 Secondly, to analyze the AFS cell microenvironment, we injected murine YFP(+) embryon
88 eature of hypoxia, as well as tumor and stem cell microenvironments, we hypothesized that pH may regu
89 pproaches had limited success in fine-tuning cell microenvironments while generating robust macroscop
90  simultaneously targeting HER2 and the tumor cell microenvironment with a therapeutic intent.
91                   The activation of the stem cell microenvironment with parathyroid hormone induced a