ライフサイエンスコーパス: cellular response

1 essment of the stimulus dependent, dynamical cellular response.

2 cs where a light source is used to trigger a cellular response.

3 perturbations is essential for understanding cellular response.

4 e prediction of cell-specific post-treatment cellular response.

5 ew binding events to successfully activate a cellular response.

6 e mode-of-cooperation capable of fine-tuning cellular response.

7 ng that this pathway represents a protective cellular response.

8 ber 5 (PGAM5) is important for the antiviral cellular response.

9 and transducing biomechanical signals into a cellular response.

10 iviral activity, whereas others regulate the cellular response.

11 Gene expression is a key determinant of cellular response.

12 M, corticosteroids and ephrins in mediating cellular response.

13 ntosaceus suppressed Ag-specific humoral and cellular responses.

14 ciated with these products that culminate in cellular responses.

15 dinated protein turnover to elicit immediate cellular responses.

16 as signaling organelles that ensure digital cellular responses.

17 r could increase both protective humoral and cellular responses.

18 ple ligands and receptors to elicit specific cellular responses.

19 rehydration requires appropriate mitigating cellular responses.

20 ns with FGFRs that control their pleiotropic cellular responses.

21 methylase 5A (KDM5A), mimics hypoxia-induced cellular responses.

22 cides with a vigorous recall of intrahepatic cellular responses.

23 conversion of external stimuli into specific cellular responses.

24 ting DNA methylation signals into downstream cellular responses.

25 e and play a key role in lipid signaling and cellular responses.

26 lement Smad signaling and thus contribute to cellular responses.

27 ling pathways must somehow engender specific cellular responses.

28 plant cells and their environment to control cellular responses.

29 Here, we review NOD1/2 stimuli and cellular responses.

30 activated Ca(2+) channels activates numerous cellular responses.

31 ntracellular targets lead to similarities in cellular responses.

32 as neurotransmitters, hormones, or drugs to cellular responses.

33 by lack of parvalbumin-specific antibody and cellular responses.

34 rganization and its modulation controls many cellular responses.

35 ge of downstream targets and elicit distinct cellular responses across cell and tissue types.

36 Some derivatives showed impressive cellular responses, allowing them to be tested as probes

37 To achieve a reproducible and specific cellular response, an increased mechanistic understandin

38 ich these changes occur are critical for the cellular response and fate decision.

39 ed expression of miRNAs may alter particular cellular responses and contribute, or lead, to the devel

40 These modulations alter host cellular responses and enable an optimal expression of v

41 networks, highlight their role in mediating cellular responses and explore their modifiability throu

42 enhance the vaccine Ag-specific humoral and cellular responses and induce homing to the intestinal a

43 logical and sensory stimuli into appropriate cellular responses and mediate the actions of one-third

44 ramework for characterizing multiple dynamic cellular responses and their coordination.

45 acterized oral HSV-1 shedding rates and host cellular response, and genotyped viral strains, in monoz

46 NThe breadth of the antibodies, the Th1-type cellular response, and killing mechanisms elicited by BP

47 elicits promising cross-subtype humoral and cellular responses, and opens up potentially simplified

48 another can be used to predict time-varying cellular responses, and provide insight on the design of

49 Gravity-sensitive cellular responses are regularly observed in both specia

50 ATF is known to be a central mediator of the cellular responses as it promotes cell proliferation and

51 Finally, we observe high cellular responses, as characterized by IFN-gamma produc

52 The p53 cellular response at single-cell resolution in the intes

53 ifaceted and complex, involving antibody and cellular responses at both systemic and mucosal levels.

54 Vaccine-specific cellular responses at days 0, 42, and 180 were assessed

55 n, thus documenting the conservation of this cellular response between insects and mammals.

56 NR not only facilitates a stimulus-specific cellular response but also might impede other NR pathway

57 omponents of S. aureus elicit TLR2-dependent cellular responses, but the exact signaling pathways act

58 by measuring Cyp c 1-specific antibodies and cellular responses by ELISA, basophil activation, spleno

59 chanical stimulation that is transduced into cellular responses by mechanotransduction mechanisms.

60 lethora of extracellular signals to specific cellular responses by using only a few second messengers

61 proteasome substrate processing and induce a cellular response characteristic of proteasome inhibitio

62 pendence and rapid adaptation of NaCl-evoked cellular responses closely resembled behavioral and affe

63 PD-1 therapy is sufficient to induce unique cellular responses compared with either monotherapy.

64 vel evidence that HD vaccine elicits greater cellular responses compared with the SD vaccine in SOT r

65 cts in interleukin-1beta (IL-1beta)-mediated cellular responses contribute to Alzheimer's disease (AD

66 ter primary challenge, enhanced antibody and cellular responses contributed to rapid clearance after

67 lie nonlinear dynamic changes in the gain of cellular response, critical for top-down control of cort

68 any cell signalling pathways that can induce cellular responses distinct from that of G proteins(2).

69 induced dose-dependent gB- and pp65-specific cellular responses, dominated by pp65-specific CD8 T cel

70 fore reveal how a chromatin modifier governs cellular responses during infection.

71 n act as a negative regulator of humoral and cellular responses during toxoplasmosis.

72 How such perturbations affect cellular response dynamics remains poorly understood.

73 enabling a more reliable characterization of cellular response dynamics.

74 r distinct niches within the LN that promote cellular responses, emphasizing the critical link betwee

75 inities are finely tuned and encode specific cellular responses, enabling multiplexed cellular functi

76 cer treatment, may increase the diversity of cellular responses, enhancing resistance to treatment.

77 tional regulation in various silicon-related cellular responses for the marine diatom Thalassiosira p

78 d signatures with the query indicate related cellular responses frequently governed by connected mech

79 In the inertial regime, we follow cellular response from (visco-)elastic through plastic d

80 Flow cytometry showed a resolving innate cellular response from 2 to 14 weeks in persistently IGR

81 scovery perspective, coupling this data with cellular responses helps to inform understanding of how

82 ess that involves inflammatory mediators and cellular responses; however, if any disturbances are pre

83 In this study, we investigated Pi-mediated cellular response in HEK293 and HeLa cells.

84 c multicellular organisms coordinate hypoxic cellular responses in a spatiotemporal manner.

85 We place cellular responses in a time- and tissue-dependent conte

86 The vv + MDV also triggers a series of cellular responses in both types of chickens.

87 In doing so, they permit cellular responses in diverse situations including where

88 tial obstacle is the myriad of unanticipated cellular responses in heterogeneous real-world environme

89 xerted antidepressant-related behavioral and cellular responses in mice, the ED(50) of (2R,6R)-HNK to

90 GI.1-specific cellular responses in peripheral blood were observed 9 d

91 are a promising analytical tool to quantify cellular responses in physiologically relevant extracell

92 evel of inflammatory monocyte and neutrophil cellular responses in the rectal route group.

93 er able to maintain durable antigen-specific cellular responses in the skin than mice immunized by th

94 le of vitreous plays in angiogenesis-related cellular responses in vitro.

95 resulting in a failure to mediate wild-type cellular responses including cell cycle arrest, senescen

96 Calcium signals drive an endless array of cellular responses including secretion, contraction, tra

97 itecture can be used to program a variety of cellular responses, including graded and proportional du

98 are bioactive, giving rise to differentiated cellular responses inducing stromal cell proliferation a

99 Underlying cellular responses is a transcriptional regulatory netwo

100 en mechanosensation, mechanotransduction and cellular responses is unclear.

101 In addition, we review the downstream cellular responses, many of which are shared by various

102 ing on the surface receptors stimulated, the cellular response may be either biphasic or monotonous.

103 ting both neutralizing antibodies (nAbs) and cellular responses may be superior.

104 In addition to identifying novel cellular responses mediated by PAR4, these data provide

105 e severity would correlate with enhanced Th2 cellular responses.Methods: Nasal aspirates were collect

106 ll tolerated and induced robust antibody and cellular responses, notably against both homologous and

107 of activated T cells, which was mirrored in cellular responses observed at 96 hr in isolated mucosal

108 ficient to account for the difference in the cellular response of the same gene in different cell typ

109 Here, we investigated long-term cellular responses of human GBM cells to ionizing radiat

110 rovide valuable models for understanding the cellular responses of human tissues to SARS-CoV-2 infect

111 ielectrode array recordings to determine the cellular responses of RGCs exposed to this mild degree o

112 sed by leukocytes and activate or suppress a cellular response once engaged with an antibody-coated t

113 d to act as coincidence detectors, promoting cellular responses only when convergent regulatory signa

114 s a central role in activation of a range of cellular responses over broad spatial and temporal bandw

115 he analysed mRNAs, thereby leaving dormant a cellular response pathway to these man-made environmenta

116 is maintained by an evolutionarily conserved cellular response pathway, the DNA damage response (DDR)

117 and introduces new possibilities in rewiring cellular response pathways.

118 lostats and that these networks predetermine cellular response prior to exposure to a stressor or dru

119 n after the fourth, with higher antibody and cellular response rates at month 18 than at month 12: Ig

120 helial cells transduce mechanical signals to cellular responses remain poorly understood.

121 onsequence of more complex multimolecular or cellular responses, remains unclear.

122 eptors and its potential to regulate diverse cellular responses.SIGNIFICANCE STATEMENT The proper for

123 whole-genome analyses to an understanding of cellular responses specific to tissue, developmental sta

124 Obesity is associated with the activation of cellular responses, such as endoplasmic reticulum (ER) s

125 C Ags become activated to elicit a myriad of cellular responses, such as target cell killing and the

126 ced more-robust initial binding antibody and cellular responses than MVA-BN-Filo dose 1 vaccination.

127 t mitophagy is a complex and multi-factorial cellular response that depends on tissue, energetic, str

128 e spinal cord transection owing to a complex cellular response that includes axon regrowth and is acc

129 These data indicate a complex cellular response that may alter retina structure and BR

130 compounds did in fact bind uPAR, and elicit cellular responses that affected invasion, migration, an

131 However, the interplay of different cellular responses that are activated following viral in

132 Genomic instability can trigger cellular responses that include checkpoint activation, s

133 nvironments are sufficient to elicit adverse cellular responses that may be ameliorated by targeting

134 s to characterize the coordinated multilevel cellular responses that occur when mitochondrial lipid b

135 he kidney tubular cells undergo a cascade of cellular responses that result in the production and acc

136 orm synaptic neurotransmitter signals into a cellular response through the binding and activation of

137 The cellular response to a single CAP treatment followed by

138 HLH-30, play an important role in mediating cellular response to a variety of stress conditions, inc

139 However, the molecular mechanisms of the cellular response to acid are not fully understood.

140 study, we investigated the mitochondrial and cellular response to alcohol in hepatoma cell line VL-17

141 Transcriptional shutoff impedes the cellular response to alphavirus replication and prevents

142 PGCC formation might represent a fundamental cellular response to Aurora kinase inhibitors and contri

143 tream transcription factor regulators of the cellular response to benzene.

144 As mithramycin affects cellular response to bile acid treatment by altering the

145 odulate transduction mechanisms that amplify cellular response to bioactive compounds.

146 FACT thus shapes the intrinsic cellular response to Cas9-based genome manipulation most

147 peutic agents elicit distinct effects on the cellular response to CDK4/6 inhibition.

148 ion of proteasome catalytic subunits and the cellular response to Cfz.

149 The Gene Ontology terms cellular response to chemical stimulus, response to exte

150 undation for understanding the mechanisms of cellular response to cisplatin.

151 suppressor proteins at an early stage of the cellular response to conflicts between DNA transcription

152 nsing transcription factor Mac1p governs the cellular response to Cu starvation by controlling Cu imp

153 r versus extracellular) and differs from the cellular response to cytokine treatment.

154 n levels have been shown to be predictive of cellular response to cytotoxic treatments.

155 A-mediated proteasome activation is a common cellular response to diverse endocrine stimuli and rapid

156 uppressor p53 is a critical regulator of the cellular response to DNA damage and is tightly regulated

157 mechanism by which tumor viruses reshape the cellular response to DNA damage by manipulating RNF168-d

158 Coordination of the cellular response to DNA damage is organised by multi-do

159 The RecA protein orchestrates the cellular response to DNA damage via its multiple roles i

160 Stress response, cellular response to DNA damage, iron ion homeostasis, u

161 es (DNMTs) are thought to be involved in the cellular response to DNA damage, thus linking DNA repair

162 he DNA damage response (DDR) encompasses the cellular response to DNA double-stranded breaks (DSBs),

163 ls, suggesting that proteins involved in the cellular response to DNA replication stress could be pot

164 The ATR kinase is a master regulator of the cellular response to DNA replication stress.

165 component analysis (PCA) is used to quantify cellular response to drug as a function of time.

166 DNA-PKcs kinase activity is required for the cellular response to DSBs immediately after their induct

167 C1) is a key metabolic hub that controls the cellular response to environmental cues by exerting its

168 gand specificity and thereby the accuracy of cellular response to environmental cues.

169 It is unclear whether the cellular response to external electrical stimuli is infl

170 ides a rich source of information on dynamic cellular response to external perturbations.

171 gulation of membrane receptor mobility tunes cellular response to external signals, such as in bindin

172 ed most down-regulated genes associated with cellular response to external stimuli, cell migration, a

173 To better understand the cellular response to FGF1 in the MBH, we sequenced >79,0

174 ed by inherited or acquired factors, impairs cellular response to genotoxic and replicative stress an

175 et medical need begin with understanding the cellular response to HHV-6 at the individual and populat

176 actor-alpha (HIF-alpha), which regulates the cellular response to hypoxia.

177 Thus, RIPK1 plays an important role in the cellular response to low energy levels and mediates AMPK

178 However, a cellular response to low oxygen levels has not been demo

179 antly, mitophagy after DNA damage is a vital cellular response to maintain mitochondrial functions an

180 1 and miR-221 upregulation is specific to RV cellular response to mechanical and hormonal stimuli ass

181 rate at the level of myosin II to govern the cellular response to mechanical inputs.

182 molecules within mechanotransduction on the cellular response to mechanical stimulation.

183 m of translation reprogramming regulates the cellular response to metabolic stress.

184 tor of INterferon Genes) mediates protective cellular response to microbial infection and tissue dama

185 erized NTHi infection model, we analyzed the cellular response to NTHi infection in the Junbo mouse m

186 differentially regulated during the earliest cellular response to oncogenic RAS(G12V) expression.

187 tion that regulates protein synthesis during cellular response to oxidative stress.

188 both reduced TORC1 signaling and altered the cellular response to P starvation.

189 e demonstrate that ProCas9 can orchestrate a cellular response to pathogen-associated protease activi

190 ic cells in vivo in order to determine their cellular response to physiological O(2) gradients as wel

191 nal platform enables integrative analyses of cellular response to radiation with drug responses and g

192 he complex molecular arrangements underlying cellular response to radiation, which is critical for no

193 h biological processes known to underpin the cellular response to radiation.

194 However, the global cellular response to ribosome collisions has not been ex

195 regulators and pathways that orchestrate the cellular response to ssUVR.

196 her applied the NanoTPOT workflow to examine cellular response to stress caused by dithiothreitol in

197 inal kinases (JNKs) are activated during the cellular response to stress signals.

198 The UPR is the cellular response to stress which results in misfolded p

199 studies are associated to ubiquitination and cellular response to stress.

200 acid metabolism, cytoskeletal proteins, and cellular response to stress.

201 ng catalyst that accelerates the rate of the cellular response to TCR stimulation, controlling the ti

202 file promoter and enhancer activities in the cellular response to TGF-beta of lung cancer cells and d

203 died were more likely to have not mounted a cellular response to the proteins.

204 and Wg, and by ecdysone, which regulates the cellular response to their signaling activities.

205 ycle progression, chromosomal stability, and cellular response to therapeutic ionizing radiation.

206 e potential explanation for the differential cellular response to these drugs is in the manner by whi

207 we address this by characterizing the early cellular response to transient T-R conflicts (TRe).

208 It is implicated in the cellular response to trauma/disease and considered to ha

209 compare outcomes (e.g., gene expression and cellular response to treatment) between individuals with

210 cently conducted a proteomic analysis of the cellular response to trivalent arsenic, a ubiquitous env

211 ndergo rapid, reversible condensation in the cellular response to ubiquitous environmental fluctuatio

212 e cycles of numerous viruses and also in the cellular response to viral infection.

213 tor of CK1alpha and has a role in modulating cellular response to Wnt signaling.

214 tion factor that plays a crucial role in the cellular response to xenobiotics.

215 ess and autophagy plays an important role in cellular responses to a wide variety of physiological an

216 We also found that the earliest cellular responses to acute S1 removal include recruitme

217 Furthermore, the cellular responses to angiotensin II and the relative ab

218 cles to replication forks, shedding light on cellular responses to anti-cancer therapies.

219 to reveal cell dynamics changes encompassing cellular responses to bioactive stimuli and optically in

220 sses, such as development, reproduction, and cellular responses to biotic and abiotic stimuli.

221 abolomic analyses were performed to evaluate cellular responses to both As(V) and As(III) stress.

222 tood how different receptors elicit distinct cellular responses to cause cell proliferation, differen

223 ile, large-scale gene expression profiles of cellular responses to chemical compounds have also recen

224 ies, have enabled fundamental discoveries of cellular responses to chemical perturbations that are of

225 ol with translational potential for studying cellular responses to Chlamydia and other sexually trans

226 To study the human cellular responses to chlamydial infection, researchers

227 aled that in vitro SLC16A5-silencing altered cellular responses to cisplatin treatment, supporting a

228 of transgenic mice to measure mesoscale and cellular responses to coherent motion.

229 ork with each other" to generate appropriate cellular responses to cues and signals.

230 ine in macrophage polarization by modulating cellular responses to cytokines such as IFN-gamma and IL

231 Characterizing cellular responses to different extrinsic signals is an

232 protein activation and is crucial for normal cellular responses to diverse extracellular signals.

233 some of the detected isoforms may influence cellular responses to drugs and represent new targets fo

234 Altering cellular responses to DSBs may rebalance editing outcome

235 nd proved very effective at detecting global cellular responses to either treatment, indicating that

236 cytoplasmic Ca(2+) changes that underlie the cellular responses to electrical activity.

237 ntegrates signaling pathways and coordinates cellular responses to environmental changes.

238 Cellular responses to environmental stress are frequentl

239 We investigated cellular responses to environmental stress at the single

240 ssary to understand the relationship between cellular responses to environmental stresses, disease pr

241 to their capability to better mimic in vivo cellular responses to external stimuli, providing excell

242 RK and MAPK/JNK pathways, crucial for normal cellular responses to extracellular stimuli, have recent

243 The brain is a genomic mosaic shaped by cellular responses to genome damage.

244 The main cellular responses to hypoxia are mediated mainly by two

245 ependent NDRG1 regulation might explain some cellular responses to hypoxia.

246 racterized by an impaired regulation of late cellular responses to IFN-I.

247 G and albumin recycling also participates in cellular responses to IgG-containing ICs.

248 surface and are loss of function and DN for cellular responses to IL-6, IL-11, LIF, and OSM.

249 communication and provides new insight into cellular responses to impaired plastid protein biosynthe

250 ere have been huge advances in understanding cellular responses to ionising radiation (IR) and DNA da

251 ell surface display of EGFR is essential for cellular responses to its ligands.

252 are prominent among mechanisms that mediate cellular responses to limited oxygen but also are induce

253 Acute phase responses, cellular responses to lipopolysaccharide, neutrophil res

254 ranscription factors that play a key role in cellular responses to low-O(2) tension.

255 These findings reveal the complexity of cellular responses to MDM2 and MDMX and suggest that MDM

256 d by mechanosensitive (MS) proteins mediates cellular responses to mechanical stimuli and osmotic str

257 aveolae to Hippo signaling in the context of cellular responses to mechanical stimuli and suggest act

258 known drug resistance pathways to reprogram cellular responses to MEKi.

259 We therefore analyzed cellular responses to membrane, nucleocapsid, and spike

260 y linked processes to determine the earliest cellular responses to misfolded proteins.

261 on of metabolic/energetic genes, programming cellular responses to nutrient and environmental adaptat

262 hiol-redox status, metabolic adaptation, and cellular responses to oxidative stress.

263 g of 16 cell lines and parallel recording of cellular responses to perturbations.

264 Cellular responses to purified protein derivative stimul

265 otein acetylation, an important regulator of cellular responses to radiation damage.

266 The major themes emerging were cellular responses to stress and signaling mechanisms li

267 To identify novel cellular responses to stress, we performed transcription

268 ow key protein-protein interactions regulate cellular responses to stress.

269 Cellular responses to structural and nonstructural viral

270 Many cellular responses to surrounding cues require temporall

271 er cell models, EIS was then used to monitor cellular responses to the DNMT and HDAC inhibitors 5-Aza

272 ed by signaling pathways that coordinate the cellular responses to the new environmental settings.

273 equired for cell expansion and implicated in cellular responses to the phytohormone auxin.

274 c signals; however, mechanisms that modulate cellular responses to these factors remain unclear.

275 signals from the tissue microenvironment and cellular responses to these physical cues, such as stiff

276 Here we further investigated the humoral and cellular responses to Tp0126 during experimental and nat

277 es of brain injury and neurodegeneration and cellular responses to treatment.

278 s to pseudoviruses TV1c8.2 and MW925.26, and cellular responses to vaccine-matched antigens (envelope

279 Finally, we demonstrated that these cellular responses toward baculovirus infection affect t

280 idual coagulation proteases induces specific cellular responses unrelated to their anticoagulant effe

281 tions and tracked the dynamics of individual cellular responses upon exposure to different concentrat

282 ocesses for explaining apparently stochastic cellular response variability and indicate that even wea

283 and-off electric fields (EFs) and monitoring cellular responses via AC electrical impedance spectrosc

284 als across the membrane, inducing downstream cellular responses via G proteins or beta-arrestin.

285 Understanding cellular responses via signal transduction is a core foc

286 Cellular responses were assessed using an ex-vivo IFN-ga

287 Cellular responses were assessed using an ex-vivo interf

288 Cellular responses were characterised by cytokine produc

289 Cellular responses were characterized by cytokine produc

290 At week 60, vaccine-induced humoral and cellular responses were detected in 51 (77%) of 66 parti

291 Humoral and cellular responses were evaluated using protein chip mic

292 Humoral and cellular responses were measured during and after immuno

293 strongest correlations between OP assays and cellular responses were observed with the antioxidant (a

294 The results show a strong apoptotic cellular response, whereby mechanical stimulation causes

295 The UPR is a highly orchestrated and complex cellular response, which is mediated through the ER chap

296 ast, inactivation of AdeB leads to a focused cellular response, which is not sensitive to the activit

297 c brain injury (TBI) results in a cascade of cellular responses, which produce neuroinflammation, par

298 Thus, ANAC017 is a master regulator of cellular responses with mitochondria acting as central s

299 e effects, depending on both the agonist and cellular response, with the biggest reductions seen in P

300 rotein-coupling profiles govern GPCR-induced cellular responses, yet receptor sequence selectivity de