ライフサイエンスコーパス: central scotoma

1 or system adjust to a loss of foveal vision (central scotoma)?

2 ned of loss of vision in the left eye with a central scotoma.

3 ch with fixation sparing and supero-temporal central scotoma.

4 us of fixation, with marked reduction of the central scotoma.

5 ound between angle alpha and the presence of central scotoma.

6 ces in subjects with and without a simulated central scotoma.

7 gulation because this would cause a blinding central scotoma.

8 ld man presented with a persistent bilateral central scotoma.

9 ion loss in both eyes, often associated with central scotomas.

10 ATION: A 34-year-old man had sudden onset of central scotoma and photopsia in the left eye.

11 pha and angle beta (+c) with the presence of central scotoma and visual field defect parameters, resp

12 cts were individuals with CN, VI adults with central scotomas and children with CVI.

13 had equal to or more than a doubling of the central scotoma area in response to a II2e test stimulus

14 ow that a new foveated ideal observer with a central scotoma correctly predicts that the human optima

15 with no visual impairment confronted with a central scotoma develop a preferred retinal locus to rep

16 -verified HFMD, a 31-year old male noticed a central scotoma, distorted lines and loss of visual acui

17 severity of Stargardt disease, likelihood of central scotoma expansion, and visual acuity deteriorati

18 Persons with central scotomas frequently use a preferred retinal locu

19 olled in this study: 30 patients affected by central scotoma, group 1, and 30 affected by peripheral

20 He complained about acute onset of central scotoma in his left eye.

21 ht volunteers (80%) successfully simulated a central scotoma in the first field and all 10 (100%) did

22 There was a central scotoma in the perimetry of the right eye and pe

23 ency department with headaches and new onset central scotoma in the right eye.

24 /63, had macular schisis with small relative central scotomas in each eye.

25 egions of the visual cortex corresponding to central scotomas in subjects with macular degeneration (

26 As a naturally occurring central scotoma, it has a large cortical representation,

27 total loss, superior peripheral defect, and central scotoma (listed in order of decreasing statistic

28 Symptoms included central scotoma, metamorphopsia, and mild to moderate vi

29 nts who showed a doubling in the size of the central scotoma over this duration.

30 al acuity and contrast sensitivity loss, the central scotoma per se delayed hazard detection even tho

31 pic B-wave amplitudes) a higher mean rate of central scotoma progression compared with those patients

32 al visual field data, 8 patients with faster central scotoma progression rates had significantly wors

33 t was found that some patients with relative central scotomas reliably used two different preferred r

34 Snellen visual acuity was 20/200, and median central scotoma size was 20.0 degrees.

35 tion; from full kinetic fields with relative central scotomas to remnant peripheral islands; from red

36 A central scotoma was noted on the Amsler chart and a loss

37 A possible laser-induced central scotoma was suspected in only 1 patient who had

38 uals presented with declining visual acuity, central scotomas, waxy disc pallor, attenuated vasculatu

39 th established macular disease and bilateral central scotomas were recruited.

40 All eyes showed large central scotomas, which were associated with visual fiel

41 Microperimetry revealed a reduction in the central scotoma with three patients recovering normal fo

42 Seven of 9 patients reported the central scotoma within the first week after surgery.