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1 ocampal volume, brain amyloid deposition and cerebral metabolism.
2 er than voluntary exercise through increased cerebral metabolism.
3  quantify cerebral autoregulation as well as cerebral metabolism.
4 onic acid (AMPA) receptors to the control of cerebral metabolism.
5 d MRS) are able to assess both perfusion and cerebral metabolism.
6 of 'isolated' mitochondrial failure on local cerebral metabolism.
7 nclear how mitochondrial dysfunction affects cerebral metabolism.
8 that may reflect compensatory alterations in cerebral metabolism.
9 hat glucocorticoids have a central action on cerebral metabolism.
10 18F]-2-fluoro-2-deoxy-D-glucose to determine cerebral metabolism.
11 bour, suggesting persisting effects on fetal cerebral metabolism.
12   Among the various techniques used to study cerebral metabolism, (13)C magnetic resonance spectrosco
13 etween re-establishment of apparently normal cerebral metabolism after HI, and the start of secondary
14  the relationship between local increases in cerebral metabolism and appropriately matched increases
15 posed approach using PET to examine regional cerebral metabolism and look for characteristic patterns
16 evious work had shown a relationship between cerebral metabolism and NMDA receptor activity.
17 conducted with fluorodeoxyglucose to measure cerebral metabolism and Pittsburgh compound B for fibril
18                      Adaptive alterations in cerebral metabolism are the rationale for the Metabolic
19 ion of the effects of general anesthetics on cerebral metabolism as revealed by imaging for therapeut
20 n signaling is critical for neuroplasticity, cerebral metabolism as well as for systemic energy metab
21 erial PCO2 /pH on heat-induced alteration in cerebral metabolism, as well as on the cerebral oxidativ
22             One possibility is that lifetime cerebral metabolism associated with regionally specific
23            The return to baseline of CBF and cerebral metabolism at normocapnia seen in our study wit
24 stages of Alzheimer's disease despite normal cerebral metabolism at rest.
25 dent effects of hyperglycemia and ketosis on cerebral metabolism, blood flow, and water distribution.
26 uronal firing patterns, neuromodulators, and cerebral metabolism change across sleep-waking states, a
27                Additionally, the increase in cerebral metabolism did not significantly impact the net
28                         Abnormal patterns of cerebral metabolism during non-REM sleep in depressed pa
29  slow waves to reduced neuronal activity and cerebral metabolism during partial seizures, but found i
30                             Abnormalities in cerebral metabolism during stimulation appeared in the f
31 repsilon4 was also associated with decreased cerebral metabolism, especially in right middle frontal
32 rameters (systemic blood pressure, anaerobic cerebral metabolism, excessive brain tissue oxygenation,
33 c magnetic resonance imaging and on regional cerebral metabolism for glucose by positron emission tom
34            In order to measure the extent of cerebral metabolism in animals with different exercise r
35                            The variations in cerebral metabolism in chronic PVS patients indicate tha
36 ermine the effect of normobaric hyperoxia on cerebral metabolism in patients with severe traumatic br
37  effects of chronic haloperidol treatment on cerebral metabolism in schizophrenic patients, 2) the re
38 yglucose (FDG) were used to measure regional cerebral metabolism in seven schizophrenic patients judg
39 s, spontaneous synaptic activity, and active cerebral metabolism in the absence of global electrocort
40 ctual ability were inversely correlated with cerebral metabolism in the prefrontal, pre-motor, and le
41 on-PDD), compared with controls, had reduced cerebral metabolism in the primary occipital cortex (BA
42 went positron emission tomography to measure cerebral metabolism in this cross-sectional study.
43 saline-treated animals, the lesion increased cerebral metabolism in typical basal ganglia regions, su
44 ises to be a useful method for investigating cerebral metabolism in vivo.
45 tter study also found significantly improved cerebral metabolism (in 3 of 6 cortical and 3 of 7 subco
46 T images of AD demonstrate focally decreased cerebral metabolism involving especially the posterior c
47     Previous imaging studies have shown that cerebral metabolism is gradually reduced at the beginnin
48                  These data demonstrate that cerebral metabolism is significantly compromised during
49 for the widely held stipulation that overall cerebral metabolism levels remain constant regardless of
50  the beneficial effects of FPSA treatment on cerebral metabolism more precisely and rapidly than ICP
51                                Elevations in cerebral metabolism necessitate appropriate coordinated
52 gulation of CBF (i.e., arterial blood gases, cerebral metabolism, neural mechanisms, and specific vas
53 nsgenic mice, the results suggest that local cerebral metabolism of beta-amyloid precursor protein ma
54                                     Regional cerebral metabolism of each patient was analyzed by dete
55                           The effects of the cerebral metabolism of the high levels of ammonia that c
56                        In addition, baseline cerebral metabolism (PCr/NTP) may identify vulnerable in
57                                   Studies of cerebral metabolism, perfusion sequences, and diffusion
58 ential for in vivo non-invasive detection of cerebral metabolism post-TBI, providing a new tool to mo
59 ower blood flow velocity, indicating reduced cerebral metabolism, predicted depressive symptoms and d
60  approximately 60-70% maximal oxygen uptake, cerebral metabolism remains elevated but perfusion in th
61 This study further determined the changes in cerebral metabolism resulting from the two methods of ex
62                 General anesthetics suppress cerebral metabolism significantly.
63                                Reductions in cerebral metabolism sufficient to impair cognition in no
64 and demonstrated marked decreases in overall cerebral metabolism to 30-40% of normal.
65 (PiB) positron emission tomography (PET) and cerebral metabolism using [(18)F]fluoro-2-deoxy-d-glucos
66                                              Cerebral metabolism was affected bilaterally in a subset
67 hourly by a parenchymal Clark electrode, and cerebral metabolism was assessed by the lactate/pyruvate
68                                Non-oxidative cerebral metabolism was calculated from the ratio of oxy
69                                 Furthermore, cerebral metabolism was depressed significantly in a tim
70                                              Cerebral metabolism was determined in 35 brain structure
71 he same divers, the impact of hypercapnia on cerebral metabolism was determined using varying levels
72 omplaint was progressive dysfluency, resting cerebral metabolism was measured using [18F]fluorodeoxyg
73 rval, 0.06-0.16), and the initial pattern of cerebral metabolism was significantly associated with th
74                               Local rates of cerebral metabolism were determined after an intravenous
75                    Furthermore, decreases in cerebral metabolism were more intense and widespread.
76                              Global rates of cerebral metabolism were significantly lower in animals
77 es were used to evaluate the associations of cerebral metabolism with CVRFs and atherosclerotic plaqu