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1 rect current stimulation (tDCS) of the right cerebellar hemisphere.
2 heta burst stimulation (cTBS) over the right cerebellar hemisphere.
3 eir climbing fiber projection to the lateral cerebellar hemisphere.
4 teral cerebral cortex with the contralateral cerebellar hemisphere.
5 mbing fibre responses were mapped across the cerebellar hemisphere.
6 is theory to include the non-dominant (left) cerebellar hemisphere.
7 ed to the mean perfusion of the full greater cerebellar hemisphere.
8 d cerebellar vermis, and mild atrophy of the cerebellar hemispheres.
9 of the left anterior and the right posterior cerebellar hemispheres.
10 arately for the whole brainstem, vermis, and cerebellar hemispheres.
11 tability, and localizing to the cerebral and cerebellar hemispheres.
12 erebellar vermis with apparent fusion of the cerebellar hemispheres.
13 right caudate, right thalamus, and bilateral cerebellar hemispheres.
14 te loss of vermis and variable hypoplasia of cerebellar hemispheres.
15 s SHH-subtype tumours are located within the cerebellar hemispheres.
16  possibly the supero-anterior aspects of the cerebellar hemispheres.
17 ncluding the medial pre-motor areas and both cerebellar hemispheres.
18 y lateralized to the right cerebral and left cerebellar hemispheres.
19 form nuclei, thalami, internal capsules, and cerebellar hemispheres.
20                       The comorbid group had cerebellar hemisphere (-1.3 SD) and vermian gray matter
21 taxia-telangiectasia had lower metabolism in cerebellar hemispheres (14%, P < 0.001), anterior vermis
22 (right premotor and parietal cortex and left cerebellar hemisphere); a different region (medial front
23 esonance imaging revealed a mass in the left cerebellar hemisphere and peduncle.
24 g the neurochemical profile of the pons, the cerebellar hemisphere and the vermis in patients with FR
25 ral misorganization of the Purkinje cells in cerebellar hemispheres and a compaction of this cell pop
26 d vermian hypoplasia and failure to fuse the cerebellar hemispheres and caudal midbrain, a phenotype
27 ontrast, the role of the lateral cerebellum (cerebellar hemispheres and dentate nuclei, DN) is less w
28 gional cerebral blood flow increases in both cerebellar hemispheres and in the left sensorimotor cort
29  in three patients, while lesions located in cerebellar hemispheres and pituitary gland in just one p
30 ilateral precentral gyrus as well as to both cerebellar hemispheres and the ipsilateral brain stem.
31 rous metastatic lesions in both cerebral and cerebellar hemispheres and the meninges.
32 ors compared the volumes of each lobe of the cerebellar hemispheres and vermis in children with ADHD
33       The levels of somatic expansion in the cerebellar hemispheres and vermis were, on average, 3.15
34 bly stable across all regions, except in the cerebellar hemispheres and vermis.
35 ccording to which the phylogenetically newer cerebellar hemispheres are involved in tasks requiring h
36 te MRTM2 pharmacokinetic analysis, using the cerebellar hemispheres as the reference region, led to t
37 hich spread to involve the bilateral lateral cerebellar hemispheres, as well as CBF increases in the
38 pe and expanded FGF14 alleles in post-mortem cerebellar hemispheres between patients and controls.
39 unctional connections with the contralateral cerebellar hemispheres, bilateral sensorimotor cortices,
40 nd, and in superior posterior regions of the cerebellar hemispheres bilaterally (Crus I and lobule VI
41 TS group demonstrated reduced volumes of the cerebellar hemispheres bilaterally that derived primaril
42 ight medial and posterior orbital gyrus, the cerebellar hemispheres bilaterally, the left nucleus acc
43 ified BP(ND), given as BP(ND-t) = C(ROI) - C(cerebellar hemispheres)/C(cerebellar hemispheres), with
44 ial structures and particularly of the whole cerebellar hemispheres can be used to distinguish patien
45 analyses found significant enrichment in the cerebellar hemisphere, cerebellum, and axonogenesis path
46 in IUGR foetuses were significantly lower in cerebellar hemispheres (CH) (1.239 vs. 1.280.5 x 10(-3)
47                                          The cerebellar hemispheres constitute a more plastic, state-
48 equence of the remarkable enlargement of the cerebellar hemispheres during the latest phase of verteb
49 oss of a Foxp1(+) subtype and the failure of cerebellar hemisphere formation.
50  transfer ratio, formalin-fixed cerebral and cerebellar hemispheres from 19 patients and six controls
51                 Later in development, mutant cerebellar hemispheres had severe foliation defects and
52              In contrast, the right inferior cerebellar hemisphere (HVIIB) exhibited this load effect
53  potential gliosis marker, was higher in the cerebellar hemispheres in FRDA relative to controls.
54 limb of the right internal capsule, and both cerebellar hemispheres in patients compared to controls.
55 osterior limbs of the internal capsules, and cerebellar hemispheres in patients compared to controls.
56  of the cerebellar vermis with fusion of the cerebellar hemispheres, in 8/10 individuals.
57 h a bias towards the left cerebral and right cerebellar hemispheres, in keeping with the left-cerebra
58  cerebellar cortex in the left and the right cerebellar hemisphere independently and found that simpl
59 and VTA predominantly originate in the right cerebellar hemisphere, interposed nucleus, and paraverma
60 oth tasks, bilateral regions of the superior cerebellar hemispheres (left superior HVIIA and right HV
61 which supports attempted GTR of cerebral and cerebellar hemisphere low-grade astrocytoma.
62 ranial fossa, characterized by fusion of the cerebellar hemispheres, medial cerebellar peduncles and
63 the putamen of all patient groups and in the cerebellar hemispheres of MSA-C and sOPCA but not MSA-P
64  have clear changes in the activation of the cerebellar hemisphere opposite the injured corticospinal
65 sociated with increased CYP2A6 expression in cerebellar hemispheres (p = 7.8 x 10(-4)).
66 ebellar map within the crus IIa folia of the cerebellar hemispheres reorganizes after deafferentation
67 ferior cerebellar artery distribution, right cerebellar hemisphere, right mesial temporal lobe, and r
68  hemisphere (THGr(Ce)) and the contralateral cerebellar hemisphere (THGr(Cb))-by its respective contr
69 or mRNA levels were determined using lateral cerebellar hemisphere tissue from both schizophrenia and
70 cells in Crus I and II of the posterior lobe cerebellar hemisphere to activation of peripheral affere
71  left frontal, anterior cingulate, and right cerebellar hemisphere to activity in Sylvian-insular cor
72                                          The cerebellar hemispheres were the major target of resultin
73 mbic system, whereas the occipital lobes and cerebellar hemispheres were unaffected.
74                        Median MD values from cerebellar hemispheres were used to discriminate patient
75 ecific, with overlap in certain areas of the cerebellar hemisphere, which showed marked atrophy in AD
76 polymicrogyria or heterotopia; (ii) atrophic cerebellar hemispheres with stunted folia, profound gran
77 ND-t) = C(ROI) - C(cerebellar hemispheres)/C(cerebellar hemispheres), with C being the averaged radio