ライフサイエンスコーパス: chemokine

1 undance of CXCL10, a pro-inflammatory T-cell chemokine.

2 t to which the receptor N terminus binds the chemokine.

3 uired to induce the expression of this major chemokine.

4 of cancer-promoting growth factors, such as chemokines.

5 recombination, coordinated by cytokines and chemokines.

6 mmunological functions such as cytokines and chemokines.

7 expression of pro-inflammatory cytokines and chemokines.

8 5) with higher plasma levels of inflammatory chemokines.

9 f immune regulatory genes and genes encoding chemokines.

10 rsor protein, cadherins, growth factors, and chemokines.

11 noglobulin free light chains; cytokines; and chemokines.

12 rently coupled with inflammation mediated by chemokines.

13 production of immune factors, cytokines and chemokines.

14 ed by site-specific directional cues such as chemokines.

15 nd STS-induced proinflammatory cytokines and chemokines.

16 production of proinflammatory cytokines and chemokines.

17 little affected by the absence of major T(M) chemokines.

18 resence or absence of blocking antibodies or chemokines.

19 Plasma C-X-C motif chemokine 10 (CXCL10) concentrations changed by mean = -

20 e correlated strongly with local C-X-C motif chemokine 10 levels.

21 appearance of proinflammatory cytokines and chemokines, along with the appearance of extracellular h

22 Highly potent chemokine analog inhibitors act via the modulation of re

23 ased endothelium had increased expression of chemokine and alarmin genes including IL33.

24 Enhanced chemokine and alarmin production, and seeding of the ski

25 ers of antigen-presenting cells and impaired chemokine and alarmin production.

26 iosynthetic enzymes integrate signals from a chemokine and cAMP to specify the spatiotemporal mobiliz

27 The chemokine and cytokine microenvironment of ES closely re

28 o compare the characteristics, outcomes, and chemokine and cytokine response in transplant recipients

29 nduced costimulatory molecule expression and chemokine and cytokine secretion compared with monocyte-

30 e-matched subjects, were collected to assess chemokine and integrin receptor levels on monocytes.

31 Further, the chemokine and neutrophil chemoattractant, CXCL1, drove t

32 easing evidence emphasizes the importance of chemokines and chemokine receptors as regulators of bone

33 K J4) in BMDC led to decreased production of chemokines and cytokines associated with the inflammator

34 rophage lineage play a key role in providing chemokines and cytokines for the localization, different

35 acrophage recruitment and activation via key chemokines and cytokines.

36 ompanied by an increase in the expression of chemokines and cytokines.

37 inflammatory and other cytokines, as well as chemokines and growth factor concentrations, were not as

38 f different forms of RD: selected cytokines, chemokines and growth factors are upregulated in the vit

39 matrix components and adhesion molecules to chemokines and growth factors.

40 I and III interferons juxtaposed to elevated chemokines and high expression of IL-6.

41 inocytes massively expressed proinflammatory chemokines and immunomodulatory proteins in a cell-auton

42 domly on the sinus floor independent of both chemokines and integrins.

43 MEGF11 upregulated the expression of various chemokines and proinflammatory cytokines via AKT activat

44 markers and elevated markers of inflammatory chemokines and proliferation.

45 onse resulted in fewer eosinophil-attracting chemokines and reduced eosinophil recruitment into the l

46 stemic production of inflammatory cytokines, chemokines and specific humoral IgM and IgG responses.

47 the increased production and release of cyto/chemokines and the development of long-lasting CD8 T cel

48 Chemokines and their receptors are orchestrators of cell

49 Chemokines and their receptors play important roles in v

50 nteractions among proinflammatory cytokines, chemokines, and cancer cell-recruited neutrophils result

51 N structure with increased HEVs, upregulated chemokines, and cell adhesion molecules, and led to grea

52 Higher levels of IL-17 receptor, CXCR2 chemokines, and CXCR2 were observed in tumors generated

53 e a swift and ample production of cytokines, chemokines, and cytotoxic molecules.

54 ammatory cytokines and neutrophil-attracting chemokines, and enhanced pulmonary leukocyte accumulatio

55 ly kill target cells by producing cytokines, chemokines, and granzymes.

56 their ability to secrete various cytokines, chemokines, and growth factors, initiating and perpetuat

57 ars) were analyzed for IgE and 33 cytokines, chemokines, and growth factors.

58 lants induced mRNAs for >40 to 50 cytokines, chemokines, and receptors.

59 njections of neutralizing antibodies against chemokines, antagonists, or control antibodies.

60 High levels of Ccr2-chemokines are a feature of regenerating aged muscle.

61 Select cytokines and chemokines are certainly important in the regulatory res

62 Chemokines are important protein-signaling molecules tha

63 trate that S100A8/A9 serum levels along with chemokines are useful in distinguishing between ATB and

64 arachidonic acid metabolites, cytokines, and chemokines as compared to healthy macrophages.

65 oV-2 infection caused striking expression of chemokines, as also seen in primary human COVID-19 pulmo

66 owed a greater capacity to secrete cytokines/chemokines associated with help for B cell activation, m

67 classical monocytes overexpressed a distinct chemokine axis, which may orchestrate inflammatory myelo

68 re we show that peptides possessing multiple chemokine-binding and anti-inflammatory activities can b

69 o Identification and characterization of the chemokine-binding interface of evasins could thus inspir

70 2 (CCL2) with an atypical chemokine receptor chemokine-binding protein 2 variant V41A (ACKR2-V41A; rs

71 ycosylated interferon-gamma fragment and the chemokine-binding protein UL22A, respectively.

72 mmatory activities can be developed from the chemokine-binding site of an evasin.

73 s ability to promote production of the CXCL1 chemokine by keratinocytes, resulting in neutrophil recr

74 tion of pro-inflammatory ROS, cytokines, and chemokines by LPS-activated neutrophils.

75 CCR4 (Ccr4), associated with high levels of chemokine C-C motif ligands 17 and 22.

76 in challenge, was accompanied by doubling of chemokine (C-C motif) ligand 21 in lung lymphatics and t

77 -regulated expression of the B cell-directed chemokine (C-C motif) ligand 5.

78 accumulation precede or are independent from chemokine (C-C motif) ligand-CCR2 signaling in the devel

79 , there was significantly increased mRNA for chemokine (C-C motif) ligands 18 and 13 and major collag

80 apeutic properties of natural and engineered chemokine (C-X-C motif) receptor 4 (CXCR4) agonists in a

81 ajor neutrophil-recruiting chemokines (e.g., chemokine [C-X-C motif] ligand 1 [CXCL1] and interleukin

82 In both cell types, chemokines can transduce signals that convert integrin a

83 sion of MEGF11 induces both a cytokine and a chemokine cascade, which will favour the tumour microenv

84 T(M), are a prodigious source for six select chemokines (CCL1/3/4/5, CCL9/10, and XCL1) that collecti

85 g) and the expression of the Treg-attracting chemokine Ccl17 by MHCII(high) tumor-associated macropha

86 Marked reductions in the chemokine Ccl2 and the pro-inflammatory cytokine Tnfalph

87 owed significantly reduced expression of the chemokine CCL2.

88 cell-derived GM-CSF as a key inducer of the chemokine CCL22 and thus, to our knowledge, identify a n

89 help by repressing expression of the T cell chemokines CCL3 and CCL4, as well as CD86 and ICAM1.

90 ine (IL-1alpha, IL-1beta, and TNF-alpha) and chemokine (CCL3 and CCL4) production.

91 to increased expression of the inflammatory chemokines CCL5 and CXCL10 in polycystic kidneys and cul

92 he secretion of Th17 and monocyte-recruiting chemokines chemokine (C-C motif) ligand (CCL)-20 and CCL

93 Inflammatory cytokine/chemokine concentrations were determined using a multipl

94 Antiretrovirals and cytokines/chemokines concentrations were compared at DS and contro

95 ositely charged residues in the receptor and chemokine, confirming the accuracy of the predicted orie

96 regulated the expression of CX3CL1, a unique chemokine constitutively produced by neurons to suppress

97 s with CYR61 in vivo point to it being a key chemokine controlling liver fibrosis and inflammation in

98 Whether plasma chemokines could also serve as biomarkers of unfavorable

99 ed increased expression of key cytokines and chemokines critical for neutrophil and monocyte recruitm

100 T cells are both responsive to various chemokine cues and a relevant source for certain chemoki

101 The soluble chemokine, CX3CL1 (fractalkine), is cleaved from membran

102 n of PAR1, CXCR4, and PAR1:CXCR4 heteromers, chemokine (CXC motif) ligand 12 stimulation reduced surf

103 ge in monocytes stimulated production of the chemokine CXCL-11, which suppressed T cell proliferation

104 produce representative cytokines (IL-6) and chemokines (CXCL-1), respectively.

105 and consequent secretion of pro-inflammatory chemokine, CXCL1.

106 We quantified serum concentrations of chemokine CXCL10 in 288 patients with measles virus (MeV

107 y cytokines (TNF-alpha, IL-2, IL-12p70), and chemokines (CXCL10, C-C motif chemokine ligand [CCL]2, C

108 Here, we report that the chemokine CXCL12 recoups both cognitive performance and

109 The chemokine CXCL13 controls the normal organization of sec

110 Here we show that the chemokine CXCL13 forms both soluble and immobilized grad

111 eased plasma levels of the B-cell-attracting chemokine CXCL13.

112 sion included those encoding proinflammatory chemokines (CXCL5, CCL20, CXCL13, and CCL18), cytokines

113 OAS1-3 and IFIT1-3 and T helper type 1 (Th1) chemokines CXCL9/10/11, as well as a reduction in transc

114 Different molecular chemokine/cytokine adjuvants effected significant altera

115 mplement, phagocytosis, Ag presentation, and chemokine/cytokine pathways.

116 ding a model Ag HIV-1 Env gp140 and selected chemokines/cytokine and boosted intravaginally with gp14

117 ne responses by activating the expression of chemokines, cytokines, and antimicrobial peptides involv

118 M expression did not regulate differences in chemokines, cytokines, or adhesion molecules associated

119 e quantification of 13 proinflammatory human chemokines/cytokines.

120 nes (B), mixed type-1/2/3 cytokines (C), and chemokines (D) that correlated with three distinct disea

121 Using CXCL12 as a model CXC chemokine, deletion of the X residue (Pro-10) had little

122 ilot-less: they are unable to migrate toward chemokines despite their normal ability to move randomly

123 ounced increase of XCL1 production capacity; chemokines dominate the earliest stages of the CD8(+)T(M

124 e but was reproduced in mice by preinfection chemokine-driven airway recruitment of neutrophils, whic

125 up-regulation of major neutrophil-recruiting chemokines (e.g., chemokine [C-X-C motif] ligand 1 [CXCL

126 reted large amounts of several cytokines and chemokines, especially tumor necrosis factor alpha (TNF-

127 types, including novel mTEC subsets, such as chemokine-expressing and ciliated TEC, which warrant fur

128 , including eosinophils, increasing cytokine/chemokine expression and mucus production, thus demonstr

129 mutation was linked to modestly impaired CXC chemokine expression and neutrophil recruitment to the i

130 aining to the temporospatial organization of chemokine expression patterns, synthesis and secretion k

131 irect role of IgE in AAA by promoting lesion chemokine expression, inflammatory cell accumulation, MA

132 -2 infection, and showed robust induction of chemokines following SARS-CoV-2 infection, similar to wh

133 e secretome demonstrated a distinct cytokine/chemokine footprint from the naive monocyte, and that TN

134 -X-C motif chemokine ligand 1 (CXCL1), a key chemokine for neutrophil infiltration (a hallmark of NAS

135 H3K4me3 marks on interleukin, defensin, and chemokine gene promoters, facilitating a rapid inflammat

136 CXC chemokine genes showed the highest degrees of connectivi

137 e CXCR4 N terminus, a major component of the chemokine-GPCR interface.

138 tages, including precis e control of defined chemokine gradients in space and time, automated quantit

139 d by potentially enhancing T cell sensing of chemokine gradients.

140 m the other clusters as being a "skin-homing chemokines/IL-1R1-dominant" cluster, whereas cluster B (

141 s differ from each other and from the parent chemokine in the extent and quality of CCR5-arrestin ass

142 TNF-alpha was the most sensitive cytokine or chemokine in this setting of impairment.

143 ls of several pro-inflammatory cytokines and chemokines in ascites fluid as compared with plasma.

144 nbiased RNA-interference screen of mammalian chemokines in co-cultures of mouse PDAC cells (K8484) an

145 Knockdown of 9 chemokines in DRG neurons significantly reduced migratio

146 al mucosa via topical vaginal application of chemokines in mice.

147 ntial profiles of inflammatory cytokines and chemokines in the UCL and NUCL lesions.

148 of different pro-inflammatory cytokines and chemokines in time- and concentration-dependent manners,

149 concentration of inflammatory cytokines and chemokines, including IL-1, TNF-alpha, IL-9, CXCL1, CCL2

150 aled up-regulation of multiple cytokines and chemokines, including IL-6 and IL-8, in response to star

151 ates the production of various cytokines and chemokines, including type I interferons (IFN-I).

152 le of Mincle for MERS-CoV-triggered cytokine/chemokine induction was established based on the results

153 e dominated by proinflammatory cytokines and chemokine induction, including interleukin 6 (IL-6), tum

154 differences in chemokine secretion including chemokines influencing the permeability of the endotheli

155 about the essential features of the receptor-chemokine interaction in which the N terminus contribute

156 critical pathogenic aspects of cytokines and chemokines involved in generation of effector T cell res

157 ed along with significantly higher levels of chemokines KC and monokine induced by gamma interferon.

158 amma burst within a proinflammatory cytokine/chemokine landscape, transactivated NK cells, increased

159 In contrast, these cytokine/chemokine levels remained unchanged in those with DHF or

160 672 that physically interacts with C-C motif chemokine ligand (CCL) 8 and synthesized a 16-mer peptid

161 Hepatic expression of C-X-C motif chemokine ligand 1 (CXCL1), a key chemokine for neutroph

162 factor alpha, interleukin-6, and C-X-C motif chemokine ligand 1 expression.

163 d an association between increased levels of chemokine ligand 2 (CCL2) with an atypical chemokine rec

164 its cognate chemokine ligands, including CC chemokine ligand 2 (CCL2), plays a central role in recru

165 at the immune cell chemoattractant C-C motif chemokine ligand 21 (CCL21) is effective as an intratumo

166 d on expression of IL4, IL5, IL13, C-C motif chemokine ligand 26 (CCL26), thymic stromal lymphopoieti

167 This study shows that potent C-C chemokine ligand 5 analogs differ from each other and fr

168 CXC-chemokine ligand 9 (CXCL9) is an interferon-inducible ch

169 IL-12p70), and chemokines (CXCL10, C-C motif chemokine ligand [CCL]2, CCL3, CCL4, and CCL13) in nasal

170 st progenitors up-regulate expression of the chemokine ligand Cxcl9l.

171 C chemokine receptor 2 (CCR2) by its cognate chemokine ligands, including CC chemokine ligand 2 (CCL2

172 sm significantly dampened C5aR1-, C3aR-, and chemokine-like receptor 1 (CMKLR1)-mediated ERK signalin

173 cAP, associated with reduction in cytokines, chemokines, liver cell death, and brain water.

174 Chemokines mediate leukocyte migration and homeostasis a

175 at multiple critical checkpoints, including chemokine-mediated T cell trafficking into lymph nodes a

176 n vitro and induce cholangiocytes to produce chemokines mediating recruitment of Th17 cells and more

177 ein 2 (MIP-2), which suggested that CXC ELR+ chemokines might be involved in neutrophil recruitment i

178 deposition associated with higher levels of chemokine mRNA and increased numbers of M2 macrophages.

179 suggest that although the interaction of the chemokine N terminus with the receptor-binding pocket is

180 that suggested the X position reorients the chemokine N terminus.

181 les for levels of inflammatory cytokines and chemokines (n=81).

182 ory peptides that therapeutically target the chemokine network in inflammatory diseases.

183 Here, we use comparative analysis of chemokine NMR structures, structural modeling, and molec

184 ligand 9 (CXCL9) is an interferon-inducible chemokine of the CXC family and is increased in cGVHD.

185 expression of pro-tumorigenic cytokines and chemokines overexpressed in ovarian cancer.

186 tion and affinity results indicated that the chemokine pair CXCR4/SDF1 may play an important role in

187 ustering approach revealed distinct cytokine/chemokine patterns, and these aligned with pathways asso

188 : secretors of proinflammatory cytokines and chemokines, presenters of autoantigens to T cells, produ

189 pression patterns, and dynamic regulation of chemokines produced by murine pathogen-specific T(M) CD8

190 tion, we have defined the entire spectrum of chemokines produced by pathogen-specific CD8(+) and CD4(

191 alone was sufficient to induce cytokine and chemokine production by macrophages and B16 tumor cells,

192 with higher granzyme B levels and increased chemokine production in response to KIR activation, sugg

193 e immune responses by elevating cytokine and chemokine production via triggering multiple signaling p

194 s and induces chemotaxis, increases cytokine/chemokine production, and enhances antimicrobial effecto

195 nstrate that FAK-depletion in CAFs increases chemokine production, which via CCR1/CCR2 on cancer cell

196 ivates their antitumor inflammatory cytokine/chemokine production.

197 ation thresholds (CCL1/3/4/5/XCL1); and T(M) chemokine profiles modulated by persisting viral Ags exh

198 resulted in reduced levels of IFN dependent chemokines Rantes (CCL5) and CXCL10.

199 l for the induction of the key thymus-homing chemokine receptor - CCR6 on Tregs.

200 Activation of CC chemokine receptor 2 (CCR2) by its cognate chemokine lig

201 C-X-C motif chemokine receptor 2 (CXCR2) is a chemokine receptor kno

202 r aim was to discover the role of IL-17, CXC chemokine receptor 2 (CXCR2) ligands, and cancer-associa

203 The monocyte chemoattractant protein-1/CCR2 (chemokine receptor 2) axis plays an important role in ab

204 to image the recruitment of proinflammatory chemokine receptor 2-positive (CCR2+) cells in multiple

205 LP), Charcot-Leyden crystal (CLC), C-C motif chemokine receptor 3 (CCR3), and CPA3.

206 Here, we identified a role for host atypical chemokine receptor 4 (ACKR4) in controlling intratumor T

207 The C-C chemokine receptor 4 (CCR4) is broadly expressed on regu

208 Here, we report that C-X-C motif chemokine receptor 4 (CXCR4) and hedgehog pathways coope

209 C-X-C chemokine receptor 4 (CXCR4) is a transmembrane chemokin

210 ells through the downregulation of C-C motif chemokine receptor 5 (CCR5) in T cells and CD4 in both T

211 C-C chemokine receptor 5 (CCR5) is a chemokine receptor belo

212 C-C chemokine receptor 5 (CCR5) is a key drug target for hum

213 tation of LXA(4) identified modulation of CC-chemokine receptor 7 (CCR7) and sphingosine 1- phosphate

214 Under the influence of CXCR1 and CXCR2 chemokine receptor agonists and other chemotactic factor

215 C-C chemokine receptor 5 (CCR5) is a chemokine receptor belonging to the G protein-coupled re

216 in transcriptional factor RORgammat, but not chemokine receptor CCR6, showed full rescue of the long-

217 f chemokine ligand 2 (CCL2) with an atypical chemokine receptor chemokine-binding protein 2 variant V

218 T cells express IL (interleukin)-17 and the chemokine receptor CXCR (C-X-C chemokine receptor)-6.

219 R-treated mice displayed lower levels of the chemokine receptor CXCR2, consistent with a reduced abil

220 Finally, chemokine receptor CXCR3 is upregulated in the expanded

221 We also demonstrate that the chemokine receptor Cxcr3.2 is expressed in a distinct su

222 t roles in development, cancer, and HIV, the chemokine receptor CXCR4 and its ligand CXCL12 have been

223 Inhibition of the chemokine receptor CXCR4 in combination with blockade of

224 e induction of the glucocorticoid-responsive chemokine receptor CXCR4, and selective blockade of CXCR

225 ammadelta T cells express high levels of the chemokine receptor CXCR6 and seed meninges shortly after

226 Chemokine receptor expression on CD4+ T cells was determ

227 lating TCR complex components and modulating chemokine receptor expression to promote dissemination o

228 CD4(+) /CD8(+) phenotype and chemokine receptor expression were analyzed by flow cyto

229 mokine receptor 4 (CXCR4) is a transmembrane chemokine receptor involved in growth, survival, and dis

230 -X-C motif chemokine receptor 2 (CXCR2) is a chemokine receptor known for its role during inflammatio

231 phocytes compartmentalize according to their chemokine receptor pattern and subsequently migrate towa

232 C-C chemokine receptor type 2 (CCR2) is expressed on monocyt

233 n)-17 and the chemokine receptor CXCR (C-X-C chemokine receptor)-6.

234 oubts remain that CXCR4 represents a classic chemokine receptor, functions assigned to ACKR3/CXCR7 ra

235 t promotes atherosclerosis through CXC-motif chemokine receptor-4 (CXCR4).

236 ctivation of Tnfa and its receptors or major chemokine receptor-ligand subsets persisted in the long

237 evasins that function through inhibition of chemokine-receptor signaling in the host.

238 Targeting a specific chemokine/receptor axis in atherosclerosis remains chall

239 (IL-17, TNF-alpha, IL-9, and IFN-gamma) and chemokine receptors (CCR1, CCR2, CCR4, CCR5, CCR6, and C

240 fferentiation 4 receptors and one of the two chemokine receptors (CCR5 and CXCR4) to gain entry in hu

241 ices, and altered mRNA expression of related chemokine receptors and ligands.

242 However, chemokine receptors are imperative for guiding cells out

243 emphasizes the importance of chemokines and chemokine receptors as regulators of bone remodeling.

244 ssing high levels of Th2 and Th17 cytokines, chemokine receptors CCR4 and CCR6, and the transcription

245 , is regulated by chemokines, which activate chemokine receptors on the leukocytes.

246 ctal cancer by impairing the function of the chemokine receptors that mediate the intratumoral accumu

247 ys were activated, and specific cytokine and chemokine receptors were up-regulated in CSF memory B ce

248 ulate various immune responses by activating chemokine receptors which belong to the G protein-couple

249 se the AT1R and certain other GPCRs (such as chemokine receptors) to adopt conformations that are cap

250 cs, suppressed matrix metalloproteinases and chemokine receptors, and the induction of CXCL11-CXCR3 a

251 Among mice deficient in chemokine receptors, CCR6(-/-) mice had 120 copies/mug R

252 Tregs reveal an enhancement in graft-homing chemokine receptors, which may be partly responsible for

253 gent for imaging both human and murine CXCR4 chemokine receptors.

254 HIV gp120 determine viral interactions with chemokine receptors; specifically, HIV-X4 viruses intera

255 These chemokines recruit MDSC and macrophages, finally enablin

256 Chemokine redundancy and ensuing network robustness has

257 accuracy of the predicted orientation of the chemokine relative to the receptor and providing insight

258 However, chemokine release (e.g. MCP-1, RANTES and TARC) was sign

259 ulation, and role of effector T cell-derived chemokines remains incompletely understood.

260 ders have an increased nasal mucosal IFN and chemokine response to the viral RNA analogue R848.

261 ls and eosinophils and systemic cytokine and chemokine responses are associated with severe C. diffic

262 larities and differences in the cytokine and chemokine responses in these two infections, we compared

263 vary proteins, called Evasins, to neutralize chemokines responsible for cell trafficking and recruitm

264 inflammatory markers including cytokines and chemokines, ROS markers, and growth factors are unchange

265 populations that largely define cytokine and chemokine secretion in human monocytes exposed to Toxopl

266 ruitment were associated with differences in chemokine secretion including chemokines influencing the

267 tion of leukocyte cell subsets, cytokine and chemokine secretion, and viremia, were assessed.

268 se-exacerbating and -protective pathways and chemokine-selectively interferes with atherosclerosis.

269 Cytokines and chemokines shape Th1 and Th17 effector responses as well

270 N-ERD macrophages included genes involved in chemokine signaling and acylcarnitine metabolism.

271 bone morphogenetic protein (BMP) signaling, chemokine signaling, and focal adhesion were activated b

272 d contribution of the receptor N terminus to chemokine signaling.

273 clusions: Profound global lymphopenia and a "chemokine signature" were observed in COVID-19 ARDS.

274 spleen that peaked by day 2, and an extended chemokine storm that was detected in both lungs and brai

275 This chemokine storm was also detected in the brain at day 6.

276 ro-10) had little to no impact on the folded chemokine structure but diminished CXCR4 agonist activit

277 IL-4, and increased expression of microglial chemokines, such as macrophage-colony-stimulating factor

278 communication network factor 1 (CYR61) as a chemokine that is up-regulated by hepatocytes during liv

279 ation-inhibitory factor (MIF) is an atypical chemokine that promotes atherosclerosis through CXC-moti

280 event the chemotaxis of pre-B cells toward a chemokine that supports B-cell trafficking and homing wi

281 N-apo AI reduced the cardiac expression of chemokines that attract neutrophils and monocytes by 60%

282 Among the chemokines that can recruit these cells into the skin, t

283 esponse relies, in part, on cues imparted by chemokines that coordinate their spatiotemporal position

284 keratinocytes and mesangial cells to produce chemokines that induce immune cell recruitment and promo

285 Ab production and may produce cytokines and chemokines that modulate inflammation.

286 -lipoxygenase-derived eicosanoids to express chemokines that recruit a combined type 2/immunoregulato

287 okine cues and a relevant source for certain chemokines themselves; yet, the actual range, regulation

288 Salivary evasins from ticks bind multiple chemokines to overcome redundancy and are effective in s

289 s but express pathogen-specific cytokine and chemokine transcripts.

290 ular signaling mechanisms activated by CCL28 chemokine via its primary receptor CCR10 in endothelial

291 In an unbiased screen of chemokines, we identified CCL21 and CXCL10 as proteins t

292 Whereas low-grade levels of plasma cytokine/chemokine were apparent in older donors (>65 y old), sur

293 eters of fibrosis and inflammatory cytokines/chemokines were determined by qRT-PCR, western, and immu

294 for B cell-specific proteins and neutrophil chemokines were elevated in patients with lower viral lo

295 Inflammatory cytokines and chemokines were examined in the sera by ELISA and in ski

296 plasma concentrations of other cytokines and chemokines were not different among the MHL and MUO grou

297 Five cytokines and chemokines were significantly lower in transplant GNB vs

298 euroimmune transcription factor, and IL-8, a chemokine, were significantly increased.

299 a hallmark of inflammation, is regulated by chemokines, which activate chemokine receptors on the le

300 mmation and regulated by the interactions of chemokines with their G protein-coupled receptors.