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1 e that arises from the head-tail symmetry of cell-to-cell interaction).
2 a process that facilitates immune evasion or cell to cell interaction.
3 that ECs can directly activate basophils via cell-to-cell interaction.
4 ors, while the CD8+ T cell response required cell-to-cell interaction.
5 hat influence survival, cell morphology, and cell-to-cell interaction.
6 des present on adjacent cells, thus creating cell to cell interactions.
7 ation during PMN vascular as well as mucosal cell to cell interactions.
8 ass of biological systems exhibiting typical cell to cell interactions.
9 se to the theoretically calculated limit and cell-to-cell interactions.
10 tumor microenvironmental patterns, including cell-to-cell interactions.
11 numerous cellular activities, processes, and cell-to-cell interactions.
12 ems that collectively make decisions through cell-to-cell interactions.
13 population structure and closer interspecies cell-to-cell interactions.
14 ear cells (PBMCs) in order to study in vitro cell-to-cell interactions.
15 feration and differentiation, through direct cell-to-cell interactions.
16 ion via stimulating both cell-to-surface and cell-to-cell interactions.
17 These changes implicate extensive cell-to-cell interactions.
18 release and CD4+ T cell proliferation during cell-to-cell interactions.
19 tracellular matrix components, bacteria tune cell-to-cell interactions.
20 hibit decreased migration speed and impaired cell-to-cell interactions.
21 nformation about cellular subpopulations and cell-to-cell interactions.
22 of cell surface receptors as well as general cell-to-cell interactions.
23 mpassing proliferation, differentiation, and cell-to-cell interactions.
24 supported by various types of spatiotemporal cell-to-cell interactions.
25 is, including cytokines, growth factors, and cell-to-cell interactions.
26 ned influence of hydrodynamic and mechanical cell-to-cell interactions.
27 s a complex network of soluble mediators and cell-to-cell interactions allowing human classically act
30 cell cultures do not resemble intercellular cell-to-cell interaction and communication; however, thr
31 on as a unique signaling system triggered by cell-to-cell interaction and have been shown to mediate
32 a necessity when considering characterizing cell-to-cell interaction and rolling dynamics during cel
34 ts: DE due to pathology, anatomical regions, cell-to-cell interactions and cellular microenvironment.
35 trafficking of cells that affect the dynamic cell-to-cell interactions and determine the outcome of i
40 chemokines, CCL5 and CCL6, was dependent on cell-to-cell interaction, and was observed only with fib
42 ell covariance and conditional distribution, cell-to-cell interactions, and short/long-scale arrangem
43 chanisms of inter-leukemic communication and cell-to-cell interactions are proposed to be important f
44 hich fail to recapitulate spatial aspects of cell-to-cell interactions as well as tissue gradients pr
45 detect and define, and make inferences about cell-to-cell interactions at single-cell resolution.
47 ed a computational method to infer potential cell-to-cell interactions based on transcriptome data of
49 bel-free, and noninvasive probing of dynamic cell-to-cell interactions between adherent and nonadhere
52 local non-MHC stimulatory factors or direct cell-to-cell interactions between the T cells themselves
53 plethora of signalling processes influencing cell-to-cell interactions between the vascular endotheli
54 istance, are required for citrate-stimulated cell-to-cell interactions, but the GraRS regulatory syst
55 s and can alter the microenvironment through cell-to-cell interactions by fusion with the plasma memb
56 s establishing the theoretical basis for how cell-to-cell interactions can influence population level
57 scriptomic analyses revealed that sequential cell-to-cell interactions direct functional maturation o
58 ng a unique insight into the dynamics of how cell-to-cell interaction drives intratumor heterogeneity
59 stem-cell behavior during its migration and cell-to-cell interaction during the process of homing.
62 ss, the implications of matrix plasticity on cell-to-cell interactions during EC outgrowth, along wit
63 ller incorporates both short- and long-range cell-to-cell interactions (e.g. via paracrine and endocr
64 odel predicts three possible benefits of the cell-to-cell interactions: First, the asymmetric interac
65 s and coupled analytical methods can recover cell-to-cell interactions, groups of spatially covarying
67 is important in establishing and maintaining cell to cell interactions in epithelial cells, thereby p
68 active eicosanoids that can be formed during cell to cell interactions in human tissues to self limit
71 spatiotemporal gene expression landscape and cell-to-cell interactions in their native microenvironme
72 identified that NOTCH signaling drives these cell-to-cell interactions in tumors, with pericytes and
73 , however, bypasses the normal mechanisms of cell-to-cell interactions in which Notch-1 participates.
75 raded when derived from a model that ignored cell-to-cell interactions, indicating the importance of
76 expression analyses, pathway enrichment and cell-to-cell interaction inferences are more accurate.
80 tic Elucidation and Assessment of Regulatory Cell-to-cell Interaction Networks (SEARCHIN) to identify
82 e three-dimensional spatial organization and cell-to-cell interactions of brain tissue that are presu
83 ine how alterations in cellular function and cell-to-cell interactions of pulmonary resident cells an
84 to better understand the composition and the cell-to-cell interactions of the alloreactive inflammato
86 ollowed by a relatively slower step in which cell-to-cell interactions predominate (the aggregation p
88 ), through deregulation of the expression of cell-to-cell interaction proteins and protein secretion.
90 d of the macroscopic patterns resulting from cell-to-cell interactions remains largely qualitative.
91 n that the bystander phenomenon must involve cell-to-cell interactions, the relevance of such single-
92 omposition, transcriptional cell states, and cell-to-cell interactions, thereby providing insight int
93 rs cell proliferation, death, migration, and cell-to-cell interaction through contact inhibition.
94 This antiapoptotic effect required direct cell-to-cell interactions, was associated with phosphory
95 bind avidly to C. albicans hyphae via direct cell-to-cell interaction, while the cariogenic pathogen
96 hat their morphology is sculpted by specific cell to cell interactions with neurons and each other.
98 evidence that migratory DCs execute targeted cell-to-cell interactions with stationary MCs before lea