ライフサイエンスコーパス: childhood obesity

1 ilk for children might not lower the risk of childhood obesity.

2 h a potential reduction of the later risk of childhood obesity.

3 disorder, to attention deficit disorder and childhood obesity.

4 pathways involved in the pathophysiology of childhood obesity.

5 ental media monitoring in efforts to prevent childhood obesity.

6 nments to make them more walkable may reduce childhood obesity.

7 is potentially a modifiable risk factor for childhood obesity.

8 tervening on multiple levels of influence on childhood obesity.

9 as a target for the effective prevention of childhood obesity.

10 strogens, flame retardants, heavy metals and childhood obesity.

11 n intakes could contribute to a reduction in childhood obesity.

12 ages 0 to 23 months is associated with early childhood obesity.

13 ic disparities exist across risk factors for childhood obesity.

14 phthalate concentrations and prevalences of childhood obesity.

15 od has a potential for primary prevention of childhood obesity.

16 re prevalent in conjunction with the rise in childhood obesity.

17 ironmental toxicant can be a risk factor for childhood obesity.

18 ectronic tool to predict an infant's risk of childhood obesity.

19 loci associated with the pathophysiology of childhood obesity.

20 magnetic fields (MFs) increases the risk of childhood obesity.

21 the scope and potency of PAC treatments for childhood obesity.

22 ected because of the increased prevalence of childhood obesity.

23 hanges may have influenced the prevalence of childhood obesity.

24 and obesity, particularly as they relate to childhood obesity.

25 eview focuses on the genetic contribution to childhood obesity.

26 dered relevant factors for the prevention of childhood obesity.

27 xamine the perinatal risk factors related to childhood obesity.

28 s and plant-based diets in the prevention of childhood obesity.

29 birth weight, was the strongest predictor of childhood obesity.

30 heavy smoking was positively associated with childhood obesity.

31 ion years and ADHD show genetic overlap with childhood obesity.

32 infant feeding method, and for their link to childhood obesity.

33 ciation between smoking during pregnancy and childhood obesity.

34 e physical and mental health consequences of childhood obesity.

35 ity plays a critical role in the increase in childhood obesity.

36 c Islanders, smoking was not associated with childhood obesity.

37 Television viewing is associated with childhood obesity.

38 ategies that promote fetal growth and reduce childhood obesity.

39 ntial in battling the increasing epidemic of childhood obesity.

40 olemia is associated with the development of childhood obesity.

41 e of early intervention in the prevention of childhood obesity.

42 mising, population-based approach to prevent childhood obesity.

43 d as weight/height, is often used to monitor childhood obesity.

44 lly contribute to reducing the prevalence of childhood obesity.

45 g, but complex approach to the prevention of childhood obesity.

46 3 loci were associated with birth weight and childhood obesity.

47 inks containing added sugar), contributes to childhood obesity.

48 ehavior in later life and is associated with childhood obesity.

49 influence risk for chronic disease including childhood obesity.

50 ention delivery and training initiatives for childhood obesity.

51 ancy is unlikely to be a main contributor to childhood obesity.

52 d make a meaningful contribution to reducing childhood obesity.

53 asthma medication use on the development of childhood obesity.

54 e, during infancy is associated with risk of childhood obesity.

55 that of underlying infection on the risk of childhood obesity.

56 rum antibiotics were associated with risk of childhood obesity.

57 ion of metabolic homeostasis contributing to childhood obesity.

58 ncreased risk of rapid growth in infancy and childhood obesity.

59 ests that air pollution is a risk factor for childhood obesity.

60 e, during infancy is associated with risk of childhood obesity.

61 FM4 associated with adult body mass index or childhood obesity.

62 s associated with an increased risk of early childhood obesity.

63 hild health and decreasing the prevalence of childhood obesity.

64 s a first step towards primary prevention of childhood obesity.

65 dered when implementing strategies to combat childhood obesity.

66 s has been associated with increased risk of childhood obesity.

67 terventions improved the quality of care for childhood obesity.

68 forcers may have a protective effect against childhood obesity.

69 ignificant contribution to the prevention of childhood obesity.

70 ring infancy was not associated with risk of childhood obesity (1.01, 0.98-1.04).

71 Childhood obesity, a major public health problem, can le

72 ciation between smoking during pregnancy and childhood obesity across race/ethnicity categories merit

73 ns of family and individual factors with the childhood obesity after adjusting for covariates.

74 ohort of 172 patients presenting with severe childhood obesity and a family history of obesity.

75 AMILIA Study was designed to genetically map childhood obesity and associated biological processes in

76 association between breastfeeding and later childhood obesity and blood pressure (BP) is inconsisten

77 Associations between childhood obesity and brain microstructural differences

78 of measures to reduce the global epidemic of childhood obesity and encourage mechanistic studies.

79 t previous studies have focused primarily on childhood obesity and have been hampered by limited cont

80 have attempted to elucidate the etiology of childhood obesity and have increased our understanding o

81 ios (ORs) (95% confidence intervals [CI]) of childhood obesity and hyperglycemia associated with diff

82 ted in improved family-centered outcomes for childhood obesity and improvements in child BMI.

83 hite matter (WM) damage has been reported in childhood obesity and in metabolic syndrome (MetS) but m

84 is implicated in clinical conditions such as childhood obesity and increased marrow adiposity in type

85 and validate equations for the prediction of childhood obesity and integrate them into a mobile phone

86 s considered the gold-standard treatment for childhood obesity and is provided to the parent and chil

87 d beverages is seen as one measure to tackle childhood obesity and is under active consideration by t

88 be implemented to decrease the prevalence of childhood obesity and its cardiovascular consequences in

89 amilia Study was designed to genetically map childhood obesity and its comorbidities in the Hispanic

90 Understanding the association between childhood obesity and maternal pre-pregnancy weight stat

91 mothers with obesity have a greater risk for childhood obesity and metabolic diseases; however, the u

92 micals may play a role in the development of childhood obesity and metabolic disorders, especially wh

93 ue to rise with the increasing prevalence of childhood obesity and metabolic syndrome.

94 pporting the role of neighborhood factors on childhood obesity and obesity-related behaviors, much wo

95 On the basis of our simulation models, childhood obesity and overweight will continue to be a m

96 Additional strategies are needed to reduce childhood obesity and related disparities among these un

97 le studies now confirm relationships between childhood obesity and specific morbidities.

98 The etiology of the development of childhood obesity and subsequent disease is poorly under

99 o smoke and NRP contribute to development of childhood obesity and suggest that combined exposures ma

100 regarding the prevention of infant and early childhood obesity and to identify research gaps and oppo

101 Given the current epidemic of childhood obesity and widespread use of artificial sweet

102 The incidence of obesity (especially childhood obesity) and its associated health-related pro

103 at IL-17(+) MAIT cells are also increased in childhood obesity, and altered MAIT cell frequencies in

104 der of importance, the main risk factors for childhood obesity are being unfit, having an obese fathe

105 emographic-socioeconomic characteristics and childhood obesity are complex in the United States.

106 mon, studies examining these same factors on childhood obesity are far fewer.

107 The prevalence and magnitude of childhood obesity are increasing dramatically.

108 Many early life risk factors for childhood obesity are more prevalent among blacks and Hi

109 Genetic and environmental contributions to childhood obesity are poorly delineated.

110 of adipokines and inflammatory molecules in childhood obesity are well documented, the contribution

111 recessive disorder that is characterized by childhood obesity associated with hyperinsulinemia, chro

112 We demonstrate that 81.3% of childhood obesity-associated heterozygous MC4R mutations

113 l surface expression of all the 14 described childhood obesity-associated MC4R missense mutations.

114 ns for the potential pharmacologic rescue of childhood obesity-associated MC4R mutations and for the

115 se mutant receptors, in addition to 11 other childhood obesity-associated MC4R mutations, indicates t

116 her examine the functional defects caused by childhood obesity-associated MC4R mutations, we develope

117 As more epidemiologic data on childhood obesity become available, researchers are face

118 9 +/- 1.5 and 12 +/- 1.5 months) for risk of childhood obesity (BMI at 2 years >91(st) centile and we

119 BPA exposure is plausibly linked to childhood obesity, but evidence is lacking to date.

120 revious studies, we addressed CNVs in common childhood obesity by examining children with a BMI in th

121 pendent African American (AA) cohort of 1479 childhood obesity cases and 1575 lean controls.

122 ohort of 1080 defined European American (EA) childhood obesity cases and 2500 lean controls (< 50(th)

123 About 5% of childhood obesity cases are caused by a defect that impa

124 ns are implicated in a significant number of childhood obesity cases.

125 ntinued to show association when two extreme childhood obesity cohorts were included (2,214 cases and

126 Childhood obesity coincides with increased numbers of ci

127 ncy was associated with an increased risk of childhood obesity compared with controls without infecti

128 The high prevalence of childhood obesity continues to persist, especially in ch

129 As the global prevalence of childhood obesity continues to rise, researchers and cli

130 he question as to whether the monocytosis in childhood obesity contributes to atherogenesis over the

131 ding children in order to address the global childhood obesity crisis.

132 Childhood obesity, defined by cutoffs based on the weigh

133 Early childhood obesity disproportionately affects Native Amer

134 Childhood obesity disproportionately impacts marginalize

135 an association between plant-based diets and childhood obesity does not mean that such diets should n

136 isorder characterized by neonatal hypotonia, childhood obesity, dysmorphic features, hypogonadism, me

137 ated for decades, the emerging links between childhood obesity, early onset puberty, and adult metabo

138 been one of the most robust risk factors for childhood obesity, effects of specific parenting influen

139 Reducing the burden of childhood obesity, eliminating health disparities, and p

140 In conclusion, childhood obesity entails monocyte gene expression alter

141 pment in genetically stable populations, the childhood obesity epidemic can be primarily attributed t

142 Addressing the childhood obesity epidemic continues to be a challenge.

143 Despite the worsening childhood obesity epidemic, the rate of inpatient bariat

144 increasingly examined for their role in the childhood obesity epidemic.

145 osures are novel contributors to the growing childhood obesity epidemic.

146 is unknown whether asthma contributes to the childhood obesity epidemic.

147 Childhood obesity, epidemic in the United States, has be

148 ansition such as China, rates of increase in childhood obesity exceed that in the West.

149 are generally cost effective for preventing childhood obesity, existing evidence suggests that nutri

150 t drop stems from the concurrent increase in childhood obesity, few longitudinal studies of growth an

151 ve previously described a women with extreme childhood obesity (Fig. 1), abnormal glucose homeostasis

152 ng was independently associated with reduced childhood obesity for both HIV-exposed and unexposed chi

153 Childhood obesity has become a global pandemic in develo

154 Severe childhood obesity has become a major health problem, and

155 Childhood obesity has become a public health challenge g

156 Concurrently, childhood obesity has become an epidemic in the United S

157 Childhood obesity has been attributed to a decline in to

158 it the most dramatic phenotype, showing that childhood obesity has consequences on intestinal IEL see

159 The study of childhood obesity has continued to grow exponentially in

160 In the past 2 decades, the prevalence of childhood obesity has increased dramatically.

161 Worldwide prevalence of childhood obesity has increased greatly during the past

162 Childhood obesity has increased significantly in recent

163 Childhood obesity has risen by more than 50% in the last

164 Prediction equations for risk of childhood obesity have been developed and incorporated i

165 Behavioral interventions to treat childhood obesity have had limited success.

166 Few existing efforts to predict childhood obesity have included risk factors across the

167 The prevalence and severity of childhood obesity have increased steadily over the past

168 e found substantially greater achievement of childhood obesity HEDIS measures in the CDS arm (adjuste

169 findings advance etiologic understanding of childhood obesity, highlighting complex effects of SES o

170 deficits and by metabolic defects including childhood obesity, hyperinsulinemia and Type 2 diabetes.

171 Given the long-term adverse sequelae of childhood obesity, identification of early life factors

172 y of smoking were positively associated with childhood obesity in a dose-response manner.

173 city-specific association of phthalates with childhood obesity in a nationally representative sample.

174 ents of pentaBDE mixture was associated with childhood obesity in a population of Latino children par

175 ng the association between breastfeeding and childhood obesity in an African setting with high HIV pr

176 use during early infancy has been linked to childhood obesity in high-income countries.

177 ion would be a promising strategy to prevent childhood obesity in humans, but more research is clearl

178 n that among adults, the rate of increase in childhood obesity in many countries has been greater tha

179 Health initiatives address childhood obesity in part by encouraging good nutrition

180 Evidence of effective treatment of childhood obesity in primary care settings is limited.

181 bute to the genetic susceptibility of common childhood obesity in subjects of both European and Afric

182 n between maternal body mass index (BMI) and childhood obesity in the offspring.

183 Although the increased prevalence of childhood obesity in the United States has been document

184 tly needed to tackle the growing epidemic of childhood obesity in the US.

185 Actions to be considered for preventing childhood obesity include the promotion of healthy mater

186 The prevalence of childhood obesity increased in the 1980s and 1990s but t

187 Childhood obesity increases the risk of obesity in adult

188 during the past year in the following areas: childhood obesity, insulin resistance, dyslipidemia, hyp

189 Despite the rising incidence of childhood obesity, international data from Eurostat show

190 Childhood obesity is a complex disorder that appears to

191 Childhood obesity is a global public health issue, which

192 Childhood obesity is a global public health problem.

193 Childhood obesity is a growing concern worldwide.

194 Childhood obesity is a growing public health concern, as

195 Childhood obesity is a growing worldwide problem.

196 Childhood obesity is a major public health issue and is

197 These results indicate that childhood obesity is a powerful predictor of development

198 Early-life intervention to prevent childhood obesity is a priority for public health, globa

199 Childhood obesity is a risk factor associated with type

200 Childhood obesity is a silent epidemic in America.

201 Childhood obesity is also growing in frequency, and the

202 Childhood obesity is an important public health problem

203 Childhood obesity is associated with a number of metabol

204 To assess whether childhood obesity is associated with airway dysanapsis (

205 Childhood obesity is associated with earlier puberty in

206 Childhood obesity is associated with increased risk of b

207 Childhood obesity is associated with type 2 diabetes mel

208 Childhood obesity is becoming more prevalent in the Unit

209 Childhood obesity is currently at its highest: recent st

210 tions, causes, evaluation, and management of childhood obesity is discussed.

211 breastfeeding to reduced risk of developing childhood obesity is inconclusive, yet previous studies

212 It is possible that the prevalence of childhood obesity is increasing across generations as a

213 Childhood obesity is increasing and is associated with a

214 The prevalence of childhood obesity is increasing and the causes of this a

215 The prevalence of childhood obesity is increasing rapidly in low- and midd

216 Childhood obesity is increasing worldwide, and all previ

217 tion of primary care interventions to reduce childhood obesity is limited.

218 In addition, childhood obesity is more prevalent among minority subgr

219 ere is now growing evidence that the risk of childhood obesity is strongly influenced by perinatal de

220 Childhood obesity is the focus of public health efforts

221 lean compared with obese women contribute to childhood obesity is unclear.

222 rly life factors related to fetal growth and childhood obesity is warranted.

223 helps to understand why, particularly during childhood, obesity is a risk factor for the development

224 acid signature significantly associated with childhood obesity, is an independent risk factor of futu

225 , hay fever or eczema), alcohol consumption, childhood obesity, low-density lipoprotein cholesterol (

226 The increase in childhood obesity mainly reflects increased energy intak

227 Childhood obesity may be a risk factor for higher mortal

228 e, genetic and environmental factors driving childhood obesity may have a lasting influence on health

229 In the context of other options for treating childhood obesity, metformin has not been shown to be cl

230 al degeneration, sensorineural hearing loss, childhood obesity, non-insulin-dependent diabetes mellit

231 mong children may partly explain the rise in childhood obesity observed in the past few years.

232 We estimate that the impact on childhood obesity of this policy may be reduced by aroun

233 Studies of the effect of childhood obesity on bone accrual during growth have yie

234 of this study was to determine the effect of childhood obesity on skeletal mass and dimensions relati

235 ng or nicotine exposure during pregnancy and childhood obesity or metabolic disorders at any age.

236 etween maternal smoking during pregnancy and childhood obesity or whether this association varies by

237 hange appears to be associated with improved childhood obesity outcomes.

238 r contributing to the world-wide epidemic of childhood obesity/overweight.

239 NPs being associated with common early-onset childhood obesity (P = 0.0003) and common adult morbid o

240 will reduce the public health burden of the childhood obesity pandemic.

241 Childhood obesity poses a significant public health chal

242 App, thereby providing proof of concept that childhood obesity prediction research can be integrated

243 gh extensive descriptive research shows that childhood obesity predisposes a person to adult obesity,

244 uring the coronavirus disease 2019 pandemic, childhood obesity prevalence has reached unprecedented l

245 data suggest that the spiralling increase in childhood obesity prevalence might be abating; increased

246 Childhood obesity prevention strategies aimed at reducin

247 mponents suggests that they hold promise for childhood obesity prevention worldwide.

248 t can be replicated in their communities for childhood obesity prevention.

249 The Childhood Obesity Project was conducted as a European mu

250 seen between infection episodes and risk of childhood obesity (ptrend <0.0001).

251 ios for obesity in adulthood associated with childhood obesity ranged from 1.3 (95 percent confidence

252 One hypothesis is that increasing childhood obesity rates may explain part of this increas

253 Childhood obesity rates remain high, especially among ad

254 l analyses (n = 10,583), the odds for severe childhood obesity reached 1.30 (P = 8.0 x 10(-11)).

255 This genetic risk expresses itself early as childhood obesity, reinforcing the notion that early pre

256 the role of truncal adiposity in endotyping childhood obesity-related asthma.

257 Childhood obesity remains a prominent public health prob

258 Childhood obesity remains a significant public health ch

259 Childhood obesity remains an epidemic in the U.S. and wo

260 However, treatment for childhood obesity remains largely ineffective.

261 Childhood obesity remains one of the most important issu

262 lues in women free of disease could mitigate childhood obesity remains unknown.

263 Lung, and Blood Institute (NHLBI)-initiated childhood obesity research.

264 ure to secondhand smoke on the prevalence of childhood obesity: results from NHANES, 2007-2010.

265 Anthropometry and childhood obesity risk data were obtained for 1868 UK-bo

266 e exposure to bisphenol A (BPA) may increase childhood obesity risk, but few prospective epidemiologi

267 haracteristics of the built environment with childhood obesity risk.

268 astfeeding is associated with a reduction in childhood obesity risk.

269 However, the common forms of childhood obesity seem to result from a predisposition t

270 y warrants further investigation, addressing childhood obesity should be an urgent public health prio

271 lic function in the potential development of childhood obesity should be further explored.

272 een PCSK1 rs6232 and obesity is stronger for childhood obesity than for adult obesity.

273 Given the current level of childhood obesity, the models predicted that a majority

274 he science for understanding the etiology of childhood obesity, the preventive interventions and trea

275 revalence, health consequences, and costs of childhood obesity, there has been substantial interest i

276 Maternal obesity is a risk factor for childhood obesity; this is a major public health concern

277 adolescents, the IOTF convened a workshop on childhood obesity to determine the most appropriate meas

278 interprets data on the early determinants of childhood obesity to provide relevant strategies for dai

279 genetic correlations have been found between childhood obesity traits and birth weight (a proxy for i

280 re identified: never obese (never-OB); early childhood obesity transitioning to non-obesity before ad

281 ssociation have released recommendations for childhood obesity treatment and prevention which include

282 ehavioral treatment (FBT) is recommended for childhood obesity treatment; however, it is not effectiv

283 tudy and 3 European cohort studies (European Childhood Obesity Trial, Norwegian Human Milk Study, and

284 cted the monocyte gene expression profile in childhood obesity using an Illumina microarray platform

285 ay of early-life environmental exposures and childhood obesity, using an exposome-wide approach.

286 ic effects of ART exposure in the context of childhood obesity warrants further investigation, addres

287 regulation of monocyte IMPDH2 and TMEM134 in childhood obesity was also observed in obese adults.

288 Childhood obesity was based on a body mass index greater

289 Childhood obesity was defined as a body-mass index at or

290 our ongoing genome-wide association study on childhood obesity, we demonstrate that ATOM increases th

291 In light of the worldwide increase in childhood obesity, we examined the association between b

292 additional genetic variants associated with childhood obesity, we performed a trans-ancestral meta-a

293 The familial risk ratio for childhood obesity when a parent is obese reaches >2.5.

294 Rapid infancy weight gain is associated with childhood obesity, whereas low infancy weight is associa

295 e the impact of maternal prepregnancy BMI on childhood obesity, which warrants further investigation

296 aring an RP intervention designed to prevent childhood obesity with a safety control.

297 5 differentially regulated monocyte genes in childhood obesity with obesity and complexity of coronar

298 ntribute to malnutrition and rising rates of childhood obesity, with effects throughout the life cour

299 etween antibiotic use in infancy and risk of childhood obesity, with implications for health-care del

300 dvise reduced milk-fat consumption to reduce childhood obesity, yet the relation between lower milk f