ライフサイエンスコーパス: cellular proliferation

1 protein kinase (MAPK) pathway and subsequent cellular proliferation.

2 oting cellular differentiation to aggressive cellular proliferation.

3 nhibiting critical cell functions, including cellular proliferation.

4 recruitment and contribute independently to cellular proliferation.

5 ide secretion also induced YAP signaling and cellular proliferation.

6 tating RUNX1 regulation of transcription and cellular proliferation.

7 TBI penumbra and hippocampus had higher cellular proliferation.

8 elomerase enables chromosome survival during cellular proliferation.

9 cycle regulators, with concomitant increased cellular proliferation.

10 ay be a response to, rather than a cause of, cellular proliferation.

11 hondrial volume, and a severe suppression of cellular proliferation.

12 elin with inflammation and myo-inositol with cellular proliferation.

13 in pro-neural gene expression and increased cellular proliferation.

14 phenotypes, including insulin secretion and cellular proliferation.

15 hat RIF is primarily associated with reduced cellular proliferation.

16 ng sites of transcription factors that drive cellular proliferation.

17 l enhancers that control gene expression and cellular proliferation.

18 y cellular processes including apoptosis and cellular proliferation.

19 utative oncogene with roles in secretion and cellular proliferation.

20 ollowed by culture in TSC medium to maintain cellular proliferation.

21 in both positively and negatively regulating cellular proliferation.

22 reased patient survival, and is required for cellular proliferation.

23 d, they were all found capable of inhibiting cellular proliferation.

24 to reduced metabolic rate and inhibition of cellular proliferation.

25 s, as well as growth retardation and reduced cellular proliferation.

26 egulation are therefore necessary for proper cellular proliferation.

27 cytoplasmic PI3K-AKT pathway and suppresses cellular proliferation.

28 forces outstanding anabolic requirements for cellular proliferation.

29 he proper temporal and spatial regulation of cellular proliferation.

30 Telomerase is essential for continuous cellular proliferation.

31 e cells in lymph nodes, indicating extensive cellular proliferation.

32 tant cell lines and plays essential roles in cellular proliferation.

33 critical transcription factor that promotes cellular proliferation.

34 T) is a functional radiotracer used to study cellular proliferation.

35 transcription factors are key regulators of cellular proliferation.

36 ts regarding the role of Mfn2 in controlling cellular proliferation.

37 ndependently associated with tumor growth or cellular proliferation.

38 iary lymphoneogenesis with active centers of cellular proliferation.

39 ads to an increase of nearly 100% in overall cellular proliferation.

40 ogenesis, at least in part by enabling rapid cellular proliferation.

41 Mitochondrial FOXM1 did not change cellular proliferation.

42 ls and transcriptional activity and promotes cellular proliferation.

43 wing acute liver injury that occurs prior to cellular proliferation.

44 de of the JNK1/2 signaling pathway, inhibits cellular proliferation.

45 lated glycolytic ATP production and restored cellular proliferation.

46 iral therapy (ART) and is maintained through cellular proliferation.

47 aNp63alpha protein levels, thereby promoting cellular proliferation.

48 tream effects through phospho-ERK levels and cellular proliferation.

49 the regulation of nuclear ERK signalling and cellular proliferation.

50 significantly reduced AR transactivation and cellular proliferation.

51 DK)/cyclin complexes drive most processes in cellular proliferation.

52 Membrane potential is also central to cellular proliferation.

53 transcription factor involved in ERK-induced cellular proliferation.

54 ing embryonic development, a period of rapid cellular proliferation.

55 endent effect on immunoblotting), stimulated cellular proliferation (about 1.8-fold increase in cell

56 ults in unopposed ERalpha mediated increased cellular proliferation, activation of the ERE and increa

57 l as the effects of PD-1 pathway blockade on cellular proliferation after T-cell receptor stimulation

58 d from A-T [M] mutant mice exhibited reduced cellular proliferation and an altered DNA damage respons

59 of Cx43, but not Cx26, significantly reduced cellular proliferation and anchorage-independent growth

60 vPK also augments cellular proliferation and anchorage-independent growth.

61 e cells' proliferation rate, suggesting that cellular proliferation and apoptosis are concurrent.

62 ions in the expression of well characterized cellular proliferation and apoptosis guards (NF-kappaB,

63 ation, epithelial barrier breach, changes in cellular proliferation and apoptosis, genome instability

64 on and degradation of multiple regulators of cellular proliferation and apoptosis, including the tumo

65 n to their well-known role in the control of cellular proliferation and cancer, cell cycle regulators

66 its tumorigenic effects in part by promoting cellular proliferation and cell survival.

67 tested 2 functional parameters of our model, cellular proliferation and chemotactic potential of cond

68 fractions (NHFs) have a remarkable effect on cellular proliferation and collective migration, and exh

69 n in the cells led to profound reductions in cellular proliferation and colony-formation capacities.

70 mality is not associated with impairments in cellular proliferation and death, but aberrantly increas

71 logical targets because of their key role in cellular proliferation and death.

72 enic HSCT and allowed visualisation of early cellular proliferation and detection of patterns of cell

73 crucial for proper retinal lamination during cellular proliferation and development.

74 Spatiotemporal balancing of cellular proliferation and differentiation is crucial fo

75 G5) functions in cell polarity and regulates cellular proliferation and differentiation via undefined

76 tential of 2-hydroxyglutarate, the impact on cellular proliferation and differentiation, and the infl

77 sion, potency, and function, particularly in cellular proliferation and differentiation, as well as i

78 tissue homeostasis, notably via controlling cellular proliferation and differentiation.

79 ly regulated biological system that controls cellular proliferation and differentiation.

80 ole as an integrator of signals that control cellular proliferation and differentiation.

81 meostasis depend on the regulated control of cellular proliferation and differentiation.

82 oducts that manipulate processes involved in cellular proliferation and differentiation.

83 Its functions include immune modulation and cellular proliferation and differentiation.

84 In response to cellular proliferation and DNA damage, proteasome and HS

85 Inhibition of BCL-2 expression can decrease cellular proliferation and enhance the efficacy of chemo

86 studies implicate Gal-9 in the regulation of cellular proliferation and epithelial restitution after

87 d by viral etiology must exhibit deregulated cellular proliferation and evade immune recognition.

88 providing a rationale for their function in cellular proliferation and for the apoptotic effect of t

89 play an important role in the regulation of cellular proliferation and functions as a tumor suppress

90 d accurate DNA replication is fundamental to cellular proliferation and genome stability.

91 hown to predict survival, clinical subtypes, cellular proliferation and genomic instability.

92 es to mRNA translation to effect facultative cellular proliferation and glucose homeostasis.

93 which is secreted by the ovaries and induces cellular proliferation and growth of the uterus and mamm

94 tches indispensable for proper regulation of cellular proliferation and growth.

95 vel mechanism how matrix stiffness regulates cellular proliferation and highlights the importance of

96 curtail transcriptomic processes involved in cellular proliferation and IL-13-induced responses, and

97 EBP1, with decreased cytokine production and cellular proliferation and increased phagocytosis activi

98 reases SASH1 protein concentrations, reduced cellular proliferation and increased sensitivity to cisp

99 ng drug, temozolomide, significantly reduces cellular proliferation and increases DNA damage.

100 ized HMECs (TRIM24 iHMECs) greatly increased cellular proliferation and induced malignant transformat

101 a co-activator and a co-repressor to enhance cellular proliferation and inhibit myogenic differentiat

102 to monitor acute effects of chemotherapy on cellular proliferation and its recovery in bone marrow,

103 These integrants appear to be maintained by cellular proliferation and longevity of infected cells,

104 expressions that result in the regulation of cellular proliferation and may contribute to ER-positive

105 GLTSCR2 controls cellular proliferation and metabolism via the transcript

106 Cells expressing the S114A/S446A mutant have cellular proliferation and migration defects.

107 vacaine reduced cell viability and inhibited cellular proliferation and migration in both cell lines.

108 suppressor let-7 family of miRNAs, inhibits cellular proliferation and migration in vitro and slows

109 methyltransferase activity of G9a inhibited cellular proliferation and migration in vitro and tumor

110 to RvE1 promoted wound repair by increasing cellular proliferation and migration through activation

111 geting expression or activity of G9a reduced cellular proliferation and motility in vitro and tumor g

112 structural alterations as well as increased cellular proliferation and motility.

113 gnaling pathway is an important regulator of cellular proliferation and organ size.

114 ant to the pathogenesis of asthma, including cellular proliferation and pathways associated with gluc

115 t, glioblastoma high immune infiltration and cellular proliferation and poor survival prognosis.

116 oreover, overexpression of HDAC5 accelerated cellular proliferation and promoted acridine mutagen ICR

117 PHOS complex activity and increased in vitro cellular proliferation and promoted tumor development in

118 C1/2 have essential and pleiotropic roles in cellular proliferation and regulate stem cell self-renew

119 f lung cancer cell lines mediated changes in cellular proliferation and sensitivity to cisplatin.

120 ough all variants induced cytokine-dependent cellular proliferation and STAT3 phosphorylation via CNT

121 ctivity as reflected by reduced drug-induced cellular proliferation and STAT3 phosphorylation.

122 of the mTOR signalling pathways and inhibits cellular proliferation and stem-cell-derived organoid fo

123 ere, we show that this modification promotes cellular proliferation and suppression of genomic instab

124 pression in glioblastoma, we found that both cellular proliferation and survival were affected upon A

125 The Hippo signaling pathway regulates cellular proliferation and survival, thus exerting profo

126 genesis, in particular to aspects other than cellular proliferation and survival, we generated three

127 y immune regulation and DNA repair, but also cellular proliferation and survival.

128 ly active kinase that plays crucial roles in cellular proliferation and survival.

129 (PTEN) is a lipid phosphatase implicated in cellular proliferation and survival.

130 de biosynthesis and plays important roles in cellular proliferation and the immune response.

131 to maintain morphology through regulation of cellular proliferation and tissue integrity.

132 s a critical factor in determining long-term cellular proliferation and tissue renewal.

133 emodeling that is characterised by increased cellular proliferation and tissue stiffness.

134 pathway, an essential event for uncontrolled cellular proliferation and transformation.

135 ine signaling, which can aberrantly modulate cellular proliferation and tumorigenesis.

136 of MYC regulated genes, leading to increased cellular proliferation and tumour burden.

137 multi-faceted complex pathophysiology, with cellular proliferation and vascular remodeling being the

138 s a cardiopulmonary disease characterized by cellular proliferation and vascular remodeling.

139 ed for HIV infection yet are dispensable for cellular proliferation and viability.

140 They had reduced cellular proliferation and were less dependent on oxidat

141 e that activates epithelial beta-catenin and cellular proliferation, and (2) the simultaneous inhibit

142 ellular molecules (p < 0.01) and accelerated cellular proliferation, and (3) significantly increased

143 o changes in gene expression, a reduction in cellular proliferation, and an inability to differentiat

144 ement of cell death, mitochondrial function, cellular proliferation, and anchorage-independent growth

145 t and chromosome segregation during mitosis, cellular proliferation, and apoptosis.

146 suppressor and regulates genomic integrity, cellular proliferation, and apoptosis; however, its role

147 al parameters of cellular diffusion, rate of cellular proliferation, and cellular tractions forces.

148 inted gene that has roles in growth control, cellular proliferation, and insulin signaling.

149 omote nuclear translocation of beta-catenin, cellular proliferation, and resistance to apoptosis.

150 ant AKT(ser473) and p70S6 signalling, slowed cellular proliferation, and reversed mitochondrial abnor

151 e being uncovered in autophagy, hypoxia, and cellular proliferation, and the lipids are now implicate

152 oxidative endurance, tumorigenic potential, cellular proliferation, and tumor growth.

153 omelanocortin transcription, ACTH secretion, cellular proliferation, and tumor invasion rates in vitr

154 In swine arteries, the BEM increases cellular proliferation, angiogenesis, and connective tis

155 remission associates to functions related to cellular proliferation, apoptosis, and inflammatory resp

156 While low-dose paclitaxel did not affect cellular proliferation, apoptosis, or cytoskeletal integ

157 rent tubular injuries but was independent of cellular proliferation as determined in physiological gr

158 ed one-carbon metabolism gene expression and cellular proliferation as well as increased global bioen

159 Furthermore, infection did not induce cellular proliferation, as it does in B cells, but inste

160 icient PAR1 was more effective at increasing cellular proliferation, associated with Gq signaling, th

161 dasatinib led to apoptosis and inhibition of cellular proliferation, associated with reduced phosphor

162 zation of p53, leading not only to continued cellular proliferation but also to further accumulation

163 types can be amplified, perhaps initially by cellular proliferation but further by limited viral repl

164 LINC00261 as a tumor suppressor that blocks cellular proliferation by activating the DNA damage resp

165 dometrial cancer show that metformin reduces cellular proliferation by inhibition of the PI3K-AKT-mTO

166 the high-risk HPV16 E7 oncogene can promote cellular proliferation by interacting with the DREAM (DP

167 pression function, participates in E2-driven cellular proliferation by modulating the expression of d

168 NEK10 loss increases cellular proliferation by modulating the p53-dependent t

169 iated regulation of DeltaNp63alpha increases cellular proliferation by promoting G(2)/M progression t

170 We demonstrate that G9a enhances cellular proliferation by two distinct mechanisms.

171 mechanism leads to a target cell size where cellular proliferation capacity is maximized.

172 opposite polarization dynamics depending on cellular proliferation capacity.

173 ein-protein interaction network that governs cellular proliferation (cell cycle) is highly complex.

174 otein kinase known to play multiple roles in cellular proliferation, cell cycle regulation, and carci

175 lution and plays a role in mRNA translation, cellular proliferation, cellular differentiation, and in

176 melanoma cells resulted in (i) a decrease in cellular proliferation, colony formation, and cellular m

177 man melanocytes, which resulted in increased cellular proliferation, colony formation, invasion, and

178 down of SEMA4C promotes adhesion and reduces cellular proliferation, colony formation, migration, wou

179 Moreover, we found that cellular proliferation counterbalanced the decay of HIV-

180 number of known ER target genes involved in cellular proliferation (cyclin D1, CDKs) and morphogenes

181 use large B-cell lymphomas (DLBCLs) inhibits cellular proliferation, decreases cholesterol biosynthes

182 an aggressive phenotype including increased cellular proliferation, deregulated G2-M checkpoint foll

183 It can provide clonal-level insights into cellular proliferation, development, differentiation, mi

184 ng through the MAP kinase cascade to control cellular proliferation, differentiation and survival(1-3

185 lation of BMP-2 production mediates rates of cellular proliferation, differentiation and tissue produ

186 oad array of biological processes, including cellular proliferation, differentiation, and apoptosis.

187 on and/or epigenetic factor, participates in cellular proliferation, differentiation, and death.

188 mutations in signaling pathways that control cellular proliferation, differentiation, and survival.

189 a ubiquitous second messenger that regulates cellular proliferation, differentiation, attachment, mig

190 eraction network analysis suggested enriched cellular proliferation/differentiation pathways in truck

191 insight into the role of APA in controlling cellular proliferation during biological processes such

192 Dnmt3a delayed tumorigenesis by suppressing cellular proliferation during disease progression.

193 or, a downstream target of K-Ras, stimulates cellular proliferation during embryogenesis, organ repai

194 While many factors regulating cellular proliferation during liver repair have been ide

195 ptionally activates target genes to suppress cellular proliferation during stress.

196 ived growth factor (PDGF)-DD, which promotes cellular proliferation, epithelial-mesenchymal transitio

197 K25 promotes YAP/TAZ activation and enhanced cellular proliferation, even under normally growth-suppr

198 gulate diverse biological processes, such as cellular proliferation, gene transcription, and tumorige

199 95% CI: 3.57 to 25.1, p = 0.031), and higher cellular proliferation (group difference: 11.3; 95% CI:

200 rmal growth factor receptor (EGFR)-dependent cellular proliferation, has been achieved.

201 RG1 plays an important role in MCPyV-induced cellular proliferation.IMPORTANCE Merkel cell carcinoma

202 f NEAT1 in hypoxia also leads to accelerated cellular proliferation, improved clonogenic survival and

203 entation is exploited to promote anisotropic cellular proliferation in 3D.

204 have investigated the dynamics of growth and cellular proliferation in a murine vestibular organ, the

205 ning BH3 mimetics and gamitrinib-TPP blunted cellular proliferation in a synergistic manner by massiv

206 creased SFRP1 mRNA expression, and decreased cellular proliferation in a trophoblast cell line.

207 iated genetic mutation resulted in increased cellular proliferation in all cell lines tested, the gen

208 orothymidine ((18)F-FLT) was used to measure cellular proliferation in bone marrow (BM), whereas MRI

209 Rapid cellular proliferation in early development and cancer d

210 039 did not affect cell cycle progression or cellular proliferation in host cells.

211 se cell cycle arrest and suppressed in vitro cellular proliferation in NSCLC cancer cells.

212 hibitors after taxanes cooperates to prevent cellular proliferation in pancreatic ductal adenocarcino

213 evealed abundant lung neovascularization and cellular proliferation in PE that was distinctly absent

214 miR-130/301 family is a master regulator of cellular proliferation in PH via regulation of subordina

215 ncogene expression only marginally increased cellular proliferation in the epidermis of Tg animals, c

216 Herein, cyclin D1 inactivation reduced cellular proliferation in the murine prostate in vivo an

217 Metformin inhibited cellular proliferation in the presence of glucose, but i

218 d the hypothesis that metformin would reduce cellular proliferation in vivo in atypical endometrial h

219 e upregulated genes, known to be involved in cellular proliferation, included c-Myc along with its ta

220 deficient embryonic stem cells have impaired cellular proliferation, increased apoptosis, defective c

221 Additionally, there was an increase in cellular proliferation, increased proteoglycan distribut

222 c signaling is an effective strategy to halt cellular proliferation, induce apoptosis and eliminate c

223 Depletion of HDAC5 by shRNA hindered cellular proliferation, induced G1 cell cycle arrest, an

224 Mostly because of its effects on metabolism, cellular proliferation, inflammation, and immunity, the

225 nsduction for multiple receptors, regulating cellular proliferation, invasion, and metastasis in huma

226 ls showed that SPZ1 was able to regulate the cellular proliferation, invasion, and tumorigenic activi

227 seem to be a primary source for rebound HIV, cellular proliferation is an important driver of HIV per

228 Cellular proliferation is antagonistically regulated by

229 Defining the genes that are essential for cellular proliferation is critical for understanding org

230 s demonstrate that within a cell population, cellular proliferation is low for small and large cells,

231 AT1 and STAT2, triple-negative breast cancer cellular proliferation is only negatively affected by ty

232 enzymatic activity, and consequently promote cellular proliferation, is compromised by mutations with

233 he STAT5A and STAT5B isoforms did not affect cellular proliferation, loss of STAT5 significantly decr

234 unostaining with antibodies against Ki-67, a cellular proliferation marker.

235 Knockdown of FAM83H-AS1 by siRNAs decreased cellular proliferation, migration and increased apoptosi

236 xpression of MBIP significantly enhances the cellular proliferation, migration and invasion of NSCLC

237 (14,15-EET), a metabolite known to influence cellular proliferation, migration, and angiogenesis.

238 dependent second messengers, with effects on cellular proliferation, migration, and invasiveness.

239 -signal-regulated kinases, and regulation of cellular proliferation (miR20b, miR10b, and miR141 throu

240 lipid sphingosine-1-phosphate (S1P) mediates cellular proliferation, mitogenesis, inflammation, and a

241 h as inflammation, resistance to cell death, cellular proliferation, neurohormonal stress, angiogenes

242 to maintain the increased rRNA synthesis and cellular proliferation observed in the absence of Arf.

243 cation factor 1 may explain the reduction in cellular proliferation observed on MINCR knockdown.

244 ulenin, a small molecule antibiotic, blocked cellular proliferation of KRAS-associated lung cancer ce

245 at domain 77 protein (WDR77) is required for cellular proliferation of lung and prostate epithelial c

246 1 inhibitor was shown to effectively inhibit cellular proliferation of NSCLC cells in a dose-dependen

247 erived exosomes exerted a positive effect on cellular proliferation of recipient RMS cells and fibrob

248 n demonstrated a synergistic decrease in the cellular proliferation of several ATC cells.

249 Abnormalities in cellular proliferation or differentiation in any of thes

250 n species (ROS), immunoinflammatory effects, cellular proliferation, or apoptosis with concomitant up

251 tial working memory performance, hippocampal cellular proliferation, or hippocampal neurogenesis.

252 n burn patients expressed Ki67, a marker for cellular proliferation (P < 0.05).

253 Cellular proliferation pathways are influenced by the ge

254 locked aspartate production and reprogrammed cellular proliferation pathways, while application of as

255 A primary in vitro drug screen assessed cellular proliferation patterns in Megf10-deficient myob

256 ease in mucin was comparable to increases in cellular proliferation (Pearson R = 0.978).

257 emin (NS) has been associated with increased cellular proliferation potential and tumor malignancy du

258 suggests that I-PTH plays a critical role in cellular proliferation, proteoglycan distribution, and m

259 ssion and mutations in TRAIP therefore limit cellular proliferation, providing a potential mechanism

260 s had prolonged S and G2 phases with reduced cellular proliferation rates.

261 Inhibiting cellular proliferation reduced Ki67 expression and HIV-1

262 sis and regeneration require coordination of cellular proliferation, regulated in part by secreted gr

263 denylation (APA) that occurs during enhanced cellular proliferation represents an important, yet poor

264 Cellular proliferation requires increased production of

265 Analysis of cellular proliferation revealed many Ki67(+) cells durin

266 indicated by induction of cytokine-dependent cellular proliferation, signal transduction, and transcr

267 1, leading to accelerated mitosis and faster cellular proliferation, similarly to SETD6-deficient cel

268 ulators, mTORC1 and tp53 and correlated with cellular proliferation status.

269 itude of genes involved in the regulation of cellular proliferation, stemness, and epithelial-mesench

270 ximal gene regulatory regions was coupled to cellular proliferation, suggesting a significant growth

271 of Etv2 resulted in a ~2.5-fold increase in cellular proliferation, supporting a proliferative role

272 ne expression is critical for the control of cellular proliferation, survival, and development.

273 spects of AML disease development, including cellular proliferation, survival, and differentiation.

274 activation of signaling pathways involved in cellular proliferation, survival, and differentiation.

275 sential process that governs many aspects of cellular proliferation, survival, and differentiation.

276 ant activation of signaling pathways driving cellular proliferation, survival, and therapeutic resist

277 (EM) cells in PB was stable, indicating that cellular proliferation that compensates for T cell loss

278 Recent evidence shows that it is not only cellular proliferation that is heavily compromised in DS

279 etastasis, and MYC is a central regulator of cellular proliferation that is upregulated in many cance

280 cluding proinflammatory cytokine release and cellular proliferation that were induced after potent T

281 n by restoring hepatic homeostasis, limiting cellular proliferation through reduced cyclinD1 expressi

282 ene expression allows the virus to stimulate cellular proliferation to support viral genome replicati

283 1 (HIV-1) reservoir and the contribution of cellular proliferation to the maintenance of the reservo

284 sory epithelium during development, confines cellular proliferation to the organ's periphery, and eve

285 The contribution of cellular proliferation to viral persistence is particula

286 ivo tumor-initiating capacity, while leaving cellular proliferation unaltered.

287 study how chronic inflammation and abnormal cellular proliferation underlie tumorigenesis and tumor

288 thin stratified epithelia included increased cellular proliferation, unscheduled DNA synthesis, incre

289 for RNA splicing, cell migration, controlled cellular proliferation, vasculogenesis, extracellular ma

290 cribed a new role for Etv2 as a regulator of cellular proliferation via Yes1 in mesodermal lineages.

291 Cellular proliferation was assessed by Ki-67 proliferati

292 Decreased cellular proliferation was associated with G0/G1 cell cy

293 Cellular proliferation was evaluated with 5-bromo-2'-deo

294 The rate of cellular proliferation was higher in mutant cells under

295 ta, matrix metalloproteinase expression, and cellular proliferation were markedly increased.

296 quantification, astrogliosis, apoptosis and cellular proliferation were performed.

297 demonstrated to be effective in stimulating cellular proliferation when the sera from mice are injec

298 has a secondary function, as a regulator of cellular proliferation, which is independent of its cata

299 ce is needed, it appears that the imaging of cellular proliferation with PET and 3'-deoxy-3'-(18)F-fl

300 hysiologic upregulation of c-Myc can lead to cellular proliferation without DNA replication stress.