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1 receptors for phrenic LTF are located in the cervical spinal cord.
2 ibres after injury to these axons in the mid-cervical spinal cord.
3 rge motoneurons in the medulla oblongata and cervical spinal cord.
4  sensory tracts from the lumbar to the upper cervical spinal cord.
5 lumbar and sacral segments and the lowest in cervical spinal cord.
6 em preceded PrP(Sc) detection in the rostral cervical spinal cord.
7 axonal decussation and projection beyond the cervical spinal cord.
8 n into the C(1) and C(2) levels of the upper cervical spinal cord.
9 resent in the lumbar spinal cord than in the cervical spinal cord.
10  scores and transverse sectional area of the cervical spinal cord.
11 ganisation of the corticospinal tract in the cervical spinal cord.
12  nervous system development, focusing on the cervical spinal cord.
13  recurrent intramedullary PNET affecting the cervical spinal cord.
14 t that projects to the ventral region of the cervical spinal cord.
15 cipal trigeminal nucleus and caudally to the cervical spinal cord.
16  applied dorsally to the C2-C6 region of the cervical spinal cord.
17 mediated by NK1R+/SST+ neurons in the dorsal cervical spinal cord.
18 cipants underwent 3.0-T MRI of the brain and cervical spinal cord.
19 n of metabolic changes in the brain stem and cervical spinal cord.
20 ury (SCI) by applying MR spectroscopy in the cervical spinal cord.
21 n-like c-Fos immunoreactivity pattern in the cervical spinal cord.
22 er afferents onto common interneurons in the cervical spinal cord.
23 athways via retrograde tracing from the high cervical spinal cord.
24  in CST cell bodies and in axon terminals in cervical spinal cord.
25 nctionally integrate in the circuitry of the cervical spinal cord.
26 he prefrontal cortex and the lamina X of the cervical spinal cord.
27 icacy after transplantation into the injured cervical spinal cord.
28 n of SCI patients suffer contusion trauma to cervical spinal cord.
29  as they ascend in the dorsal columns of the cervical spinal cord.
30 nt of their physiological actions within the cervical spinal cord.
31 aptic responses require a bilaterally intact cervical spinal cord.
32 bral hemispheres, brainstem, cerebellum, and cervical spinal cord.
33 n content was elevated in the Gaa(-/-) mouse cervical spinal cord.
34 ion of both the white and grey matter of the cervical spinal cord.
35  MRI abnormalities were predominantly in the cervical spinal cord (103/118).
36 s into the contralesional gray matter of the cervical spinal cord administered 28 d after stroke indu
37 to restrict corticospinal sprouting in mouse cervical spinal cord after unilateral pyramidotomy.
38 ntrol subjects to undergo 7 T imaging of the cervical spinal cord and brain as well as conventional 3
39 cit movements and had few connections to the cervical spinal cord and distinctive cyto- and immunoarc
40 es, we demonstrated similar pathology in the cervical spinal cord and greater accumulation of glycoge
41  better corticospinal tract integrity in the cervical spinal cord and improved neurological outcomes
42 e growth of corticospinal tract axons in the cervical spinal cord and then use specific optogenetic a
43 e degeneration of motoneurons, mostly in the cervical spinal cord, and in the brain stem cranial moto
44              Thirty of them were acquired in cervical spinal cord, and the remaining 18 spectra were
45 stem-spinal and segmental projections to the cervical spinal cord are present at birth, skilled forel
46 in the midbrain/pons, medulla oblongata, and cervical spinal cord as defined on the individual's MRI
47 on tensor imaging of the mouse brainstem and cervical spinal cord at 117 mum x 117 mum in-plane resol
48  subjects were prospectively acquired in the cervical spinal cord at C2 above the level of injury bet
49                     Strikingly, we find that cervical spinal cord Atoh1-lineage neurons form mainly t
50 s of MWF and f(M) in healthy human brain and cervical spinal cord (available online) and compared the
51 ctive for Zif268 protein were not present in cervical spinal cord before postnatal day (P) 6.
52 ted that corticospinal axons reach the lower cervical spinal cord by 24 weeks post-conceptional age (
53 enshaw cells, identified in human postmortem cervical spinal cord by their morphology, location and c
54 R mRNA in the adult rat and mouse brains and cervical spinal cords by using ISHH with novel cRNA prob
55 R and LE immunoreactivities in the adult rat cervical spinal cord (C3-C5).
56 BDA-labeled terminals in the ventral horn of cervical spinal cord (C4-C5) were immunoreactive for enk
57 lly sectioned dorsal column afferents in the cervical spinal cord (C4-C6) in adult squirrel monkeys.
58 sly that epidural stimulation of the lateral cervical spinal cord can evoke tactile sensations percei
59 ocal glucose hypermetabolism at the level of cervical spinal cord compression may predict an improved
60  humans, and we hypothesized that EES of the cervical spinal cord could antagonize opioid-induced res
61 vated by designer drugs (DREADDs) in the mid-cervical spinal cord could effectively stimulate phrenic
62 l and neuronal injury in mouse brainstem and cervical spinal cord could improve our understanding of
63  acquire functional images of both brain and cervical spinal cord (CSC) simultaneously and examined t
64                                       In the cervical spinal cord, CSP axons from S2 and M1 partly co
65          Purpose To identify MRI features of cervical spinal cord damage that could help predict disa
66 al column somatosensory pathway in the upper cervical spinal cord deactivates neurons in the hand reg
67 activity is similarly plastic in human lower cervical spinal cord development, with many changes occu
68 al nerve 12) and lateral motor column of the cervical spinal cord, displaying differential vulnerabil
69 asures and macromolecular MRI metrics in the cervical spinal cord, distal to the site of injury.
70                                              Cervical spinal cord, DRG, and forepaw skin were removed
71 nd strengthening of existing circuits in the cervical spinal cord due to a combination of afferent ta
72 ally applied electrical stimulation over the cervical spinal cord during structured rehabilitation.
73 at C3, with or without a transplant of fetal cervical spinal cord (embryonic day 14); unoperated pups
74  deficit; in contrast to forebrain, fyn(-/-) cervical spinal cord exhibited no reduction in myelin co
75 Epidural electrical stimulation (EES) at the cervical spinal cord facilitated motor activity in roden
76  to map functional connectivity of the human cervical spinal cord from C1 to C4 at 1 x 1 x 3-mm resol
77 he volume of brainstem, cerebellum and upper cervical spinal cord from three-dimensional MRIs acquire
78 d structural and quantitative imaging of the cervical spinal cord from which we calculated magnetizat
79                                              Cervical spinal cord GM atrophy is an accurate predictor
80 pendent predictors of EDSS score in PMS were cervical spinal cord GM CSA and brain GM volume (R(2) =
81      Logistic regression analysis identified cervical spinal cord GM CSA and T2 lesion volume as inde
82                                   An optimal cervical spinal cord GM CSA cut-off value of 11.1 mm(2)
83                                              Cervical spinal cord GM lesions may subsequently cause G
84 al funiculi FA, normalized brain volume, and cervical spinal cord GM T2 lesion volume (R(2) = 0.51).
85 multivariable analysis identified phenotype, cervical spinal cord gray matter (GM) cross-sectional ar
86  differed at various CNS regions in that the cervical spinal cord had the greatest rate of influx, wh
87                                              Cervical spinal cord imaging at 7 T was used to segment
88  distinct upper extremity movements onto the cervical spinal cord in accordance with established myot
89  show that a single session of TESS over the cervical spinal cord in individuals with incomplete chro
90 egional changes of glucose metabolism of the cervical spinal cord in patients with degenerative cervi
91 s clinico-pathological study we examined the cervical spinal cord in patients with primary and second
92  corticospinal tracts from the cortex to the cervical spinal cord in patients with various disease ph
93  electrodes over motor cortex and the dorsal cervical spinal cord in rats; motor evoked potentials (M
94 , we found neurons in the dorsal horn of the cervical spinal cord in which c-Fos and pseudorabies wer
95 ather patterns could predict the severity of cervical spinal cord injuries (CSCI) across the United S
96 ncy is the leading cause of death after high-cervical spinal cord injuries (SCIs).
97 es recovery of manual dexterity in rats with cervical spinal cord injuries.
98                To investigate the effects of cervical spinal cord injury (C-SCI) on cough reflex sens
99  lower limb muscles after chronic incomplete cervical spinal cord injury (iSCI), but the underlying m
100 -70 years with acute, complete or incomplete cervical spinal cord injury (neurological level of injur
101                                              Cervical spinal cord injury (SCI) alone did not affect b
102                                    Traumatic cervical spinal cord injury (SCI) can result in debilita
103                                              Cervical spinal cord injury (SCI) causes devastating los
104 ger muscle in humans with chronic incomplete cervical spinal cord injury (SCI) compared with age-matc
105                Respiratory dysfunction after cervical spinal cord injury (SCI) has not been examined
106                             High-thoracic or cervical spinal cord injury (SCI) is associated with sev
107                                              Cervical spinal cord injury (SCI) leads to permanent imp
108 accuracy of prognostication in patients with cervical spinal cord injury (SCI) needs to be improved.
109                                  Humans with cervical spinal cord injury (SCI) often recover voluntar
110                        Most individuals with cervical spinal cord injury (SCI) show increased muscle
111 s, as well as in monkeys subjected to either cervical spinal cord injury (SCI), Parkinson's disease (
112                                        After cervical spinal cord injury (SCI), people often recover
113                 By 2 months after unilateral cervical spinal cord injury (SCI), respiratory motor out
114 ortical representations reorganize following cervical spinal cord injury (SCI).
115  preserved in humans with incomplete chronic cervical spinal cord injury (SCI).
116  of diaphragm paralysis associated with high cervical spinal cord injury (SCI).
117 ts with subcortical damage due to incomplete cervical spinal cord injury (SCI).
118 ss of T1-weighted images in 10 subjects with cervical spinal cord injury and 16 controls.
119 ls in 15 individuals with chronic incomplete cervical spinal cord injury and 17 uninjured participant
120 ped a low-cost portable BMI for survivors of cervical spinal cord injury and investigated it as a mea
121 mediated respiratory recovery following high cervical spinal cord injury and that activation of intra
122 ensory cortex of two human participants with cervical spinal cord injury and varied the stimulus ampl
123  This effect was just as strong in rats with cervical spinal cord injury as in uninjured rats, demons
124                                              Cervical spinal cord injury at birth permanently disrupt
125 te whether baclofen use and paralysis due to cervical spinal cord injury change the contractile prope
126                                     Neonatal cervical spinal cord injury disrupts not only locomotion
127 6-year-old man with tetraplegia secondary to cervical spinal cord injury enrolled in the ongoing Brai
128 d replacement from 7 weeks to 7 months after cervical spinal cord injury in four adult rhesus monkeys
129 erent acute anti-inflammatory treatments for cervical spinal cord injury in rats.
130  study were to determine the consequences of cervical spinal cord injury on forelimb motor function a
131                                The extent of cervical spinal cord injury was measured on MR images ob
132  20, 2022, 463 patients with acute traumatic cervical spinal cord injury were screened, 334 were deem
133 indings of an individual with traumatic high-cervical spinal cord injury who coordinated reaching and
134 as investigated in an individual with a high cervical spinal cord injury, a 5-year absence of nearly
135 People with chronic tetraplegia, due to high-cervical spinal cord injury, can regain limb movements t
136 ese observations suggest that after neonatal cervical spinal cord injury, embryonic transplants suppo
137 rgo lesion-dependent plasticity after either cervical spinal cord injury, Parkinson's disease-like sy
138                     Here we show that, after cervical spinal cord injury, the expression of chondroit
139 ween elbow flexor and extensor muscles after cervical spinal cord injury.
140  of the diaphragm is a severe consequence of cervical spinal cord injury.
141 intains recovery for prolonged periods after cervical spinal cord injury.
142  of skilled forelimb activity after neonatal cervical spinal cord injury.
143 c avenue for people with chronic, incomplete cervical spinal cord injury.
144 S-evoked percepts in three participants with cervical spinal cord injury.
145  function in people with chronic, incomplete cervical spinal cord injury.
146 n a study participant with quadriplegia from cervical spinal cord injury.
147 targeted multisegmental cell delivery to the cervical spinal cord is a promising therapeutic strategy
148                Electrical stimulation of the cervical spinal cord is gaining traction as a therapy fo
149                               In the rostral cervical spinal cord, labelled axons projecting to the l
150                Three-day-old rats received a cervical spinal cord lesion at C3, with or without a tra
151        We also measured brain lesion volume, cervical spinal cord lesion number and cross-sectional a
152 ng neurotrophin-3 (NT-3) within and beyond a cervical spinal cord lesion site grafted with autologous
153 n atrophy (whole brain and gray matter), and cervical spinal cord lesions (T2LV) and atrophy.
154                    Despite the prevalence of cervical spinal cord lesions and atrophy, brain patholog
155 tissue bridges at the epicenter of traumatic cervical spinal cord lesions in 24 subacute tetraplegic
156 netic resonance imaging of the brainstem and cervical spinal cord lesions resulting from damage to LM
157                                              Cervical spinal cord lesions were mapped voxel-wise as a
158  relevant pain circuitry locations following cervical spinal cord level (C)5/6 contusion (using both
159 tion activated SST-expressing neurons in the cervical spinal cord, likely interneurons, that communic
160 gravir and tenofovir), CSF (lamivudine), and cervical spinal cord/meninges (efavirenz); the lowest we
161  2-fold more CST axons decussated across the cervical spinal cord midline (approximately 12,000 axons
162                         Here we characterise cervical spinal cord morphometric abnormalities in SCA1,
163 gulation nor neuronal death of brainstem and cervical spinal cord motor neurons and have markedly red
164                                   Conclusion Cervical spinal cord MRI involvement has a central role
165                                              Cervical spinal cord MRI was performed with three-dimens
166 ists transactivate TrkB receptors in the rat cervical spinal cord near phrenic motoneurons, thus indu
167 njections in lateral rectus eye muscle label cervical spinal cord neurons closely connected to abduce
168  investigated the functional response in the cervical spinal cord of 18 healthy human subjects (aged
169 heximide) were injected intrathecally in the cervical spinal cord of anesthetized rats.
170 stigated using immunocytochemical methods in cervical spinal cord of neonatal and adult rats.
171  tactile and nociceptive heat stimuli in the cervical spinal cord of nonhuman primates.
172 e the grey and white matter pathology in the cervical spinal cord of patients with early relapsing-re
173  brain hypermetabolism in the brain stem and cervical spinal cord of patients within the amyotrophic
174 ium bromide into the dorsal funiculus of the cervical spinal cord of the rat to induce zones of demye
175 of differentiating into astrocytes--into the cervical spinal cord of WT rats to reveal how mutant ast
176 nstrated that after transplantation into the cervical spinal cords of adult mice with severe combined
177 o astrocytes and reduced microgliosis in the cervical spinal cords of SOD1(G93A) rats.
178 thway were made in adult rats in the rostral cervical spinal cord or caudal medulla.
179                Three-day-old rats received a cervical spinal cord overhemisection with or without tra
180 nistration of the same doses of DAMGO to the cervical spinal cord produces a suppression of withdrawa
181 us system; necrotizing focal myelitis in the cervical spinal cord; radiculitis; neuritis and demyelin
182       From postmortem studies of eight human cervical spinal cords ranging in age from 11 to 35 weeks
183 stem cell transplantation into lumbar and/or cervical spinal cord regions in amyotrophic lateral scle
184 ys after dorsal column lesions (DCLs) in the cervical spinal cord relies on neural rewiring in the cu
185                  We transplanted GRPs around cervical spinal cord respiratory motor neuron pools, the
186     Persons with long-standing injury to the cervical spinal cord resulting in complete or partial pa
187 termittent hypoxia induces plasticity in the cervical spinal cord, resulting in enhanced inspiratory
188 oracic segment and below, transection of the cervical spinal cord results in severely compromised exp
189 his, we utilised a collection of post-mortem cervical spinal cord samples and investigated seven DCM
190  objective of this study was to characterize cervical spinal cord (SC) changes in asymptomatic C9orf7
191 21 [49.2%], including one lesion observed at cervical spinal cord [SC] MRI) lesions.
192                               Compression of cervical spinal cord secondary to cervical spondylosis o
193  Microinjections of glutamate into the upper cervical spinal cord significantly reduced (to 57% of co
194 ol involves similar circuits, located in the cervical spinal cord, suggesting that EES could also imp
195 with access to spinal motoneurons in primate cervical spinal cord that receive inputs from the periph
196 somatostatin (SST)-expressing neurons in the cervical spinal cord that were activated (c-Fos-positive
197                                       In the cervical spinal cord the number of corticospinal axons o
198  of ascending dorsal column afferents in the cervical spinal cord, the hand representation in the con
199 al nucleus and dorsal horns of the C(1/)C(2) cervical spinal cord: the trigeminocervical complex.
200 ontralateral red nucleus and the ipsilateral cervical spinal cord; this axonal plasticity is enhanced
201 occurred onto medial motoneuron pools in the cervical spinal cord; this sprouting was paralleled by f
202 inations of persons with acute injury to the cervical spinal cord to examine the time post-injury at
203 hondroitinase ABC was microinjected into the cervical spinal cord to induce localized plasticity of f
204 upper thoracic spinal segments relays in the cervical spinal cord to inhibit activity of lumbar spino
205  of nongenomic estrogen signaling within the cervical spinal cord to recover respiratory neuroplastic
206 , and the trigeminal sensory system from the cervical spinal cord to the midbrain.
207                              In decerebrate, cervical spinal cord-transected male rats, the lumbosacr
208          Spinal L6-S2 dorsal horn neurons of cervical spinal cord-transected, decerebrate female rats
209 after 6.2 pmol), and completely abolished by cervical spinal cord transection at the C6 level.
210                                    Following cervical spinal cord transection, NaCN and also dinitrop
211 othetical scenario of a 2-yr old with a high cervical spinal-cord transection.
212 D), direct spinal connections from the upper cervical spinal cord (UC; C1 and C2) and the cervical en
213 esonance images from the mouse brainstem and cervical spinal cord using the actively decoupled, anato
214 iated virus type 8 (AAV8)-Gfa2 vector to rat cervical spinal cord ventral horn for targeting focal as
215                     Reducing ephrinB2 in the cervical spinal cord ventral horn via viral-mediated shR
216 es respiratory-related rhythms recorded from cervical spinal cord ventral roots.
217 whether NPCs can be delivered to the injured cervical spinal cord via lumbar puncture using a mixed p
218                                        Human cervical spinal cord was derived at autopsy from 54 pati
219  tracts of the dorsolateral funiculus of the cervical spinal cord was investigated in cats.
220              The volume of the upper (C1-C3) cervical spinal cord was significantly correlated with a
221 tion of the dorsal white matter tract of the cervical spinal cord, we found that both lesioned dorsal
222 structural MRI of both the brainstem and the cervical spinal cord, we were able to identify a number
223                             MT images of the cervical spinal cord were collected parallel to the inte
224 irst experiments, slices of embryonic day 16 cervical spinal cord were cultured for one, two or three
225 numbers of neurons in the dorsal horn of the cervical spinal cord were labeled, especially ipsilatera
226 produces homogeneous delivery throughout the cervical spinal cord white and gray matter and brain mot
227 ing-state functional MR imaging of the human cervical spinal cord with a 3.0-T clinical MR imaging un
228 re, we combined a computational model of the cervical spinal cord with experiments in macaque monkeys
229                         Neurons in the upper cervical spinal cord, with descending propriospinal proj
230 ibution of multiple sclerosis lesions in the cervical spinal cord, with respect to clinical status.
231 97% in the lumber spinal cord and 91% in the cervical spinal cord, without changing the number of Opr
232 that pairing stimulation of motor cortex and cervical spinal cord would strengthen motor responses th

 
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