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1 cts were induced in a virulent strain of the chestnut blight fungus Cryphonectria parasitica (Murr.)
4 patibility (vic)] loci were disrupted in the chestnut blight fungus Cryphonectria parasitica using an
5 ion of one of two dicer genes, dcl-2, of the chestnut blight fungus Cryphonectria parasitica was rece
6 virulence attenuation (hypovirulence) of the chestnut blight fungus Cryphonectria parasitica were use
9 virulence attenuation (hypovirulence) of the chestnut blight fungus Cryphonectria parasitica, could s
10 V-1/Euro7, and CHV-1/EP721, which infect the chestnut blight fungus Cryphonectria parasitica, differ
11 virulence attenuation (hypovirulence) of the chestnut blight fungus Cryphonectria parasitica, encodes
12 silencing antiviral defense response in the chestnut blight fungus Cryphonectria parasitica, is indu
13 on and sporulation by the infected host, the chestnut blight fungus Cryphonectria parasitica, while b
18 hat was decimated by the introduction of the chestnut blight fungus (Cryphonectria parasitica) in the
19 which was isolated from strain NB631 of the chestnut blight fungus (Cryphonectria parasitica), a mod
20 nce attenuation) observed for strains of the chestnut blight fungus, Cryphonectria parasitica, harbor
22 We now report that DI RNA production in the chestnut blight fungus, Cryphonectria parasitica, persis
23 st characterized of a number of genes in the chestnut blight fungus, Cryphonectria parasitica, that a
24 itor global transcriptional responses of the chestnut blight fungus, Cryphonectria parasitica, to inf
25 irus 1 (CpMV1) from U.S. strain NB631 of the chestnut blight fungus, Cryphonectria parasitica, was th
26 ed phenotype, such as the hypoviruses of the chestnut-blight fungus, have been studied for their pote
27 he oah gene in Cryphonectria parasitica, the chestnut blight fungus, reduces the ability of the fungu
28 viruses (hypoviruses or Hypoviridae) of the chestnut blight fungus, the Sclerotinia sclerotiorum ssD