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1 tes the neurotoxic and nephrotoxic byproduct chloroacetaldehyde.
2 f an etheno ring after reaction of ADPR with chloroacetaldehyde.
3  caused by exogenous chemical agents such as chloroacetaldehyde.
4 r cells under the influence of the cytotoxin chloroacetaldehyde.
5                    For toxicity profiling of chloroacetaldehyde, 428 and 317 metabolite features were
6           Monochloramine quickly reacts with chloroacetaldehyde, a DBP formed during primary disinfec
7 1, treated with 10 and 35 mumol.liter(-1) of chloroacetaldehyde, a metabolite of the anti-cancer drug
8 rated with the isophosphoramide mustard; and chloroacetaldehyde, a neurotoxic and nephrotoxic compoun
9 ly, treatment of cultured human cells with 2-chloroacetaldehyde, a reactive metabolite of vinyl chlor
10 -butyl hydroperoxide, N-hydroxylamine, alpha-chloroacetaldehyde and glutaraldehyde.
11 ADPR was formed by the reaction of ADPR with chloroacetaldehyde at 80 degrees C and pH 4.0.
12                                              Chloroacetaldehyde (CA) is a nephrotoxic and neurotoxic
13 esized that inhibition of complex I (C-I) by chloroacetaldehyde (CAA), a metabolite of IFO, is the ch
14                        Chemical probing with chloroacetaldehyde, dimethyl sulfate, and potassium perm
15 tions using OsO4, diethyl pyrocarbonate, and chloroacetaldehyde probes.
16 ective condensation of aminotriazine 15 with chloroacetaldehyde provided the desired imidazotriazine
17 adducts produced by chloroethylene oxide and chloroacetaldehyde, reactive metabolites of vinyl chlori
18      As a model for this, we have shown that chloroacetaldehyde reacts more than 18-fold faster with
19 elease of the active entity and limiting the chloroacetaldehyde release.
20 nt acyl etheno CoA esters in the presence of chloroacetaldehyde, separated by ion-paired reversed-pha
21 substrate; furthermore, each mutant oxidized chloroacetaldehyde slower than propionaldehyde, and a pr
22 lboronate agarose columns and incubated with chloroacetaldehyde to convert the adenosine present in t
23 dissociation to deacylation was finding that chloroacetaldehyde was oxidized more rapidly than acetal
24                                  Reaction of chloroacetaldehyde with F-ddA triphosphate in extracts f