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1 ative agent was a previously-uncharacterised cestode.
2 e instead of definitive hosts, as in taeniid cestodes?
3 tention to the situation that the genomes of cestodes and trematodes have lost the piwi and vasa gene
6 a, one microsporidium, five nematodes, three cestodes, and one trematode) were included in the negati
9 d by the tissue-dwelling larval stage of the cestode Echinococcus granulosus, is characterised by nor
11 e-threatening zoonotic disease caused by the cestode Echinococcus vogeli, is endemic in the Amazon.
12 phenotype that contributes to suppression of cestode growth and viability but also imposes a substant
15 mogenate and excretory/secretory products of cestode larvae have an acute excitatory effect on neuron
17 al disease produced by this newly recognised cestode may not be limited to immunodeficient hosts.
18 er (LL), which protects the larval stages of cestodes of the genus Echinococcus We show that particle
21 or similar reasons, its coevolution with the cestode parasite Schistocephalus solidus, its interactio
24 tates of human infection with the intestinal cestode Raillietina, a parasite primarily of rodents and
27 s revealed that the larval homogenate of the cestodes Taenia crassiceps and Taenia solium contained h
29 with Hodgkin disease, we identified a larval cestode with a previously uncharacterized 18S rDNA seque