戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 paired balance, which may be associated with cerebellar dysfunction.
2 yeblink conditioning was used as a marker of cerebellar dysfunction.
3 e display behavioral defects consistent with cerebellar dysfunction.
4 cts had ataxia or other detected evidence of cerebellar dysfunction.
5 g of the behavioral deficits associated with cerebellar dysfunction.
6 type attributable to motor hyperactivity and cerebellar dysfunction.
7 ponses in the IN were suppressed, suggesting cerebellar dysfunction.
8 emor in limbs when immobile, consistent with cerebellar dysfunction.
9 , 9 of 16 positive control patients also had cerebellar dysfunction.
10 s, to the later appearance of vestibular and cerebellar dysfunction.
11 companied by an intention tremor, typical of cerebellar dysfunction.
12 bit deficits in motor learning indicative of cerebellar dysfunction.
13 ese difficulties may be attributable to mild cerebellar dysfunction.
14 d between cerebellar K1 and the intensity of cerebellar dysfunction.
15 ss, to more pronounced signs and symptoms of cerebellar dysfunction.
16 tative measurements are required to evaluate cerebellar dysfunction.
17 dents, atypical neurodevelopment, or both in cerebellar dysfunction.
18 dination and intention tremor, suggestive of cerebellar dysfunction.
19 o Purkinje cell degeneration and progressive cerebellar dysfunction.
20 ) is a rare disease that is characterized by cerebellar dysfunction.
21 de insights into disease processes involving cerebellar dysfunction.
22 = 16), followed by cranial nerve palsies and cerebellar dysfunction.
23 resent in Rett syndrome arise, in part, from cerebellar dysfunction.
24 sorders characterised by incoordination from cerebellar dysfunction.
25 errations, immunodeficiency, and progressive cerebellar dysfunction.
26  levels, completely 'normalizing' the fronto-cerebellar dysfunctions.
27 e from a combination of cerebellar and extra-cerebellar dysfunctions.
28 sorders characterized by clinically variable cerebellar dysfunction and accompanied by involvement of
29                The clinical symptoms include cerebellar dysfunction and associated signs from dysfunc
30 ly unidentified role for Bmal1 disruption in cerebellar dysfunction and autistic-like behaviors.
31 ta-III spectrin function appears to underpin cerebellar dysfunction and degeneration in both diseases
32 ercent], P=0.007), even after adjustment for cerebellar dysfunction and for stratification variables
33   Here, we investigated PS1-E280A-associated cerebellar dysfunction and found that it occurs early in
34 taxia type 1 presents with brief episodes of cerebellar dysfunction and persistent neuromyotonia and
35 disorder characterised by brief paroxysms of cerebellar dysfunction and persistent neuromyotonia.
36 anges in DISC1 and unc5H3 may be relevant to cerebellar dysfunction and schizophrenia respectively, i
37             Three patients had brainstem and cerebellar dysfunction, and one had dysphagia and motor
38 n, cognitive impairment, pyramidal tract and cerebellar dysfunction, and white matter loss in the cen
39            While ataxia and other aspects of cerebellar dysfunction are the core features of these di
40     Acquired forms are often associated with cerebellar dysfunction arising due to instability of the
41           Together, these findings establish cerebellar dysfunction as a pathogenic driver of neurode
42 ant potential implications for patients with cerebellar dysfunction as well as to motor control studi
43                                              Cerebellar dysfunction, as measured with the CCFS and SA
44 A1), a neurological disease characterized by cerebellar dysfunction, ataxic attacks, persistent myoky
45 mature pattern resembles that of people with cerebellar dysfunction, because we know that this adapta
46                              The presence of cerebellar dysfunction before treatment was associated w
47                                      Primary cerebellar dysfunction can produce social deficits, and
48 individuals who attended hospital for any of cerebellar dysfunction, encephalopathy, or peripheral ne
49 t of several gait parameters consistent with cerebellar dysfunction from 40- to 80-d-old mice.
50                                              Cerebellar dysfunction has been demonstrated in autism s
51            These findings suggest that early cerebellar dysfunction in EA2 results from the intrinsic
52  to gain insight into the pathophysiology of cerebellar dysfunction in EAE.
53 IF1C cause hereditary spastic paraplegia and cerebellar dysfunction in human patients.(5-8) In contra
54        Chronic cannabis exposure can lead to cerebellar dysfunction in humans, but the neurobiologica
55 y be important factors in the genesis of the cerebellar dysfunction in KSS.
56 evidence that a channelopathy contributes to cerebellar dysfunction in MS.
57                                              Cerebellar dysfunction in multiple sclerosis (MS) contri
58 ay a significant role in the pathogenesis of cerebellar dysfunction in PCA.
59                     Experimental evidence of cerebellar dysfunction in preterm subjects, however, is
60 light complex mechanisms converging onto the cerebellar dysfunction in the phenotypic model and provi
61 ic exercise ameliorates, at least partially, cerebellar dysfunction in the sHW rat, an excellent mode
62 ies have investigated the pathophysiology of cerebellar dysfunction in this neuroinflammatory disorde
63 .2 vs 1.7+/-1.7, P<0.001), indicating slower cerebellar dysfunction indexes for FRDA than for SCA.
64                                              Cerebellar dysfunction is strongly implicated in both co
65                                              Cerebellar dysfunction leads to motor, learning, emotion
66 arked psychomotor retardation, and prominent cerebellar dysfunction manifested by nystagmus, intentio
67 ry is vulnerable to Thap1 mutations and that cerebellar dysfunction may be a primary cause of tremor
68                              We propose that cerebellar dysfunction may induce deregulation of tonic
69                     The relationship between cerebellar dysfunction, motor symptoms, and neuronal los
70 res (MSA-P), 8 MSA patients with principally cerebellar dysfunction (MSA-C), and 6 sporadic olivopont
71 taxia score (SARA, a standardized measure of cerebellar dysfunction on clinical examination, scores r
72 nts (26%) reported episodes of brainstem and cerebellar dysfunction or persistent vertigo several mon
73 Posterior fossa syndrome is characterized by cerebellar dysfunction, oromotor/oculomotor apraxia, emo
74    Although, in EA2 the emphasis has been on cerebellar dysfunction, patients also exhibit episodic,
75 tion, and their absence leads to PC loss and cerebellar dysfunction similar to that observed in AT pa
76 a clinical syndrome dominated by progressive cerebellar dysfunction that might elude standard diagnos
77  Consistent with this behavioral evidence of cerebellar dysfunction, there is increased death of gran
78                                      Despite cerebellar dysfunction, they can improve motor performan
79 tion as the intermediary in the pathway from cerebellar dysfunction to psychopathology.
80 or neuropsychiatric symptoms that arise from cerebellar dysfunction underscores the importance of elu
81 c neuropathy, particularly, but not only, if cerebellar dysfunction, vestibular involvement and cough
82 rological mutants, detected initially due to cerebellar dysfunction, was identified as models for epi
83 gest a mechanism for tremor generation under cerebellar dysfunction, which may explain the origin of
84 ice expressing mutant beta-III spectrin have cerebellar dysfunction with altered mGluR1alpha localiza
85 k for translational efforts to connect early cerebellar dysfunction with the origins of neurodevelopm