ライフサイエンスコーパス: cervical

1 ovements were observed for trunk (53.2%) and cervical (50.5%) dystonia, with less clinical impact on

2 fever, aphthous stomatitis, pharyngitis, and cervical adenitis (PFAPA) syndrome is the most common pe

3 La-derived KB cell line, a cellular model of cervical adenocarcinoma, where FKBP51 is highly overexpr

4 team, who believed that the parotid mass and cervical adenopathy were technically resectable but that

5 A woman with mild Covid-19 developed cervical adenopathy, being diagnosed of Epstein-Barr vir

6 For HPV genotyping, cervical and anal samples were obtained at baseline and

7 (64)Cu-ATSM signal and redox state in human cervical and colon cancer cells.

8 fluencing choice of therapy and prognosis in cervical and endometrial cancers; therefore, the explora

9 rences in recurrence and patient outcomes in cervical and head and neck cancers.

10 Within the cervical and lumbar spinal enlargements, central pattern

11 ely (baseline) and up to one year (Y1) after cervical and lumbar spine surgery.

12 pillomavirus (HPV) is the causative agent of cervical and other epithelial cancers.

13 Half of HPV positive cervical and pharyngeal cancers comprised a subtype with

14 pillomavirus (HPV) is the causative agent of cervical and some other epithelial cancers.

15 and extensive adenopathy in the ipsilateral cervical and submental chains (Figs 1A and 1C), without

16 inct predominant TCR clones were detected in cervical and TLSB specimens in a woman with frequent HSV

17 Cross-sectional area of the cervical and upper thoracic cord (down to T3 level) was

18 0.89, 0.97) for pancreatic, kidney, uterine, cervical, and esophageal cancer and melanoma, respective

19 nhanced cell membrane penetration in breast, cervical, and multiple myeloma cancer cells.

20 egnancy was associated with a higher risk of cervical artery dissection (incidence rate ratio [IRR],

21 ncy was twice as common among 826 women with cervical artery dissection compared with the 826 matched

22 e women 12-42 years of age hospitalized with cervical artery dissection, defined using validated diag

23 termine whether pregnancy is associated with cervical artery dissection.

24 Activity was greater in patients with cervical, as compared to patients with lumbar spine dise

25 High resolution cervical auscultation is a very promising noninvasive me

26 on both brain (T2-FLAIR or T2-weighted) and cervical (axial T2- or T2*-weighted) MRI scans.

27 yo location along the longitudinal oviductal-cervical axis of murine uteri.

28 We collected endometrial and cervical biopsies from women using the levonorgestrel-re

29 es, provider-collected cervical samples, and cervical biopsy samples were obtained from all enrolled

30 Typical herpes lesion site biopsy (TLSB) and cervical biopsy specimens were collected at the end of t

31 ired antiviral defense is a driving force in cervical, bladder and head-and-neck carcinoma.

32 onset progressive dystonia with predominant cervical, bulbar, orofacial, and upper limb involvement.

33 contributed more than a third of the global cervical burden, with 106 000 cases in China and 97 000

34 calculated at three reference points at the cervical (C), middle (M), and occlusal (O) regions of th

35 rica, 63.8% (95% CI 58.9-68.1) of women with cervical cancer (9200 new cases, 95% CI 8500-9800) were

36 Cervical cancer (CC) remains highest in the mortality of

37 ication among patients with locally advanced cervical cancer (LACC) and paraaortic lymph node (PALN)

38 We estimate global cervical cancer 5-year net survival as 42.1% (95% uncert

39 cancer survival, we estimated stage-specific cervical cancer 5-year net survival in 200 countries and

40 ncluding self-sampling-has potential to make cervical cancer a rare disease in the decades to come.

41 We estimated the pooled risk of cervical cancer among women living with HIV across four

42 are needed for HSIL to reduce the burden of cervical cancer among women living with HIV.

43 Approximately 570 000 cases of cervical cancer and 311 000 deaths from the disease occu

44 her odds of pancreatic, kidney, uterine, and cervical cancer and lower odds of esophageal cancer and

45 elial neoplasia of grade 2 or 3 and cases of cervical cancer and noncervical HPV-associated cancer by

46 cination and the subsequent risk of invasive cervical cancer are lacking.

47 study aimed to assess the existing burden of cervical cancer as a baseline from which to assess the e

48 hape and monitor the initiative to eliminate cervical cancer as a major public health problem.

49 ng, and treatment interventions to eliminate cervical cancer as a public health problem during the 21

50 al has issued a call for action to eliminate cervical cancer as a public health problem.

51 ith a substantially reduced risk of invasive cervical cancer at the population level.

52 with approximately 6.5% of women developing cervical cancer before age 75 years.

53 n living with HIV and to estimate the global cervical cancer burden associated with HIV.

54 d updates to demography, disability weights, cervical cancer burden estimates resulted in a 26% incre

55 obal Burden of Disease (GBD) 2017 study, and cervical cancer burden from the Global Cancer Incidence,

56 Africa, where a substantial HIV-attributable cervical cancer burden has added to the existing cervica

57 hods for demography, disability weights, and cervical cancer burden, and generated revised estimates

58 ical cancer burden has added to the existing cervical cancer burden.

59 We calculated the number of cervical cancer cases and deaths in a given country, dir

60 Globally, 5.8% (95% CI 4.6-7.3) of new cervical cancer cases in 2018 (33 000 new cases, 95% CI

61 Health impact was presented in terms of cervical cancer cases, deaths, or disability-adjusted li

62 te of irinotecan (SN-38), on Trop-2 positive cervical cancer cell lines and a xenograft model.

63 r loss of expression of the two microRNAs in cervical cancer cell lines and primary tumors, indicatin

64 sing MKN-45 gastric or FOLR1-expressing HeLa cervical cancer cells confirmed noninterference of the a

65 In cervical cancer cells, human papilloma virus (HPV) prote

66 TRL-positive cancer bearing G-CSF expressing cervical cancer cells.

67 uorescent cation, Hoechst 33258 (H33258), in cervical cancer cells.

68 Cervical cancer continues to be a major public health pr

69 Australia, Centre for Research Excellence in Cervical Cancer Control, Canadian Institute of Health Re

70 Australia Centre for Research Excellence in Cervical Cancer Control.

71 en benchmarked using cell line data and TCGA cervical cancer data.

72 umans study of PLG in women with uterine and cervical cancer demonstrates its feasibility and its abi

73 PV vaccination coverage of girls can lead to cervical cancer elimination in most LMICs by the end of

74 The WHO Cervical Cancer Elimination Modelling Consortium (CCEMC)

75 The WHO Cervical Cancer Elimination Modelling Consortium (CCEMC)

76 n regions in which all countries can achieve cervical cancer elimination with girls-only vaccination,

77 mavirus (HPV) infection is the main cause of cervical cancer has resulted in the development of proph

78 eductions and will be necessary to eliminate cervical cancer in countries with the highest burden.

79 tion in the rate of premature mortality from cervical cancer in LMICs is possible, contributing to th

80 dicted to reduce the median age-standardised cervical cancer incidence in LMICs from 19.8 (range 19.4

81 ds examined were an average age-standardised cervical cancer incidence of four or fewer cases per 100

82 predefined criteria, projected reductions in cervical cancer incidence over time in 78 LMICs for thre

83 Cervical cancer is among the most common preventable can

84 ination, supporting the notion that invasive cervical cancer is preventable.

85 Cervical cancer is the fourth most common cancer among w

86 Cervical cancer is the most common cancer affecting sub-

87 f hsa-miR-34a and hsa-miR-449a expression in cervical cancer leads to overexpression of PACS-1 and su

88 We discuss the role of imaging in cervical cancer management and estimate the potential su

89 the 90-70-90 triple-intervention targets on cervical cancer mortality and deaths averted over the ne

90 of the WHO elimination strategy would reduce cervical cancer mortality by almost 99% and save more th

91 Reductions in age-standardised rates of cervical cancer mortality in 78 low-income and lower-mid

92 In 2020, the estimated cervical cancer mortality rate across all 78 LMICs was 1

93 ccination alone would have minimal impact on cervical cancer mortality, leading to a 0.1% (0.1-0.5) r

94 For estimation of cervical cancer mortality, we used the WHO mortality dat

95 fective cancer treatment is needed to reduce cervical cancer mortality.

96 represent a novel targeted therapy option in cervical cancer patients overexpressing Trop-2.

97 Clearance of surgical margins in cervical cancer prevents the need for adjuvant chemoradi

98 Cervical cancer ranked in the top three cancers affectin

99 We aimed to investigate cervical cancer risk among women living with HIV and to

100 in the self-reported lifetime prevalence of cervical cancer screening among countries within regions

101 sting may be a promising approach to improve cervical cancer screening coverage, especially among wom

102 HPV vaccination and cervical cancer screening for women living with HIV are

103 gic exams are two of the biggest barriers to cervical cancer screening for women.

104 en and treat is the recommended approach for cervical cancer screening in low-resource settings, but

105 tion of the cervix with acetic acid (VIA) as cervical cancer screening strategy in resource-poor sett

106 tion (Cervex-Brush or Cytobrush/spatula) for cervical cancer screening.

107 in the self-reported lifetime prevalence of cervical cancer screening.

108 f care could improve 5-year net survival for cervical cancer to 62.5% (57.7-67.8).

109 will also be needed to successfully scale up cervical cancer treatment worldwide.

110 Methods: Patients with cervical cancer underwent baseline and repeat (18)F-FDG

111 udy period, we evaluated girls and women for cervical cancer until their 31st birthday.

112 The cumulative incidence of cervical cancer was 47 cases per 100,000 persons among w

113 Globally, the average age at diagnosis of cervical cancer was 53 years, ranging from 44 years (Van

114 The global average age at death from cervical cancer was 59 years, ranging from 45 years (Van

115 Cervical cancer was diagnosed in 19 women who had receiv

116 umab for patients with recurrent or advanced cervical cancer was safe and treatment-related adverse e

117 Cervical cancer was the fourth most common cancer in wom

118 Cervical cancer was the leading cause of cancer-related

119 ASIRs of HIV-attributable cervical cancer were more than 20 per 100 000 in six cou

120 Mouse models of cervical cancer were used to evaluate the relationship b

121 d we assumed that 50% of women with invasive cervical cancer would receive appropriate surgery, radio

122 or advanced HPV-positive (HPV-16 or HPV-18) cervical cancer, and who had progressed after available

123 standardised incidence and mortality rate of cervical cancer, indirectly standardised incidence ratio

124 ients with recurrent or advanced, inoperable cervical cancer, who were aged 18 years or older with Ea

125 for the treatment of recurrent or metastatic cervical cancer, with an overall response rate of 14.3%.

126 of hypoxia levels in patients with tumors of cervical cancer.

127 d cancer registries to estimate incidence of cervical cancer.

128 omaviruses (HPV) are the causative agents of cervical cancer.

129 However, not all infected women develop cervical cancer.

130 and treatment for pre-invasive and invasive cervical cancer.

131 NCT02285192) in 23 patients with uterine or cervical cancer.

132 t a novel therapeutic target for HPV-induced cervical cancer.

133 ting dysregulation of these two microRNAs in cervical cancer.

134 women aged 20-29 years who were screened for cervical cancer.

135 al hysterectomy in patients with early stage cervical cancer.

136 umour activity against recurrent or advanced cervical cancer.

137 to influence the likelihood of conversion to cervical cancer.

138 port of having ever had a screening test for cervical cancer.

139 preservation of women treated surgically for cervical cancer.

140 l hysterectomy for patients with early stage cervical cancer.

141 ng the association between HIV infection and cervical cancer.

142 r patients with pancreatic, renal, lung, and cervical cancers showed that high-level expression of MC

143 HPV16 causes half of cervical cancers worldwide; for unknown reasons, most in

144 l immunisation, and screening for breast and cervical cancers) and four treatment indicators (skilled

145 Out of 147 primary cervical cancers, 113 were squamous cell carcinomas (SCC

146 n many forms of cancer, including breast and cervical cancers, were significantly increased during th

147 E6 oncoprotein plays a crucial role in cervical carcinogenesis and commonly cause the dysregula

148 ndependent, dynamic models of HPV infection, cervical carcinogenesis, screening, and precancer and in

149 ing the primary target for HPV infection and cervical carcinogenesis.

150 EC3-induced mutations in viral clearance and cervical carcinogenesis.

151 HPV was assessed in cervical cell samples by using general primer GP5+/GP6+-

152 us (HPV) genotyping assays were performed on cervical cells from 149 women with CIN2 in a 2-year coho

153 At each visit, clinicians collected cervical cells from sexually active women for cytology a

154 ability to associate with human vaginal and cervical cells in vitro but did not contribute to coloni

155 In contrast to the results from cancerous cervical cells, K(Ca)3.1-dependent H33258 uptake was rar

156 ms around the anterior articular surfaces of cervical centra and bifurcated anterior dorsal neural sp

157 increased corticospinal axon density in the cervical cord below the level of the injury relative to

158 ns (LAPNs) that inter-connect the lumbar and cervical CPGs disrupts left-right limb coupling of each

159 In order to achieve this aim, the cranio-cervical CT angiography images of patients who were refe

160 r of differentiation 38 (CD38) expression by cervical cytobrush-derived CD4+ T cells was assessed by

161 eported in monkeys that following a combined cervical dorsal root/dorsal column lesion (DRL/DCL), bot

162 inated against HPV have a lower incidence of cervical dysplasia compared to unvaccinated women.

163 ry of any anogenital cancer or treatment for cervical dysplasia, had no hysterectomy, and were not pr

164 k (22.4%) for blepharospasm, hand (3.5%) for cervical dystonia and neck for hand (12.8%) and laryngea

165 was observed in 50% of blepharospasm, 8% of cervical dystonia, 17% of hand dystonia and 16% of laryn

166 asis of gynaecological malignancies, such as cervical, endometrial and ovarian cancers, through direc

167 ular penetrations into 89 motoneurons in the cervical enlargement of four terminally anesthetized fem

168 xynol-9, as a surrogate for any mechanism of cervical epithelial damage).

169 ur results highlight the crucial role of the cervical epithelium as a barrier against ascending infec

170 sophago-gastrostomy and side-to-side stapled cervical esophagogastric anastomosis after esophagectomy

171 Side-to-side stapled cervical esophagogastric anastomosis may reduce operatio

172 sophago-gastrostomy and side-to-side stapled cervical esophagogastric anastomosis.

173 mes can lead to unnecessary biopsies or even cervical excisions.

174 mary somatosensory cortex that synapses with cervical excitatory neurons and modulates the lumbar loc

175 ck muscle fat infiltration (MFI) in the deep cervical extensor muscles (multifidus and semispinalis c

176 centrated in the medial portions of the deep cervical extensors regardless of WAD recovery, but the m

177 12], Functional Assessment of Cancer Therapy-Cervical [FACT-Cx], EuroQoL-5D [EQ-5D], and MD Anderson

178 ating over 90% of latent virus from superior cervical ganglia.

179 y, the nodose ganglion (NG) and the superior cervical ganglion (SCG) in a cohort of C57BL/6J mice.

180 Increasing superior cervical ganglion activity by activating Gq-coupled desi

181 Using unilateral superior cervical ganglionectomy in wild-type C57BL/6 mice, we sh

182 nt activity and antiproliferative effects on cervical (HeLa) and colon (HT-29) cancer cells.

183 t modes of reticulospinal fiber growth after cervical hemisection, local rewiring of axotomized proje

184 Prevalence of cervical hrHPV DNA was measured in 3 studies at 3 differ

185 ected cervicovaginal, and provider-collected cervical hrHPV results; 83 women (27%) had CIN2+.

186 in Johannesburg, South Africa diagnosed with cervical HSIL by colposcopic biopsy.

187 accination improves response to treatment of cervical HSIL.

188 gnostic and management feedback on patients' cervical images, which were reviewed weekly by nurses.

189 lascope camera meets visual requirements for cervical imaging.

190 n males and females with and without chronic cervical incomplete SCI.

191 TEMENT Although a number of individuals with cervical incomplete spinal cord injury show limited func

192 a grade 3 or worse associated with HPV 16/18 cervical infection detected at colposcopy referral.

193 We demonstrate that cervical infections with a greater burden of somatic HPV

194 using vaginal inoculation combined with mild cervical injury induced by a common spermicide (Nonoxyno

195 mmercial approaches to helping the patient's cervical instability and resulting 'head-drop' had previ

196 Pseudo-occlusion (PO) of the cervical internal carotid artery (cICA) can be caused by

197 ke projection into the lumen of the proximal cervical internal carotid artery without evidence of cal

198 n, human papillomavirus (HPV) infection, and cervical intraepithelial neoplasia (CIN); however, causa

199 (HIV) plasma viral load (PVL) on high-grade cervical intraepithelial neoplasia (CIN2+) detection at

200 Sensitivity of detecting cervical intraepithelial neoplasia grade 2 or greater in

201 of these types) were informative to predict cervical intraepithelial neoplasia grade 2 or greater.

202 cervicovaginal samples for the detection of cervical intraepithelial neoplasia grade 2 or higher (CI

203 vaccine for preventing HPV 16/18-associated cervical intraepithelial neoplasia grade 2 or worse (CIN

204 ed as histopathologically confirmed CIN2+ or cervical intraepithelial neoplasia grade 3 or worse asso

205 number of diagnoses of anogenital warts and cervical intraepithelial neoplasia of grade 2 or 3 and c

206 e is no prognostic test to ascertain whether cervical intraepithelial neoplasias (CINs) regress or pr

207 Cervical length (RR(adj) 4.52 95% CI [1.08-19.01]) was i

208 infection, and its sequelae of precancerous cervical lesions and their subsequent treatment, have be

209 virus (HPV) vaccine in preventing high-grade cervical lesions have been shown.

210 nates healthy from premalignant and invasive cervical lesions with high accuracy and can improve onco

211 four quartiles (Q1-Q4; medial to lateral) at cervical levels C4 through C7 were included.

212 The cervical LNs had a greater tumor burden and infiltration

213 x2 regulates formation of the Sox2(+) labial cervical loop (LaCL) stem cell niche in concert with two

214 ology of the labial and lingual sides of the cervical loop during early developmental stages, both at

215 n stage, highlighting the difference between cervical loop morphology during early development and di

216 a positive domain on the lingual side of the cervical loop overlapping with the SOX2 domain.

217 acromolecules and immune cells from CSF into cervical lymph nodes (CLNs).

218 Tregs from cervical lymph nodes had reduced Foxp3 expression (> 25%

219 rainage of Gd-DOTA to submandibular and deep cervical lymph nodes was demonstrated as 25-50% T1 reduc

220 mory T-helper 17 (Th17) cells in the retina, cervical lymph nodes, inguinal lymph nodes, and spleen.

221 priming of CD8 T cells in the draining deep cervical lymph nodes, migration of CD8 T cells into the

222 n patients with PTLD in the Waldeyer's ring, cervical lymph nodes, or small bowel with either nondest

223 well as drainage to the deep and superficial cervical lymph nodes.

224 proliferation of both Treg and Th17 cells in cervical lymph nodes.

225 inges drain interstitial fluid into the deep-cervical lymph nodes.

226 peripheral desquamation, strawberry tongue, cervical lymphadenopathy.

227 Indications were chylothorax (n = 19; 76%), cervical lymphatic fistula (n = 2; 8%), and combined chy

228 inspiratory motor potential amplitude and by cervical magnetic simulation.Measurements and Main Resul

229 CI}, 1.27-29.18], P = .02) and with positive cervical methylation (aOR, 6.49 [95% CI, 1.66-25.35], P

230 However, these maturative processes occur in cervical motoneurons prior to lumbar motoneurons, correl

231 ver, these characteristics mature earlier in cervical motoneurons.

232 going decompression surgery for degenerative cervical myelopathy.

233 In principle, variability in levels of cervical OS has the potential to influence the likelihoo

234 standard practice for treating patients with cervical precancer in see-and-treat programmes in low-in

235 eness of qHPV vaccination against high-grade cervical precancerous lesions, among women vaccinated wi

236 important role in the clinical management of cervical radiculopathy (CR).

237 creased epithelial cell proliferation at the cervical region of the molar in K14-Cre;Wnt10a(flox/flox

238 that these mutations are overrepresented in cervical relative to urethral isolates.

239 lected cervicovaginal and provider-collected cervical samples (both 94% [95% CI, 89 to 99%]).

240 samples (kappa = 0.58) or provider-collected cervical samples (kappa = 0.54) was moderate.

241 All women had cervical samples collected for Xpert HPV (an assay that

242 anoscopy and anal cytology and had anal and cervical samples collected.

243 d cervicovaginal samples, provider-collected cervical samples, and cervical biopsy samples were obtai

244 d (C(T) ) established for provider-collected cervical samples, but sensitivity remained below the est

245 found in 69 (95%) anal samples and 19 (26%) cervical samples.

246 s, we recorded neural activity in M1 and the cervical SC during a visuomotor tracking task, in which

247 cord in individuals with incomplete chronic cervical SCI influenced in parallel the excitability cor

248 hypothesis that the recovery of biceps after cervical SCI results, at least in part, from increased r

249 females with and without chronic incomplete cervical SCI.

250 Cervical screening involved HPV testing at age 35 years,

251 Cervical screening involved HPV testing once or twice pe

252 n routine screen-and-treat clinics providing cervical screening using visual inspection with acetic a

253 Mixed anogenital swabs and cervical secretions were self-collected by nine HIV/HSV-

254 Unexpectedly, we found in cervical segments a population of fluorescent neurons ha

255 ratinocytes isolated and cultured from these cervical specimens also displayed high variability in RO

256 acquire functional images of both brain and cervical spinal cord (CSC) simultaneously and examined t

257 ntrol subjects to undergo 7 T imaging of the cervical spinal cord and brain as well as conventional 3

258 Purpose To identify MRI features of cervical spinal cord damage that could help predict disa

259 Cervical spinal cord GM atrophy is an accurate predictor

260 pendent predictors of EDSS score in PMS were cervical spinal cord GM CSA and brain GM volume (R(2) =

261 Logistic regression analysis identified cervical spinal cord GM CSA and T2 lesion volume as inde

262 An optimal cervical spinal cord GM CSA cut-off value of 11.1 mm(2)

263 Cervical spinal cord GM lesions may subsequently cause G

264 Cervical spinal cord imaging at 7 T was used to segment

265 show that a single session of TESS over the cervical spinal cord in individuals with incomplete chro

266 corticospinal tracts from the cortex to the cervical spinal cord in patients with various disease ph

267 ween elbow flexor and extensor muscles after cervical spinal cord injury.

268 Despite the prevalence of cervical spinal cord lesions and atrophy, brain patholog

269 Cervical spinal cord lesions were mapped voxel-wise as a

270 Conclusion Cervical spinal cord MRI involvement has a central role

271 Cervical spinal cord MRI was performed with three-dimens

272 ys after dorsal column lesions (DCLs) in the cervical spinal cord relies on neural rewiring in the cu

273 MT images of the cervical spinal cord were collected parallel to the inte

274 produces homogeneous delivery throughout the cervical spinal cord white and gray matter and brain mot

275 tion of the dorsal white matter tract of the cervical spinal cord, we found that both lesioned dorsal

276 ent CT of the abdomen or pelvis or CT of the cervical spine or neck with unsuspected findings highly

277 the abdomen or pelvis and 18 CT scans of the cervical spine or neck.

278 or more of the following criteria: previous cervical surgery, cerclage, premature rupture of membran

279 t of cytokine levels; primary endpoints were cervical T cell activation, vaginal microbial diversity

280 red endogenous hormone levels and associated cervical Th17 cell frequencies to a greater extent than

281 combined oral contraceptive pills (COCPs) on cervical Th17 cells and cytokines were compared.

282 adeno-associated virus (AAV) throughout the cervical, thoracic and lumbar spinal cord, as well as br

283 nty-five individuals with chronic incomplete cervical, thoracic, and lumbar spinal cord injury were r

284 ure was significantly greater than TFV-DP in cervical tissue and was correlated with abundance of Lac

285 ctocervix and transformation zone of healthy cervical tissue.

286 e between healthy, preinvasive, and invasive cervical tissue.

287 ge biomarker 8-oxodG in normal non-cancerous cervical tissues and cells obtained from women with uter

288 7-fold greater than TFV-DP concentrations in cervical tissues.

289 was correlated with 3TC-TP concentrations in cervical tissues.

290 ificant difference in NADPH was seen between cervical tumor orthotopic implants in vivo, without a co

291 Cervical tumorigenesis is characterized by a multifactor

292 op-2 expression was evaluated in 147 primary cervical tumors by immunohistochemistry, real-time polym

293 tures extracted from (18)F-FDG PET images of cervical tumors.

294 CS-1, a cytosolic sorting protein in primary cervical tumors.

295 s had an examination including collection of cervical/vaginal and anal specimens, followed by high-re

296 with up to 16 recording carbon fibers in the cervical vagus nerve of 22 isoflurane-anesthetized rats.

297 ite being composed of only 13 hyper-elongate cervical vertebrae [1-8].

298 so, elongated prezygapophyseal facets on the cervical vertebrae and a specialized first dorsal verteb

299 own for their very characteristic, elongated cervical vertebrae.

300 8, generalised; 34, segmental; and 32, focal cervical) were included in this comprehensive analysis.