ライフサイエンスコーパス: central amygdala

1 modulated BOLD responding to cat fur in the central amygdala.

2 the bed nucleus of the stria terminalis and central amygdala.

3 d increased oxytocin receptor binding in the central amygdala.

4 ons in the basal ganglia and basolateral and central amygdala.

5 tractus solitarius, parabrachial nucleus and central amygdala.

6 y of a specific population of neurons in the central amygdala.

7 olateral amygdala or lateral division of the central amygdala.

8 of data paralleled by cFos expression in the central amygdala.

9 bed nucleus of the stria terminalis and the central amygdala.

10 basal amygdala and the medial portion of the central amygdala.

11 ethanol-stimulated GABA release in the mouse central amygdala.

12 eive disproportionally higher input from the central amygdala.

13 cohol-induced ERK-phosphorylation in the rat central amygdala, a region involved in regulation of alc

14 wed an increase in Hcrtr2 mRNA levels in the central amygdala, a stress-related brain region, of LgA

15 revealed by chemogenetic manipulation of the central amygdala, a stress-sensitive nucleus that forms

16 Chronic stress suppressed central amygdala activity and phenocopied the effects of

17 ling has common roles in the hippocampus and central amygdala, affecting synaptic function, systemic

18 alpha5, beta1, beta3, and delta, and in the central amygdala alpha1, alpha2, beta3, and gamma2 were

19 aventricular nucleus of the hypothalamus and central amygdala, although no effect of the drug on 2-DG

20 cortex (Fctx), dorsal hippocampus (DHPC) and central amygdala (Amyg).

21 stic effects on the expression of CRH in the central amygdala and antagonizes GR-mediated reduction i

22 GAD67 mRNA was increased in the hippocampus, central amygdala and dorsomedial hypothalamus in pups te

23 on of the VTA provoked afterdischarge in the central amygdala and enhanced kindling rate.

24 in hyperkatifeia-associated regions (ie, the central amygdala and infralimbic cortex) during acute wi

25 not only failed to elevate FLI expression in central amygdala and insular cortex, but also failed to

26 This pattern was seen in central amygdala and insular cortex, but not in basolate

27 nce c-Fos-like immunoreactivity (FLI) in the central amygdala and insular cortex.

28 not induce Fos or iNOS in stress-responsive central amygdala and paraventricular hypothalamic neuron

29 in several other limbic sites, including the central amygdala and paraventricular nuclei of the hypot

30 Through non-overlapping projections to the central amygdala and pre-Botzinger complex, these two su

31 its direct output to the medial part of the central amygdala and the hypothalamic 'aggression area',

32 teral and ventromedial hypothalamus), in the central amygdala and various mid- and hindbrain structur

33 subthalamic nucleus, lateral division of the central amygdala, and Barrington's nucleus (Bar).

34 ers of the limbic system (e.g., hippocampus, central amygdala, and both medial and lateral habenula),

35 the well-described action of oxytocin in the central amygdala, and demonstrates that self-defense sup

36 ssion in bed nucleus of stria terminalis and central amygdala, and dopamine, CRF, kappa/dynorphin, an

37 al pons, raphe nuclei, lateral hypothalamus, central amygdala, and insular cortex.

38 n subregions of the neocortex, caudoputamen, central amygdala, and parasubthalamic nucleus while it d

39 lated gamma-aminobutyric acid release in the central amygdala, and reduced ethanol consumption when a

40 ventricular nucleus of the hypothalamus, the central amygdala, and the bed nucleus of the stria termi

41 cingulate cortex, the ventral striatum, the central amygdala, and the bed nucleus of the stria termi

42 nd ERC; moderate strength connections in the central amygdala; and weak connections in the cingulate

43 ections from the parabrachial nucleus to the central amygdala are implicated in pain transmission.

44 ections to the ventromedial hypothalamus and central amygdala are necessary for these behaviours.

45 glutamatergic connection from basolateral to central amygdala (BLA-CeA) develops rapidly during the f

46 Central amygdala (but not prefrontal cortex) infusion of

47 ouble dissociation, AM251 infusions into the central amygdala, but MJN110 infusions into the basolate

48 ion of nicotine-cue-activated Fos neurons in central amygdala, but not orbitofrontal cortex, decrease

49 c-fos expression was also induced in the central amygdala by i.c.v. CRF, unlike the vehicle-injec

50 significantly greater in the BNST, PAG, and central amygdala (Ce) following the microinjection of AV

51 ing Fos immunocytochemistry suggest that the central amygdala (Ce) might be a component of this neura

52 ico-limbic loop between insular cortex (IC), central amygdala (CE), and nucleus basalis of Meynert (N

53 The central amygdala (Ce), particularly its medial sector (C

54 ory inputs to DRN 5-HT neurons projecting to central amygdala (Ce).

55 BAergic neurons activated by GA in the mouse central amygdala (CeA(GA) neurons).

56 general-anesthesia-activated neurons in the central amygdala (CeA(GA)) that can potently suppress pa

57 rk stressed the differing involvement of the central amygdala (CeA) and bed nucleus of the stria term

58 ossibility by studying the roles of the BLA->central amygdala (CeA) and BLA->nucleus accumbens (Acb)

59 In contrast, the central amygdala (CeA) and its rostral extension receive

60 one, alone, increased FLI in VTA, NAC shell, central amygdala (ceA) and laterodorsal bed nucleus of t

61 ed dendritic spine density (DSD) in both the central amygdala (CeA) and medial amygdala (MeA) but not

62 ropeptide Y (NPY) were innately lower in the central amygdala (CeA) and medial amygdala (MeA), but no

63 ithin the insular cortex (IC), targeting the central amygdala (CeA) and nucleus accumbens (NAc), resp

64 xy-5-methyl-4-isooxazole receptor (AMPAR) in central amygdala (CeA) and phosphorylation of AMPAR GluA

65 The pathway between central amygdala (CeA) and ventrolateral periaqueductal

66 asing factor (CRF)-expressing neurons in the central amygdala (CeA) antagonize the extinction memory

67 orticotropin-releasing hormone gene (Crh) in central amygdala (CeA) are implicated in threat regulati

68 cent dextran amines identifies the X. laevis central amygdala (CeA) as a target for ascending auditor

69 The central amygdala (CeA) displays elevated GABA release fo

70 ections to the basolateral amygdala (BLA) or central amygdala (CeA) during safety recall.

71 ency of chloroquine-activated neurons in the central amygdala (CeA) for both itch sensation and assoc

72 o the bed nucleus stria terminalis (BNST) or central amygdala (CEA) generates an aversive memory.

73 The central amygdala (CeA) has a key role in learning and ex

74 otropin releasing factor (CRF) system in the central amygdala (CeA) has been implicated in the effect

75 otropin releasing factor (CRF) system in the central amygdala (CeA) has been implicated in the effect

76 fear conditioning.SIGNIFICANCE STATEMENT The central amygdala (CeA) has been implicated in the establ

77 The central amygdala (CeA) has emerged as an important brain

78 The central amygdala (CeA) is a bilateral hub of pain and em

79 The central amygdala (CeA) is a critical brain region as dys

80 The central amygdala (CeA) is a crucial hub in the processin

81 The central amygdala (CeA) is a hub for stress and AUD, whil

82 gulating mood, and OTergic signalling in the central amygdala (CeA) is a key mechanism that controls

83 The central amygdala (CeA) is a key structure at the limbic-

84 The central amygdala (CeA) is an important neuronal hub that

85 The central amygdala (CeA) is critically involved in a range

86 D1-cerebellin 1 (Cbln1) signaling within the central amygdala (CeA) is crucial for neuroplasticity an

87 The central amygdala (CeA) is implicated in a range of menta

88 The central amygdala (CeA) is important for fear responses t

89 The central amygdala (CeA) is known to mediate a panoply of

90 on of the tachykinin 2 (Tac2) pathway in the central amygdala (CeA) is necessary and sufficient for t

91 The central amygdala (CeA) is positioned as a key integrator

92 We hypothesized that CRF-CRFR1 signaling in central amygdala (CeA) mediates stress-induced hyperalge

93 ovided new means of mapping functionality in central amygdala (CeA) neuron populations based on their

94 eral periaqueductal gray [vlPAG])-projecting central amygdala (CeA) neurons using whole-cell electrop

95 tudies show that the GABAergic system in the central amygdala (CeA) nucleus has a complex role in the

96 The central amygdala (CeA) nucleus, a subcortical structure

97 or conditionally overexpress (OE) CRF in the central amygdala (CeA) of adult mice.

98 cts of local mifepristone infusions into the central amygdala (CeA) on yohimbine-induced reinstatemen

99 (NTS), and activation of cell bodies in the central amygdala (CeA) or axons in the vBNST, PBN, and N

100 The central amygdala (CeA) orchestrates adaptive responses t

101 The central amygdala (CeA) orchestrates defensive behaviors

102 rticolimbic structures, resulting in reduced central amygdala (CEA) output.

103 mission in the parabrachial nucleus (PBN) to central amygdala (CeA) pathway has been reported in rode

104 Activity of central amygdala (CeA) PKCdelta expressing neurons has b

105 The central amygdala (CeA) plays a central role in physiolog

106 xcessive drinking.SIGNIFICANCE STATEMENT The central amygdala (CeA) plays a critical role in the deve

107 The central amygdala (CeA) plays a crucial role in defensive

108 The central amygdala (CeA) plays a role in the relationship

109 vious work using a rat model showed that the central amygdala (CeA) plays an important role in avoida

110 received ibotenic acid lesions in the MeA or central amygdala (CeA) prior to cat-odor exposure.

111 croglia in the basolateral amygdala (BLA) or central amygdala (CeA) produces elevated anxiety.

112 tested 1 d later, whereas silencing the VTA-central amygdala (CeA) projection had no effect.

113 We found that ADRB1(+) neurons in the central amygdala (CeA) sent projections to the LC, and s

114 Pharmacological inactivation of the central amygdala (CeA) severely impaired acquisition and

115 ealed that cFos expression in neurons of the central amygdala (CeA) showed the greatest difference be

116 tion potential (AP) firing in neurons of the central amygdala (CeA) that project to the dlBnST, incre

117 Prolonged exposure of the central amygdala (CeA) to elevated corticosteroids (CORT

118 e brain [i.e., neurons that project from the central amygdala (CeA) to the lateral hypothalamus (LH)]

119 Here, we found that GABAergic neurons in the central amygdala (CeA) were highly activated during seve

120 , are controlled by neuronal circuits in the central amygdala (CeA)(2); however, the source of neuron

121 ledge of the circuit organization within the central amygdala (CeA), a critical regulator of emotiona

122 The central amygdala (CeA), a key brain region for pain, thr

123 ts of stress- and fear-relevant genes in the central amygdala (CeA), a locus that regulates stress-me

124 nses, GABAergic synaptic transmission in the central amygdala (CeA), and circulating cytokine levels

125 messenger RNA expression was measured in the central amygdala (CeA), and DYN-expressing CeA neurons w

126 (NAc) shell and core, basolateral (BLA) and central amygdala (CeA), and medial prefrontal cortex (mP

127 itazone in the ventral tegmental area (VTA), central amygdala (CeA), and nucleus accumbens (NAc) shel

128 bed nucleus of the stria terminalis (BNST), central amygdala (CeA), and posterior basolateral amygda

129 stems are important stress mechanisms in the central amygdala (CeA), bed nucleus of stria terminalis

130 A dose of 5 mg DF/kg activated the central amygdala (CeA), bed nucleus of the stria termina

131 xpression are highly colocalized in both the central amygdala (CeA), composed of mostly GABAergic neu

132 In the central amygdala (CeA), ethanol acts via corticotrophin-

133 omedial amygdala (CeM), a subdivision of the central amygdala (CeA), is believed to be the main outpu

134 prague Dawley rats were cannulated targeting central amygdala (CeA), medial amygdala (MeA), or basola

135 d nucleus of the stria terminalis (BNST) and central amygdala (CEA), naltrexone increased FLI in ad l

136 The central amygdala (CeA), once viewed as a passive relay b

137 y in the basolateral amygdala (BLA), but not central amygdala (CeA), reversed deactivation of mPFC py

138 Here we studied the role of central amygdala (CeA), ventral medial prefrontal cortex

139 BLA neurons project to the central amygdala (CeA), which also participates in negat

140 alimbic prefrontal cortex (ilPFC) to inhibit central amygdala (CeA)-mediated Pavlovian reactions.

141 have focused on neuroadaptations within the central amygdala (CeA).

142 taste processing, gustatory cortex (GC) and central amygdala (CeA).

143 tic inhibition in fear output neurons of the central amygdala (CEA).

144 nal groups of rats were given lesions in the central amygdala (CeA).

145 sular (CeC) and lateral (CeL) nucleus of the central amygdala (CeA).

146 expressed significantly more c-Fos IR in the central amygdala (CeA).

147 n was decreased in the nucleus accumbens and central amygdala (CeA).

148 ain signals to the brain regions such as the central amygdala (CeA).

149 in stress system dysfunction, including the central amygdala (CeA).

150 s in altering excitatory transmission in the central amygdala (CeA).

151 lateral parabrachial nucleus (LPB), and the central amygdala (CeA).

152 n areas that regulate anxiety, including the central amygdala (CEA).

153 s (IGF1Rs) in the hippocampus (Hippo-DKO) or central amygdala (CeA-DKO) by stereotaxic delivery of AA

154 rtex - but not to the lateral nucleus of the central amygdala (CEAl) - were found in the DsRed-positi

155 A population of neurons in the lateral central amygdala (CeAL) expressing corticotropin releasi

156 indicate that the lateral subdivision of the central amygdala (CeL) is essential for fear learning.

157 ar processing in the lateral division of the central amygdala (CeL), a structure that orchestrates fe

158 s, located in the lateral subdivision of the central amygdala (CEl), which express protein kinase C-d

159 ronal excitability in rat lateral nucleus of central amygdala (CeL).

160 astrocytes in the medial subdivision of the central amygdala (CeM) determine the synaptic and behavi

161 V(1a) receptors in the medial nucleus of the central amygdala (CeM) increases neuronal excitability,

162 eptor 1 (CRFR1) in the medial nucleus of the central amygdala (CeM) is known to increase anxiety-like

163 uronal excitability in the medial nucleus of central amygdala (CeM) V(1a) receptor activation excites

164 neurons inhibit output neurons in the medial central amygdala (CEm), and also make reciprocal inhibit

165 he activity of the medial subdivision of the central amygdala (CeM), the canonical amygdala output to

166 dial amygdala and the medial division of the central amygdala (CeM), with little AT(2)R-eGFP expressi

167 septum (LS), lateral amygdala (LatAmyg), and central amygdala (CenAmyg) than less social, naive LD fe

168 ikely form differential connections with the central amygdala (controlling freezing), this process wo

169 emory whereas insulin/IGF-1 signaling in the central amygdala controls thermogenesis via regulation o

170 In the central amygdala, CRF mRNA levels were increased in both

171 In slices of the central amygdala, DCUK-OEt acted primarily on extrasynap

172 ion of neurotensin-expressing neurons in the central amygdala decreases intake of and preference for

173 contrast, no sex difference was seen in the central amygdala-dependent acquisition or expression of

174 s induction, DREADD-mediated inhibition of a central amygdala-dependent behavior, and DREADD-mediated

175 However, inactivation of the central amygdala disrupted only the expression, but not

176 efflux in the basolateral amygdala, whereas central amygdala efflux remained unchanged.

177 In central amygdala, elevated Tacr1 expression was accompan

178 ndent increases in the responsiveness of the central amygdala ERK pathway to cocaine cues.

179 After 1 d of withdrawal, stimulation of central amygdala ERK phosphorylation increased cocaine s

180 The pain-recipient neurons in the central amygdala expressing CGRP receptors are also crit

181 activation of ventromedial ITCs that inhibit central amygdala fear output.

182 phenotype and combined MR up-regulation and central amygdala GR deficiency.

183 xacerbated both acute freezing responses and central amygdala hyperexcitability via cannabinoid recep

184 cortex, bed nucleus of the stria terminalis, central amygdala, hypothalamic paraventricular nucleus (

185 nd feeding-induced tuning of anxiety via the central amygdala in rats.

186 role of PSTN Tac1 neurons projecting to the central amygdala in the hindrance of feeding initiation.

187 e found an up-regulation of CART mRNA in the central amygdala induced by acute but not chronic stress

188 Mice receiving acute intra-central amygdala injections of the selective AT(2)R agon

189 ay be related to the increase in the NTS and central amygdala inputs leading to inhibition of DMN neu

190 projections from anterior insular cortex to central amygdala is critical to relapse after the cessat

191 rons onto midbrain-projecting neurons in the central amygdala is necessary for context-dependent retr

192 nase-Cdelta (PKCdelta)-expressing neurons in central amygdala lateral division (CeL).

193 ia nigra pars reticulata, entorhinal cortex, central amygdala, lateral amygdala, arcuate nucleus, and

194 accumbens, bed nucleus of stria terminalis, central amygdala, lateral hypothalamus, and ventral hipp

195 the medial preoptic area, nucleus accumbens, central amygdala, lateral septum, and cortex.

196 Intra-central amygdala m-CLD had no effect on WS-evoked behavi

197 The presence of the hormone oxytocin in the central amygdala makes a mother rat willing to put her l

198 females were treated with intra-LC or intra-central amygdala mannosylated liposomes containing clodr

199 Our results identify dissociable central amygdala mechanisms of abstinence-dependent expr

200 os, and double-labeled expression in CeL and central amygdala medial division (CeM).

201 Minor projections also go to central amygdala, mediodorsal thalamus, dorsal raphe, an

202 riven GABAergic synaptic activity within the central amygdala microcircuit of HF rats.

203 itional effects to decrease accumbens GR and central amygdala MR expression.

204 tion increased the number of basolateral and central amygdala neurons activated during sucrose and ox

205 ap at the level of defined subpopulations of central amygdala neurons and demonstrate that persistent

206 neurons inhibited a subpopulation of medial central amygdala neurons and shunted excitation from the

207 Here we show that PKC-delta-expressing central amygdala neurons are essential for the synaptic

208 use, we identify a specific subpopulation of central amygdala neurons expressing protein kinase C del

209 Central amygdala neurons expressing protein kinase C-del

210 distinct behavioral modules and suggest that central amygdala neurons instruct predatory hunting acro

211 Here, we categorize central amygdala neurons that express secretagogin (Scgn

212 In rat central amygdala neurons, deletion of Tlr4 altered GABAA

213 n failed to change in CA1 pyramidal neurons, central amygdala neurons, pyramidal neurons of layer II/

214 rain regions, including the parabrachial and central amygdala neurons; and sympathetic efferent neuro

215 These data show a role for GABA release from central amygdala neurotensin neurons in modulating consu

216 we show that knockdown of GABA release from central amygdala neurotensin neurons using a Nts-cre-dep

217 found that the most robust projection of the central amygdala neurotensin neurons was to the parabrac

218 nal tracers in the basolateral, lateral, and central amygdala nuclei of adult macaques and used brigh

219 Japanese and moor macaques in the basal and central amygdala nuclei when controlling for neuron dens

220 ergic system in neurons of slices of the rat central amygdala nucleus (CeA), a brain region thought t

221 neurons were highest in the BSTLcn, lateral central amygdala nucleus (CeLcn), and medial central amy

222 central amygdala nucleus (CeLcn), and medial central amygdala nucleus (CeM) (74%, 73%, and 85%, respe

223 eus of the hypothalamus, supraoptic nucleus, central amygdala, nucleus tractus solitarius and area po

224 eased neuropeptide Y (NPY) expression in the central amygdala of alcohol-preferring rats, causing hig

225 positive reinforcement effects of OT in the central amygdala of mice and rats.

226 optogenetic and chemogenetic stimulation of central amygdala of mice elicited predatory-like attacks

227 tor binding was significantly greater in the central amygdala of socially experienced hamsters than i

228 Conversely, excitation of either the central amygdala or parasympathetic vagal neurons activa

229 t CeM AT(2)R-expressing neurons can modulate central amygdala outputs that play a role in fear expres

230 geminal, C1/A1 group, ventrolateral medulla, central amygdala, parabrachial nucleus, cuneate nucleus,

231 follicular compared with midcycle timing in central amygdala, paraventricular and ventromedial hypot

232 However, how the central amygdala participates in such a learning process

233 in the nuclei of neurons within hippocampus, central amygdala, piriform cortex, and striatum, brain r

234 ala (BLA) paraventricular thalamus (PVT) and central amygdala PKCdelta-expressing cells (CeA(PKCdelta

235 dministration and that neuronal ensembles in central amygdala play a critical role in this incubation

236 of prepronociceptin-expressing cells in the central amygdala (Pnoc(CeA)) that are activated by palat

237 We identified the anterior insular cortex-to-central amygdala projection as a new addiction- and moti

238 ning lethal bites on prey, was mediated by a central amygdala projection to the reticular formation i

239 Blocking GABAergic signaling in the central amygdala re-established normal anxiety levels in

240 Given that neurons in the central amygdala responding to DCUK-OEt were recently id

241 FKBP51 in the basolateral amygdala (BLA) or central amygdala resulted in increased anxiety-related b

242 but not vasopressin, administration into the central amygdala reversed the social incompetence of the

243 he mPFC of KO rats and increased activity of central amygdala somatostatin-positive neurons, putative

244 ypothalamus into areas within the medial and central amygdala, terminating at the medial border of th

245 d in H/S pups, in which fewer neurons in the central amygdala, the bed nucleus of the stria terminali

246 ts that could trigger hunting, including the central amygdala, the lateral hypothalamus, and the zona

247 Acting within the central amygdala, the neuropeptide oxytocin regulates em

248 leus of the stria terminalis, the medial and central amygdala, the periaqueductal gray, the dorsal ra

249 TA activation and neural excitability in the central amygdala, the present results are consistent wit

250 In the central amygdala, the release of OT modulates inhibitory

251 These data suggest that this central amygdala to parabrachial nucleus projection infl

252 Conversely, the projection from the central amygdala to the dorsomedial striatum mediates ha

253 at the TRH analog, RX 77368, acts within the central amygdala to vagally stimulate gastric contractil

254 f this gut-brain vagal circuit modulated the central amygdala transcriptome in both sexes but selecti

255 solateral amygdala neurons projecting to the central amygdala, uncovering an amygdala circuit that me

256 synapses from the lateral subdivision of the central amygdala via A2A receptor activation.

257 ion, an indirect pathway with a relay in the central amygdala was also observed that is similar in it

258 e, we show the infralimbic projection to the central amygdala was necessary for suppressing fear cue-

259 Y1 receptor ir was evident in the central amygdala, whereas both Y1- and Y5-immunoreactive

260 ocalized within the parabrachial nucleus and central amygdala, which constitute part of the 'emergenc