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1 ion of the type I interferon (IFN)-regulated chemokine CXCL10.
2 ent of the chemokine receptor CXCR3 with the chemokine CXCL10.
3 cking such a reentry by neutralizing the key chemokine CXCL10.
4 , but with increased mRNA for the T-cell Th1 chemokine CXCL10.
5 hment, which was mediated by the LEC-derived chemokine CXCL10.
6  and tumor immunity by preserving functional chemokine CXCL10.
7 ntrol by the cytokine interleukin-13 and the chemokine CXCL10.
8 response genes, IFN-induced GTPases, and the chemokine CXCL10.
9 xpressed cytokine interleukin-27 (IL-27) and chemokine CXCL10.
10 interferons (IFN-lambda1, lambda2/3) and the chemokine CXCL10.
11  interferon regulatory factor, IRF1, and the chemokine, CXCL10.
12 f IL-12, IFN-gamma and the IFN-gamma induced chemokine, CXCL10.
13 cterized by significant up-regulation of the chemokine, CXCL10.
14  the infection, notably interferon-inducible chemokine, CXCL10.
15 ion of an IFN-inducible and STAT-1-dependent chemokine, CXCL10.
16 N-gamma transcript and the IFN-gamma-induced chemokine, CXCL10.
17 latelet-derived growth factor (PDGF) and the chemokine, CXCL10.
18 on of the transcription factor IRF1, and the chemokine, CXCL10.
19 e highly upregulated downstream of CCL2; the chemokine CXCL10 and its cognate receptor, CXCR3, stood
20 crobial peptides beta-defensin 3, CRAMP, and chemokine CXCL10 and its receptor CXCR3 in corneal epith
21 ed that blocking the interaction between the chemokine CXCL10 and its receptor CXCR3 on activated CTL
22 ementia and demonstrated the presence of the chemokine CXCL10 and its receptor, CXCR3, in the neurons
23 ease of proinflammatory cytokines (IL-6) and chemokines (CXCL10 and CCL5).
24 BC), neopterin levels, and concentrations of chemokines CXCL10 and CCL2.
25 decreased concentrations of pro-inflammatory chemokines CXCL10 and CCL5 in infected tissues incubated
26 essential for IL-1-induced expression of the chemokines CXCL10 and CCL5, which recruit mononuclear ce
27 e abundance is associated with IFN-inducible chemokines CXCL10 and CCL5.
28 Immature myeloid cells in dLNs expressed the chemokines CXCL10 and CXCL16 in an IFN-gamma-dependent m
29 ymphocytes are recruited into the CNS by the chemokines CXCL10 and CXCL9.
30 nstrate that estradiol induces expression of chemokines CXCL10 and/or CXCL11 within human endometrium
31 osphorylation, production of type I IFN, the chemokine CXCL10, as well as caspase-9-mediated tumor ce
32 ne of CA-MRSA, and the related SPA-releasing chemokine CXCL10 bound to both cell wall and cell membra
33 us studies have shown that expression of the chemokine CXCL10 by West Nile virus (WNV)-infected neuro
34 y cytokines (TNF-alpha, IL-2, IL-12p70), and chemokines (CXCL10, C-C motif chemokine ligand [CCL]2, C
35 atory cytokines (TNF-a, IL-2, IL-12p70), and chemokines (CXCL10, C-C motif chemokine ligand [CCL]2, C
36 patients with active cryptosporidiosis, four chemokines (CXCL10, CCL11 [eotaxin], CCL5 [RANTES], and
37 ha [TNF-alpha] and interleukin-6 [IL-6]) and chemokines (CXCL10, CCL2, CCL3, and CCL5) correlated wit
38 essed the expression of the genes encoding 3 chemokines, CXCL10, CCL2, and CCL20, and repressed CXCL1
39                                          The chemokine, CXCL10, chemotactic for NK cells, activated T
40                                Levels of the chemokines CXCL10, CXCL11, CCL4, and CCL5 increased in b
41 fined by expression of the T cell-attracting chemokines CXCL10/CXCL11 and abundant T cells.
42 gans and to bind with nanomolar affinity the chemokines CXCL10, CXCL12, and CXCL13.
43             Expression of the Th1-associated chemokines CXCL10, CXCL9, CCL5, and CXCL11 was elevated
44  alpha-toxin in the induction of Th1-related chemokine CXCL10 diversely, which could favour the recru
45 type with the secretion of a proinflammatory chemokine CXCL10 due to activation of STAT1 signaling.
46 ti-photon microscopy to demonstrate that the chemokine CXCL10 enhances the ability of CD8+ T cells to
47                                          The chemokine CXCL10 has been associated with several autoim
48  report, we investigated the role of the CXC chemokine CXCL10 (IFN-gamma-inducible protein-10) in the
49        We quantified serum concentrations of chemokine CXCL10 in 288 patients with measles virus (MeV
50 dition, the metastatic IECs also induced the chemokine CXCL10 in a TLR3-, TRIF-, and IRF3-dependent m
51  during viral infection through induction of chemokine CXCL10 in central nervous system epithelial an
52 ction of IFNgamma and the IFNgamma-inducible chemokine CXCL10 in healthy skin.
53 hibition of EZH2 induces reexpression of the chemokine CXCL10 in hepatic tumor cells.
54 ionally, coculture induced expression of the chemokine CXCL10 in MSCs and the cognate receptor CXCR3
55 ced an overactivation of the proinflammatory chemokine CXCL10 in PBMC from NCGS patients, and that th
56           Various cell types can produce the chemokine CXCL10 in response to IFN-gamma stimulation.
57 ts demonstrate an important role for the CXC chemokine CXCL10 in the recruitment and accumulation of
58  that PND patients harbor high levels of the chemokine CXCL10 in their CSF.
59      Mass spectrometry identified the potent chemokine, CXCL10, in the EVs, which was markedly enrich
60                                          The chemokines CXCL10/interferon-gamma-inducible protein of
61 itor of the monocyte-derived proinflammatory chemokine CXCL10 (IP-10) and other CXCR3 ligands, except
62                                          The chemokine CXCL10 (IP-10) was significantly elevated (> 2
63  infection by upregulating expression of the chemokine CXCL10 (IP-10).
64                                          The chemokine CXCL10/IP-10 facilitates recruitment of Th1-ty
65  additional secreted proteins, including the chemokine CXCL10/IP10.
66                                          The chemokine CXCL10 is expressed early after virus infectio
67 eviously reported that the IFN-gamma-induced chemokine CXCL10 is expressed in lesional skin from viti
68                                          The chemokine CXCL10 is expressed within the CNS in response
69 e (AD) patients and in AD animal models, the chemokine CXCL10 is found in high concentrations, sugges
70                                          The chemokine CXCL10 is produced by many inflammatory cells
71 ht to investigate the mechanism by which the chemokine CXCL10 mediates bactericidal activity against
72 ion plasmids; 2) by inhibiting DMXAA-induced chemokine (CXCL10) mRNA and protein production in the AB
73                              The non-ELR-CXC chemokine, CXCL10 or IP-10, is chemotactic for monocytes
74                                    Of these, chemokine CXCL10, previously linked to cardiovascular di
75 in inflammatory responses, including reduced chemokine (CXCL10) production, reduced chemokine recepto
76 otropic TG delivery of the T cell-attracting chemokine CXCL10 (pull), boosted the number and function
77           In contrast, the T-cell attractant chemokine CXCL10 showed an almost 10-fold higher express
78 ptor type 6 (CXCR6) on MSCs and MSCs secrete chemokine CXCL10 that binds to receptor CXCR3 on BCCs, d
79 lyses suggest the interferon-gamma-inducible chemokine CXCL10 to be a potentially causal mediator for
80                Remarkably, T-cell recruiting chemokine CXCL10 turns out to be the most abundant infla
81                        The expression of the chemokine CXCL10 was initially detected in neurons as ea
82 cell-deficient mice with the IFN-I-dependent chemokine, CXCL10 was also sufficient to improve sepsis
83 sponse to infection-induced elevation of the chemokine CXCL10, which attracted CXCR3(+) memory CD8(+)
84 or levels of IFN-gamma and the IFN-inducible chemokine CXCL10, which is a ligand for CXCR3.
85 es leads to transcriptional induction of the chemokine CXCL10, which is considered an interferon (IFN
86 sponse to WNV infection, neurons secrete the chemokine CXCL10, which recruits effector T cells via th