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1 e, cannulas were placed in the right lateral cerebral ventricle.
2 ntagonist, BMS-200261 into the right lateral cerebral ventricle.
3 s to the infusion of oleic acid in the third cerebral ventricle.
4 al inhibitors of its activity into the third cerebral ventricle.
5 nd administered this directly into the third cerebral ventricle.
6 e brain through an infusion into the lateral cerebral ventricle.
7 orpus callosum, and enlargement of the third cerebral ventricle.
8 a 5-HT1A receptor agonist, into the lateral cerebral ventricle.
9 subthreshold for effect if injected into the cerebral ventricles.
10 spatial order from germinal cells lining the cerebral ventricles.
11 al cells were observed in the linings of all cerebral ventricles.
12 3) included studies of the skull, brain, and cerebral ventricles.
13 e olfactory bulb, and in cells bordering the cerebral ventricles.
14 ary neuronal damage in regions bordering the cerebral ventricles.
15 ood intake in the rat when injected into the cerebral ventricles.
16 e developed criteria for leakage of PRV into cerebral ventricles.
17 duces obesity in rats when injected into the cerebral ventricles.
18 that results in the active expansion of the cerebral ventricles.
19 ology resulting in abnormal expansion of the cerebral ventricles.
20 istration of ATA by injection into the right cerebral ventricle 1 h before (but not 6 h after) bilate
21 0.3, 3.0, or 10 nmol) into the left lateral cerebral ventricle 5 min before a smaller non-aggressive
22 ulating proteins, infused within the lateral cerebral ventricle (a primary site of CSF production).
23 l fluid (CSF) continuously flows through the cerebral ventricles, a process essential for brain homeo
25 afish larva as an in vivo model for studying cerebral ventricle and blood-brain barrier function.
26 ted in the germinal zones near the embryonic cerebral ventricle and migrate radially to the overlying
27 id plexus epithelial cells extends into each cerebral ventricle and secretes signaling factors into t
28 Cerebrospinal fluid (CSF) is produced in the cerebral ventricles and circulates within the subarachno
29 advance in understanding the genetics of the cerebral ventricles and demonstrates the potential utili
30 velopment leads to marked enlargement of the cerebral ventricles and destruction of brain tissue, due
32 ical disorder that leads to expansion of the cerebral ventricles and is associated with a high rate o
33 a critical role in the development of normal cerebral ventricles and neuroependymal integrity as well
34 nd venous catheters, a catheter in the third cerebral ventricle, and ultrasonic flow probes on the le
36 al longitudinal anastomotic vessel, enlarged cerebral ventricles, and pericardial edema, in addition
38 ular (ICV) cannula was placed in the lateral cerebral ventricle at 12 weeks of age for ICV infusion o
40 nding the anteroventral portion of the third cerebral ventricle (AV3V) of the preoptic recess is crit
42 and/or endoscopic removal of clots from the cerebral ventricles could transform the outcome of infan
43 ) mice, and leptin infusion into the lateral cerebral ventricles decreases feeding with short latency
44 tely smaller and have elevated incidences of cerebral ventricle dilation but otherwise appear normal.
45 gic hydrocephalus (PHH), an expansion of the cerebral ventricles due to CSF accumulation following in
46 ore, the infusion of oleic acid in the third cerebral ventricle failed to decrease the expression of
48 l disorder that is characterized by enlarged cerebral ventricles, gait difficulty, incontinence, and
49 in the subependymal (SZ) zone of the lateral cerebral ventricle generate neuroblasts that migrate in
52 in or an active cysteine mutant in the third cerebral ventricle (icv) or in the mediobasal hypothalam
54 , we directly infused the ASO into a lateral cerebral ventricle in adult mice expressing a human SMN2
56 rain arise from progenitors located near the cerebral ventricles in a temporally segregated manner su
58 ormal mice resistin delivered in the lateral cerebral ventricle increased endogenous glucose producti
61 thesis, injection of alpha2-antiplasmin into cerebral ventricles markedly ameliorated HI-induced dama
62 w dose of leptin (3.5 microg) into the third cerebral ventricle (n = 15) during a 40-h period of fast
63 e (2 microl) was administered into the third cerebral ventricle of age- and weight-matched male and f
64 and administration of insulin into the third cerebral ventricle of fasted rats increased expression o
65 ion of 60 fmol of gamma-MSH into the lateral cerebral ventricle of hypertensive mice also lowered MAP
67 to c-fos, rncfosr115, infused into the left cerebral ventricle of male Long-Evans rats and the effec
68 ed continuously for 2 weeks into the lateral cerebral ventricle of male Wistar rats rendered obese by
69 hat chronic resistin infusion in the lateral cerebral ventricle of normal rats markedly affects both
70 tracerebroventricularly (ICV) into the third cerebral ventricle of ovariectomized, estradiol benzoate
71 d that administration of hUCB cells into the cerebral ventricle of presymptomatic Naglu mice had a be
72 -6 to brain organotypic cultures or into the cerebral ventricles of adult rats did not activate the J
73 ving rise to both neurons and glia, line the cerebral ventricles of all adult animals, including huma
74 cycle exit in mammalian neocortex--into the cerebral ventricles of chicks at embryonic day (ED) 4.
75 ique system for maintaining catheters in the cerebral ventricles of conscious mice to test whether th
76 Fetal human brain cells implanted into the cerebral ventricles of embryonic rats incorporate indivi
77 al populations, and infusion of CRF into the cerebral ventricles of infant rats produces severe age-d
78 ar injection of Abeta1-42 oligomers into the cerebral ventricles of mice, a validated Alzheimer's dis
80 ctor, called satietin, was injected into the cerebral ventricles of rats that were either fed ad libi
81 lantation of glial cell progenitors into the cerebral ventricles of the embryonic myelin-deficient ra
83 ive inhibitor of CPT1A activity in the third cerebral ventricle or in the mediobasal hypothalamus (MB
84 mplanted with cannulas to either the lateral cerebral ventricle or the ventromedial hypothalamic nucl
85 administration of PNU120596 either into the cerebral ventricles or directly into the mediodorsal tha
87 small-molecule insulin mimetic in the third cerebral ventricle suppressed glucose production indepen
88 onist dizocilpine (100 nmol infused into the cerebral ventricles) suppressed drinking following eithe
89 to the contralateral hemisphere, or into the cerebral ventricles), the donor cells migrate through no
92 subpopulation of ependymal cells lining the cerebral ventricle wall, and other nonneuronal cells.
93 plets were asymmetrically distributed in the cerebral ventricle walls, mainly located in the lateral
94 a-MSH), or antagonist, SHU9119, in the third cerebral ventricle while food intake was maintained cons