ライフサイエンスコーパス: central vision

1 many positions throughout a wide portion of central vision.

2 tients with diabetic retinopathy and loss of central vision.

3 ng to photoreceptor degeneration and loss of central vision.

4 ittle attention has been paid to deficits in central vision.

5 specialization between the mouse and primate central vision.

6 can lead to permanent loss of peripheral or central vision.

7 his left eye that progressed to include his central vision.

8 y packed with cones and mediates high-acuity central vision.

9 experiment 1, participants viewed stimuli in central vision.

10 n of disorders causing a progressive loss of central vision.

11 tients (61.5%) demonstrated deterioration of central vision.

12 y in a foveal-sparing pattern with preserved central vision.

13 e light/dark ratios of visual performance in central vision.

14 the part of the retina responsible for human central vision.

15 moreover, optical defocus can cause blur in central vision.

16 closely associated with the deterioration of central vision.

17 retinal pigment epithelium leads to loss of central vision.

18 reported neither for near peripheral nor for central vision.

19 slowly over time, eventually causing loss of central vision.

20 representation of letter feature position in central vision.

21 tive disorder associated with severe loss in central vision.

22 on, common only in normal peripheral but not central vision.

23 omplication of diabetes resulting in loss of central vision.

24 of family C who experienced markedly reduced central vision.

25 eneration (AMD), which causes severe loss of central vision.

26 a small region of the retina responsible for central vision.

27 the impact of the scotomata on the patient's central vision.

28 clinical stages are accompanied by impaired central vision.

29 lium (RPE) and the retina leading to loss of central vision.

30 ily rods and secondarily cones, that mediate central vision.

31 The wet form leads to severe loss of central vision.

32 hotophobia, loss of color vision and reduced central vision.

33 ium is associated with a progressive loss of central vision.

34 ive to vertical - disparities that occurs in central vision.

35 scularization, which leads to severe loss of central vision.

36 Here, we study how loss of central vision affects lateral feedback to spared areas

37 There was no change in central vision after 6 months of lutein supplementation,

38 There was no change in central vision after 6 months of lutein supplementation.

39 AMD results in loss of central vision and a dependence on low-resolution periph

40 s besides retinal degeneration are affecting central vision and causing this increased risk.

41 importance of investigating emotions beyond central vision and demonstrate commonalities and differe

42 Patients with MAK-RP maintain functional central vision and have a favorable prognosis.

43 trophy due to AMD, the PRIMA system restored central vision and led to a significant improvement in v

44 cular dystrophy leads to progressive loss of central vision and shows symptoms similar to age-related

45 milies included progressive deterioration of central vision and subsequently night vision, mild photo

46 patients could simultaneously use prosthetic central vision and their remaining peripheral vision in

47 ched in the macula, which is responsible for central vision and visual acuity.

48 lly treatable nAMD patients had already lost central vision and were legally blind.

49 was typical CORD with photophobia, decreased central vision, and dyschromatopsia.

50 thy (LHON) is a syndrome of subacute loss of central vision associated with mutations in mitochondria

51 Such large fields and inclusion of central vision at nearly all sites precluded retinotopic

52 Both maps represent central vision at the posterior end of the border betwee

53 dystrophy and is characterized by decreased central vision, atrophy of the macula and underlying ret

54 tion, and this likely reflects the fact that central vision benefits most from the increased visual a

55 nd 20 patients with RP who had retained good central vision (better than 20/32).

56 In central vision, both schemes are capable of producing co

57 Humans exploit the high resolution of central vision by actively moving their eyes three to fo

58 Epiretinal membrane (ERM) can impair central vision by forming a pre-retinal fibrous layer on

59 , progressing to severe disease with loss of central vision by the third decade in affected males.

60 ionnaire measuring 7 unidimesnional domains: central vision, color vision, contrast sensitivity, scot

61 performed between; Cone-Function Anxiety and Central Vision controlling for better eye visual acuity,

62 Cone-Function Anxiety correlated with Central Vision controlling for visual acuity, Rod-Functi

63 It often presents unilaterally with central vision disruption, and typically resolves with v

64 and 5 males) with a primary presentation of central vision disturbance.

65 when this information is present outside of central vision, emotion perception studies typically foc

66 lateral, the severe and irreversible loss of central vision experienced by affected persons has been

67 Severe loss of central vision frequently occurs with the exudative (wet

68 Given the prominence of central vision in humans, it has been assumed that visua

69 escence, side vision in young adulthood, and central vision in later life because of progressive loss

70 or (VEGF)-neutralising proteins can preserve central vision in many patients with neovascular age-rel

71 rdinal orientations in the representation of central vision in owl monkey V1 was relatively small and

72 Because of the prominence of central vision in primates, it has generally been assume

73 reasing visual field loss, with concerns for central vision increasing, whereas those for outdoor mob

74 We demonstrate that the loss of central vision induces functional mobilization of motion

75 locus in normally sighted individuals whose central vision is blocked by an artificial scotoma.

76 is well preserved, or late disease, in which central vision is lost.

77 serves as the preferred fixation locus when central vision is lost.

78 On the other hand, unrestricted central vision is not sufficient to ensure normal refrac

79 (AMD), a blinding disorder that compromises central vision, is characterized by the accumulation of

80 study has found that artificial occlusion of central vision leads to rapid emergence, and long-term m

81 hange visual sensory processing, introducing central vision loss (a scotoma).

82 -6.88; P < 0.001), greater odds of bilateral central vision loss (aOR, 3.43; 95% CI, 1.49-8.99; P = 0

83 reduced baseline BCVA and longer duration of central vision loss (P < 0.001).

84 wet, age-related macular degeneration causes central vision loss and represents a major health proble

85 Most patients with AMD-related central vision loss continue to drive, but demonstrate s

86 Our data suggested that age and mild central vision loss did not affect significantly a subje

87 -year-old White man presented with bilateral central vision loss due to foveal lesions consisting of

88 mally-sighted controls and participants with central vision loss due to macular degeneration (MD).

89 A 62-year-old male presented with bilateral central vision loss due to neovascular AMD.

90 's hereditary optic neuropathy (LHON) causes central vision loss from bilateral optic neuropathy.

91 In patients with central vision loss from ERM foveoschisis, vitrectomy wi

92 can be used to screen for moderate to severe central vision loss from glaucoma.

93 e-related macular degeneration, which causes central vision loss globally.

94 2-fold (MS4) increased instantaneous risk of central vision loss in CORD compared with rod-cone patie

95 nderstood about the prevalence and nature of central vision loss in early glaucoma.

96 As one of the leading causes of central vision loss in elderly population, worldwide cas

97 mpetent woman presented with sudden painless central vision loss in her left eye (best corrected visu

98 r degeneration (AMD) is the leading cause of central vision loss in older adults.

99 acular degeneration (AMD)-a leading cause of central vision loss in the elderly.

100 scope of visual cortex plasticity following central vision loss is essential both for clarifying the

101 ch humans adapt eye movements in response to central vision loss is still not well understood and car

102 These results suggest that central vision loss may give rise to cortical thinning,

103 ry cone photoreceptor dysfunction triggering central vision loss of these patients, we model the stim

104 Therefore we examined the impact of central vision loss on motion perception using random do

105 Here, we explore the possible effects of central vision loss on the optimal saccades during a fac

106 bservers (mean age, 73.8) with long-standing central vision loss practiced an oral sentence-reading t

107 utational model to predict where humans with central vision loss should direct their eye movements in

108 in most patients, although in 1 patient with central vision loss such change was absent.

109 Clinical observations of patients with central vision loss suggest a lengthy adjustment period,

110 Central vision loss was a common presenting symptom in C

111 e reading speed in people with long-standing central vision loss was evaluated.

112 Full-thickness macular hole can cause central vision loss, and outcomes depend on hole size an

113 and height of foveal detachment, duration of central vision loss, and RRD extent (P < 0.001).

114 underscore the need to recognize the role of central vision loss, and vision loss more broadly, in mo

115 ss adaptive gait kinematics in subjects with central vision loss, older controls, and younger control

116 the height of foveal detachment, duration of central vision loss, RRD extent, and development of ERM/

117 Age, but not mild central vision loss, significantly affected a subject's

118 Thus, a central vision loss-specific mobility questionnaire may

119 e photoreceptor degeneration, which leads to central vision loss.

120 nerve degeneration and progressive bilateral central vision loss.

121 ilize visual fixation in adult patients with central vision loss.

122 RD patients at markedly higher risk of early central vision loss.

123 f retinal break formation and to prevent the central vision loss.

124 -onset complex retinal disease that leads to central vision loss.

125 that results in progressive and irreversible central vision loss.

126 ble rehabilitation regimen for patients with central vision loss.

127 cell and photoreceptor degeneration leads to central vision loss.

128 2 (LCA2) characterized by the early onset of central vision loss.

129 enhancing visual performance for people with central vision loss.

130 up of disorders characterized by progressive central vision loss.

131 s more prominent in parts of MT representing central vision (< or =10 degrees).

132 All patients had myopia, reduced central vision, nystagmus, and electroretinographic evid

133 The effects on MP and central vision of 6 months of lutein supplementation at

134 ritical factor limiting object perception in central vision of individuals with neurodegeneration of

135 characterized by crowding and poor acuity in central vision of the affected eye.

136 Specifically, an emphasis on central vision over peripheral vision results in pupil c

137 e a variety of maculopathies that can reduce central vision permanently.

138 Central vision ranged from normal to reduced in the firs

139 The PRL was located outside the compromised central vision region, typically near the edge of the sc

140 low 24 degrees or 37 degrees of unrestricted central vision, respectively.

141 retained in such conditions, restoration of central vision should not jeopardize the surrounding hea

142 Central vision typically remains preserved at least unti

143 ld-to-severe hearing loss and generally good central vision until late adulthood.

144 ith the relative amount of cortex devoted to central vision varying by more than a factor of 2.

145 ERG a- and b-waves, central vision, vestibulomotor function, the spiking pro

146 ients who presented with unexplained loss of central vision, visual field defects, and/or photopsia w

147 ion selectivity, vestibulomotor function and central vision was compared between the D2 and B6 mouse

148 Central vision was unchanged after supplementation.

149 Central vision was unchanged after the period of supplem

150 Changes in MPOD and central vision were determined in a subset of patients r

151 foveated: we can see fine spatial details in central vision, whereas resolution is poor in our periph

152 AMD affects central vision which impairs one's ability to drive, rea

153 uration may enable functional restoration of central vision with acuity better than 20/100 for millio

154 To restore central vision without compromising the residual periphe