ライフサイエンスコーパス: central administration

1 c-Fos expression in GnRH neurons only after central administration.

2 ing hormone (LH) release after peripheral or central administration in Kiss1- or Gpr54 (Kiss1r)-null

3 g hormone (GnRH) release after peripheral or central administration in mice.

4 ervation of an acute orexigenic effect after central administration in mice.

5 hypothalamic signaling molecules, but their central administration induces different effects on hepa

6 sive rats, high-resolution echocardiography, central administration (intracerebroventricular infusion

7 ciception with reduced adverse effects after central administration (intrathecal or intracerebroventr

8 vity of relatively short duration whereas on central administration it had only a KOR-antagonist acti

9 In addition, chronic central administration of 1,25D3 dramatically decreased

10 Central administration of 1-10 microg of peptide elicits

11 Central administration of 14 did not produce locomotor e

12 Central administration of 17beta-estradiol (E2) decrease

13 o extend the dissimilar efficacy profiles of central administration of a CRF agonist, r/h CRF(1-41),

14 We tested this theory by central administration of a gapmer antisense oligonucleo

15 Continuous central administration of a neutralizing antibody to the

16 ic leptin overexpression was induced through central administration of adeno-associated virus-encodin

17 restingly, NPY-/- mice are hypersensitive to central administration of AgRP(83-132), yet exhibit a no

18 Finally, central administration of alphaCGRP to adult hybrid mice

19 Following central administration of an ASO to rodents, we observe

20 sion in the mediobasal hypothalamus and that central administration of an NPY Y(1) receptor antagonis

21 Central administration of analogue 2 in rats increased f

22 resonance imaging (MRI) it was observed that central administration of Ang-(1-7) following transient

23 rain and peripheral cholinergic systems, and central administration of anticholinergic drugs in depen

24 (1) or delta opioid antagonists, and through central administration of antisense oligodeoxynucleotide

25 Central administration of bicuculline (a GABA(A) recepto

26 MJN110 (which selectively elevates 2-AG) and central administration of both MJN110 and the CB1 antago

27 The central administration of C75, a potent inhibitor of the

28 iogenic effects in male C57BL/6J mice, while central administration of CART reduced c-Fos in 5-HT(DRN

29 Central administration of CGRP activates the hypothalamo

30 Test the hypothesis that central administration of CMT-3 would inhibit microglial

31 Central administration of corticotropin-releasing hormon

32 However, the effect of central administration of CRF in these rat strains has y

33 Central administration of CRH results in endocrinologica

34 In contrast, central administration of DHT, 3beta-diol, and the ERbet

35 Central administration of DPDPE (delta-selective agonist

36 neral opioid antagonist, naltrexone, through central administration of either general, mu, mu(1), kap

37 Here, we show that the central administration of estradiol rapidly increases th

38 After central administration of excess Ang II, the reduction o

39 ormones compared with glucose ingestion, and central administration of fructose provokes feeding in r

40 food deprivation are reduced by systemic and central administration of general opioid antagonists.

41 We previously found that systemic and central administration of GHSR antagonists reduced binge

42 Central administration of GLP-1 augmented glucose-stimul

43 Central administration of GLP-1 increases plasma cortico

44 Central administration of glucagon like peptide-1 (GLP-1

45 Central administration of glucagon-like peptide-1 (GLP-1

46 nts indicate that the anorexigenic effect of central administration of glucose was blunted by coinjec

47 Similar results were observed following central administration of GM-CSF in mice.

48 We demonstrate that central administration of gp120 (4 microg) significantly

49 Similar to insulin, central administration of IGF-1 can suppress hepatic glu

50 Central administration of IL-1 (1 or 2 ng/mouse) depress

51 Central administration of IL-1beta (10 ng) also signific

52 Central administration of IL-1beta suppresses natural ki

53 Finally, both peripheral and central administration of IL-1beta, itself, induced sick

54 In addition, in conscious rats, central administration of IMD results in increased plasm

55 Systemic or central administration of leptin alone did not alter GP,

56 Central administration of leptin to peripherally leptin-

57 Central administration of leucine increases hypothalamic

58 Chronic central administration of MCH and adenoviral vectors inc

59 agonist, inhibited food intake stimulated by central administration of MCH, reduced consumption of pa

60 e been observed in antinociception following central administration of morphine into either the later

61 Central administration of MTII for 4 days (10 nmol/day)

62 ression in the brain was analyzed, following central administration of N6-cyclohexyladenosine.

63 In vivo, central administration of nesfatin-1 reduces motivation

64 Central administration of neuropeptide Y (NPY) potently

65 Central administration of NPS increases locomotor activi

66 Central administration of NPY (10 microg) also enhanced

67 d the metabolic and anorectic effects of the central administration of oleic acid.

68 In normal rats, central administration of orexin or exposure to certain

69 In rodent studies, central administration of orexin peptides increases food

70 Central administration of orexin-A acutely suppressed ca

71 level dependent (BOLD) fMRI to test whether central administration of oxytocin 45-60 min before fMRI

72 Beginning in 1979 with the first report that central administration of oxytocin stimulates maternal b

73 se and alcohol-seeking, we demonstrated that central administration of peptide antagonists for relaxi

74 hey also have a severely blunted response to central administration of peptide YY (PYY).

75 d (b) to further characterize the effects of central administration of QRFP(26) on energy balance in

76 Recognizing that central administration of QRFP26 and QRFP43 increases hi

77 Central administration of recombinant CTRP13 suppressed

78 Central administration of recombinant protein suppressed

79 Finally, central administration of ROS alone increased c-fos and

80 We showed that central administration of senktide (NK3R agonist) induce

81 related cardiovascular responses elicited by central administration of the cannabinoid receptor (CB(1

82 esponse to solicitous males in females after central administration of the D1-like agonist SKF38393 a

83 Central administration of the endozepine octadecaneurope

84 The central administration of the fatty acid synthase (FAS)

85 Central administration of the long-chain fatty acid olei

86 ing schedule attenuated rats' sensitivity to central administration of the melanocortin agonist MTII.

87 Central administration of the melanocortin receptor agon

88 th this compound in MC4-R knockout mice, and central administration of this compound had no effect on

89 earlier vasopressor initiation, the need for central administration of vasopressors has been question

90 Central administration of WIN 55,212-2 significantly inc

91 knockout (KO) induces alcohol drinking, and central administrations of CRF reduce alcohol intake.

92 Central-administration of oxytocin antagonist attenuated

93 iabetic rodent models through peripheral and central administration routes.