ライフサイエンスコーパス: cell receptor

1 erference with the virus docking to the host cell receptor.

2 omplexes including the CD19 subunit of the B-cell receptor.

3 een anti-CD20 antibodies and lymphoma cancer cell receptors.

4 ood samples to interact with lymphoma cancer cell receptors.

5 many cancers are not fully recognized via NK cell receptors.

6 specific glycan determinants exposed on host cell receptors.

7 nstant alpha and beta domains of the human T-cell receptor, a technology known as bispecific engageme

8 n BTN3A1 inhibits tumor-reactive alphabeta T cell receptor activation by preventing segregation of N-

9 We found that mimicking T-cell receptor activation in Jurkat leukemia cells induce

10 ng up to ~25-30 nm monotonically increases B-cell receptor activation.

11 g the roles of B cell precursor frequency, B cell receptor affinity for antigen, antigen avidity, and

12 f thresholds for B cell selection based on B cell receptor affinity.

13 l TCRs (e.g., immune-mobilizing monoclonal T cell receptors against cancer [ImmTAC] molecules), where

14 Immune mobilizing monoclonal T Cell receptors against virus (ImmTAV) molecules represen

15 ntra- and inter-method reproducibility for T cell receptor alpha (TRA) and T cell receptor beta (TRB)

16 based single-cell RNA sequencing of paired T cell receptor alpha and beta chain sequences show pronou

17 staining and coexpression of CD161 and the T-cell receptor alpha variable gene TRAV1-2 were strongly

18 es of immunoglobulin H (IgH), Igkappa, and T cell receptor-alpha (TCRalpha) loci during B lymphopoies

19 In the presence of target cell receptors, an immunocomplex was formed between anti

20 es of immune cells, coupled with assembled T cell receptor and B cell receptor sequences, we analyzed

21 thways affected in these disorders include T cell receptor and B cell receptor signaling, cytokine si

22 s solely responsible for binding to the host cell receptor and facilitating fusion between the viral

23 -cell compartment, and B-cell responses to B-cell receptor and IL-21 receptor engagement.

24 g of automatically prepared libraries from T-cell receptor and immunoglobulin gene rearrangements in

25 le, transcriptional activation of both the T-cell receptor and programmed cell death protein 1 pathwa

26 lope protein called spike (S) to engage host cell receptors and catalyze membrane fusion.

27 e cell therapy, including chimeric antigen T-cell receptors and other novel T-cell receptor-based the

28 Interactions between immune cell receptors and proteins that determine disease susce

29 y low frequencies of appropriate precursor B cell receptors and the complex maturation pathways requi

30 on, such as the targeting of pathways for NK cell receptors and their ligands, apoptosis, and cytokin

31 ction between its Spike protein and the host cell receptor angiotensin-converting enzyme 2 (ACE2).

32 , but OT1 mice fail to do so because their T cell receptors are engineered to recognize a single oval

33 eus and their delivery to intracellular host cell receptors are not well understood.

34 ands for stimulation and co-stimulation of T-cell receptors are presented via the fluid, synthetic me

35 amily tyrosine kinase (LYN), zeta chain of T-cell receptor-associated protein kinase 70 (ZAP-70), and

36 ct phosphorylation of Zap70 (zeta chain of T cell receptor-associated protein kinase 70).

37 raction; and (2) limiting the amount of stem cell receptors available for niche signal reception.

38 This study delineated a stromal Lama5-T cell receptor axis that can be targeted for immune toler

39 c antigen T-cell receptors and other novel T-cell receptor-based therapies.

40 F) and next-generation sequencing of B and T cell receptor (BCR and TCR) repertoires, we demonstrate

41 repository of currently >14 million B and T cell receptor (BCR and TCR) sequences from the blood of

42 At high levels of B-cell receptor (BCR) activation, which may occur in indiv

43 he antigen-binding variable regions of the B cell receptor (BCR) and of antibodies are encoded by exo

44 B cell receptor (BCR) and T cell receptor (TCR) repertoire

45 However, engagement of the B cell receptor (BCR) induced both expression of IFITM3 an

46 B-cell receptor (BCR) knock-in (KI) mouse models play an i

47 2A (LMP2A), which has been described as a B cell receptor (BCR) mimic promoting malignant transforma

48 ) of more than 180 million human and mouse B-cell receptor (BCR) repertoire sequences.

49 Donor B cell receptor (BCR) repertoires identified two bNAb line

50 Analysis of the B cell receptor (BCR) repertoires in six IMDs provides ins

51 Next generation sequencing of B cell receptor (BCR) repertoires offers an additional sou

52 ivation markers, and are hyporesponsive to B-cell receptor (BCR) restimulation in vitro.

53 ame initial V(D)J rearrangement, but their B cell receptor (BCR) sequence may differ due to the accum

54 These processes require B cell receptor (BCR) signaling and occur in bone marrow,

55 lymphoma cell proteins and inhibits early B-cell receptor (BCR) signaling events critical for surviv

56 e B cell lymphoma (DLBCL), which relies on B cell receptor (BCR) signaling for survival.

57 , PKCbetaI and PKCbetaII, functions in the B cell receptor (BCR) signaling pathway and contributes to

58 B-cell receptor (BCR) signaling pathways and interactions

59 re B lineage selection have been linked to B cell receptor (BCR) signaling strength and environmental

60 d PI3K signaling consistent with activated B cell receptor (BCR) signaling, although they do not expr

61 We surmise that, unlike B cell receptor (BCR) signaling, MYD88/IRAK signaling is c

62 V-mutated with no restricted IGHV usage or B-cell receptor (BCR) stereotypy.

63 ncreasingly used to query the antibody, or B-cell receptor (BCR), sequence repertoire, and the amount

64 from that of conventional B cells, through B cell receptor (BCR)-dependent positive selection of feta

65 ng valency and mode of presentation to the B-cell receptor (BCR).

66 igen (HLA) receptors as well as B cell and T cell receptors (BCR and TCR).

67 On antigen binding, B cell receptors (BCR) cluster on the plasma membrane and

68 to external signals, such as in binding of B cell receptors (BCR) to antigen, which initiates signali

69 rface of an antigen-presenting cell (APC), B cell receptors (BCRs) are gathered into microclusters th

70 Animal models of human antigen-specific B cell receptors (BCRs) generally depend on "inferred germ

71 alk between Toll-like receptors (TLRs) and B cell receptors (BCRs) in the TI B cell immunity, we here

72 h affinity antibodies, the soluble form of B cell receptors (BCRs), that bind to and neutralize invad

73 ific engagement by antibodies based on the T-cell receptor (BEAT).

74 ency, which prompted us to investigate the T cell receptor beta (TCRbeta) repertoire in the CP and co

75 In this study, we performed T cell receptor beta (TCRbeta) sequencing of virus-specifi

76 bility for T cell receptor alpha (TRA) and T cell receptor beta (TRB) TCR chains.

77 of IDH1 mutants to the CDR3 domain of the T-cell receptor beta chain (TRB).

78 r patients, with a focus on studies of the T-cell receptor beta chain locus.

79 We studied the T-cell receptor beta-chain (TCRbeta) usage and phenotypes

80 ociated with oligoclonality and restricted T-cell receptor beta-chain V-J pairing in CD8(+) but not C

81 Host cell receptor binding causes conformational changes in t

82 class T epitope on SEB overlapped with the T-cell receptor binding site, whereas other evidence sugge

83 I complex stability, beta167-169 RGD loop, T-cell receptor binding, formation of homodimer of alpha-b

84 rcinoma (RCC) tumors were stained for the NK cell receptors CD56, NKp30, and NKp46 to determine expre

85 The dendritic cell receptor Clec9A facilitates processing of dead cell

86 re ICI are effective, with concomitant low T-cell receptor clonalities.

87 By mapping T cell receptor clonality, we find that responding patient

88 and regulatory T-cell subsets with diverse T-cell receptor clonotypes in the periphery.

89 Thymus structure, immunophenotyping, T-cell receptor clonotypes, T-cell function, immune respon

90 rearranged Igh allele assembles into a PreB cell receptor complex (PreBCR) to generate signals to in

91 round anchor pockets 1 and 9, as potential T-cell receptor contacts, in the areas for CD4 binding and

92 T cell differentiation by ensuring normal T cell receptor density and signaling.

93 east 91 unique clones expressing different T-cell receptors) directed against HLA*02:01-restricted pe

94 Additionally, T-cell receptor diversity, cytolytic activity score (CYT),

95 tic and biosynthetic programs and licences T cell receptor driven proteome reprogramming.

96 e multi-phenotypic impact of disrupting 25 T cell receptor-driven kinases.

97 cal benefit demonstrated overexpression of T cell receptor-encoding genes.

98 ior in mice and was partially dependent on T cell receptor engagement and commensal-derived signals.

99 hed, control C57BL/6 mouse LNSC suppressed T-cell receptor engagement by anti-CD3/CD28 via MHC-indepe

100 recombinant CHIKV strain encoding a CD8(+) T cell receptor epitope from ovalbumin, as well as a viral

101 ototypical celiac patient-derived anti-TG2 B cell receptor equally reactive to human and mouse TG2.

102 severe combined immunodeficiency using the T-cell receptor excision circle assay.

103 ed TCR V-beta repertoire usage and diluted T-cell receptor excision circles confirm that DN T cells f

104 arises from a small subset of gamma/delta T-cell receptor-expressing lymphocytes.

105 invasion and identify SR-B1 as the airway M cell receptor for Mtb.

106 man transferrin receptor 1 (hTfR1) as a host cell receptor for virus entry.

107 cile infection showed increased IL-17A and T cell receptor gamma chain expression, and IL-17 producti

108 but molecular testing identified the same T-cell receptor gamma rearrangement present in the gastric

109 Molecular analysis revealed a clonal T-cell receptor gamma rearrangement.

110 unctional avidity of selected gamma9delta2 T cell receptors (gamma9delta2TCRs), was not associated wi

111 rase chain reaction-based investigation of T-cell receptor gene rearrangement to detect clonality.

112 le possess the most diverse repertoire of NK cell receptor genes among all mammals studied to date.

113 ll clones through paired sequencing of the T cell receptor genes and high-dimensional single-cell spa

114 Killer cell receptor genes encoded within the NK complex and ki

115 mbination assembles and diversifies Ig and T cell receptor genes in developing B and T lymphocytes.

116 rthermore, we isolated a number of cognate T cell receptor genes with tumor reactivity.

117 a background of high genetic diversity of NK cell receptor genes, this KLRA allele fixation points to

118 ation critically enhanced signaling by the T cell receptor in the formation of functional immune syna

119 Crystal structures of T cell receptors in complex with HLA-DQ2.5 bound to two di

120 al-transduction protein STAT5)(2-4) or pre-B-cell receptors in more mature cells (via activation of t

121 ing deep single-cell sequencing of RNA and T cell receptors in patients with different types of cance

122 iles of various populations of T cells and T cell receptors in tumours, normal adjacent tissue, and p

123 Signals downstream of the T cell receptor influence whether individual clones bearin

124 A live-cell receptor internalization assay was used to test the

125 ll cytotoxicity and expression of certain NK cell receptors involved in NK cell development and funct

126 The effectiveness of pathogen detection by T cell receptor is limited by chemical similarity of forei

127 he antigenic specificity of the gammadelta T cell receptors is incompletely understood.

128 design of effective immunogens to activate B cell receptors leading to protective HIV-1 antibodies wi

129 reach the stationary concentration of the T-cell-receptor-ligand-activated complex, which transfers

130 in and residues involved in binding the host-cell receptor, lipolysis-stimulated lipoprotein receptor

131 nd fatty acid oxidation, without affecting T cell receptor-mediated activation.

132 y triggers that can enhance or even bypass T cell receptor-mediated signals-substantially broadening

133 can, in principle, be discovered against any cell receptors; moreover, the aptamers can be replaced b

134 ILCs was not dependent on the natural killer cell receptor (NCR1), since NCR1-deficient mice still in

135 were generated that bridge the activating NK cell receptor NKp30 on NK cells with epidermal growth fa

136 napse is classically described between the T-cell receptor of CD4-positive lymphocytes and MHC II on

137 Activating precursor B cell receptors of HIV-1 broadly neutralizing antibodies

138 As well as binding T cell receptors on CD8(+) T cells, HLA class I molecules

139 f activation mediated by engagement of the T-cell receptor or of CD16.

140 ng and visualizing glycans and protein-based cell receptors overexpressed in certain diseases, which

141 Inhibition of the B-cell receptor pathway, and specifically of Bruton tyrosi

142 T cell receptor phosphorylation by Lck is an essential ste

143 Our theoretical method models T cell receptor phosphorylation events as a sequence of st

144 Lowering R(in) will reduce the hair cells receptor potential and presumably moderate the sys

145 thymocyte development, requires myriad pre-T cell receptors (preTCRs) and alphabetaTCRs.

146 ination and subsequent selection of T- and B-cell receptors provide useful tools to analyse and compa

147 T-cell receptor repertoire analyses showed similar clonal

148 UC) using single-cell transcriptomics with T-cell receptor repertoire analysis and mass cytometry.

149 se, profound immune exhaustion with skewed T cell receptor repertoire and broad T cell expansion.

150 We evaluated the T-cell receptor repertoire associated with 72 primary Merk

151 self-antigen representation and increased T cell receptor repertoire diversity.

152 l carcinomas and correlated metrics of the T-cell receptor repertoire with clinicopathologic characte

153 mmune-monitoring reveals remodeling of the T-cell receptor repertoire with immunodominant clones and

154 ansport into the endoplasmic reticulum and T-cell receptor repertoire, also contribute to the immunog

155 cludes critical variables, such as MHC and T cell receptor repertoire, is needed.

156 sets and systemic remodeling of peripheral T cell receptor repertoires.

157 Activating NK cell receptors represent promising target structures to

158 ability to induce c-Jun/AP-1 expression on T cell receptor restimulation, a mechanism that may contri

159 e-like T cells expressing a semi-invariant T cell receptor restricted to the non-classical MHC class

160 hese two viruses likely compete for the same cell receptor(s).

161 T cell receptor sequence comparison between patients ident

162 coupled with assembled T cell receptor and B cell receptor sequences, we analyzed the functional prop

163 B cell receptor sequencing confirmed that meningeal IgA(+)

164 tical modeling and statistical analyses of T cell receptor sequencing data, we develop a quantitative

165 T cell receptor sequencing monitored repertoires during tr

166 nrichment of immune-related processes, and T cell receptor sequencing revealed increased clonality in

167 umors, we conducted whole genome, RNA, and T-cell receptor sequencing, immunohistochemistry and rever

168 Previous studies suggested that chronic B-cell receptor signaling and increased NF-kappaB activati

169 re, we review the current understanding of T-cell receptor signaling and their intersection with IC s

170 t activates the PI3K pathway downstream of B cell receptor signaling in B cells and Toll-like recepto

171 lve the spatial and temporal resolution of T cell receptor signaling in the context of immune respons

172 mphoma-2, and inhibitors of kinases in the B-cell receptor signaling pathway, like Bruton tyrosine ki

173 Unique attached oligomannoses activate B-cell receptor signaling pathways after engagement with c

174 er, the relative contributions of IL-2 and T cell receptor signaling to this process are unknown.

175 s, including posttranscription regulation, T-cell receptor signaling, and metabolic function.

176 isingly, NFAT activation is independent of B-cell receptor signaling, but mediated by an increased ca

177 hese disorders include T cell receptor and B cell receptor signaling, cytokine signaling, skin barrie

178 yrosine phosphosites that mediate proximal T cell receptor signaling, cytoskeletal organization, and

179 cellular processes, including deregulated B cell receptor signaling, which we also identified in hum

180 uting to IgE class switch recombination or B-cell receptor signaling.

181 and tryptophan metabolism, autophagy, and B cell receptor signaling.

182 nd FCRL2-5 activation along with increased B cell receptor signaling.

183 -kinase/AKT/mTOR activation independent of T-cell receptor signaling.

184 to vaccine induced antibody durability and B-cell receptor signaling.

185 Antibody blockade of these host cell receptors significantly reduced NTHI adherence.

186 The immune cell receptor SLAMF7 has been shown to be upregulated in

187 biting T cell proliferation in response to T cell receptor stimulation and mediating fibroblast cell

188 T cell receptor stimulation induces depletion of the endop

189 in TILs was induced by the coordination of T cell receptor stimulation, microenvironmental stressors

190 ermore, we were able to isolate and verify T cell receptors targeting KRASG12D mutation.

191 T cell receptor (TCR) activation is modulated by mechanism

192 ily kinases (pSFK), LAT and PLC-gamma over T cell receptor (TCR) alone.

193 iencies received 155 mismatched grafts: 30 T-cell receptor (TCR) alphabeta/CD19-depleted grafts, 43 c

194 nalyzing the cytokine production following T cell receptor (TCR) and co-receptor stimulation with a c

195 vation by enabling interaction between the T-cell receptor (TCR) and human leukocyte antigen class II

196 The assembly of T cell receptor (TCR) and immunoglobulin (Ig) genes by V(D

197 Virtually all T-PLL expressed a T-cell receptor (TCR) and/or CD28-coreceptor without overr

198 Two genes encoding the endogenous T cell receptor (TCR) chains, TCRalpha (TRAC) and TCRbeta

199 t a subpopulation of T cells expresses two T cell receptor (TCR) clonotypes, though the extent and fu

200 Crystal structures of the MAIT T cell receptor (TCR) complexed with MR1-DB28 and MR1-NV18

201 -23)) specificity, and mutation of the key T-cell receptor (TCR) contact residue within the epitope p

202 eptide elution, mixed lymphocyte reaction, T-cell receptor (TCR) deep sequencing, tetramer-guided nai

203 We found that T cell receptor (TCR) engagement induces the selective pho

204 ive selection in the thymus when their new T cell receptor (TCR) engages and signals in response to s

205 and clonality index of T cells by means of T-cell receptor (TCR) immunosequencing in a discovery data

206 Knockout (KO) of the endogenous T-cell receptor (TCR) in CD19-CAR-T cells may be a promisi

207 However, the role of T cell receptor (TCR) in this process remains unclear.

208 cell maturation and activation depend upon T cell receptor (TCR) interactions with a wide variety of

209 Mechanosensing by T cells through the T cell receptor (TCR) is at the heart of immune recognitio

210 eric antigen receptor (CAR) or a classical T cell receptor (TCR) is revolutionizing cancer treatment,

211 to study OT-1 T cells, revealing that the T cell receptor (TCR) mechanically samples antigens carryi

212 A T cell receptor (TCR) mediates antigen-induced signaling t

213 mediated immunity is governed primarily by T cell receptor (TCR) recognition of peptide-human leukocy

214 CRISPR-Cas9 screening to establish that a T cell receptor (TCR) recognized and killed most human can

215 ct T cell differentiation trajectories and T cell receptor (TCR) recombination kinetics.

216 TILs, TSA-reactive TILs possess a distinct T cell receptor (TCR) repertoire and unique gene features.

217 spond to nickel but the involved alphabeta T cell receptor (TCR) repertoire has not been comprehensiv

218 Monitoring the T cell receptor (TCR) repertoire in health and disease can

219 clone size distribution of the human naive T-cell receptor (TCR) repertoire is an important determina

220 but its influence on the antigen-specific T cell receptor (TCR) repertoire is unknown.

221 rted by a combination of immunophenotypic, T cell receptor (TCR) repertoire, functional, and transcri

222 ultifaceted impact on the exhausted CD8(+) T cell receptor (TCR) repertoire.

223 CD4(+) conventional T cells with a diverse T cell receptor (TCR) repertoire.

224 peptidome variation contributes to shaping T cell receptor (TCR) repertoires and hence individual imm

225 B cell receptor (BCR) and T cell receptor (TCR) repertoires are generated through so

226 2-V5 single-genome amplification (SGA) and T-cell receptor (TCR) repertoires assessed.

227 The study of T cell receptor (TCR) repertoires has generated new insigh

228 The diversity of T-cell receptor (TCR) repertoires is achieved by a combina

229 s parameterized by deep neural networks to T cell receptor (TCR) repertoires.

230 Activation of the T cell receptor (TCR) results in binding of the adapter pr

231 ude selecting an appropriate repertoire of T cell receptor (TCR) self-affinities in the thymus, regul

232 T cell receptor (TCR) sequence analysis demonstrated that

233 th a substantial increase in the number of T-cell receptor (TCR) sequences and their cognate antigens

234 e utilized the complementary approaches of T cell receptor (TCR) sequencing and cytometry by time-of-

235 e assessed with single-cell RNA and paired T cell receptor (TCR) sequencing of 30,604 T cells from 7

236 rein, we perform genome-wide DNA, RNA, and T cell receptor (TCR) sequencing on 29 cutaneous gammadelt

237 Transcriptional profiling coupled with T cell receptor (TCR) sequencing reveal lineage connection

238 this issue of the JCI, Ogongo et al. used T cell receptor (TCR) sequencing to characterize unconvent

239 Both the T cell receptor (TCR) signal and environmental cues contri

240 Pre-T-cell receptor (TCR) signal transduction is required for

241 The suppressor of T-cell receptor (TCR) signaling (Sts) proteins Sts-1 and S

242 ified an interaction between Trib1 and the T cell receptor (TCR) signaling activator, MALT1, which di

243 It is known that antigen recognition and T cell receptor (TCR) signaling depend on forces applied b

244 To determine how LAIR-1 affects T-cell receptor (TCR) signaling, we compared 1) T cells fr

245 horylating substrates involved in proximal T cell receptor (TCR) signaling.

246 , single-cell RNA sequencing revealed that T cell receptor (TCR) signalling was enhanced in these cel

247 cardiac autoantigen and that T cells with T cell receptor (TCR) specificity to MYHCA acquired a Treg

248 T cell receptor (TCR) transductants and BTN3-deficient hum

249 In marked contrast to the biased T cell receptor (TCR) usage associated with HLA-DQ2.5-medi

250 tegrates signal strength downstream of the T cell receptor (TCR) within activated thymocytes and peri

251 we report that, similar to the endogenous T cell receptor (TCR), antigen engagement triggers the for

252 through multiple receptors, including the T-cell receptor (TCR), co-receptors, and cytokine receptor

253 The T cell receptor (TCR)-CD3 complex comprises a diverse alph

254 The T-cell receptor (TCR)-CD3 complex is composed of a diverse

255 e Qa-1, which disrupts Qa-1 binding to the T cell receptor (TCR)-CD8 complex and impairs the CD8 Treg

256 adelta T cell population that respond in a T cell receptor (TCR)-dependent manner to phosphoantigens

257 votal regulators of immune tolerance, with T cell receptor (TCR)-driven activated T reg (aT reg) cell

258 zens of unique barcoded templates into the T cell receptor (TCR)-locus revealed gene constructs that

259 g., virus-infected cells, which depends on T cell receptor (TCR)-mediated activation of beta(2)-integ

260 Adoptively transferred T cell receptor (TCR)-transgenic T cells (TCR-T cells) are

261 ized, in part, by the TRAV1-2(+) alphabeta T cell receptor (TCR).

262 sing MHC-related protein-1 (MR1) via their T cell receptor (TCR).

263 related to the chains that constitute the T cell receptor (TCR).

264 olecular modeling, we established that the T cell receptor (TCR):CD3 complex is required for USSN-ind

265 T-cell receptors (TCR) have considerable potential as ther

266 eering chimeric antigen receptors (CAR) or T cell receptors (TCR) helps create disease-specific T cel

267 The diverse repertoire of T-cell receptors (TCR) plays a key role in the adaptive im

268 The regulation of T cell receptor Tcra gene rearrangement has been extensive

269 the binding of pathogenic superantigens to T cell receptors (TCRs) and/or major histocompatibility co

270 T-cell receptors (TCRs) are immune proteins that primarily

271 lls engineered to express antigen-specific T cell receptors (TCRs) are potent therapies for viral inf

272 Recombinant T cell receptors (TCRs) can be used to redirect naive T ce

273 stocompatibility complex (pMHC) ligand for T cell receptors (TCRs) is inactive from solution yet capa

274 Four T cell receptors (TCRs) made up the TIL infusion and recog

275 iation (CD)8(+) T cells, by means of their T cell receptors (TCRs) recognizing intracellular targets

276 cultured T cells expressing MCPyV-specific T cell receptors (TCRs) show increased cytokine production

277 of Vdelta1, three CD1b-specific gammadelta T cell receptors (TCRs) showed clear differences in the su

278 We studied oligoclonal T-cell receptors (TCRs) that recognize a shared neoepitope

279 T cells express clonotypic T cell receptors (TCRs) that recognize peptide antigens in

280 hreats in an antigen-specific manner using T cell receptors (TCRs) that recognize short peptide antig

281 y described GLIPH, an algorithm to cluster T-cell receptors (TCRs) that recognize the same epitope an

282 distinctive nature of DURTs and gammadelta T cell receptors (TCRs) to investigate the involvement of

283 The binding of T cell receptors (TCRs) to their target peptide MHC (pMHC)

284 High-avidity CBFB-MYH11 epitope-specific T cell receptors (TCRs) transduced into CD8+ T cells confe

285 Studying the composition of T cell receptors (TCRs) using next-generation sequencing (

286 the associations of particular gammadelta T cell receptors (TCRs) with specific anatomical sites.

287 neered T cells expressing antigen-specific T-cell receptors (TCRs), is an appealing therapeutic appro

288 CD1d antibodies that block CD1d binding to T-cell receptors (TCRs).

289 is influenced by clonotypically expressed T-cell receptors (TCRs).

290 n other cells, much like MHC molecules and T cell receptors (TCRs).

291 red for N-glycosylation of key T cell and NK cell receptors that can account for some of the clinical

292 (MHC) class I, MHC class II and alphabeta T cell receptors, the antigenic specificity of the gammade

293 For immunology, the binding of a T cell receptor to an antigen-presenting pMHC initiates do

294 mented expression of CEACAM1 and ICAM1, host cell receptors to which NTHI binds via engagement of mul

295 variability of the immunoglobulin (Ig) and T cell receptor (TR) gene loci.

296 ate rigidity, implying that early steps in T cell receptor triggering are not mechanosensitive.

297 To address this, sequencing of the T-cell receptor variable-region beta-chain was performed o

298 ed erythrocytes bind to specific endothelial cell receptors via members of the PfEMP1 family exported

299 es such as IFNA, MRC1, immunoglobulins and T-cell receptors which contribute to defend against pathog

300 ic strategy combining an affinity-enhanced T Cell receptor with an anti-CD3 T Cell-activating moiety.