ライフサイエンスコーパス: cerebral blood volume

1 were maps of apparent diffusion and relative cerebral blood volume.

2 part by changes in intracranial pressure and cerebral blood volume.

3 brain tissue, cerebrospinal fluid (CSF) and cerebral blood volume.

4 nd abolished the post-stimulus undershoot in cerebral blood volume.

5 rectly correlated to regional differences in cerebral blood volume.

6 e values are inversely proportional to local cerebral blood volume.

7 , 11-40] vs 14 [range, 6-22] mL/100 mL/min), cerebral blood volume (2.4 [range, 1.6- 4.2] vs 3.9 [ran

8 ative cerebral blood flow (16-53% decrease), cerebral blood volume (6-33% decrease), and tissue mean

9 showed an average 60% decrease in normalized cerebral blood volume (adults P < 0.05; children P < 0.0

10 First, we used MRI to map cerebral blood volume, an established correlate of basal

11 and radiologic (Gaussian normalized relative cerebral blood volume and apparent diffusion coefficient

12 hemodynamic properties of gliomas including cerebral blood volume and blood flow, vascular permeabil

13 , respiratory, and cardiovascular systems on cerebral blood volume and cerebral perfusion and intracr

14 ating increased Gaussian-normalized relative cerebral blood volume and Gaussian-normalized relative c

15 flow, prolonged mean transit time, elevated cerebral blood volume and high mean transit time/cerebra

16 ilate the cerebral vasculature, and increase cerebral blood volume and intracranial pressure while de

17 inflow and cerebral venous pressure and thus cerebral blood volume and intracranial pressure.

18 istically significant difference in relative cerebral blood volume and metabolic ratios between high-

19 maging technique that independently measures cerebral blood volume and oxygenation, continuously, in

20 ntributed to the IL-1beta-induced changes in cerebral blood volume and the ADC of brain water.

21 ale rats had greater vessel density, greater cerebral blood volumes and performed better on a neuromo

22 nel for imaging hemoglobin absorption (i.e., cerebral blood volume); and a laser speckle contrast (LS

23 s regional oxygen extraction fraction (OEF), cerebral blood volume, and an index of cerebral blood fl

24 lution, layer-specific measurements of BOLD, cerebral blood volume, and cerebral blood flow in region

25 synchronized oscillations in norepinephrine, cerebral blood volume, and cerebrospinal fluid (CSF) as

26 cal parameter such as cerebral blood flow or cerebral blood volume, and direct mapping of neural acti

27 rophysiological rhythms, vasomotor activity, cerebral blood volume, and glymphatic flow.

28 longer duration, less focal increase in CBF, cerebral blood volume, and hyperoxygenation, the duratio

29 diffusion coefficients, cerebral blood flow, cerebral blood volume, and intratumoral susceptibility s

30 5%, 5.0%, and 1.9%, for cerebral blood flow, cerebral blood volume, and mean transit time, respective

31 eate images of relative blood flow, relative cerebral blood volume, and mean transit time.

32 hod that uses ultrasound to track changes in cerebral blood volume as an indirect readout of neuronal

33 hy had an average 80% decrease in normalized cerebral blood volume at the core of the lesion (P < 0.0

34 sing optical imaging of light scattering and cerebral blood volume, autofluorescence flavoprotein ima

35 total hemoglobin concentration, i.e. in the cerebral blood volume, by -7+/-3 microM, as opposed to a

36 xygenation level-dependent (BOLD) signal and cerebral blood volume (CBV) and blood flow (CBF), which

37 ogenetic-induced tonic LC activity decreased cerebral blood volume (CBV) and glucose uptake and incre

38 vity during voluntary behaviors by measuring cerebral blood volume (CBV) and neural activity in the s

39 d 570 and 610 nm optical signals to estimate cerebral blood volume (CBV) and oxygenation, respectivel

40 imaging modalities that can measure in vivo cerebral blood volume (CBV) and post mortem vascular str

41 Simultaneously, we measured changes in cerebral blood volume (CBV) as a proxy of drug effects o

42 ctional magnetic resonance imaging (fMRI) of cerebral blood volume (CBV) at 7 Tesla.

43 rmed at 570 nm to provide functional maps of cerebral blood volume (CBV) changes and at 610 nm to est

44 imaging (fUSi) to investigate sound-induced cerebral blood volume (CBV) changes in the Field L compl

45 tudies with blood oxygen level-dependent and cerebral blood volume (CBV) contrasts at 9.4 tesla, as w

46 blood oxygenation level-dependent (BOLD) and cerebral blood volume (CBV) fMRI signal.

47 through changes in arteriole diameter, i.e., cerebral blood volume (CBV) fMRI.

48 Changes in total cerebral blood volume (CBV) have been hypothesized to dr

49 ndent (BOLD), cerebral blood flow (CBF), and cerebral blood volume (CBV) HRF to ultrashort forelimb s

50 s used to continuously monitor variations in cerebral blood volume (CBV) in +65 brain regions/hemisph

51 ation of (18)F-FDG uptake, permeability, and cerebral blood volume (CBV) in children with pediatric b

52 sly measuring neural activity and changes in cerebral blood volume (CBV) in the somatosensory cortex

53 ubjects, baseline hippocampal activity using cerebral blood volume (CBV) mapping was measured.

54 collected prospectively, with guidance from cerebral blood volume (CBV) MR imaging measurements.

55 genesis and test whether MRI measurements of cerebral blood volume (CBV) provide an imaging correlate

56 ter (GM) versus white matter (WM), (2) GM/WM cerebral blood volume (CBV) ratio close to the histologi

57 fUS is an imaging technique that measures cerebral blood volume (CBV) temporal changes.

58 Perihematomal cerebral blood volume (CBV) was inversely associated wit

59 Images of regional cerebral blood volume (CBV) were generated from echo-pla

60 Absolute cerebral blood volume (CBV), absolute cerebral blood flo

61 enerating maps of cerebral blood flow (CBF), cerebral blood volume (CBV), and mean transit time (MTT)

62 Comparisons of mean CBF, cerebral blood volume (CBV), and mean transit time (MTT)

63 Cerebral blood volume (CBV), cerebral blood flow (CBF),

64 15)O-H(2)O to generate parametric images for cerebral blood volume (CBV), cerebral blood flow (CBF),

65 the dynamic spatiotemporal relationships of cerebral blood volume (CBV), deoxygenated hemoglobin (Hb

66 m measurements of cerebral blood flow (CBF), cerebral blood volume (CBV), oxygen extraction fraction

67 s a significant, acute reduction (15-30%) in cerebral blood volume (CBV), which is dependent on TNF-a

68 trocyte pathway, enhances the specificity of cerebral blood volume (CBV)-weighted fMRI signals to cor

69 Here we show that cerebral blood volume (CBV)-weighted fMRI with a blood p

70 potential to study CAR in neonates based on cerebral blood volume (CBV).

71 l based thresholds for mean transit time and cerebral blood volume (CBV).

72 th mean tissue cerebral blood flow (CBF) and cerebral blood volume (CBV); venous and arterial peak en

73 nges in receptor occupancy and hemodynamics [cerebral blood volume (CBV)] in the domains of space, ti

74 for blood-oxygen-level-dependent (BOLD) and cerebral-blood-volume (CBV)-based laminar fMRI and used

75 ssel blood-oxygen-level-dependent (BOLD) and cerebral-blood-volume (CBV)-fMRI from individual venules

76 umor-to-blood [T/B] ratio), vascularization (cerebral blood volume [CBV]), and vascular permeability

77 pped the temporal trajectories of arteriolar cerebral blood volumes (CBVa) using inflow-based vascula

78 Cerebral blood flow, cerebral blood volume, cerebral oxygen metabolism (CMRO2

79 gh mean transit time/cerebral blood flow and cerebral blood volume/cerebral blood flow ratios.

80 n tomography imaging of cerebral blood flow, cerebral blood volume, CMRO2, and oxygen extraction frac

81 A maximum relative cerebral blood volume cutoff of 3.672 provided 90% sensi

82 anges in cerebral blood flow (DeltaCBF/CBF), cerebral blood volume (DeltaCBV/CBV), and transverse rel

83 the brain is able to assess local changes in cerebral blood volume during cognitive tasks, with suffi

84 Change in quantitative relative cerebral blood volume early poststimulation was a median

85 Temporoparietal cerebral blood volume, expressed as a percentage of the

86 d to marijuana-induced alteration in resting cerebral blood volume/flow or downregulation of cannabin

87 which were obtained in contrast agent-aided cerebral blood volume fMRI and total hemoglobin-based op

88 pathologies of hippocampal dysfunction-focal cerebral blood volume, focal atrophy, and evidence of el

89 nd REM sleep, mice showed large increases in cerebral blood volume ([HbT]) and arteriole diameter rel

90 ere is the characterization of changes in of cerebral blood volume, heart rate and behaviour that occ

91 Here, we use optical techniques to measure cerebral blood volume, hemoglobin oxygenation (S(t)O(2))

92 e range in Pa(CO2), cortical blood flow, and cerebral blood volume in animals studied using vertical

93 t shows predictive timing, with increases of cerebral blood volume in anticipation of trial onsets ev

94 sm, transient decreases in cell swelling and cerebral blood volume in the surround are consistent wit

95 e child with arrested progression normalized cerebral blood volume in this region.

96 The measured relative cerebral blood volumes in the peritumoral region in high

97 Relative cerebral blood volumes in these regions were calculated

98 ting, and has been linked to hypoxia-induced cerebral blood volume increases, inflammation and relate

99 ata revealed that DMS inactivation decreased cerebral blood volume levels in DMS and several distant

100 VLCBV was defined as cerebral blood volume<2.5th percentile of brain contrala

101 The clinical applications of cerebral blood volume maps obtained with perfusion MR im

102 bility contrast magnetic resonance perfusion cerebral blood volume maps were co-registered, segmented

103 n areas predominately exhibit a reduction of cerebral blood volume mirrored by suppression of cortica

104 raction between Gaussian-normalized relative cerebral blood volume (nrCBV) and Gaussian-normalized re

105 rmality volume, Gaussian-normalized relative cerebral blood volume (nrCBV), Gaussian-normalized relat

106 V(max) cutoff of 4.66 and a maximum relative cerebral blood volume of 3.67 on MRI provided 100% sensi

107 23) were associated with increasing relative cerebral blood volume of NER (rCBVNER), which was higher

108 high glutamate/glutamine and elevated focal cerebral blood volume on functional magnetic resonance i

109 Decreased cerebral blood volume on PCT was the most accurate predi

110 esis that LVV fluctuations, driven by global cerebral blood volume oscillations, regulate CSF movemen

111 e properties including chemical composition, cerebral blood volume, perfusion, vascular permeability,

112 Relative cerebral blood volume (rCBV) (maximum rCBV [rCBV(max)] a

113 The hypothesis was that a change in relative cerebral blood volume (rCBV) 1 month after RT-TMZ is pre

114 take and physiologic MRI, including relative cerebral blood volume (rCBV) and apparent diffusion coef

115 Relative cerebral blood volume (rCBV) and blood flow (rCBF) maps

116 Relative cerebral blood volume (rCBV) and flow (rCBF) maps were a

117 sisting of concomitant increases of relative cerebral blood volume (rCBV) and LFPs.

118 The current study evaluated both relative cerebral blood volume (rCBV) and VSI(MRI) in eleven pati

119 xin injection caused an increase in regional cerebral blood volume (rCBV) around the lesion site afte

120 ied to MR imaging-based whole-tumor relative cerebral blood volume (rCBV) histograms.

121 i.v.) in control monkeys increased relative cerebral blood volume (rCBV) in a number of brain region

122 cant differences were noted in age, relative cerebral blood volume (rCBV) in contrast-enhanced region

123 Relative cerebral blood volume (rCBV) maps were created from anal

124 -(18)F-fluorothymidine ((18)F-FLT), relative cerebral blood volume (rCBV) maps, and uptake of (18)F-f

125 lity contrast (DSC) MRI measures of relative cerebral blood volume (rCBV) play a central role in moni

126 Relative cerebral blood volume (rCBV) ratio is one of the best no

127 ffusion coefficient (ADC) with high relative cerebral blood volume (rCBV) represented elevated cholin

128 Regional cerebral blood volume (rCBV) was determined by using dyn

129 Tumor relative cerebral blood volume (rCBV) was estimated from each DSC

130 , a group with intratumor values of relative cerebral blood volume (rCBV)>3.0 has shown a significant

131 R imaging (PWI), especially maps of regional cerebral blood volume (rCBV), may provide similar diagno

132 educed perfusion values in terms of relative cerebral blood volume (rCBV).

133 ntrast-enhanced perfusion-weighted (relative cerebral blood volume [rCBV]) imaging were evaluated in

134 Significantly higher fMRI signals [relative cerebral blood volumes (rCBVs)] and atrophy were observe

135 etic stimulation of TH(VTA) neurons enhanced cerebral blood volume signals in striatal target regions

136 2 to 12.61, p = 0.01), standardized relative cerebral blood volume (srCBV) (HR = 1.61, 95% CI 1.09 to

137 tic theory, yields quantitative estimates of cerebral blood volume that reflect the underlying microv

138 n emerging technique that detects changes of cerebral blood volume triggered by brain activation.

139 cerebral lesion was present, and normalized cerebral blood volume values were analysed using a Food

140 brain incorporation rate of 11C-AA (K*) and cerebral blood volume (Vb), as well as CBF, were generat

141 Very low cerebral blood volume (VLCBV), diffusion, and hypoperfus

142 In an MRI substudy, hippocampal cerebral blood volume was mapped.

143 njection of IL-1beta (1 ng in 1 microliter), cerebral blood volume was significantly increased, the b

144 ng global hemodynamic changes using BOLD and cerebral blood volume-weighted (CBVw) fMRI.

145 In addition, we use cerebral blood volume-weighted functional magnetic reson

146 Cerebral blood flow and cerebral blood volume were determined with intravenous H

147 an 8-month-old infant, mean transit time and cerebral blood volume were low relative to cerebral bloo

148 al flow was normal but mean transit time and cerebral blood volume were low, consistent with perfusio

149 ptical intrinsic signal recording to measure cerebral blood volume, which under baseline conditions i

150 ound (fUS) neuroimaging to record changes in cerebral blood volume with 100 um resolution.

151 across the PPC by recording local changes in cerebral blood volume within PPC as two male monkeys per