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1 on using sequence polymorphisms induced by a chemical mutagen.
2 increased to within an order of magnitude of chemical mutagens.
3 xposed to ionizing radiation and a number of chemical mutagens.
4 g the utility of non-null alleles created by chemical mutagens.
5 including via the handling and metabolism of chemical mutagens.
6 erfering particles, (ii) the presence of the chemical mutagen 5-fluorouracil (5-FU), or (iii) an incr
7 12 times in MRC-5 cells in the presence of a chemical mutagen, 5-fluorouracil.
8 op skin lesions following treatment with the chemical mutagen 9,10-dimethyl-1,2-benzanthracene, or th
9 or investigating the effects of physical and chemical mutagens and for exploring the potential of mut
10 hotriester (PTE) lesions that are induced by chemical mutagens and refractory to DNA repair have not
11 f mutational changes can be used to identify chemical mutagens and to explore the molecular mechanism
12           Rat2 fibroblasts were treated with chemical mutagens, and individual virus-resistant clones
13                     We performed an unbiased chemical mutagen-based genetic suppressor screen to iden
14 - to 30-fold in cells by gamma radiation and chemical mutagens but not by UV treatment.
15                Mutations that are induced by chemical mutagens can occur anywhere in the genome.
16 ethal alleles created by either radiation or chemical mutagens (Ednrb27Pub, Ednrb17FrS, Ednrb1Chlc, a
17 e treated with physical mutagen (gamma-rays) chemical mutagen (EMS) and their combinations.
18 proximately one-ninth that observed with the chemical mutagen ENU in zebrafish.
19        Phenotype-driven approaches using the chemical mutagen ethylnitrosourea (ENU) represent a pote
20 re an insufficiently characterized source of chemical mutagens for consumers.
21 r stomatitis virus (VSV) upon treatment with chemical mutagens have been investigated.
22                Forward genetic screens using chemical mutagens have been successful in defining the f
23 unodeficiency virus type 1 (HIV-1) by use of chemical mutagens [i.e., 5-azacytidine (5-AZC)] as well
24 exposure of each of these strains to several chemical mutagens in the lab revealed that strains vary
25 oses of alpha radiation, gamma radiation, or chemical mutagens in the presence and absence of caffein
26                                         Four chemical mutagens, including ethyl methane sulfonate, et
27 , the killing of a microbial pathogen with a chemical mutagen, is a potential broad-spectrum antivira
28 reen of recessive mutations generated by the chemical mutagen n-ethyl-n-nitrosourea (ENU) mapped a ne
29 f spermatogonial (premeiotic) cells with the chemical mutagen N-ethyl-N-nitrosourea (ENU).
30 ations was isolated after treatment with the chemical mutagen N-methyl-N'-nitro-N-nitrosoguanidine.
31                                          The chemical mutagen of choice for mouse has been N-ethyl-N-
32 be generated with ultraviolet (UV) light and chemical mutagens, or by insertional mutagenesis.
33 in wild-type E. coli in <24 h, outperforming chemical mutagens, ultraviolet light and the mutator str
34 iently by densely ionizing radiation than by chemical mutagens, x-rays, or endogenous aging processes