ライフサイエンスコーパス: central action

1 -induced insulin resistance is driven by its central actions.

2 derstanding the mechanism of its pleiotropic central actions.

3 However, extensive efforts to evaluate the central actions and therapeutic efficacy of IN-OT may be

4 However, in an in vivo test of central action (i.e., stimulus generalization with rats

5 NGF also has central actions in the brain, where it regulates the phe

6 s also a brain neuropeptide that has diverse central actions, including inhibition of food and water

7 In addition to its central actions, leptin exerts biological effects by act

8 ed remission of hyperglycemia induced by the central action of FGF1 involves both preservation of bet

9 nnectivity and its establishment through the central action of Lim-HD governed programs.

10 effect may, in fact, be mediated by a direct central action of nAChR antagonists.

11 Inhibition of cyclooxygenases, the central action of nonsteroidal antiinflammatory drugs (b

12 reinstate cocaine-seeking behavior through a central action of the stress-associated neurohormone cor

13 Although a central action of these agents involving direct perturba

14 the primary mediator of clinically important central actions of alpha2AR agonists and suggest that th

15 n of AT(1) antagonists can effectively block central actions of Ang II, regulating blood pressure and

16 Recent work suggests that the central actions of angiotensin II via the circumventricu

17 To prevent the central actions of angiotensin II, we created anterovent

18 brain c-fos expression may be related to the central actions of angiotensin II.

19 utyric acid (GABA) is a major target for the central actions of cocaine and lidocaine, which can resu

20 Central actions of estrogen (E2) include, among others,

21 t implications with respect to the potential central actions of FGF21.

22 It is suggested that determination of the central actions of glucocorticoids in mediating fast-fee

23 Blocking central actions of histidine and denervating peripheral

24 Despite discoveries of central actions of insulin, in vivo molecular mechanisms

25 The central actions of insulin, on galanin (GAL) and neurope

26 a provide a foundation for understanding the central actions of kinins and their putative role in med

27 The central actions of leptin are essential for homeostatic

28 us (DMN) may serve as a relay center for the central actions of leptin on thyrotropin-releasing hormo

29 jor contributor, at least in part due to the central actions of leptin.

30 Among the central actions of nicotine on body weight, much attenti

31 The central actions of NPY and CRF have opposing functions i

32 y used to distinguish between peripheral and central actions of opiates.

33 uggest that a balance between peripheral and central actions of PDE5A inhibitors on bone formation to

34 In vivo, central actions of polyphosphate at the level of the bra

35 Central actions of proinflammatory cytokines, in particu

36 CRH might act as one of the mediators of the central actions of the melanocortin system.

37 e in any species that GAL can antagonize the central actions of VP.

38 s have suggested that glucocorticoids have a central action on cerebral metabolism.

39 levels of plasma fatty acids, negating their central action on hepatic glucose fluxes through (i) inh

40 ctions of nicotine, which precede its direct central actions, serve as a conditioned interoceptive cu

41 Nonetheless, these central actions should be taken into account alongside p

42 vated by leptin and mediate part of leptin's central actions to influence energy balance.

43 nylethylamine itself, is without significant central action when administered at moderate doses, but

44 CoA generation and attempts to integrate its central actions with its peripheral antilipotoxic action