ライフサイエンスコーパス: cerebral artery occlusion

1 rtery occlusion and 360 with isolated middle cerebral artery occlusion).

2 l carotid, basilar, and M1 segment of middle cerebral artery occlusions).

3 80 male rats underwent 90-min middle cerebral artery occlusion.

4 were subjected to ischemic stroke by middle cerebral artery occlusion.

5 weeks into the treatment by transient middle cerebral artery occlusion.

6 bra using a mouse model of reversible middle cerebral artery occlusion.

7 r bundles from degeneration following middle cerebral artery occlusion.

8 brains 6 h or 7 days after transient middle cerebral artery occlusion.

9 neonatal and adult rats by transient middle cerebral artery occlusion.

10 t cavity for 7 d, beginning 7 d after middle cerebral artery occlusion.

11 d into rat brains 6 h after transient middle cerebral artery occlusion.

12 nal infarcts such as those induced by middle cerebral artery occlusion.

13 arge peri-infarct cortex region after middle cerebral artery occlusion.

14 emic brain damage caused by permanent middle cerebral artery occlusion.

15 ously or intra-arterially after right middle cerebral artery occlusion.

16 ct cortex before and after unilateral middle cerebral artery occlusion.

17 ed ischemia by transient or permanent middle cerebral artery occlusion.

18 me 11 for collateral remodeling after middle cerebral artery occlusion.

19 larly starting 3 days after 30 min of middle cerebral artery occlusion.

20 me in aged rats following a transient middle cerebral artery occlusion.

21 e and oxidative stress in response to middle cerebral artery occlusion.

22 t rats were subjected to a 90-minutes middle cerebral artery occlusion.

23 to stroke rats from days 6 to 9 after middle cerebral artery occlusion.

24 when initiated both before and after middle cerebral artery occlusion.

25 nuated in CD36-null mice subjected to middle cerebral artery occlusion.

26 ype animals after a 90 min reversible middle cerebral artery occlusion.

27 lesion volume induced by a transient middle cerebral artery occlusion.

28 ring and subjected to transient (2 h) middle cerebral artery occlusion.

29 to affect brain damage from transient middle cerebral artery occlusion.

30 in the ischemic penumbra region after middle cerebral artery occlusion.

31 in vivo rat stroke model of transient middle cerebral artery occlusion.

32 We used the stroke model of temporary middle cerebral artery occlusion.

33 ic cortex of rats 7 days after distal middle cerebral artery occlusion.

34 pidly upregulate STC2 after transient middle cerebral artery occlusion.

35 Cerebral ischemia was induced by middle cerebral artery occlusion.

36 ischemia was induced by 30 minutes of middle cerebral artery occlusion.

37 in the brains of 2D2 mice 14 d after middle cerebral artery occlusion.

38 tes AhR activation in the brain after middle cerebral artery occlusion.

39 nctional recovery following permanent middle cerebral artery occlusion.

40 ittermates received sham or transient middle cerebral artery occlusion.

41 sults CT scans from 100 patients with middle cerebral artery occlusion (44 women [mean age +/- standa

42 85% reduction of infarct volume after middle cerebral artery occlusion; 54% rescue of low skeletal mu

43 inflammatory cytokine profile in the middle cerebral artery occlusion-affected right brain hemispher

44 H have improved outcomes after 60-min middle cerebral artery occlusion and 24-h reperfusion.

45 We used rodent models of middle cerebral artery occlusion and cell-culture models of neu

46 ) of Niaspan, starting 24 hours after middle cerebral artery occlusion and daily for 14 days.

47 ere subjected to 60 min of reversible middle cerebral artery occlusion and evaluated for infarct volu

48 Imaging revealed left middle cerebral artery occlusion and left transverse and sigmoi

49 Adipor gene expression in mice after middle cerebral artery occlusion and lipopolysaccharide injecti

50 n status, is increased following both middle cerebral artery occlusion and oxygen-glucose deprivation

51 e model of focal cerebral ischemia by middle cerebral artery occlusion and reperfusion (I/R) in male

52 In vivo, transient middle cerebral artery occlusion and reperfusion in kinase-dead

53 ion in brain infarct size after acute middle cerebral artery occlusion and reperfusion, strongly supp

54 icient Rag1(-/-) mice after 60 min of middle cerebral artery occlusion and reperfusion.

55 l or energy-restricted diets prior to middle cerebral artery occlusion and reperfusion.

56 ficit, and infarct size at 24 h after middle cerebral artery occlusion and reperfusion.

57 ts were subjected to right hemisphere middle-cerebral artery occlusion and reperfusion.

58 were subjected to 90 min of transient middle cerebral artery occlusion and treated with either a cont

59 ed significantly in rats subjected to middle cerebral artery occlusion and treated with no intravenou

60 Adult male rats were subjected to middle cerebral artery occlusion and were treated with or witho

61 ed in mice (by permanent or transient middle cerebral artery occlusion) and rats (by 3-vessel occlusi

62 trophic lateral sclerosis (SOD1G93A), middle cerebral artery occlusion, and multiple mini-strokes.

63 after (late post-ischemia) 60 min of middle cerebral artery occlusion, and were killed 10 days after

64 ns, while applying a remote transient middle cerebral artery occlusion as a model for ischemic stroke

65 ng the EcoHIV infection model and the middle cerebral artery occlusion as the ischemic stroke model i

66 patients who had ischaemic stroke and major cerebral artery occlusion beyond 3 h of symptom onset.

67 In rats with middle cerebral artery occlusion, both the recovery of motor sk

68 its and poststroke inflammation after middle cerebral artery occlusion by preventing microglia polari

69 were subjected to 2 hrs of transient middle cerebral artery occlusion by the intraluminal occlusion

70 .5%) rats were subjected to permanent middle cerebral artery occlusion by the intraluminal occlusion

71 Following transient middle cerebral artery occlusion, ck2beta(-/-) mice displayed s

72 e scores at those times, and proximal middle cerebral artery occlusion demonstrated prior to treatmen

73 calculated in patients with proximal middle cerebral artery occlusion (derivation cohort) with known

74 of either sex subjected to transient middle cerebral artery occlusion developed dramatically smaller

75 either sex challenged with transient middle cerebral artery occlusion developed significantly smalle

76 male mice underwent unilateral distal middle cerebral artery occlusion (dMCA) occlusion and were imag

77 , 5 and 7 days after permanent distal middle cerebral artery occlusion (dMCAO) in mice compared to ve

78 Ischaemia was induced by distal middle cerebral artery occlusion (dMCAO) in normoxic (30% inhal

79 e rats received a 90-min right distal middle cerebral artery occlusion (dMCAo).

80 initiated at 48 h after mouse distal middle cerebral artery occlusion (dMCAO).

81 volume of mice subjected to transient middle cerebral artery occlusion even up to 3 to 5 hours after

82 rious microvascular settings, such as middle cerebral artery occlusion, femoral artery clipping, and

83 and wild-type (WT) mice to 1 hour of middle cerebral artery occlusion followed by 23 hours of reperf

84 ivo focal ischemic model, 2 h of left middle cerebral artery occlusion followed by 24 h of reperfusio

85 ate controls were subjected to 1 hour middle cerebral artery occlusion followed by 28-day reperfusion

86 and hyperlipidemic mice to transient middle cerebral artery occlusion, followed by measurement of st

87 Male Swiss Webster mice underwent middle cerebral artery occlusion for 1 h followed by reperfusio

88 We performed right middle cerebral artery occlusion for 3 hours, administered reco

89 Mice were subjected to middle cerebral artery occlusion for 40 min, followed by reperf

90 ebral ischemia was induced in mice by middle cerebral artery occlusion for 60 minutes and s-NSCs were

91 othelin receptors following permanent middle cerebral artery occlusion for 7 days.

92 Sprague-Dawley rats were subjected to middle cerebral artery occlusion for 70 min followed by reperfu

93 bral ischemia was produced in rats by middle cerebral artery occlusion for 90 min.

94 Animals were subjected to transient middle cerebral artery occlusion for 90 mins.

95 A 60 minute middle cerebral artery occlusion in C57 mice resulted in over 5

96 We induced permanent middle cerebral artery occlusion in female mice that previously

97 ne the role of AhR in stroke, we used middle cerebral artery occlusion in mice and oxygen-glucose dep

98 the severity of stroke in a model of middle cerebral artery occlusion in mice.

99 spontaneous functional recovery after middle cerebral artery occlusion in mice.

100 y, and cerebral edema formation after middle cerebral artery occlusion in mice.

101 owing acute focal ischemia induced by middle cerebral artery occlusion in normotensive rats.

102 apsulated clodronate before transient middle cerebral artery occlusion in postnatal day 7 rats.

103 nvestigated the effects of VEGF after middle cerebral artery occlusion in rats using a series of beha

104 mbra when administered six hours post middle cerebral artery occlusion in rats.

105 e ET(B) receptors following permanent middle cerebral artery occlusion in rats.

106 e, and clinical outcome in a model of middle cerebral artery occlusion in rats.

107 he ischemic brain following transient middle cerebral artery occlusion in rats.

108 and until completion of 15 min distal middle cerebral artery occlusion in spontaneously hypertensive

109 Middle cerebral artery occlusion in the rat brain resulted in a

110 After transient middle cerebral artery occlusion in the rat brain, elevated upt

111 al ischaemia was induced by transient middle cerebral artery occlusion in the rat using the filament

112 ilized two model systems of ischemia, middle cerebral artery occlusion in vivo and oxygen-glucose dep

113 umbilical cord blood cells 48 h after middle cerebral artery occlusion increased Akt phosphorylation

114 ologically induced excitotoxicity and middle cerebral artery occlusion-induced brain damage.

115 ainst NMDA-induced excitotoxicity and middle cerebral artery occlusion-induced stroke in mice.

116 We found that after transient middle cerebral artery occlusion, inhibiting PlexinD1 signaling

117 Infarct volume following middle cerebral artery occlusion is dramatically diminished in

118 ggression; (3) larger infarcts in the middle cerebral artery occlusion ischemic stroke model; and (4)

119 erebral ischemia induced by transient middle cerebral artery occlusion it selectively dilates arterio

120 in the ischemic brain after transient middle cerebral artery occlusion leading to increased intracran

121 ld-type mice were subjected to 1 h of middle cerebral artery occlusion (MCAO) and 24-72 h of reperfus

122 nhibitor, in a rat model of transient middle cerebral artery occlusion (MCAO) and an in vitro model o

123 s consist of rats subjected to 2 h of middle cerebral artery occlusion (MCAo) and at 24 h after MCAo

124 severe ischemic injury, as induced by middle cerebral artery occlusion (MCAo) and if this protection

125 ts (n=57) were subjected to permanent middle cerebral artery occlusion (MCAo) and injected intravenou

126 barrier (BBB) permeability following middle cerebral artery occlusion (MCAo) and intrastriatal trans

127 unction and tissue injury response to middle cerebral artery occlusion (MCAO) and reperfusion.

128 on are attenuated following transient middle cerebral artery occlusion (MCAO) and reperfusion.

129 he animals were subjected to a 2-hour middle cerebral artery occlusion (MCAO) and sacrificed at 24 ho

130 odel encompasses a combination of the middle cerebral artery occlusion (MCAO) and spatial restraint s

131 acute brain injury: stroke caused by middle cerebral artery occlusion (MCAO) and traumatic brain inj

132 reventing brain damage from transient middle cerebral artery occlusion (MCAO) as was estrone.

133 (reproductive senescence) shows that middle cerebral artery occlusion (MCAo) causes a larger cortica

134 f age, rat pups underwent a permanent middle cerebral artery occlusion (MCAo) coupled with a temporar

135 l of permanent and transient (45 min) middle cerebral artery occlusion (MCAO) during the hyperacute,

136 to the ischemic site after transient middle cerebral artery occlusion (MCAO) followed by reperfusion

137 -Tg) littermates underwent reversible middle cerebral artery occlusion (MCAO) for 1 hour followed by

138 riments, rats were subjected to right middle cerebral artery occlusion (MCAO) for 2 h under ketamine/

139 study, the model of reversible right middle cerebral artery occlusion (MCAO) for 2 h was used.

140 t male C57BL/6 mice were subjected to middle cerebral artery occlusion (MCAO) for stroke induction.

141 for the 30-min, 60-min and permanent middle cerebral artery occlusion (MCAO) group, respectively.

142 We also tested H(2) in middle cerebral artery occlusion (MCAO) in adult rats (MCAO n=9

143 Following transient middle cerebral artery occlusion (MCAO) in adult rats, expressi

144 o the cerebral cortex before a 60-min middle cerebral artery occlusion (MCAo) in adult rats.

145 emia and 2) infarct volumes 24h after Middle Cerebral Artery Occlusion (MCAO) in all 3 types of mice.

146 d preconditioning following 90 min of middle cerebral artery occlusion (MCAO) in male Wistar rats.

147 Since we previously demonstrated that middle cerebral artery occlusion (MCAO) in mice induces sheddin

148 evaluated various outcomes following middle cerebral artery occlusion (MCAO) in ovariectomised femal

149 addition, the functional recovery to middle cerebral artery occlusion (MCAO) in rats and hMCT2 trans

150 roved behavioral outcome after distal middle cerebral artery occlusion (MCAO) in rats.

151 peutic effect in a model of permanent middle cerebral artery occlusion (MCAo) in rats.

152 followed by continuous decline after middle cerebral artery occlusion (MCAO) in the mouse brain.

153 Ischemia induced either by embolic middle cerebral artery occlusion (MCAO) in vivo or by oxygen an

154 tion of Ang-(1-7) following transient middle cerebral artery occlusion (MCAO) increased the amount of

155 Experimental middle cerebral artery occlusion (MCAO) increases tPA activity

156 ane and subjected to 2 h of temporary middle cerebral artery occlusion (MCAo) induced by means of a p

157 We found that middle cerebral artery occlusion (MCAO) induces microglial acti

158 Middle cerebral artery occlusion (MCAO) is a popular model in e

159 ue-Dawley rats using the intraluminal middle cerebral artery occlusion (MCAO) method.

160 prove sensorimotor functions in a rat middle cerebral artery occlusion (MCAO) model after a single in

161 tion were investigated in a permanent middle cerebral artery occlusion (MCAO) model in the adult mous

162 We used transient rat middle cerebral artery occlusion (MCAO) model of brain ischemia

163 cle-treated groups in a 12h permanent middle cerebral artery occlusion (MCAO) model of focal ischemia

164 e intravenous (IV) injection in a rat middle cerebral artery occlusion (MCAO) model of ischemia/reper

165 AE) model of multiple sclerosis and a middle cerebral artery occlusion (MCAO) model of stroke, LSR wa

166 Using an in vivo middle cerebral artery occlusion (MCAO) model only the 57kDa fr

167 lized a murine transient endovascular middle cerebral artery occlusion (MCAO) model to examine the in

168 focal cerebral ischemia in the mouse middle cerebral artery occlusion (MCAO) model.

169 mpact of gemfibrozil in two permanent middle cerebral artery occlusion (MCAO) models in young adult m

170 n injury in mouse photothrombotic and middle cerebral artery occlusion (MCAo) models.

171 on ischemic stroke was studied in the middle cerebral artery occlusion (MCAo) mouse model.

172 hemia was induced by permanent distal middle cerebral artery occlusion (MCAO) on day 14 of vehicle or

173 UCHL1 C152A KI and WT mice underwent middle cerebral artery occlusion (MCAO) or sham surgery.

174 ale rats were subjected to a two-hour middle cerebral artery occlusion (MCAO) procedure.

175 ague-Dawley rats undergoing permanent middle cerebral artery occlusion (MCAO) received three intraven

176 late as 5 h after 60 min of transient middle cerebral artery occlusion (MCAO) reduced infarct volume

177 1620-treated rats following permanent middle cerebral artery occlusion (MCAO) showed significant impr

178 tudy, rats were sacrificed 24 h after middle cerebral artery occlusion (MCAO) stroke and gene transcr

179 Using the mouse middle cerebral artery occlusion (MCAO) stroke model, we have e

180 immune cell populations in mice after middle cerebral artery occlusion (MCAO) strongly implicates a m

181 itioning stimulus in a mouse model of middle cerebral artery occlusion (MCAO) to examine whether impr

182 dified nylon suture in a rat model of middle cerebral artery occlusion (MCAO) under two anesthesia re

183 size of penumbra in mice subjected to middle cerebral artery occlusion (MCAO) using a genome-wide app

184 Two weeks later, permanent middle cerebral artery occlusion (MCAO) was induced by intralum

185 Infarct sizes 72 h after 60 min middle cerebral artery occlusion (MCAo) were on average 30 +/-

186 greater in diabetic animals following middle cerebral artery occlusion (MCAO) when compared to non-di

187 GRN(+/-) and PGRN(-/-) mice underwent middle cerebral artery occlusion (MCAO) with monitoring of cere

188 Studies were conducted following middle cerebral artery occlusion (MCAO) with or without reperfu

189 pite smaller infarcts after transient middle cerebral artery occlusion (MCAO) with the suture model.

190 This study utilized middle cerebral artery occlusion (MCAO) with tissue plasminogen

191 replacement were subjected to 2 h of middle cerebral artery occlusion (MCAO), and phosphorylated STA

192 One week later the animals received a middle cerebral artery occlusion (MCAO), and T(2)-weighted MRI

193 of stroke, via transient intraluminal middle cerebral artery occlusion (MCAO), or SHAM surgery.

194 In a mouse model of middle cerebral artery occlusion (MCAO), p38 MAPK activation wa

195 ion in stroke volume in rats with the middle cerebral artery occlusion (MCAO), provided the BDNF is c

196 ia induced by permanent and transient middle cerebral artery occlusion (MCAO), we observed an initial

197 eraction between tPA and LRP plays on middle cerebral artery occlusion (MCAO)-induced NF-kappaB-media

198 erfusion after a 90-minute unilateral middle cerebral artery occlusion (MCAO).

199 a was induced by a transient (90 min) middle cerebral artery occlusion (MCAO).

200 ts with experimental stroke caused by middle cerebral artery occlusion (MCAO).

201 proteins, following a 1-h reversible middle cerebral artery occlusion (MCAO).

202 T) mice were subjected to a transient middle cerebral artery occlusion (MCAO).

203 ittermate were subjected to 45 min of middle cerebral artery occlusion (MCAO).

204 ed to permanent, 60-min and 30-min of middle cerebral artery occlusion (MCAO).

205 a focal stroke caused by a transient middle cerebral artery occlusion (MCAO).

206 schemia produced in rats by permanent middle cerebral artery occlusion (MCAO).

207 rological deficits after the onset of middle cerebral artery occlusion (MCAO).

208 l of focal ischemic stroke induced by middle cerebral artery occlusion (MCAO).

209 ats infused with HUCB cells 48h after middle cerebral artery occlusion (MCAO).

210 with ATX or vehicle prior to a 60-min middle cerebral artery occlusion (MCAo).

211 outcome were assessed after permanent middle-cerebral artery occlusion (MCAO).

212 injury (CCI) and another to transient middle cerebral artery occlusion (MCAO).

213 ly with HNG before being subjected to middle cerebral artery occlusion (MCAO).

214 N in the intraluminal suture model of middle cerebral artery occlusion (MCAo).

215 ats were subjected to permanent right middle cerebral artery occlusion (MCAo).

216 BL/6 mice were subjected to transient middle cerebral artery occlusion (MCAO).

217 reperfusion injury in a rat model of middle cerebral artery occlusion (MCAO).

218 of the integrity of the NVU following middle cerebral artery occlusion (MCAO).

219 d to behavioral tests after 90 min of middle cerebral artery occlusion (MCAO).

220 es) was induced by intraluminal right middle cerebral artery occlusion (MCAO).

221 Rats in the model groups underwent middle cerebral artery occlusion (MCAO).

222 129/SV mice were subjected to 30-min middle cerebral artery occlusion (MCAo)/reperfusion and serial

223 in were investigated with a permanent middle cerebral artery occlusion model in the rat.

224 eases infarct volume in the transient middle cerebral artery occlusion model of stroke.

225 al model of hypoxia/ischemia and in a middle cerebral artery occlusion model of transient focal ische

226 ave improved cerebral blood flow in a middle cerebral artery occlusion model than mice expressing an

227 We used a transient middle cerebral artery occlusion model to induce stroke and exa

228 A transient middle cerebral artery occlusion model was used to investigate

229 In the middle cerebral artery occlusion model, the volume and fraction

230 cerebrovascular protection in a mouse middle cerebral artery occlusion model.

231 e C57BL/6 mice using the intraluminal middle cerebral artery occlusion model.

232 ase in stroke volume in the permanent middle cerebral artery occlusion model.

233 en used the murine suture and embolic middle cerebral artery occlusion models of stroke to investigat

234 oglial activation in infarcted distal middle cerebral artery occlusion mouse brain tissue more accura

235 groups: 30-min, 60-min, and permanent middle cerebral-artery occlusion (n=12 rats for each group).

236 hemic stroke and in mice subjected to middle cerebral artery occlusion, natural killer (NK) cells dis

237 n females, we evaluated the effect of middle cerebral artery occlusion on infarct size and peripheral

238 y rats (12 months old) with permanent middle cerebral artery occlusion or sham operations on multiple

239 Methods: After distal middle cerebral artery occlusion or sham surgery, mice underwen

240 ensive rats and were subjected to 1-h middle cerebral artery occlusion or sham surgery.

241 thrombotic cortical injury, transient middle cerebral artery occlusion, or neonatal hypoxic-ischemic

242 Animals were subjected to permanent middle cerebral artery occlusion (pMCAo) or a sham surgical pro

243 55/262/279/282A) (MK4) on a permanent middle cerebral artery occlusion (pMCAO) stroke model.

244 es in transient (tMCAO) and permanent middle cerebral artery occlusion (pMCAO) stroke models using Ex

245 1 and in most cases 2 h of permanent middle cerebral artery occlusion (pMCAO), mild sensory stimulat

246 Following transient middle cerebral artery occlusion, progesterone was administered

247 At 6 hrs following middle cerebral artery occlusion, rats were treated in a blinde

248 At 6 hrs following middle cerebral artery occlusion, rats were treated in a blinde

249 against cerebral ischemia induced by middle cerebral artery occlusion, reducing percent hemispheric

250 In the middle cerebral artery occlusion-reperfusion model of transient

251 deficit in a hyperglycemic rat right middle cerebral artery occlusion/reperfusion (MCAO/R) model.

252 gical impairment in mice subjected to middle cerebral artery occlusion/reperfusion.

253 istration of PAN-811 i.c.v. 1 h after middle cerebral artery occlusion resulted in a 59% reduction in

254 e analyses of rat brains subjected to middle cerebral artery occlusion revealed marked increases in e

255 rcted tissue at 72 h after reversible middle cerebral artery occlusion (rMCAo) in adult rats.

256 xcitatory neurotoxicity in reversible middle cerebral artery occlusion (rMCAO) model in vivo.

257 Following right middle cerebral artery occlusion (rMCAo) using an intraluminal

258 infarction using a stroke model with middle cerebral artery occlusion (see figure).

259 4-Fc decoy receptor immediately after middle cerebral artery occlusion significantly reduced infarct

260 TAT-C1aB in mice following transient middle cerebral artery occlusion significantly reduced ischemic

261 In the model of transient middle cerebral artery occlusion stroke Gna(i2)(fl/fl)/PF4-Cre

262 this study, using a murine transient middle cerebral artery occlusion stroke model, a novel therapeu

263 ved functional outcome in a transient middle cerebral artery occlusion stroke model.

264 ment of blood flow anomaly in a mouse middle cerebral artery occlusion stroke model.

265 s implanted with CTX-DP 4 weeks after middle cerebral artery occlusion stroke prompted investigation

266 r evaluated in vivo using a transient middle cerebral artery occlusion (t-MCAO) model of stroke.

267 We investigated in a murine model of middle cerebral artery occlusion the effect of blocking SIDS by

268 In a mouse model of thrombin-induced middle cerebral artery occlusion, the efficacy of the diabody w

269 mouse brains following 1 h transient middle cerebral artery occlusion (tMCAO) and measured real-time

270 ed neuronal death following transient middle cerebral artery occlusion (tMCAO) by up to 90% with an e

271 trating myeloid cells after transient middle cerebral artery occlusion (tMCAO) in neonatal mice of bo

272 Furthermore, by transient middle cerebral artery occlusion (tMCAO) in rats, the transcrip

273 Transient middle cerebral artery occlusion (tMCAO) in spontaneously hyper

274 We induced transient middle cerebral artery occlusion (tMCAO) in T2D/obese mice (aft

275 A transient middle cerebral artery occlusion (tMCAO) model was used to esta

276 ronic diaschisis by using a transient middle cerebral artery occlusion (tMCAO) rat model.

277 Transient middle cerebral artery occlusion (tMCAO) significantly increase

278 rial thrombosis models: the transient middle cerebral artery occlusion (tMCAO) stroke model and tail

279 rct volumes 3 and 7 d after transient middle cerebral artery occlusion (tMCAo), independent of changi

280 injury in a mouse model of transient middle cerebral artery occlusion (tMCAO).

281 anxiety) in rats following transient middle cerebral artery occlusion (tMCAO).

282 corresponding controls, to transient middle cerebral artery occlusion (tMCAO).

283 .) 15 min before undergoing transient middle cerebral artery occlusion (tMCAO; 2 h occlusion) with re

284 Following middle cerebral artery occlusion to induce stroke in mice, immu

285 ients with internal carotid artery or middle cerebral artery occlusions transferred over an 11-month

286 l vascular endothelial cell death and middle cerebral artery occlusion-triggered cerebrovascular dama

287 fecal transplant gavage 3 days after middle cerebral artery occlusion using young donor biome (2-3 m

288 The infarct produced by middle cerebral artery occlusion was 49% smaller in CD36-null m

289 ere reperfusion after photothrombolic middle cerebral artery occlusion was increased in Klkb1(-/-) mi

290 Middle cerebral artery occlusion was induced for 1 hour (follow

291 ternal carotid artery or the proximal middle cerebral artery occlusions we found that an infarct core

292 (BM) chimeras subjected to transient middle cerebral artery occlusion, we found that CD36(-/-) mice

293 Following middle cerebral artery occlusion, we observed a rapid increase

294 ver, using a mouse model of transient middle cerebral artery occlusion, we observed that cerebral inf

295 Using two distinct models of acute middle cerebral artery occlusion, we show by next-generation se

296 lumes following a 60-minute transient middle cerebral artery occlusion were determined in adult male

297 Rats subjected to 2h of transient middle cerebral artery occlusion were studied temporally over 1

298 Rats subjected to embolic middle cerebral artery occlusion were treated with atorvastatin

299 ollowing experimental stroke, using a middle cerebral artery occlusion with reperfusion model.

300 Middle cerebral artery occlusion with reperfusion was performed