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1 eatitis-like changes noted after addition of cerulein.
2 or rats were injected intraperitoneally with cerulein.
3 osed to a supramaximally stimulating dose of cerulein.
4 concentrations of the cholecystokinin analog cerulein.
5 d with 5 daily intraperitoneal injections of cerulein.
6  by administration of lipopolysaccharide and cerulein.
7 investigated in mice after administration of cerulein.
8 ory response, immediately after injection of cerulein.
9 ed in these mice during induction of AP with cerulein.
10  1 hr and 6 hrs after the first injection of cerulein (10 mg/kg, intraperitoneally).
11 uced by hourly intraperitoneal injections of cerulein (50 mug/kg x 6).
12 mation was induced by 8 hourly injections of cerulein (50 mug/kg).
13 in adult male C57Bl/6J mice by administering cerulein (8 injections of 50 ug/kg I.P., spaced an hour
14  and groups 2, 3, and 4 received intravenous cerulein (8.5 microg x kg(-1) x h(-1)).
15                  Pancreatitis was induced by cerulein administration (50 microg/kg, 7 intraperitoneal
16                                              Cerulein administration led to more BrdU(+) cells in PDG
17                                        After cerulein administration, Ptf1a-Cre(ERTM);LSL-Kras(G12D)
18 y 50 microg/kg intraperitoneal injections of cerulein and a single intraperitoneal injection of Esche
19                Pancreatitis was induced with cerulein and by retrograde injection of bile salts.
20 revented the inflammatory response caused by cerulein and decreased the cell damage.
21 pothermia on signaling pathways initiated by cerulein and GTL was studied in acinar cells.
22 ministration of the cholecystokinin analogue cerulein and interfered with acinar cell regeneration.
23 oplasmic reticulum stress markers induced by cerulein and reduced the pancreatic damage.
24 ethyl pyruvate just before the first dose of cerulein and then injected with a second 40 mg/kg dose 6
25 ancreatitis induced by supramaximal doses of cerulein, and in isolated pancreatic acini.
26 B-deleted) were stimulated with supramaximal cerulein, and the cytosolic activity of cathepsin B and
27 he effects of pancreatic PTP1B deficiency on cerulein- and arginine-induced acute pancreatitis.
28  reveal a novel role for pancreatic PTP1B in cerulein- and arginine-induced acute pancreatitis.
29 effects of sEH pharmacological inhibition on cerulein- and arginine-induced AP using the selective sE
30 nd after induction of pancreatitis mitigated cerulein- and arginine-induced AP.
31 rmore, panc-PTP1B KO mice exhibited enhanced cerulein- and arginine-induced NF-kappaB inflammatory re
32 f pancreatitis was associated with decreased cerulein- and arginine-induced nuclear factor-kappaB inf
33                                    Moreover, cerulein- and arginine-induced serum amylase and lipase
34             We noted obesity to convert mild cerulein AP to SAP with greater cytokines, unsaturated f
35                               Secretagogues (cerulein, carbachol, and bombesin) can induce protease a
36 hat use calcium as a second messenger (e.g., cerulein, carbamylcholine, and bombesin) but not to thos
37                                   When given cerulein, Ctrl-KO mice exhibited lower intrapancreatic c
38 ting dose of cholecystokinin or its analogue cerulein develop acute pancreatitis with acinar cell inj
39 otransferase measured 10 hrs after the first cerulein dose were significantly lower than in mice with
40 ts received a 6-hour intravenous infusion of cerulein either alone or after treatment with ANS, BN520
41                                              Cerulein elicited higher and more sustained trypsin acti
42     Mice imaged with (18)F-FDG PET/CT showed cerulein-enhanced pancreatic uptake in addition to a mod
43                                     Combined cerulein/enterokinase infusions resulted in marked TAP i
44 eparate model, mice were given injections of cerulein for 10 weeks to induce chronic pancreatitis.
45                                 Injection of cerulein for 2 days induced progressive pancreatitis in
46  was induced by intraperitoneal injection of cerulein (hourly x5, 50 mug/kg).
47 another chemokine, mcp-1, were induced after cerulein hyperstimulation in vivo.
48                                    We used a cerulein hyperstimulation model of acute pancreatitis an
49 d band was consistently observed early after cerulein hyperstimulation.
50 ndardized intraductal bile acid infusion and cerulein hyperstimulation.
51 by retrograde injection of bile salts and by cerulein in acinar cells in vitro.
52        Chronic pancreatitis was induced with cerulein in C57BL/6 mice, which were contrasted with sal
53 issues and primary acinar cells treated with cerulein in combination with ethanol (50 mmol/L) and cig
54 f experimental acute pancreatitis induced by cerulein in mice.
55  supramaximally stimulating concentration of cerulein in vitro.
56 le supramaximal intraperitoneal injection of cerulein, in C57Bl6 (control) and C5-deficient mice.
57    An acute acid load given in vivo enhanced cerulein-induced (50 microg/kg) trypsinogen activation a
58 with either a single episode or a repetitive cerulein-induced (50 mug/kg, 6 hourly i.p. injections) p
59 nhibition of GSK-3beta reduces the degree of cerulein-induced acute pancreatitis and the associated m
60 ld-type (WT) littermates were exposed to the cerulein-induced acute pancreatitis model, and acute (1-
61 the altered protease activation, severity of cerulein-induced acute pancreatitis was similar in Ctrl-
62 in-null pancreata were highly susceptible to cerulein-induced acute pancreatitis, displaying an enhan
63 e acinar cells exhibit increased severity of cerulein-induced acute pancreatitis.
64  in vivo efficacy study in a murine model of cerulein-induced acute pancreatitis.
65 nar cells and is potently protective against cerulein-induced acute pancreatitis.
66 icacy in reducing the inflammatory damage in cerulein-induced AP in mice.
67                                 In rats with cerulein-induced AP, pancreatic cooling decreased pancre
68 er in vitro and in vivo studies in models of cerulein-induced AP.
69 pHe significantly increased the amplitude of cerulein-induced Ca(2+) signals.
70 57BL/6J being more susceptible to repetitive cerulein-induced CP as assessed by pancreatic atrophy, p
71 primary pancreatic stellate cells (PSCs) and cerulein-induced CP.
72 ne, 4-phenolbutyrate, was protective against cerulein-induced death.
73 tf1a(Cre/+) neoplasia model and subjected to cerulein-induced experimental pancreatitis.
74 irreversible cathepsin B inhibitor, prevents cerulein-induced in vitro trypsinogen activation.
75  regeneration of the exocrine pancreas after cerulein-induced injury through cell autonomous mechanis
76       TRPV1 and TRPA1 antagonists attenuated cerulein-induced pain behaviors and pancreatic inflammat
77  we show that EZH2 is up-regulated following cerulein-induced pancreatic injury and is required for t
78 hophysiological and neurological sequelae of cerulein-induced pancreatitis in mice, which provides su
79 in isoform that plays no significant role in cerulein-induced pancreatitis in mice.
80     Here we investigated the role of CTRL in cerulein-induced pancreatitis in mice.
81  following injury, we examined the course of cerulein-induced pancreatitis in plg-deficient and -suff
82                   SAM was also protective in cerulein-induced pancreatitis in the rat, but the protec
83 sumption accelerates fibrosis in response to cerulein-induced pancreatitis in the rat.
84 thin the pancreas during the early stages of cerulein-induced pancreatitis reflects activation of try
85      Immunofluorescence and western blots on cerulein-induced pancreatitis samples from pancreatic ac
86 indings indicate that the protection against cerulein-induced pancreatitis that follows culture-induc
87                                              Cerulein-induced pancreatitis was characterized by neutr
88 lein pancreatitis induced once (P1), or with cerulein-induced pancreatitis weekly for 3 weeks (P3).
89 E-stained tissue sections, responsiveness to cerulein-induced pancreatitis, and immunohistochemical i
90 noma (PDAC) model, Ube2f deletion suppresses cerulein-induced pancreatitis, and progression of acinar
91                                 In rats with cerulein-induced pancreatitis, concentrations of trypsin
92 results in changes similar to those noted in cerulein-induced pancreatitis, i.e., intra-acinar cell t
93 -deficient mice also are more susceptible to cerulein-induced pancreatitis.
94 70 (HSP70) expression and protection against cerulein-induced pancreatitis.
95 mice also showed increased susceptibility to cerulein-induced pancreatitis.
96 uced a mild improvement in the parameters of cerulein-induced pancreatitis.
97  APK) and without KRAS(G12D) were exposed to cerulein-induced pancreatitis.
98 ouse strains had different susceptibility to cerulein-induced pancreatitis.
99 ly, their loss has detrimental effects after cerulein-induced pancreatitis.
100 le signaling pathways in acinar cells during cerulein-induced pancreatitis.
101  exocrine pancreas during regeneration after cerulein-induced pancreatitis.
102 gene is associated with enhanced response to cerulein-induced pancreatitis.
103    Treatment with ANS increased apoptosis in cerulein-infused animals.
104 in treatment alone and a 14-fold increase in cerulein-infused, neutrophil-depleted animals.
105 50,000 U intraperitoneal IL-10 2 hours after cerulein infusion and every 3 hours thereafter.
106 000 U of intraperitoneal IL-10 1 hour before cerulein infusion and every 3 hours thereafter.
107                   Induction of CP in mice by cerulein injection does not require intra-acinar activat
108  at the acute phase (7 hours after the first cerulein injection) or during recovery (at 2, 4, and 7 d
109                  Pancreatitis was induced by cerulein injection, and multiple pathological parameters
110 Serum was collected 6 or 18 h after the last cerulein injection, pancreatic tissue was collected 2 an
111 ed by cyclosporine pretreatment, followed by cerulein injections (50 mug/kg, 6 intraperitoneal inject
112                            T7K24R mice given cerulein injections had increased pancreatic activation
113 ne-deficient-ethionine-supplemented diet and cerulein injections), ST2-deficient mice (Il1rl1(-/-)) p
114 ild-type control animals received repetitive cerulein injections, and a detailed histologic analysis
115 acute pancreatitis was induced with repeated cerulein injections, FVB/N mice had more severe pancreat
116 se activity compared with control mice given cerulein injections.
117 as induced by lipopolysaccharide (LPS) or by cerulein injections.
118 scue of impaired exocrine regeneration after cerulein injury.
119 corresponding with mitigated pancreatitis on cerulein insult.
120 ating human disease is repeated injection of cerulein into mice.
121 ute pancreatitis was induced by injection of cerulein into wild-type and Trpc3-/- mice.
122       Hyperstimulation with the secretagogue cerulein is a commonly used experimental model to study
123                             Using a model of cerulein-mediated injury and repair, we demonstrate that
124 inhibited apoptosis in a modification of the cerulein model of pancreatitis which is associated with
125 AF antagonist (BN52021) were measured in the cerulein model of pancreatitis.
126 atitis was induced by i.p. administration of cerulein or by intraductal administration of sodium taur
127 ither supramaximal doses of the secretagogue cerulein or feeding a choline-deficient, ethionine-suppl
128 uced in Wistar rats by the administration of cerulein or glyceryl tri-linoleate (GTL).
129 dels of acute pancreatitis induced by either cerulein or IL-12 + IL-18.
130 sing the Cre/locus of cross-over P strategy; cerulein or L-arginine were used to induce AP.
131 n activation in mice after administration of cerulein or L-arginine.
132 l3(-/-) mice by intraperitoneal injection of cerulein or pancreatic infusion of sodium taurocholate.
133 loped more severe AP after administration of cerulein or sodium taurocholate than control mice; pancr
134 creatitis was induced in Bmi1(-/-) mice with cerulein; pancreatic cell regeneration, differentiation,
135 tis was induced in mice by administration of cerulein; pancreatic tissues were collected, analyzed by
136  that caspases were greatly activated during cerulein pancreatitis in the rat but not mouse.
137 were studied without pancreatitis (P0), with cerulein pancreatitis induced once (P1), or with cerulei
138 ic inflammation, alleviated allodynia in the cerulein pancreatitis model, and improved glucose homeos
139                                    Injury by cerulein pancreatitis resulted in regeneration of normal
140                                    For this, cerulein pancreatitis was induced in lean and obese mice
141                                           In cerulein pancreatitis, new evidence supports the idea th
142                                           In cerulein pancreatitis, trypsinogen levels increased prom
143 zed the differences between the rat model of cerulein pancreatitis, with relatively high apoptosis an
144 ats with a secretory or supermaximal dose of cerulein produced an acidic shift in hsp27, indicating a
145 duced pancreatitis, rPAF-AH given before the cerulein reduced hyperamylasemia, acinar cell vacuolizat
146                             The injection of cerulein resulted in acute necrotizing pancreatitis.
147 ents to a supramaximally stimulating dose of cerulein results in changes similar to those noted in ce
148  supramaximally stimulating concentration of cerulein results in trypsinogen activation and acinar ce
149 ancreatic NG and DRG neurons from mice given cerulein revealed increased responses to TRP agonists.
150                    The results indicate that cerulein stimulates pancreatic production of PAF.
151 s determined in vitro by FM1-43 loading with cerulein stimulation.
152  higher in pancreata from control mice given cerulein than from mice without AP, and were higher in b
153                                          For cerulein, this sensitization was seen over a range of co
154 ion was impaired following administration of cerulein to Bmi1(-/-) mice.
155 ed with a supramaximally stimulating dose of cerulein to induce mild pancreatitis.
156 signaling pathway), along with injections of cerulein to induce pancreatitis.
157 7BL/6N (control) mice repeated injections of cerulein to induce pancreatitis.
158 ver, p65 transgenic mice given injections of cerulein, to induce acute pancreatitis, had higher level
159                   Histopathology of aged and cerulein-treated mice were assessed by histology and imm
160  samples revealed enhanced features of AP in cerulein-treated panc-PTP1B KO mice compared with contro
161 eatic tissue showed a ninefold increase with cerulein treatment alone and a 14-fold increase in cerul
162 f diabetes and chronic pancreatitis on PDGs, cerulein was injected i.p., repetitively over 10 weeks,
163  trypsinogen in response to stimulation with cerulein was quantitated in isolated rat pancreatic acin
164  supramaximally stimulating concentration of cerulein, which is known to inhibit secretion.

 
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