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1 ced osteoclast conditioned media-induced hMS chemokinesis.
2  is a common feature both for chemotaxis and chemokinesis.
3 calpain, induced neutrophil polarization and chemokinesis.
4 chemotaxis, but dimeric TFF1 stimulated some chemokinesis.
5 active CREB decreased both proliferation and chemokinesis.
6 ated that SPP stimulates both chemotaxis and chemokinesis.
7 d monocytes induced by CC chemokines, termed chemokinesis.
8 ns not because of chemotaxis, but because of chemokinesis.
9        Sema7A orchestrated MO chemotaxis and chemokinesis, activated MO differentiation and polarizat
10 gher for faster cells, and was compounded by chemokinesis, an increase in speed with resource concent
11 tively active CREB abolished the increase in chemokinesis and cell cycle progression induced by eithe
12                          Our model simulates chemokinesis and chemotaxis in a variety of chemical and
13 oxidative concentrations of H2O2 also impede chemokinesis and chemotaxis of previously activated huma
14 nsient overexpression of PLD1 increased both chemokinesis and chemotaxis toward IL-8 and FMLP but not
15                      Complement C5a-mediated chemokinesis and chemotaxis were almost completely aboli
16 (PLD1) and PLD2 reduced cell migration (both chemokinesis and chemotaxis) by approximately 60% and >8
17 says do not definitively distinguish between chemokinesis and chemotaxis, single-cell chemotaxis assa
18  sufficient to promote SMC migration by both chemokinesis and chemotaxis, which was inhibited by prot
19 f transendothelial migration or interstitial chemokinesis and chemotaxis.
20 ming of stem cells to this site by mediating chemokinesis and chemotaxis.
21 ve stress decrease SMC CREB content increase chemokinesis and entry into the cell cycle, which is blo
22 se decreased CREB function but increased SMC chemokinesis and entry into the cell cycle.
23 was biologically active, as it induced vitro chemokinesis and faster scratch assay wound healing by a
24 by endothelial activation with IL-4 improved chemokinesis and lateral migration toward a monocyte che
25 /neogenin interactions augment CD4(+) T cell chemokinesis and promote cellular infiltration in associ
26                  We show that GM-CSF induces chemokinesis and promotes eosinophil survival in vitro,
27 ed, AMPK increased microtubule synthesis and chemokinesis and provided adaptation to energy demand du
28 dependent PIP3 accumulation, PKB activation, chemokinesis and reactive oxygen species (ROS) formation
29 lly probe directed (chemotaxis), undirected (chemokinesis), and 3D amoeboid cell migration in these f
30 show defective chemokine-induced chemotaxis, chemokinesis, and adhesion to integrin ligands.
31 -mediated actin rearrangement and eosinophil chemokinesis, and impairs eosinophil viability.
32 predominantly stimulated chemotaxis and some chemokinesis, and it was chemotactic for a variety of hu
33 -/-) mice exhibited an increased chemotaxis, chemokinesis, and transendothelial migration in vitro.
34 te cell migration in chemotaxis, haptotaxis, chemokinesis, and wound healing assays.
35             Increases in DC accumulation and chemokinesis are partially dependent on ccr6, a crucial
36 d of similar magnitude during chemotaxis and chemokinesis, at 18 +/- 1.4 and 16 +/- 1.3 nN/cell, resp
37                In addition, thrombin induced chemokinesis, but not chemotaxis, of CD8(+) T cells, whi
38 thionylleucylphenylalanine (FMLP)-stimulated chemokinesis by >60%, markedly reduced polar morphology,
39 une reactions to the joint through leukocyte chemokinesis, cell-cell adhesion, and matrix specializat
40 of PA and EF) significantly impair human PMN chemokinesis, chemotaxis, and ability to polarize.
41 eased stem cell migration when compared to a chemokinesis control.
42 s (motion up or down a chemical gradient) or chemokinesis (dependence of speed on chemical concentrat
43 taxis induced by IL-8 and fMLP but decreased chemokinesis for neutrophils.
44                                              Chemokinesis has been most thoroughly studied in the per
45 transport, epilepsy, behavioral abnormality, chemokinesis, histone deacetylation, and immunity.
46 ld involve chemotaxis toward infected cells, chemokinesis (i.e., increased motility) combined with CT
47                     However, UDP-G increased chemokinesis in eosinophils and enhanced their response
48 d cells, indicating that F. alocis increases chemokinesis in human neutrophils.
49                Lysophosphatidic acid induced chemokinesis in T cells.
50 neutrophil adhesion, polarization, and rapid chemokinesis in the absence of exogenous activators.
51  model, and experiments bolsters the role of chemokinesis in this system.
52 ne CCL21, a ligand of CCR7, strongly induces chemokinesis in vitro, and T cell motility in LNs from C
53 reas micromolar GABA predominantly initiated chemokinesis (increased random movement).
54 n contrast to IL-8-induced chemokinesis, the chemokinesis induced by calpain inhibition was not reduc
55                                              Chemokinesis is distinct from chemotaxis in that no dire
56  PE increased cell velocity, suggesting that chemokinesis may be at least partly responsible for dire
57 ndicated that LPA causes both chemotaxis and chemokinesis of beta1-replete cells.
58 y by astrocytes, promoted the chemotaxis and chemokinesis of cancer cells via their C-C chemokine rec
59 ll m.w. cytokines that induce chemotaxis and chemokinesis of leukocytes.
60           LPA stimulates both chemotaxis and chemokinesis of ovarian cancer cells and LPA-stimulated
61                                PGE2-elicited chemokinesis of RNK-16 cells across a Matrigel model bas
62              Furthermore, this dependence of chemokinesis on PI(3)Kgamma activity is context dependen
63                                              Chemokinesis on VCAM-1 appeared to be associated with tr
64 e constitutively active Ras has no effect on chemokinesis or chemotaxis.
65 sures true macrophage chemotaxis rather than chemokinesis or fugetaxis.
66 (2)O(2)) demonstrated a 3.5-fold increase in chemokinesis (p < 0.05) and accelerated entry into cell
67 with ET causes an additive inhibition of PMN chemokinesis, polarization, chemotaxis, and FMLP (N-form
68  PCNs utilized FGF-2 found in situ to induce chemokinesis, potentiate SDF-1alpha chemotactic recruitm
69 r-1alpha (SDF-1alpha)-induced chemotaxis and chemokinesis (random migration).
70 ne airways primarily by increasing velocity (chemokinesis) rather than directional migration (chemota
71 vidual eggs reveals that both chemotaxis and chemokinesis significantly promote contacts.
72                                              Chemokinesis stimulated by IL-8 was inhibited in the pre
73 PLD1 isoform, led to an abolishment of basal chemokinesis that could not be rescued with chemoattract
74                  In contrast to IL-8-induced chemokinesis, the chemokinesis induced by calpain inhibi
75 work suggests a novel strategy of exploiting chemokinesis to effect accumulation of motile colloids i
76 cteria use chemically regulated motility, or chemokinesis, to sense host-emitted cues that trigger ac
77  analysis indicated that both chemotaxis and chemokinesis took place.
78 we describe reduced chemotaxis but preserved chemokinesis toward a range of inflammatory stimuli in m
79                                              Chemokinesis was not mediated by the enzymatic activity
80                               Chemotaxis and chemokinesis were markedly impaired, but staphylococcal
81                     Simulated DCs undergoing chemokinesis were measured to have a speed of 5.1 +/- 0.
82 sulted in elevation of both S6K activity and chemokinesis, whereas PLD silencing was inhibitory.
83 s only, is a potent stimulator of macrophage chemokinesis, whose activity is enhanced by yeast cell w