ライフサイエンスコーパス: cell movement

1 ma, achieve remarkable speeds of directional cell movement.

2 exclusion limits and promoting virus cell-to-cell movement.

3 namics, thereby determining the direction of cell movement.

4 ntrol of cytoskeleton remodeling involved in cell movement.

5 proliferation, in contrast to its effects on cell movement.

6 HDAC9 in GT1-7 cells increased apoptosis and cell movement.

7 r-789 phosphorylation to positively regulate cell movement.

8 C cells, AMPK blockade resulted in increased cell movement.

9 gulators of PIP3-sensitive mRNAs involved in cell movement.

10 sufficient for this dynamic coordination and cell movement.

11 tes to alter plasmodesmata for virus cell-to-cell movement.

12 nduced apoptosis and impaired basal neuronal cell movement.

13 (ECM) proteins serve as cues for directional cell movement.

14 egulate the fundamental processes that drive cell movement.

15 y that enables proper neuronal signaling and cell movement.

16 vision, but that the number increases due to cell movement.

17 y promoting cell adherence and by inhibiting cell movement.

18 teins needed for virion assembly and cell-to-cell movement.

19 extracellular matrix, processes critical for cell movement.

20 ridge, abolished virion assembly and cell-to-cell movement.

21 g necessary and sufficient for SRBP1 cell-to-cell movement.

22 in ratio that drives membrane protrusion and cell movement.

23 surface beta1 integrin required for forward cell movement.

24 but not cell velocity or the persistence of cell movement.

25 events that define the context and instruct cell movement.

26 cluding cell division) with the mechanics of cell movement.

27 dissociation accompanied by FA turnover and cell movement.

28 nce of cell morphology and the regulation of cell movement.

29 ion number, cell-cell adhesion and polarized cell movement.

30 led similarities to cancer, development, and cell movement.

31 g axis in the dynamic control of coordinated cell movement.

32 e lineage boundary at the MHB by restricting cell movement.

33 chanisms that take place during CLas cell-to-cell movement.

34 of complex protein networks associated with cell movement.

35 anchored proteases that could contribute to cell movement.

36 ns and is well characterized for its role in cell movement.

37 amental energy requirements during mammalian cell movement.

38 ilamentous actin staining, indicating active cell movement.

39 lk4 and Arp2 in mediating Plk4-driven cancer cell movement.

40 which we now show involves concerted active cell movement.

41 vascular development, cell proliferation and cell movement.

42 ional tension and inhibiting intraepithelial cell movement.

43 on, or enhanced virion stability and cell-to-cell movement.

44 tion site, which is required for Plk4-driven cell movement.

45 ion for efficient leading edge formation and cell movement.

46 ing coordinated cytoskeletal dynamics during cell movement.

47 roid hormone metabolism with early embryonic cell movements.

48 ing the possibility that durotaxis underlies cell movements.

49 skeleton and power the membrane machinery of cell movements.

50 for organised organ growth in the absence of cell movements.

51 WNT5A ligand, leading to JNK activation and cell movements.

52 es place via a series of dramatic collective cell movements.

53 in individual cell behaviors and collective cell movements.

54 ipital tissues participate in well-conserved cell movements.

55 s as a direct and essential mediator for C&E cell movements.

56 organization underlying PCP and gastrulation cell movements.

57 ich it regulates cell polarity and polarized cell movements.

58 irectionality as well as the coordination of cell movements.

59 n levels, suggesting a role in morphogenetic cell movements.

60 actin filaments is a major driving force for cell movements.

61 ombination of changes in gene expression and cell movements.

62 ophila pupal eye is characterized by precise cell movements.

63 ncreased mitochondrial trafficking and tumor cell movements.

64 the 3V wall is not static and is amenable to cell movements.

65 rved across species and driven by collective cell movements.

66 ses within the tissue to regulate collective cell movements.

67 ial reprogramming and suppressed sEV-induced cell movements.

68 ut not XTRPM7 disrupted radial intercalation cell movements.

69 f patterning signals and major morphogenetic cell movements.

70 sotropic apical constriction and coordinated cell movements.

71 ses) are master regulators of cell shape and cell movement [1].

72 The coordination of cell movements across spatio-temporal scales ensures pre

73 -cell signaling, which consequently prevents cell movements across the MHB.

74 We analyze 2 complementary mechanisms of cell movement: actin-driven and water-driven.

75 n (BMP) signalling together with mesenchymal cell movement, acting in a coordinated reaction-diffusio

76 The mechanisms facilitating cell movement against the gradient have not been defined

77 that coordinate cytoskeletal events to bias cell movement along a chemoattractant gradient.

78 recent observations of extensive endothelial cell movement along growing blood vessels.

79 hs in the matrix and the temporal control of cell movements along these paths.

80 oth flow of cytoplasmic organelles and whole-cell movement-analogous to the autonomous motility displ

81 receptor for CXCL12, exhibit defective GnRH cell movement and a significant reduction in their numbe

82 revealed MASTL-regulated genes implicated in cell movement and actomyosin contraction, including Rho

83 renewed interest in the relationship between cell movement and affinity maturation.

84 in controlling FAK activation during planar cell movement and amoeboid motility during extracellular

85 ism and also cytoskeletal functions, such as cell movement and attachment.

86 during different stages of enamel formation; cell movement and attachment; regulation of ion and prot

87 , beta-catenin-independent pathway modulates cell movement and behavior.

88 studies indicate that DeltaNp63alpha affects cell movement and can reverse the increase of cell motil

89 he central role of cAMP in the regulation of cell movement and cell differentiation.

90 a stable demarcation line can form when both cell movement and cell growth cease at low nutrient leve

91 in the immune system have been implicated in cell movement and cell-cell interaction during the cours

92 Cell movement and cytokinesis are facilitated by contrac

93 plete, implicating a potential separation of cell movement and differentiation during this process.

94 We quantified defects in cell lifetime, cell movement and division axis in end-3(-) embryos, whi

95 ong to group I of the PAK family and control cell movement and division.

96 GF)-beta family ligand Nodal, regulates both cell movement and EMT during embryonic development.

97 ficient Tregs lost the ability to modulate T cell movement and failed to inhibit the T cell-dendritic

98 sion of about 1300 genes, mostly involved in cell movement and growth, and specifically affected meta

99 f the cytosolic protein actin is critical to cell movement and host cell invasion by the malaria para

100 ommonly increased in both GSC lines involved cell movement and included a number of genes that have b

101 infiltration, we analyzed the dynamics of T cell movement and interactions within individual islets

102 ally different behaviour, one showing robust cell movement and intercalation (in which the AVE migrat

103 that coordinate Dictyostelium morphogenetic cell movement and is highly expressed at the organizing

104 poptosis rates were observed based solely on cell movement and local conditions.

105 the underlying mechanisms that regulate PDAC cell movement and metastasis remain little understood.

106 INC00313 overexpression negatively regulates cell movement and migration pathways, and enrichment of

107 characteristics, including overexpression of cell movement and migration-associated genes in the Spin

108 d one that is static, with relatively little cell movement and mixing.

109 ngrailed 1 (En1) as a necessary regulator of cell movement and NeuC/Mes lineage boundary positioning

110 ical information emerges from self-propelled cell movement and not force transfer between cells.

111 The ratio between the distance of red blood cell movement and plasma separation is the criterion for

112 owed the role of Rho-GDI2 in regulating both cell movement and proliferation.

113 the processes of birth, death and diffusion (cell movement and public good distribution).

114 Its modular structure, the lack of cell movement and relative accessibility to microscopic

115 ievable with soluble EGF, we examined single-cell movement and signaling in human immortalized HaCaT

116 s IIa HDAC9 with Class IIb HDAC6 to modulate cell movement and survival in GnRH neurons.

117 often lacks real-time monitoring of vertical cell movement and systematically controlled chemotactic

118 CV) was necessary for efficient TVCV cell-to-cell movement and systemic infection in Nicotiana bentha

119 ts that occur during gastrulation, including cell movement and the activation of some endodermal targ

120 quired for TGB1 self-interaction and cell-to-cell movement and the amino-terminal domain required for

121 y distributed, often as gradients, to direct cell movement and the mechanisms by which they steer cel

122 Furthermore, subtle relationships between cell movement and the positive and negative interactions

123 clude that microtubules act both to restrain cell movement and to establish directionality.

124 ut dysregulates a subset of genes related to cell movement and transport.

125 air, and disease largely rely on coordinated cell movements and are controlled by the tissue environm

126 d used time lapse microscopy to characterize cell movements and behavior in wild type and mutant tail

127 ces forces that are essential to driving the cell movements and cell shape changes that generate tiss

128 in endocytosis is essential for both dynamic cell movements and establishment of stable tissue archit

129 signaling plays a pivotal role in regulating cell movements and lineage induction during gastrulation

130 Moreover, coordinated cell movements and physical cell-cell interactions are r

131 ial cues that induce systematically oriented cell movements and promote tissue elongation.

132 Actin filaments play an essential role in cell movement, and many posttranslational modifications

133 arized protrusive cell activity, directional cell movement, and oriented cell division and is crucial

134 promotes cell-cell interaction, limits lone cell movement, and slows swarm expansion.

135 -mediated vesicle induction to virus cell-to-cell movement are discussed.

136 rate gastrulation, convergence and extension cell movements are coordinated with the anteroposterior

137 Coordinated cell movements are crucial for vertebrate gastrulation a

138 Moreover, cell movements are highly correlated and in phase with E

139 Collective cell movements are integral to biological processes such

140 Cell movements are often coordinated by supracellular ne

141 These cell movements are spearheaded by the occipital lateral

142 quiring human intervention to compensate for cell movement as a patch pipette approaches a targeted n

143 t TAZ, is strictly required for WSS-enhanced cell movement, as blockade of YAP1, TEAD1-4 or the YAP1-

144 in transcript splicing were also detected in cell movement associated genes including Cd44.

145 and a resultant failure of hearts to undergo cell movements associated with cardiac formation.

146 e metabolite concentration caused changes in cell movements at gastrulation that also altered the tis

147 chemokine receptors (cCKRs) directly control cell movement; atypical chemokine receptors (ACKRs) regu

148 tERF53 also has a function to regulate guard-cell movement because the stomatal aperture of AtERF53 o

149 Cell movement begins with a leading edge protrusion, whi

150 pathogens, and they define requirements for cell movement between parenchyma and SCS in what we spec

151 In the spleen, B cell movement between the blood-rich marginal zone and f

152 Cell movement biased by a chemical gradient, or chemotax

153 r resulted in an additive effect to increase cell movement but did not alter the acetylation of alpha

154 es that AprA affects the directional bias of cell movement, but not cell velocity or the persistence

155 er Pyr or Ths was sufficient to redirect CVM cell movement, but only when the endogenous source of th

156 to regulate both ciliogenesis and collective cell movement, but the underlying mechanism is unknown.

157 e T4P is the S motility motor, and it powers cell movement by retraction.

158 strategy that automatically compensates for cell movement by tracking cell position and adjusting pi

159 cally, we show that Celsr1 regulates dynamic cell movements by inhibiting stabilization of VE-cadheri

160 We show that glioblastoma cell-cell movement can be described as Brownian motion biased

161 of the models showed that cytokine induced T cell movement can explain the very slow decline of CD4+

162 his arrangement is the result of coordinated cell movements, cell shape changes, and the organisation

163 gination occurs through coordinated vertical cell movement: cells towards the periphery of the placod

164 is established during early gastrulation via cell movement characteristics that are predominantly det

165 grams in MCCs, regulating genes that control cell movement, ciliogenesis, and cilia function.

166 OCM depends instead on a complex set of cell movements coordinated between the prospective neura

167 ll transplantation experiments show that the cell movement defect is cell autonomous.

168 minus of CP functions as a dedicated cell-to-cell movement determinant.

169 dentified, but are thought to control either cell movement directly or the patterning of their axonal

170 Fundamental cellular processes such as cell movement, division or food uptake critically depend

171 ransition (EMT) is a core process underlying cell movement during embryonic development and morphogen

172 lates cell alignment required for collective cell movement during embryonic development.

173 The mechanisms of coordinated cell movement during embryonic wound closure also drive

174 0b, is required for convergent and extension cell movement during gastrulation.

175 or tyrosine kinases that control directional cell movement during various biological processes, inclu

176 n algorithms to track and analyze individual cell movements during expansion of P. aeruginosa biofilm

177 are able to track cell lineages and dynamic cell movements during germ cell differentiation.

178 Directional cell movements during morphogenesis require the coordina

179 adhesion, cell proliferation, cell death and cell movements during otic development.

180 he primary regulator of convergent extension cell movements during vertebrate development, but the ro

181 elease of particles is abolished by blocking cell movement (either by depolymerizing actin with latru

182 usly un-recognized role for an Rfx factor in cell movement, finding that Rfx2 cell-autonomously contr

183 and work in concert to coordinate individual cell movements for collective migration.

184 scopy to monitor PI3K signaling dynamics and cell movements for extended periods.

185 T and B cell movement from blood into lymph nodes is reduced in

186 ases of precursor and migratory neural crest cell movements from the neural keel stage to times of ac

187 rs, and cell-cell cohesion during collective cell movements, further highlight that tension-dependent

188 echanistically, BMAL1 regulated a network of cell movement genes, 148 of which were within 100 kb of

189 MMP20 cleaves cadherins and may facilitate cell movement, however MMP20 is not known to cleave tigh

190 TGB1 self-interaction is needed for cell-to-cell movement, importin-alpha-mediated nucleolar targeti

191 Chemokines like CXCL12 regulate cell movement in a biphasic pattern, with peak migration

192 n and the basis of all cell shape change and cell movement in development.

193 standing changing metabolic requirements for cell movement in different conditions.

194 ary approach to further our understanding of cell movement in epithelia.

195 mune deficiency illustrate the importance of cell movement in host defense.

196 tina in vivo and in correlation with dynamic cell movement in mouse embryonic stem cell-derived sprou

197 een responses to differentiation signals and cell movement in patterning based on 'salt and pepper' d

198 echanisms of Gram-negative bacterial cell-to-cell movement in plant phloem.

199 that Dab1 activation is sufficient to orient cell movement in the absence of other signals.

200 dure to study marginal zone and follicular B-cell movement in the live mouse spleen.

201 ti-strip model) was developed to investigate cell movement in the presence and absence of the EGF che

202 igration, we develop a mathematical model of cell movement in the VE.

203 Matrix metalloproteinases (MMPs) facilitate cell movement in various tissues during development, and

204 anisms by which actomyosin drives collective cell movement in vertebrate embryos.

205 To identify new genes that regulate cell movement in vivo, we screened lethal mutations on c

206 nal analyses in zebrafish, we establish that cell movements in the developing optic cup require neura

207 ify a contractile force gradient that drives cell movements in the hindgut-forming endoderm, enabling

208 ow that Ret/Etv4 signaling promotes directed cell movements in the ureteric bud tips, and suggest a m

209 the whole animal level, they regulate early cell movements in zebrafish development.

210 a more than twofold increase in the rate of cell movement, in part due to a significant increase in

211 ll cortex and disrupted events essential for cell movement, including actin dynamics, lamellipodia pr

212 ction mice, we demonstrated that mesothelial cell movement into the lung requires the direct action o

213 The WASF3 gene has been implicated in cancer cell movement, invasion, and metastasis by regulating ge

214 Instead, T cell movement is better described as a correlated random

215 ed analysis has definitively shown whether T cell movement is consistent with Brownian motion.

216 Collective cell movement is critical to the emergent properties of

217 echniques, we demonstrate that the vector of cell movement is regulated by localised epidermal growth

218 Cell movement is thought to involve motor complexes comp

219 Directional cell movement is universally required for tissue morphog

220 isting knowledge on the molecular control of cell movements, it is unclear how the different observed

221 which generates actomyosin-based tension and cell movement, JNK signaling is required to establish mi

222 cs has significant influence on the speed of cell movement, kinetics of mutation propagation, and sen

223 n fixing the body plan: it controls epiblast cell movements leading to primitive streak formation, ge

224 bud tips, and suggest a model in which these cell movements mediate branching morphogenesis.

225 ocesses, such as cytoskeletal rearrangement, cell movement, microtubule dynamics, signal transduction

226 mplexes present on ameloblasts to foster the cell movement necessary for formation of the decussating

227 zing mitotic profiles, we found that neither cell movement nor oriented cell division could explain t

228 derm) from a single sheet, while large scale cell movements occur across the entire embryo.

229 Extensive cell movements occur during zebrafish optic vesicle morp

230 Zebrafish gastrulation cell movements occur in the context of dynamic changes i

231 light cues are processed to regulate cell-to-cell movement of auxin to allow establishment of a trans

232 tory effect of RFA expression on the cell-to-cell movement of Bean dwarf mosaic virus, a single-stran

233 V failed to complement the defective cell-to-cell movement of BMV.

234 P kinase signalling is important for cell-to-cell movement of invasive hyphae in M. oryzae.

235 that the replication of TuMV and the cell-to-cell movement of its replication vesicles are impaired i

236 Demonstrating cell-to-cell movement of mitochondria reconstructs the evolution

237 We report cell-to-cell movement of mitochondria through a graft junction.

238 lation of biological processes that included cell movement of monocytes, migration of dendritic cells

239 Our data show that cell-to-cell movement of plant rhabdoviruses is highly specific

240 LP5, which functions to restrict the cell-to-cell movement of signals via plasmodesmata, is induced b

241 Here, we present evidence for cell-to-cell movement of the entire 161-kb plastid genome in the

242 Directed cell-to-cell movement of the plant growth hormone auxin is often

243 ve distinct rhabdoviruses to support cell-to-cell movement of two positive-stranded RNA viruses by us

244 In marked contrast, cell-to-cell movement of two recombinant plant rhabdoviruses was

245 matic morphogenetic processes, including the cell movements of gastrulation, epiboly and dorsal conve

246 In addition to cell divisions, coordinated cell movements of the progeny allow the rapid expansion

247 Also, by considering the kinetics of the cell movement, our model predicts a biphasic invasivenes

248 tastasis through down-regulation of multiple cell movement pathways by regulating transcript levels a

249 Collective cell movements play a central role in embryonic developm

250 his requires dramatic cell shape changes and cell movements, powered by the contractile force generat

251 caffold protein is an essential regulator of cell movement processes required for normal eye developm

252 Tight regulation of Schwann cell movement, proliferation and differentiation is esse

253 rce are present, increased directionality of cell movement promotes chemotaxis.

254 r of the plant cell wall by encoding cell-to-cell movement proteins (MPs), which direct newly replica

255 nuclear localization, whereas the effects on cell movement required a cytoplasmic site of action.

256 Coordinated cell movement required the matrix protein RbmA, without

257 s reveal that high temperature-induced guard cell movement requires components involved in blue light

258 Cell movement requires the coordinated reception, integr

259 Efficient cell movement requires the dynamic regulation of focal a

260 gration because perturbations to these early cell movements result in the alteration of specific fate

261 zation and dynamics that fail to drive rapid cell movements, resulting in defects in epithelial morph

262 icate that lamellipodia are not required for cell movement, suggesting an alternate mechanism.

263 virion assembly but are required for cell-to-cell movement, suggesting that the C terminus of CP func

264 and ubiquitous production of Toddler promote cell movement, suggesting that Toddler is neither an att

265 of Xenopus Amer2 blocks convergent extension cell movements, suggesting that the Amer2-EB1-APC comple

266 ding motility, a substrate-dependent form of cell movement that underpins the protozoan parasite's ab

267 tern tissues in the context of the extensive cell movements that drive gastrulation.

268 precursor motility pathway, identify dynamic cell movements that generate posterior hypaxial and fin

269 ermal-myocardial communication can guide the cell movements that initiate cardiac morphogenesis.

270 of the cytoskeleton, accounting for precise cell movements that organize the functional retinal fiel

271 talloproteinase/PTK7 axis are detrimental to cell movements that shape the body plan and that chz rep

272 w that the nucleus dictates the direction of cell movement through mechanical guidance by its environ

273 for export to the cell periphery and cell-to-cell movement through plasmodesmata.

274 Directional cell movement through tissues is critical for multiple b

275 ind that the hindgut is formed by collective cell movements through a stationary CIP, rather than by

276 cover previously unappreciated long-distance cell movements throughout the life cycle of the hair fol

277 is requisite for TSWV infection and cell-to-cell movement; thus, this behavior is most likely to est

278 ssed, but, in normal conditions, facilitates cell movement to locally prepatterned sources of FGF.

279 Medulla cortex cells follow two patterns of cell movements to acquire their final position: first, n

280 Integrating individual cell movements to create tissue-level shape change is es

281 ange of defects, from aberrant morphogenetic cell movements to failure to correctly orient structures

282 structure, while also driving and organizing cell movements to promote rapid repair.

283 es, from fast subcellular rearrangements and cell movements to slow structural changes at the whole-o

284 electron shuttle and an attractant to direct cell movement toward local sources of insoluble electron

285 ion in which, for increasing time intervals, cell movement transitions from a ballistic to a diffusiv

286 HTR-8/SVneo trophoblast cell line to measure cell movement under the influence of EGF (chemoattractan

287 The genes/proteins associated with cell movement were highlighted in breast cancer brain me

288 and function for the WRAMP structure during cell movement were not determined.

289 acellular components could be easily imaged, cell movements were mostly classified by different types

290 of PIK3CA may also stimulate intraepithelial cell movement, which could contribute to spread of cells

291 ed for efficient virion assembly and cell-to-cell movement, while the C-terminal 65 amino acids are d

292 In brief, cell movement will lead to a significant technical (samp

293 on and branching of their leading processes, cell movement with axon specification and extension, swi

294 Coordination of cell movement with cell differentiation is a major feat

295 Inhibiting basal-type cell movement with clinically relevant drugs blocked inv

296 We show that the direction of cell movement with respect to the cell-cell contact is c

297 tes is driven by a proximodistal gradient of cell movement, with WNT and FGF activities controlling d

298 and may modulate intercellular adhesion and cell movement within in epithelia during development and

299 t previously unknown, stimulus for directing cell movement within porous extracellular matrix.

300 pplied to more complex systems of collective cell movement without prior knowledge of the cellular ma