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1 en negative imprinting of virions and target cell protection.
2 PR37 located in the plasma membrane provides cell protection.
3 y terms like oxidative stress, transport and cell protection.
4 tumor cells to co-opt pathways used for stem cell protection.
5 cribed to these mutants beyond impaired self-cell protection.
6 FITMs and about its relationship with target cell protection.
7 presents a novel strategy for enhancing beta-cell protection.
8 for SF stimulation of NF-kappaB activity and cell protection.
9 tudied Src signaling pathways involved in SF cell protection.
10 ty of SF to stimulate NF-kappaB activity and cell protection.
11 to SF stimulation of NF-kappaB signaling and cell protection.
12  functions are in membrane stabilization and cell protection.
13 ty of SF to stimulate NF-kappaB activity and cell protection.
14 iological roles of EPO and its mechanisms of cell protection.
15  a small but significant reduction in the SF cell protection.
16 Akt-regulated genes that might contribute to cell protection.
17 pleen, liver, and lung, as well as, CD4(+) T-cell protection.
18 tream target of NAD replenishment leading to cell protection.
19 tant in regulating underlying stromal immune cell protection.
20 h as regional delivery or hematopoietic stem-cell protection.
21 nfluence neurovirulence by inhibiting CD8+ T cell protection.
22 f the initial candidates under testing for B-cell protection.
23  glucose uptake, insulin secretion, and beta-cell protection.
24 d Renalase (Rnls) as a novel target for beta-cell protection.
25        The anti-FH Abs perturbed FH-mediated cell protection (100%), inhibited FH interaction with C3
26 imaging have been explained as the result of cell protection (acclimation) mechanisms to unfavorable
27 ated the mechanism of sinusoidal endothelial cell protection after ischemic preconditioning.
28 oth functional and morphologic photoreceptor cell protection against acute light-induced damage, most
29  IRS1 could be a therapeutic target for beta-cell protection against ER stress-mediated cell death by
30  that Sirt3-ROS signaling axis mediated hair cell protection against gentamicin by adjudin, at least
31                           Induction of islet cell protection against inflammation could therefore be
32 ATP) channels activity that is essential for cell protection against ischemia.
33 ration, leading to cell-cycle inhibition and cell protection against oxidative challenge.
34 chiral analog have nearly the same degree of cell protection against ricin as (+/-)-Retro-2(cycl).
35 o the former, which is a racemate and showed cell protection against RIPs.
36 dicate that PC extract is capable of colonic cells' protection against the adverse effects of oxidati
37                                Without these cells, protection against lethal challenge was essential
38  but significantly inhibited HGF/SF-mediated cell protection and DNA repair.
39 e most mutations concomitantly affect target cell protection and negative imprinting, a region in the
40 strogen increases LRH-1 expression conveying cell protection and proliferation.
41 ing pathway is a potential approach for beta-cell protection and regeneration therapies.
42 hanistic understanding of STX4 function in B-cell protection and warrant further investigation of STX
43 ion of incoming virions in endosomes (target cell protection) and with the production of virion parti
44 t the cellular partners of PTPN4 involved in cell protection are unknown.
45 ase or p21-activated kinase eliminates basal cell protection as well as the elevated migration and pr
46  a phenotypic screening approach utilizing a cell protection assay identified a series of azaindole b
47 lls, the possibility that mesalamine confers cell protection by increasing intestinal epithelial hsp7
48 creased cAMP mediates sinusoidal endothelial cell protection by ischemic preconditioning.
49                       This demonstrates that cell protection can be effected by directly binding pro-
50 l applications are also discussed, including cell protection, cell labeling, targeted delivery and in
51 odes-of-action to infer pathways of neuronal cell protection connected to drug mechanism.
52 ibition of sustained c-Akt activation and of cell protection did not require the Gab1 pleckstrin homo
53 -1 played a key role in the BCR/ABL-mediated cell protection from apoptosis.
54         The role of the NF-kappaB pathway in cell protection from CDC was examined.
55 omponent promoting both metabolic and immune cell protection from colitis.
56 drial heat shock protein 70, plays a role in cell protection from complement-dependent cytotoxicity (
57  and proteins that play an important role in cell protection from different damaging factors includin
58 ological characteristics required for CD8+ T cell protection from liver-stage malaria dictate that mo
59  such, serve an important role in epithelial cell protection from mechanical and non-mechanical stres
60 nd Sirt3 may be of therapeutic value in hair cell protection from ototoxic insults.
61  mitochondrial translocation and endothelial cell protection from the intrinsic pathway of apoptosis,
62 g a direct immunoregulatory effect on immune cells, protection from oxidative stress and improved gut
63                  Over the past decades, beta cell protection has been extensively investigated in rod
64 role of cell surface-expressed FasL in tumor cell protection has recently become controversial.
65 balance of competing animal processes toward cell protection, health, and longevity.
66    Finally, reduced CS-specific memory CD8 T cell protection in P. yoelii-infected BALB/c or P. bergh
67 refore represent a promising target for beta-cell protection in type 1 diabetes mellitus.
68 us studies have established that SF-mediated cell protection involves antiapoptotic signaling from it
69                                      In PC12 cells, protection is also conferred by a c-Jun mutant la
70 earch on the processes underlying human beta-cell protection, loss, and replacement in type 1 diabete
71         Study 2 deployed an animation of the cell-protection model (Model 2), alone, and in combinati
72                           By analysis of the cell protection produced by some small molecules, a diph
73  current encapsulation technologies focus on cell protection rather than cell regulation that is esse
74                            First, collective cell protection results from inhibition of phage transpo
75 rucial role in regulating hematopoietic stem cell protection, self-renewal, and regeneration.
76             Using DeltaSHAPE, we identify in-cell protection sites that correspond with previously de
77 leptic tolerance is an endogenous program of cell protection that can be activated in the brain by pr
78                                              Cell protection that exhibits a memory (preconditioning)
79                    While Gcn2 is central for cell protection to nutrient stress and its depletion in
80 ss of wall distensibility blunts endothelial cell protection to oxidant stress-induced apoptosis.
81 od, are concentration-dependent ranging from cell protection to toxicity.
82 s accumulate sucrose and trehalose, offering cell protection under anhydrobiotic conditions.
83 of the heat shock protein family that exerts cell protection under several stress-related conditions.
84 at encodes dehydrin, a protein implicated in cell protection under stress conditions.
85   Because SPF mice exaggerated humoral and T cell protection upon influenza vaccination, reconstituti
86 ve effects, and 3) STAT3 confers endothelial cell protection via both HO-1-dependent and independent
87                                Memory CD4+ T cell protection was enhanced through synergy with naive
88                                              Cell protection was more prominent in the superior retin
89                                            B-cell protection was the strongest correlate of the long-
90                 To explore the mechanisms of cell protection, we examined the effects of cariporide o
91  protein-coupled receptor activation elicits cell protection (without mitochondrial swelling or durab