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1 ell neoplasm that often expresses the CD4+ T cell surface marker.
2 cells has been difficult without a defining cell surface marker.
3 major glycoprotein 2 (GP2) as a PP-specific cell surface marker.
4 nto Th17 cells in vitro and in vivo via CD25 cell surface marker.
5 re limited by the lack of available specific cell surface markers.
6 e ISCs from mouse and human tissues based on cell surface markers.
7 al nerve, and immunofluorescence staining of cell surface markers.
8 4, KDR) and osteoblastic (osteocalcin [OCN]) cell surface markers.
9 her coagulation, inflammatory, or lymphocyte cell surface markers.
10 subpopulation characterized previously using cell surface markers.
11 arly-outgrowth colony-forming unit assay and cell surface markers.
12 ke cysts that selectively incorporate apical cell surface markers.
13 eased up-regulation of activation-associated cell surface markers.
14 xpression of both DC and monocyte/macrophage cell surface markers.
15 iling cytokines, intracellular molecules and cell surface markers.
16 st cells as shown by cellular morphology and cell surface markers.
17 an be identified by the presence of specific cell surface markers.
18 et been achieved due to the lack of specific cell surface markers.
19 ls that previously identified based on other cell surface markers.
20 y Ab responses due to an absence of specific cell surface markers.
21 using flow cytometry to determine lymphocyte cell surface markers.
22 protein assays for cytokine, chemokine, and cell surface markers.
23 as electrical activity, gene expression, and cell surface markers.
24 TNF-alpha(+)CD4(+) T cells expressing naive cell surface markers.
25 positive cells and increased stem/progenitor cell surface markers.
26 lliliter of blood and the fold expression of cell surface markers.
27 human fetal pancreatic differentiation using cell surface markers.
28 ytometry together with various hematopoietic cell surface markers.
29 he result of context-dependent expression of cell surface markers.
30 ined challenging due to the lack of specific cell surface markers.
31 oskeleton regulators and the localization of cell surface markers.
32 pol mRNA, intracellular p24 Gag protein, and cell surface markers.
33 ypically purified from the bone marrow using cell surface markers.
34 Fab:Fab interactions in targeting oligomeric cell-surface markers.
35 ession profiles and identification of unique cell-surface markers.
36 lacked mature arterial, venal, and lymphatic cell-surface markers.
37 mass cytometry revealed expression of unique cell-surface markers.
38 roid progenitor cells that express analogous cell-surface markers.
39 expression of APC (macrophages and dendritic cells) surface markers.
40 ngle-cell mass cytometric measurements of 14 cell surface markers, 20 signaling/cell cycle proteins,
41 unbiased manner a panel of all commonly used cell surface markers (280 genes) from individual cells.
42 ogenase-bright cells expressed CD34 or CD133 cell surface markers (57.0% and 27.1%, respectively), co
46 nfection of DC with live 35000HP caused less cell surface marker activation than infection with heat-
47 also expressed higher levels of more mature cell surface markers, additionally linking inflammasome
48 ses were assessed by measuring expression of cell surface markers (adhesion molecules, fibrinogen-lik
50 ated with respect to endocytosis properties, cell surface markers, allostimulatory activity, and cyto
53 ary, we identified GD2 as a new CSC-specific cell surface marker and GD3S as a potential therapeutic
55 thelial cells expressed specific endothelial cell surface markers and also exhibited the capacity for
56 bpopulations of CSCs, characterized by their cell surface markers and colony morphology, which can se
58 We used a combination of flow cytometry for cell surface markers and enzyme-linked immunospot method
59 to different sub-populations on the basis of cell surface markers and examine their function in an in
60 comprised of distinct subsets with different cell surface markers and functional characteristics and
61 ntional dendritic cells (cDCs) with distinct cell surface markers and functions exist in mouse and hu
62 ancer cell line, which we first confirmed by cell surface markers and gene profiling to be highly enr
63 otypes, express telomerase activity, express cell surface markers and genes that characterize human E
65 s include (1) the identification of distinct cell surface markers and other cellular properties in he
66 stinct subsets can be distinguished based on cell surface markers and pathophysiological function.
68 d expression of CD80, CD86, CD40, and MHC-II cell surface markers and production of proinflammatory c
69 single skeletal stem cell population through cell surface markers and the development of single-cell
71 this likeness extended into the non-ISCT MSC cell surface markers and trilineage differentiation, whi
72 ubsets based on their expression of specific cell surface markers and used them in our adoptive trans
75 display significantly altered expression of cell-surface markers and produce increased inflammatory
76 ly, neutrophil morphology (nucleus shape and cell-surface markers) and functions (phagocytosis, degra
77 nology for measuring cell entity, evaluating cell surface marker, and peculiarly in the field of stem
78 xpress characteristic transcription factors, cell surface markers, and cytokines, including glycoprot
79 ion of apoptosis, acquisition of tumorigenic cell surface markers, and epithelial-mesenchymal transit
80 including those encoding effector cytokines, cell surface markers, and key transcription factors.
81 ptor (uPAR), a uniquely overexpressed cancer cell-surface marker, and facilitating the immune-mediate
84 wed AME downregulates the expression of such cell surface markers as CD80, CD86, and major histocompa
86 al cells in part by increasing the levels of cell surface markers associated with mesenchymal stem ce
88 effector T cells, and caused an increase in cell surface markers associated with T(Regs) such as Fox
89 nsplants in regenerative medicine depends on cell-surface marker-based characterization and/or purifi
90 eration of reagents that specifically target cell-surface markers, because transmembrane proteins are
92 and FACS analyses demonstrate that specific cell surface markers can be used to discriminate prostat
95 ed high Wnt activity was associated with the cell surface markers CD133, CD166, and CD29, but not CD2
97 ive L3 for 48 h showed no alterations in the cell surface markers CD14, CD86, CD83, CD207, E-cadherin
98 ammatory T cells into tissues, or target the cell surface marker CD20 (rituximab; Rituxan for hematol
99 data in K562 leukemic cells, we identify the cell surface marker CD24 as co-varying with chromatin ac
102 (MLN), spleen and thymus were labeled for T cell surface markers (CD3, CD4, CD8) and intracellular F
104 including CTNND1 and the early hematopoietic cell surface marker CD34, resulted in reduced leukemic g
106 nterferon-gamma with a unique combination of cell surface markers (CD4(+)CD25(-)CD44(hi)CD62L(lo)) an
110 ese pancreatic cancer stem cells express the cell surface markers CD44, CD24, and epithelial-specific
111 on of pancreatic cancer cells expressing the cell surface markers CD44, CD24, and epithelial-specific
112 stage transitions, marked by changes in the cell-surface markers CD44 and ICAM1, and a Nanog-enhance
113 Here, we explored this hypothesis by using 2 cell surface markers, CD44 and CD137, to isolate antitum
114 hat express IL-10, as well as Tr1-associated cell surface markers, CD49b and LAG-3, and transcription
115 rences between these profiles, we identified cell surface markers, CD69 and CD36, whose genes were di
117 seq data, we defined a novel set of possible cell surface markers (Cd74 and Cd81) for these candidate
119 n the low nanomolar range, we identified the cell surface marker CD86 as a sensitive surrogate biomar
120 ls based on a variety of phenotypes, such as cell surface markers, cell proliferation and drug respon
121 B in vitro, as measured by the expression of cell surface markers, cellular signaling events, and cyt
122 man MDDCs exposed to galectin-1 up-regulated cell surface markers characteristic of DC maturation (CD
123 ubsets of HPCs examined, including HPCs with cell surface markers consistent with immature hematopoie
124 ssion signatures, morphological changes, and cell surface markers consistent with myeloid maturation.
125 e cells (CSCs) from DCIS.com cell line using cell surface markers (CS24(-)CD44(+)ESA(+)) and found th
129 blood frequencies of MAIT cells, defined by cell surface markers, decline during tuberculosis (TB) d
130 etailed study has been hampered by a lack of cell surface markers defining tumor-specific dysfunction
132 is approach is that the presence of specific cell surface markers does not directly reflect the trans
133 umor cell (CTC) detection strategies rely on cell surface marker EpCAM and intracellular cytokeratins
134 XCL9, CXCL10, CXCL12, CXCL13 and CXCL16) and cell surface marker expression (CD3, CD4 and CXCR3) in p
135 eripheral blood, cutaneous mTregs had unique cell surface marker expression and cytokine production.
137 lar trilineage differentiation potential and cell surface marker expression as bone marrow hMSCs.
138 ted control iDCs to WT capsule did not alter cell surface marker expression but did elicit IL-8.
139 onal profiling, TCR repertoire analyses, and cell surface marker expression indicate that Dock2-defic
140 normal, displaying no obvious compromise in cell surface marker expression or antibody production ei
142 tro proliferation responses, alloreactivity, cell surface marker expression, and antibody production.
143 T cells showed convergence in the pattern of cell surface marker expression, cytokine profiles, and g
144 n of MEK1/2 did not reduce CT-B induction of cell surface marker expression, it did attenuate CT-B-me
145 nd, when isolated from skeletal muscle using cell surface marker expression, these cells showed compa
148 isolation, cells were characterized through cell-surface marker expression and lineage-specific diff
149 and, regardless of methodology for harvest, cell-surface marker expression of CD73, CD90, CD105, and
151 lectively increased polyploidization, mature cell-surface marker expression, and apoptosis of maligna
153 transmembrane glycoprotein, is an important cell surface marker for both stem cells and cancer stem
156 xpression of CD83 (previously described as a cell surface marker for mature dendritic cells) on CD4 T
159 matic stem cells, and it is widely used as a cell surface marker for the isolation and characterizati
162 Our analysis identified 24 new/potential cell surface markers for murine fetal hepatic stem cells
163 ss I molecules offers unique cancer-specific cell surface markers for the identification and targetin
167 nd proteomic approaches to identify specific cell-surface markers for cardiac PW1(+) cells and found
168 In this study, we attempted to identify cell-surface markers for leukemia-initiating cells in FA
170 of commercial antibodies, we have identified cell-surface markers for the separation of pancreatic ce
171 cently, a simple phenotype for HSCs based on cell surface markers from the signaling lymphocyte activ
172 ood, we acquired an immunological profile of cell-surface markers from healthy control and untreated
173 ytosis activity, viability and expression of cell-surface markers, from tens of thousands of single i
174 ssessment, nitro-blue tetrazolium reduction, cell-surface markers, genome-wide patterns of gene expre
175 h all the hallmarks of stem cells, including cell surface markers, global gene expression profiles, a
178 allowing detection of increasing numbers of cell surface markers, has challenged the traditional tec
180 rious combinations of antibodies directed to cell surface markers have been used to isolate human and
182 the basis of the differential expression of cell-surface markers, here we identify a mesenchymal str
183 ific membrane antigen (PSMA), a prototypical cell surface marker highly overexpressed in prostate can
184 utrophils, fibroblasts, and lymphocytes; and cell surface markers, ie, F4/80, CD11b, CD11c, and Ly-6C
185 ify and purify anergic T cells by a distinct cell surface marker in an autoimmune disease and paves t
186 he up-regulation of macrophage/hematopoietic cell surface markers in a large proportion of NIH 3T3 ce
189 o studied expression of the identified novel cell surface markers in fetal rat liver progenitor cells
190 In this work, we studied progenitor/oval cell surface markers in the liver of rats subjected to 2
194 ells that exhibit differential expression of cell surface markers, including CD105 (or endoglin), Thy
195 ethod was reported, using the SLAM family of cell-surface markers, including CD150 (SlamF1), to offer
197 The transcriptional profile identified 2 cell-surface markers, ITGA6 and NGFR, which can be used
198 n inflammatory cell numbers and cytokine and cell-surface marker levels on monocytes and macrophages.
199 gland stem cells (MaSCs) using combinatorial cell surface markers (Lin(-)CD24(+)CD29(h)CD49f(h)) has
200 lation based on the expression profiles of 2 cell-surface markers LNGFR (CD271) and THY-1 (CD90).
201 re the identification of TIM3 as an AML stem cell surface marker more highly expressed on multiple sp
203 We have isolated rare cells bearing the NK cell surface marker NK1.1, as well as the dendritic cell
204 rapidly upregulate the expression of the NK cell-surface marker NK1.1 in response to MSU crystals bu
206 e attenuates acute loss of the developing OL cell surface marker O1 and the mature OL marker MBP (mye
207 ir expression of CD4, CD8, naive, and memory cell-surface markers, occupy distinct homeostatic compar
212 11 receptor alpha subunit (IL-11Ralpha) as a cell surface marker of tumor progression that correlates
213 ets showed that effector memory pathways and cell surface markers of activation and proliferation in
214 ukocytes (VLC), due to sharing functions and cell surface markers of both dendritic cells and endothe
215 protein resulted in increased expression of cell surface markers of DC maturation and an increase in
216 that a group of immature myeloid cells with cell surface markers of Gr-1(+) CD11b(+) are highly enri
217 ion, cytokine production, proliferation, and cell surface markers of immune cells between GA-treated
218 ry CD8 T cells from naive old mice expressed cell surface markers of memory in addition to receptors
219 MSCs) were established and characterized for cell surface markers of mesenchymal stem cell origin in
220 also known as CD143), a recently identified cell-surface marker of adult human hematopoietic stem ce
224 S, and microglial activation was assessed by cell surface marker or phospho-MAPK immunofluorescence.
226 ls (CSCs) can be identified by expression of cell surface markers or enzymatic activity, but these me
229 haracteristics such as cell cycle status and cell surface marker phenotype, they respond to different
230 These results demonstrate that CD96 is a cell surface marker present on many AML-LSC and may serv
231 ese results demonstrate that IL-3Ralpha is a cell-surface marker present on FA-AML leukemia-initiatin
232 vity was confirmed by examining cytokine and cell surface marker production in bone-marrow-derived de
233 n cytokine-supplemented medium changed their cell surface marker profile and gene expression pattern
236 OV-1 antigen, a6 integrin, and connexin 43), cell surface markers recently identified by us (CD44, CD
237 in rat tissue, they distinctively express a cell surface marker recognized by the SE-1 antibody.
238 ation; SP) in conjunction with canonical HSC cell-surface markers (Sca-1, c-Kit, and lineage markers)
243 identical sets of T(EM)-associated genes and cell surface markers shown to be associated with latency
245 n is reliant on the presence of well-defined cell surface markers specific for diverse progenitor pop
247 cells as demonstrated by expression of stem cell surface markers such as aldehyde dehydrogenase 1, s
250 ed by a remarkable up-regulation of specific cell surface markers, suggesting that LPS stimulation le
251 n of MSC with both endothelial and pericytic cell surface markers suppresses the homing of cancer cel
252 olobus purpureas agglutinin (TPA) as a novel cell surface marker that allows for such delineation.
255 eceptor superfamily member OX40 (CD134) is a cell surface marker that is highly specific for activate
257 e and chemokine responses, and expression of cell surface markers that are related to T cell activati
258 nitive endoderm with the goal of identifying cell surface markers that can be used to track the devel
259 t LSC, one potential strategy is to identify cell surface markers that can distinguish LSC from norma
260 ssociated with subclonal mutations, and find cell surface markers that could be used to purify subclo
263 acteristics, as well as the precise panel of cell surface markers that uniquely define this newly des
264 ha,and IL-2, and up-regulation of numerous T cell surface markers that would promote T-T Ag presentat
266 , zymosan, heat-killed or live bacteria, and cell-surface markers that coexpress with FR were identif
268 ptor type I and II (CD121a/CD121b) as unique cell-surface markers that distinguish activated Tregs fr
269 lls have been hampered by a lack of suitable cell-surface markers that specifically enable their puri
270 cell subsets that display either CD4 or CD8 cell surface markers, the leukemic cell is exclusively o
271 ltiple defects in the expression of effector cell surface markers, the synthesis of cytokines, and in
274 op an assay based on loss of expression of a cell surface marker to monitor epigenetic instability at
276 ling core-2 O-glycosylation and identified a cell surface marker to quantify Notch signals in multipl
278 ins on fibroblast cells, which are potential cell surface markers to differentiate endothelial and fi
279 tential in part independent of commonly used cell surface markers to discriminate effector and memory
281 nhanced green fluorescent protein (eGFP) and cell surface markers to FACS-isolate DeltaSox2-eGFP(+) G
282 is due, in part, to the difficulty of using cell surface markers to identify CD4(+)CD25(+) T reg cel
283 we combine H2B-GFP-based pulse-chasing with cell-surface markers to distinguish quiescent from proli
284 ave used flow cytometry and a defined set of cell-surface markers to identify and subsequently isolat
285 ly carried out by quantification of multiple cell surface markers, transcription factors and cytokine
287 eries of heterogeneous subpopulations in its cell surface markers, tumorigenicity, invasion and metas
295 he differential expression patterns of these cell surface markers were dependent on Ly49H recognition
297 cently identified myeloid lineage restricted cell surface marker, which is overexpressed in over 90%
298 Cs and a germline seminoma that share a CD38 cell-surface marker, which collectively defines likely p
300 r-, costimulation-, or activation-associated cell surface markers yet were associated with lower Ly6C